ライフサイエンスコーパス: coeliac

1 tential to mitigate the problems confronting coeliacs.

2 duced by subdiaphragmatic or by coeliac plus coeliac accessory branch vagotomy.

3 erent activity is carried in the coeliac and coeliac accessory branches of the subdiaphragmatic vagus

4 e results indicate that afferent activity in coeliac and accessory coeliac vagal branches is involved

5 is vagal afferent activity is carried in the coeliac and coeliac accessory branches of the subdiaphra

6 In patch-clamp studies, nodose, coeliac and superior cervical ganglia (SCG) neurones fro

7 e wheat flour and develop bread suitable for coeliacs and gluten intolerant individuals.

8 of the descending aorta at the level of the coeliac arteries, a stimulus that elevated blood pressur

9 (i) Interruption of the coeliac branches mimicked the effect of total subdiaphra

10 subsets that may be safely incorporated into coeliac diets.

11 ad positive antibody results, of whom 14 had coeliac disease (11 EMA positive, three EMA negative).

12 ildren with IgE-mediated wheat allergy (WA), coeliac disease (CD) and Helicobacter pylori infection (

13 The consequences of subclinical coeliac disease (CD) in Type 1 diabetes mellitus (T1DM)

14 Coeliac disease (CD), an enteropathy caused by cereal gl

15 Coeliac disease (CD), due to its protean clinical manife

16 Despite the considerable health impact of coeliac disease (CD), reliable estimates of the impact o

17 ed by several clinical conditions, including Coeliac Disease (CD).

18 ant microbiota may play a pathogenic role in coeliac disease (CD).

19 o a gluten-free diet (GFD) for patients with coeliac disease (CD).

20 s been proposed to play a pathogenic role in coeliac disease (CD).

21 loci shared between two autoimmune diseases: coeliac disease (CeD) and rheumatoid arthritis (RA).

22 such as inflammatory bowel disease (IBD) and coeliac disease (CeD).

23 l syndrome was significantly associated with coeliac disease (p=0.004, odds ratio=7.0 [95% CI 1.7-28.

24 osed with active CD, CD on a GFD, Refractory coeliac disease (RCD) type I and II, and enteropathy ass

25 A strong HLA association is seen in coeliac disease [specifically to the DQ(alpha1*0501,beta

26 lude the association of Down's syndrome with coeliac disease and Alzheimer's disease, and improved me

27 enes at HLA-unlinked loci also predispose to coeliac disease and are probably stronger determinants o

28 nded to occur rapidly after the diagnosis of coeliac disease and cumulative survival dropped in the f

29 dietary gluten intake in conditions such as coeliac disease and dermatitis herpetiformis.

30 falls of vitamin A supplementation in active coeliac disease and have enabled identification of oat a

31 ng, the well established association between coeliac disease and insulin dependent diabetes mellitus,

32 ptibility to type 1 diabetes (T1D) with both Coeliac disease and multiple sclerosis.

33 isk to people affected by conditions such as coeliac disease and non-coeliac gluten sensitivity.

34 understanding of the complex pathogenesis of coeliac disease and novel therapeutic targets.

35 tic susceptibilities that are both unique to coeliac disease and overlap with other autoimmune diseas

36 mall bowel Crohn's disease, complications of coeliac disease and surveillance of polyposis syndromes.

37 e the possible concealed burden of untreated coeliac disease and the effects of a gluten-free diet.

38 sidue at position beta57 are associated with coeliac disease and type I diabetes.

39 5-positive patients aged 18-70 years who had coeliac disease and were on a gluten-free diet.

40 designed for the selective amplification of coeliac disease associated alleles (DQA1*05, DQB1*02, DQ

41 l criterion to compare duodenal histology in coeliac disease before and after gluten withdrawal.

42 Crohn's disease and coeliac disease both demonstrate considerable overlap in

43 gliadin antibodies are a marker of untreated coeliac disease but can also be found in individuals wit

44 eurological syndromes may be associated with coeliac disease but it is unclear whether these are dire

45 (age and sex matched) were investigated for coeliac disease by analysis of serum IgA antigliadin, Ig

46 Although coeliac disease can be misdiagnosed as irritable bowel s

47 The number of people diagnosed with coeliac disease continues to rise, and this article crit

48 y of the mechanism of the immune response in coeliac disease could provide insight into the mechanism

49 Coeliac disease develops in genetically susceptible indi

50 The long-term implications of active coeliac disease emphasize the need for early detection a

51 We have examined these regions in 28 coeliac disease families by linkage analysis.

52 iopsy did not identify any causes other than coeliac disease for iron deficiency anaemia, suggesting

53 in developing new strategies for preventing coeliac disease has motivated efforts to identify cereal

54 manifestations in patients with established coeliac disease have been reported since 1966, it was no

55 in two patients, and changes compatible with coeliac disease in 11.

56 Worldwide awareness of coeliac disease in all ages continues to grow.

57 barley and rye, or gluten protein, can cause coeliac disease in individuals not tolerating gluten.

58 g offers additional sensitivity in detecting coeliac disease in individuals who have self-prescribed

59 a measure of cryptic gluten sensitivity, and coeliac disease in neurological patients.

60 om group 1 revealed histological evidence of coeliac disease in nine (35%), non-specific duodenitis i

61 candidate locus conferring susceptibility to coeliac disease in some families.

62 Coeliac disease is a common enteropathy characterized by

63 Coeliac disease is a genetically-determined chronic infl

64 Complicated coeliac disease is an extremely serious condition with a

65 Epidemiological studies have shown that coeliac disease is as common in parts of Asia, Africa an

66 Coeliac disease is caused by a genetically determined, s

67 The natural history of complicated coeliac disease is characterised by two different types

68 Coeliac disease is characterised by villous atrophy, whi

69 ease, but further work into the treatment of coeliac disease is needed.

70 Coeliac disease is the prototypical gluten-sensitive dis

71 Epidemiological studies have shown that coeliac disease is very common and affects about one in

72 ion is difficult because occult sub-clinical coeliac disease occurs commonly and background prevalenc

73 Coeliac disease occurs in about 1% of people in most pop

74 macodynamics of the vaccine in patients with coeliac disease on a gluten-free diet.

75 In our material, 8/245 (3,2 %) coeliac disease patients presented inflammatory bowel di

76 Evidence suggests that many coeliac disease patients suffer from persistent clinical

77 a cytokine greatly upregulated in the gut of coeliac disease patients, retinoic acid rapidly activate

78 Patients with established coeliac disease referred for neurological opinion show s

79 - 13 years (range 19-64)) with biopsy proven coeliac disease referred for neurological opinion.

80 Our findings on coeliac disease replicate the previous SNP results and s

81 Coeliac disease serology was positive in all cases.

82 of course: patients with a new diagnosis of coeliac disease that do not improve despite a strict glu

83 Although the evidence for linkage of coeliac disease to chromosome 15q26 is not strong, the w

84 Coeliac disease was associated with excessive health car

85 In 7/8 patients the diagnosis of coeliac disease was made first and inflammatory bowel di

86 significant evidence in favour of linkage to coeliac disease was obtained for chromosomes 3q27, 5q33.

87 gical dysfunction is a known complication of coeliac disease we have investigated the frequency of an

88 We aimed to assess the association of coeliac disease with irritable bowel syndrome in patient

89 orwegian Human Milk Study, and Prevention of Coeliac Disease) that collaborate in the European Union-

90 opsy samples from 42 patients with untreated coeliac disease, 37 treated patients, and 18 controls, w

91 gluten-free diet is the only means to manage coeliac disease, a permanent immune intolerance to glute

92 icanus larvae for allergic rhinitis, asthma, coeliac disease, and multiple sclerosis.

93 or all immune recognition of wheat gluten in coeliac disease, and to explore if the tissue transgluta

94 ases such as systemic lupus erythematosus or coeliac disease, antibodies to specific membrane targets

95 esis and improving diagnostic strategies for coeliac disease, but further work into the treatment of

96 s have mainly been studied: Turner syndrome, coeliac disease, cystic fibrosis, growth hormone deficie

97 n our genetic and immunological knowledge of coeliac disease, early introduction of a gluten-free die

98 ociated with systemic lupus erythematosus or coeliac disease, have not in general disclosed consisten

99 nded our knowledge of the long-term risks of coeliac disease, in addition to excluding infertility as

100 Within our cohort of patients with coeliac disease, inflammatory bowel disease was signific

101 Coeliac disease, or gluten-sensitive enteropathy, is onl

102 present in cereal proteins that do not cause coeliac disease, Shan and colleagues suggest that genera

103 data, clinical presentation and treatment of coeliac disease, time and place of diagnosis and presenc

104 Conditions such as coeliac disease, type 1 diabetes, Crohn's disease and ul

105 diseases including asthma, Crohn's disease, coeliac disease, vitiligo, multiple sclerosis and type 1

106 A notable exception is coeliac disease, where genetically susceptible individua

107 time and place of diagnosis and presence of coeliac disease-associated or other co-morbidities.

108 e critically the recent research advances in coeliac disease.

109 onhuman leucocyte antigen genetic factors in coeliac disease.

110 he molecular pathways involving cytokines in coeliac disease.

111 tive gluten peptides presented by HLA-DQ8 in coeliac disease.

112 mune system is central to the development of coeliac disease.

113 sponsiveness, and tissue transglutaminase in coeliac disease.

114 3) known to induce small intestine damage in coeliac disease.

115 P kinase might have the potential to control coeliac disease.

116 an adaptive immune response in patients with coeliac disease.

117 ents with this disorder are investigated for coeliac disease.

118 ry care should be investigated routinely for coeliac disease.

119 ontrols, both of whom were EMA positive, had coeliac disease.

120 uodenal biopsy to confirm the possibility of coeliac disease.

121 the commonest neurological manifestation of coeliac disease.

122 l biopsy for those tested positive to detect coeliac disease.

123 ology found no causes for anaemia other than coeliac disease.

124 rapeutic vaccine, Nexvax2, designed to treat coeliac disease.

125 first months after diagnosis of complicated coeliac disease.

126 in resolving long-lasting health problems in coeliac disease.

127 sed symptoms and impaired quality of life in coeliac disease.

128 nt of this potential therapeutic vaccine for coeliac disease.

129 dditional primary care costs associated with coeliac disease.

130 proximately 1% of the population suffer from coeliac disease.

131 gnostic approach to reduce underdiagnosis of coeliac disease.

132 e recent scientific and clinical advances in coeliac disease.

133 selected members of the Swedish Society for Coeliacs, divided into equal-sized age- and sex strata;

134 nosis and presence of thyroidal disease, non-coeliac food intolerance or gastrointestinal co-morbidit

135 in isolated superior cervical ganglia (SCG), coeliac ganglia (CG), and superior mesenteric ganglia (S

136 sympathetic neurons acutely dissociated from coeliac ganglia of the guinea-pig.

137 Neurones from mouse aortic-renal and coeliac ganglia were identified as either 'tonic' or 'ph

138 Truncal vagotomy or treatment of the coeliac ganglia with capsaicin did not significantly aff

139 Truncal vagotomy or treatment of the coeliac ganglia with capsaicin did not significantly aff

140 DiI crystals were placed bilaterally on the coeliac ganglia, labeled piriform and fusiform pregangli

141 dominal sympathetic (mesenteric) nerves, the coeliac ganglia, or on the rostral three somatic spinal

142 y conditions such as coeliac disease and non-coeliac gluten sensitivity.

143 be a potential alternative for reduction of coeliac immunological activities in gluten proteins.

144 tomach (nucleus gelatinosus); 5) hepatic and coeliac nerves (nucleus subpostrema); and 6) carotid bod

145 se-specific mortality compared to those with coeliac node metastasis (HR 0.71, 95% CI 0.40-1.27).

146 It is uncertain whether coeliac node metastasis precludes long-term survival in

147 patients with distal oesophageal cancer with coeliac node metastasis seem to have a similarly poor su

148 The prognosis in patients with coeliac node metastasis was compared with patients with

149 Among 446 patients, 346 (77.6%) had no coeliac node metastasis, 56 (12.6%) had coeliac node met

150 d no coeliac node metastasis, 56 (12.6%) had coeliac node metastasis, and 44 (9.9%) had more distant

151 Compared to coeliac node negative patients, coeliac node positive pa

152 Compared to coeliac node negative patients, coeliac node positive patients were at a 52% increased r

153 Siblings of coeliac patients carry a high risk, but those found to h

154 diet (type A cases) and previously diagnosed coeliac patients that initially improved on a gluten-fre

155 Untreated coeliac patients used primary health care services more

156 ationwide cohort of 700 newly detected adult coeliac patients were prospectively evaluated.

157 Duodenal biopsies from coeliac patients were retrospectively reviewed to compar

158 Clinical and laboratory data from coeliac patients who later developed complications (A an

159 ons (A and B cases) and sex- and age-matched coeliac patients who normally responded to a gluten-free

160 s health concerns in long-term treated adult coeliac patients.

161 nhibition produced by subdiaphragmatic or by coeliac plus coeliac accessory branch vagotomy.

162 how unequivocally that: (a) receptors in the coeliac-portal circulation are more sensitive in amplify

163 diet remains paramount as the recognition of coeliac related complications increases.

164 ties and cost effectiveness of pre-endoscopy coeliac screening with Simtomax in anaemia; 3) whether o

165 high risk, but those found to have negative coeliac serology are very unlikely to develop the diseas

166 International guidelines recommend coeliac serology in iron deficiency anaemia, and duodena

167 ical, stellate, paravertebral chain ganglia, coeliac/superior mesenteric and inferior mesenteric gang

168 ositive in 8/9 first-degree relatives having coeliac-type mucosal lesions of grade Marsh 2 (n = 3) or

169 t afferent activity in coeliac and accessory coeliac vagal branches is involved in the regulation of

170 n help improve the total dietary intake of a coeliac while not negating on the quality properties of