ライフサイエンスコーパス: coeliac disease

1 ents with this disorder are investigated for coeliac disease.

2 ry care should be investigated routinely for coeliac disease.

3 ontrols, both of whom were EMA positive, had coeliac disease.

4 uodenal biopsy to confirm the possibility of coeliac disease.

5 the commonest neurological manifestation of coeliac disease.

6 l biopsy for those tested positive to detect coeliac disease.

7 ology found no causes for anaemia other than coeliac disease.

8 rapeutic vaccine, Nexvax2, designed to treat coeliac disease.

9 first months after diagnosis of complicated coeliac disease.

10 in resolving long-lasting health problems in coeliac disease.

11 sed symptoms and impaired quality of life in coeliac disease.

12 dditional primary care costs associated with coeliac disease.

13 proximately 1% of the population suffer from coeliac disease.

14 gnostic approach to reduce underdiagnosis of coeliac disease.

15 nt of this potential therapeutic vaccine for coeliac disease.

16 e recent scientific and clinical advances in coeliac disease.

17 e critically the recent research advances in coeliac disease.

18 onhuman leucocyte antigen genetic factors in coeliac disease.

19 he molecular pathways involving cytokines in coeliac disease.

20 tive gluten peptides presented by HLA-DQ8 in coeliac disease.

21 mune system is central to the development of coeliac disease.

22 sponsiveness, and tissue transglutaminase in coeliac disease.

23 3) known to induce small intestine damage in coeliac disease.

24 P kinase might have the potential to control coeliac disease.

25 an adaptive immune response in patients with coeliac disease.

26 ad positive antibody results, of whom 14 had coeliac disease (11 EMA positive, three EMA negative).

27 opsy samples from 42 patients with untreated coeliac disease, 37 treated patients, and 18 controls, w

28 gluten-free diet is the only means to manage coeliac disease, a permanent immune intolerance to glute

29 lude the association of Down's syndrome with coeliac disease and Alzheimer's disease, and improved me

30 enes at HLA-unlinked loci also predispose to coeliac disease and are probably stronger determinants o

31 nded to occur rapidly after the diagnosis of coeliac disease and cumulative survival dropped in the f

32 dietary gluten intake in conditions such as coeliac disease and dermatitis herpetiformis.

33 falls of vitamin A supplementation in active coeliac disease and have enabled identification of oat a

34 ng, the well established association between coeliac disease and insulin dependent diabetes mellitus,

35 ptibility to type 1 diabetes (T1D) with both Coeliac disease and multiple sclerosis.

36 isk to people affected by conditions such as coeliac disease and non-coeliac gluten sensitivity.

37 understanding of the complex pathogenesis of coeliac disease and novel therapeutic targets.

38 tic susceptibilities that are both unique to coeliac disease and overlap with other autoimmune diseas

39 mall bowel Crohn's disease, complications of coeliac disease and surveillance of polyposis syndromes.

40 e the possible concealed burden of untreated coeliac disease and the effects of a gluten-free diet.

41 sidue at position beta57 are associated with coeliac disease and type I diabetes.

42 5-positive patients aged 18-70 years who had coeliac disease and were on a gluten-free diet.

43 icanus larvae for allergic rhinitis, asthma, coeliac disease, and multiple sclerosis.

44 or all immune recognition of wheat gluten in coeliac disease, and to explore if the tissue transgluta

45 ases such as systemic lupus erythematosus or coeliac disease, antibodies to specific membrane targets

46 designed for the selective amplification of coeliac disease associated alleles (DQA1*05, DQB1*02, DQ

47 time and place of diagnosis and presence of coeliac disease-associated or other co-morbidities.

48 l criterion to compare duodenal histology in coeliac disease before and after gluten withdrawal.

49 Crohn's disease and coeliac disease both demonstrate considerable overlap in

50 gliadin antibodies are a marker of untreated coeliac disease but can also be found in individuals wit

51 eurological syndromes may be associated with coeliac disease but it is unclear whether these are dire

52 esis and improving diagnostic strategies for coeliac disease, but further work into the treatment of

53 (age and sex matched) were investigated for coeliac disease by analysis of serum IgA antigliadin, Ig

54 Although coeliac disease can be misdiagnosed as irritable bowel s

55 ildren with IgE-mediated wheat allergy (WA), coeliac disease (CD) and Helicobacter pylori infection (

56 The consequences of subclinical coeliac disease (CD) in Type 1 diabetes mellitus (T1DM)

57 Coeliac disease (CD), an enteropathy caused by cereal gl

58 Coeliac disease (CD), due to its protean clinical manife

59 Despite the considerable health impact of coeliac disease (CD), reliable estimates of the impact o

60 ed by several clinical conditions, including Coeliac Disease (CD).

61 ant microbiota may play a pathogenic role in coeliac disease (CD).

62 o a gluten-free diet (GFD) for patients with coeliac disease (CD).

63 s been proposed to play a pathogenic role in coeliac disease (CD).

64 loci shared between two autoimmune diseases: coeliac disease (CeD) and rheumatoid arthritis (RA).

65 such as inflammatory bowel disease (IBD) and coeliac disease (CeD).

66 The number of people diagnosed with coeliac disease continues to rise, and this article crit

67 y of the mechanism of the immune response in coeliac disease could provide insight into the mechanism

68 s have mainly been studied: Turner syndrome, coeliac disease, cystic fibrosis, growth hormone deficie

69 Coeliac disease develops in genetically susceptible indi

70 n our genetic and immunological knowledge of coeliac disease, early introduction of a gluten-free die

71 The long-term implications of active coeliac disease emphasize the need for early detection a

72 We have examined these regions in 28 coeliac disease families by linkage analysis.

73 iopsy did not identify any causes other than coeliac disease for iron deficiency anaemia, suggesting

74 in developing new strategies for preventing coeliac disease has motivated efforts to identify cereal

75 manifestations in patients with established coeliac disease have been reported since 1966, it was no

76 ociated with systemic lupus erythematosus or coeliac disease, have not in general disclosed consisten

77 in two patients, and changes compatible with coeliac disease in 11.

78 Worldwide awareness of coeliac disease in all ages continues to grow.

79 barley and rye, or gluten protein, can cause coeliac disease in individuals not tolerating gluten.

80 g offers additional sensitivity in detecting coeliac disease in individuals who have self-prescribed

81 a measure of cryptic gluten sensitivity, and coeliac disease in neurological patients.

82 om group 1 revealed histological evidence of coeliac disease in nine (35%), non-specific duodenitis i

83 candidate locus conferring susceptibility to coeliac disease in some families.

84 nded our knowledge of the long-term risks of coeliac disease, in addition to excluding infertility as

85 Within our cohort of patients with coeliac disease, inflammatory bowel disease was signific

86 Coeliac disease is a common enteropathy characterized by

87 Coeliac disease is a genetically-determined chronic infl

88 Complicated coeliac disease is an extremely serious condition with a

89 Epidemiological studies have shown that coeliac disease is as common in parts of Asia, Africa an

90 Coeliac disease is caused by a genetically determined, s

91 The natural history of complicated coeliac disease is characterised by two different types

92 Coeliac disease is characterised by villous atrophy, whi

93 ease, but further work into the treatment of coeliac disease is needed.

94 Coeliac disease is the prototypical gluten-sensitive dis

95 Epidemiological studies have shown that coeliac disease is very common and affects about one in

96 ion is difficult because occult sub-clinical coeliac disease occurs commonly and background prevalenc

97 Coeliac disease occurs in about 1% of people in most pop

98 macodynamics of the vaccine in patients with coeliac disease on a gluten-free diet.

99 Coeliac disease, or gluten-sensitive enteropathy, is onl

100 l syndrome was significantly associated with coeliac disease (p=0.004, odds ratio=7.0 [95% CI 1.7-28.

101 In our material, 8/245 (3,2 %) coeliac disease patients presented inflammatory bowel di

102 Evidence suggests that many coeliac disease patients suffer from persistent clinical

103 a cytokine greatly upregulated in the gut of coeliac disease patients, retinoic acid rapidly activate

104 osed with active CD, CD on a GFD, Refractory coeliac disease (RCD) type I and II, and enteropathy ass

105 Patients with established coeliac disease referred for neurological opinion show s

106 - 13 years (range 19-64)) with biopsy proven coeliac disease referred for neurological opinion.

107 Our findings on coeliac disease replicate the previous SNP results and s

108 Coeliac disease serology was positive in all cases.

109 present in cereal proteins that do not cause coeliac disease, Shan and colleagues suggest that genera

110 A strong HLA association is seen in coeliac disease [specifically to the DQ(alpha1*0501,beta

111 of course: patients with a new diagnosis of coeliac disease that do not improve despite a strict glu

112 orwegian Human Milk Study, and Prevention of Coeliac Disease) that collaborate in the European Union-

113 data, clinical presentation and treatment of coeliac disease, time and place of diagnosis and presenc

114 Although the evidence for linkage of coeliac disease to chromosome 15q26 is not strong, the w

115 Conditions such as coeliac disease, type 1 diabetes, Crohn's disease and ul

116 diseases including asthma, Crohn's disease, coeliac disease, vitiligo, multiple sclerosis and type 1

117 Coeliac disease was associated with excessive health car

118 In 7/8 patients the diagnosis of coeliac disease was made first and inflammatory bowel di

119 significant evidence in favour of linkage to coeliac disease was obtained for chromosomes 3q27, 5q33.

120 gical dysfunction is a known complication of coeliac disease we have investigated the frequency of an

121 A notable exception is coeliac disease, where genetically susceptible individua

122 We aimed to assess the association of coeliac disease with irritable bowel syndrome in patient