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1 r cell differentiation within the developing cochlear duct.
2 on of ectopic sensory patches underneath the cochlear duct.
3 sh sensory and nonsensory territories in the cochlear duct.
4 is vestibular-like hair cells located in the cochlear duct.
5 paracrine Wnt signaling predominates in the cochlear duct.
6 ause of displaced hair cells and a shortened cochlear duct.
7 circular canals, and a malformed saccule and cochlear duct.
8 e down-regulated in the other regions of the cochlear duct.
9 neither a vestibular apparatus nor a coiled cochlear duct.
10 essed in adjacent, nonsensory regions of the cochlear duct.
11 ve rise to ectopic vestibular patches in the cochlear duct.
12 aintain sensory/nonsensory boundaries in the cochlear duct.
13 roperties of the basilar membrane within the cochlear duct.
14 gamma are initially expressed throughout the cochlear duct.
15 nating mosaic that extends the length of the cochlear duct.
16 were more susceptible to RA effects than the cochlear duct.
17 ducts and semicircular canals and have short cochlear ducts.
18 inner ear development including a shortened cochlear duct, a decrease in the total number of cochlea
21 ells, and Reissner's membrane throughout the cochlear duct and had complete inner and outer hair cell
22 rosensory domain that runs the length of the cochlear duct and is bounded by two nonsensory domains,
23 tly, ventral otic derivatives, including the cochlear duct and saccule, fail to form, and dorsal otic
24 By contrast, the formation of the proximal cochlear duct and saccule, which requires less Shh signa
25 cts in differentiation and patterning of the cochlear duct and sensory epithelium and loss of the dor
26 Frzb (SFRP3) is confined to the nonsensory cochlear duct and the lagena macula, whereas SFRP2 is ma
27 egulating the differential growth within the cochlear duct and thus, its proper outgrowth and coiling
28 ranscripts were both widely expressed in the cochlear duct and utricle in an overlapping pattern, sug
30 ation of vestibular formation, length of the cochlear duct, and the number of cochlear hair cells.
31 ory domain that lies along the length of the cochlear duct, and which forms before the onset of hair
32 the sensory and secretory epithelium of the cochlear duct appear normal in the Hmx3 null animals.
34 ithelial tissues, including cells lining the cochlear duct, at embryonic day 18.5 and postnatal day 5
35 regionalization, convergent extension of the cochlear duct, cell fate specification, synaptogenesis,
37 ng conducted over the time course of chicken cochlear duct development reveals that Wnt-related gene
38 found in the same post-mitotic region of the cochlear duct during early stages of cochlear developmen
39 ature semicircular canals, utricle, saccule, cochlear duct, endolymphatic duct and sac, and neurons o
40 s with shifted rhombomeres showed defects in cochlear duct formation indicating that signaling from r
43 t Id1, Id2, and Id3 are expressed within the cochlear duct in a pattern that is consistent with a rol
44 cochlea, as inhibiting the outgrowth of the cochlear duct increases the number of Atoh1-lineage cell
46 utcome of the intricate K+ regulation in the cochlear duct is the endocochlear potential (EP), approx
48 ve severe vestibular defects and a shortened cochlear duct, markers of the prosensory domain appear a
49 ound that cells and tissue components of the cochlear duct may be labelled by fluorescent markers wit
50 along the anteroposterior axis also impacts cochlear duct morphogenesis but has little effect on the
54 Pax2 affects tissue specification within the cochlear duct, particularly regions between the sensory
56 omotes ventral fates such as the saccule and cochlear duct, possibly by restricting Otx2 expression.
57 thin the prosensory domain of the developing cochlear duct relies on the temporal and spatial regulat
63 e RAR and RXR receptors within the embryonic cochlear duct were determined by in situ hybridization.
64 ne of the known exceptions in mammals is the cochlear duct, where the potential is approximately 80-1
65 pillar cells) along the entire length of the cochlear duct, with the most extreme abnormalities found
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