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1 the other modulates the excitability of the cochlear nerve.
2 s form ribbon synapses with terminals of the cochlear nerve.
3 ve terminals and delayed degeneration of the cochlear nerve.
4 unrecognized embryologic malformation of the cochlear nerve.
5 ts NM neurons by preventing formation of the cochlear nerve.
6 cues by direct electrical stimulation of the cochlear nerve.
7 hearing loss by electrically stimulating the cochlear nerve.
8 rally during formation of the vestibular and cochlear nerves.
11 Nav1.6 and Nav1.2 in the cochlear ganglion, cochlear nerve, and organ of Corti, including the type I
13 the basilar papilla and NM is established as cochlear nerve axons arrive in the NM prior to the onset
16 nts examined the arrival and organization of cochlear nerve axons in the primary auditory brainstem n
18 f cochlear afferent synapses and progressive cochlear nerve degeneration in noise-exposed ears with r
19 nts who demonstrate abnormal function of the cochlear nerve despite typical function of sensory cells
20 relationship between stimulus intensity and cochlear nerve discharge rate (the rate-intensity functi
21 ional model of a single mammalian myelinated cochlear nerve fiber coupled to a stimulator-electrode-t
22 ses spontaneous and sound-evoked activity in cochlear nerve fibers and helps control noise-induced ex
23 ral reduction in spontaneous activity in the cochlear nerve fibers caused by the acoustic injury to t
24 ory epithelium, where functional subtypes of cochlear nerve fibers differ in threshold sensitivity, a
25 opy to count synapses between hair cells and cochlear nerve fibers, and using measurement of auditory
26 pses between hair cells and the terminals of cochlear nerve fibers, as seen in confocal analysis of t
27 D1 and D2 immunolabeling was localized to cochlear nerve fibers, near the first nodes of Ranvier (
38 mice demonstrate a significant reduction in cochlear nerve sound-evoked activity compared with wild-
39 o determine an energy-efficient waveform for cochlear nerve stimulation, we used a genetic algorithm
41 erented animals, there was up to 40% loss of cochlear nerve synapses and a corresponding decline in t
42 auditory processing compensate for a loss of cochlear nerve synapses by increasing the gain on remain
43 ate excitotoxicity has been characterized in cochlear nerve terminals, but much less is known about w
44 s postnatal day 3 (P3) in the portion of the cochlear nerve that innervates the base of the modiolus.
45 ent likely contributes to the differences in cochlear nerve threshold and SR seen on the two sides of
46 s, used to predict the response of the human cochlear nerve to vowels coded by a cochlear implant.
47 e length and width of the bony canal for the cochlear nerve were significantly smaller in patients wi
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