ライフサイエンスコーパス: codeletion

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1 letion of chromosome arms 1p and 19q (1p/19q codeletion).

2 eletion is further potentiated by Hif-1alpha codeletion.

3 utated in oligodendroglial tumors with 1p19q codeletion.

4 ic remodeling from such coamplifications and codeletions and delineate genes driving cancer metabolis

5 oligodendroglial tumors (109 with the 1p19q codeletion) and analyzed patients' outcome.

6 (IDH1/2 wt, IDH1/2 mut, and IDH1/2 mut 1p19q codeletion) and the four subtypes of GBMs (classical, me

7 and the long arm of chromosome 19, or 1p19q codeletion; and (c) methylguanine methyltransferase prom

8 ade gliomas with IDH mutations and no 1p/19q codeletion had mutations in TP53 (94%) and ATRX inactiva

9 rade gliomas with an IDH mutation and 1p/19q codeletion had the most favorable clinical outcomes.

10 letion of chromosome arms 1p and 19q (1p/19q codeletion) have been implicated as clinically relevant

11 Homozygous codeletion occurred in eight of the 13 (62%) metastatic,

12 and predictive marker in glioblastoma; (ii) codeletion of 1p and 19q differentiating oligodendroglio

13 Codeletion of Adrb2 and Cox10, a gene encoding a cytochr

14 Codeletion of Apc and Fak strongly reduced proliferation

15 terial phagocytosis that can be abolished by codeletion of Bim.

16 In contrast, codeletion of both Fbw7 and p53 causes highly penetrant,

17 rs sustained either deletion of p16 alone or codeletion of both p16 and p15 or amplification of CDK4.

18 Codeletion of both Palb2 and Tumor protein 53 (Trp53) ac

19 We showed that codeletion of CDKN2A and CDKN2C serves as a strong predi

20 in the TERT promoter, mutations in IDH, and codeletion of chromosome arms 1p and 19q (1p/19q codelet

21 Mutations in IDH, TP53, and ATRX and codeletion of chromosome arms 1p and 19q (1p/19q codelet

22 ies of these cell lines for the incidence of codeletion of genes located near p16, in particular, the

23 e p16, p15 or CDK4 genes, whereas homozygous codeletion of p16 and p15 was observed in both primary a

24 f both animal models was entirely rescued by codeletion of p53.

25 als rescues adipose tissue browning, as does codeletion of PGC-1beta.

26 contiguous gene syndrome caused by germline codeletion of PKD1 and TSC2 genes.

27 Codeletion of Pten and Trp53 resulted in fully penetrant

28 Codeletion of pVHL and HIF-2alpha in JG cells completely

29 However, heterozygous deletion of Sav1 or codeletion of Rassf1a and Sav1 produced liver tumors wit

30 rget malignant cells that have undergone the codeletion of such genes.

31 Codeletion of TFE3 in adipose-specific FLCN knockout ani

32 e severity of this defect was exacerbated by codeletion of the Gcn5 acetyltransferase within SAGA.

33 pha,-beta gene cluster; one cell line bore a codeletion of the IFNA1 gene but retained the IFNB1 locu

34 TAP deficiency in cancer is primarily due to codeletion of the MTAP and p16 genes.

35 Here we show that codeletion of the p53 and alphav integrin genes in mouse

36 way, because the cell death was rescued with codeletion of the proapoptotic proteins Bax and Bak.

37 Upon codeletion of TRF1 and TRF2, the telomere volume increas

38 Codeletion of VHL together with HIF2A but not with HIF1A

39 iomas with an IDH mutation either had 1p/19q codeletion or carried a TP53 mutation.

40 In the 80 patients with a 1p/19q codeletion, OS was increased, with a trend toward more b

41 iomas with strong EGFR expression and 1p/19q codeletion showed reduced survival, compared with their

42 for AOA (3 v 4); and modified grade, 1p/19q codeletion status, and oligodendroglial histology for th

43 In Cox models that included codeletion status, the adjusted OS for all patients was

44 ood mononuclear cells from patients with SS, codeletion with CDKN2A occurred in 18% of samples but lo

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