ライフサイエンスコーパス: cobalamin deficiency

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1 strongly influenced by both renal status and cobalamin deficiency.

2 s identified the entity of mild, preclinical cobalamin deficiency.

3 not all abnormal metabolite results reflect cobalamin deficiency.

4 seem at increased risk for mild, preclinical cobalamin deficiency.

5 plasma MMA values have been used to diagnose cobalamin deficiency.

6 y years before the establishment of clinical cobalamin deficiency.

7 In cobalamin deficiency, 2 mg of cyanocobalamin administere

8 ed to ascertain the prevalence of folate and cobalamin deficiencies and hyperhomocysteinemia in Bangl

9 tHcy is a marker of folate and cobalamin deficiencies and is also related to several li

10 row mononuclear cells of eight patients with cobalamin deficiency and compared this with that found i

11 chemical basis for neurologic dysfunction in cobalamin deficiency and the frequent divergence between

12 Ethnic differences in cobalamin deficiency and the Hcys patterns associated wi

13 unusual states of neurologically symptomatic cobalamin deficiency are being recognized, such as nitro

14 were increased and only GSH was decreased in cobalamin deficiency as a whole, compared with 17 contro

15 mes more common in the elderly than clinical cobalamin deficiency but also differs from it in arising

16 nborn errors of metabolism and indicators of cobalamin deficiency in older persons.

17 The importance of treating cobalamin deficiency in pregnancy is considered.

18 y and bariatric surgery rates, prevalence of cobalamin deficiency in pregnancy is rising.

19 determine the demographic characteristics of cobalamin deficiency in the elderly and its role in thei

20 the very common problem of mild, preclinical cobalamin deficiency in the elderly await further clarif

21 Mild cobalamin deficiency is most common in elderly white men

22 Cobalamin deficiency is relatively common, but the great

23 Because cobalamin deficiency is routinely treated with parentera

24 ile megaloblastic anemia due to vitamin B12 (cobalamin) deficiency is caused by intestinal malabsorpt

25 The challenges in medical management of cobalamin deficiency lie in attention to the unique path

26 Vitamin B-12 (cobalamin) deficiency may produce severe neurologic and

27 to the unique pathophysiology that underlies cobalamin deficiency, more than in the mechanics of ther

28 In the diagnosis of juvenile cobalamin deficiency, mutational analysis of the CUBN, A

29 re of this knockout and the lack of systemic cobalamin deficiency point to other mechanisms for cellu

30 individual components: correctly diagnosing cobalamin deficiency, reversing it, defining its underly

31 es in epidemiologic surveys have subclinical cobalamin deficiency (SCCD), not classical clinical defi

32 consequences, and management of subclinical cobalamin deficiency (SCCD), which affects many elderly

33 Cobalamin deficiency (serum cobalamin <258 pmol/L and MM

34 entrations or the mild metabolic evidence of cobalamin deficiency so often seen in the elderly.

35 cohort of elderly with a high prevalence of cobalamin deficiency to determine whether SAH, SAM, or t

36 ave broadened and complicated the picture of cobalamin deficiency while providing greater opportuniti

37 Cobalamin deficiency with elevated serum MMA concentrati

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