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1 is of glutathione, phospholipids, NADPH, and coenzyme Q10.
2 OQ2 and is essential for the biosynthesis of coenzyme Q10.
3 ebral cortex mitochondrial concentrations of coenzyme Q10.
4 wed that TQ is reduced more efficiently than coenzyme Q10.
5 eatine, 66 received minocycline, 71 received coenzyme Q10, 71 received GPI-1485, and 138 received pla
6                               Treatment with coenzyme Q10, an essential cofactor of the electron tran
7 reased antioxidant defenses, a water-soluble coenzyme Q10 analog (Qter) was used.
8 cise duration remained unchanged in both the coenzyme Q10 and placebo groups.
9 outcome of a recent clinical trial examining coenzyme Q10 and remacemide in HD patients.
10                The combined treatment, using coenzyme Q10 and remacemide together, was more efficacio
11 ched control subjects and that the levels of coenzyme Q10 and the activities of complex I and complex
12 administration of the mitochondrial cofactor coenzyme Q10 and the NMDA antagonist remacemide.
13 c acid) or oil-soluble (vitamin E acetate or Coenzyme Q10) antioxidants.
14                        Hereditary defects of coenzyme Q10 biosynthesis cause steroid-resistant nephro
15  we have identified 6 different mutations in coenzyme Q10 biosynthesis monooxygenase 6 (COQ6) in 13 i
16  mutations in genes that function within the coenzyme Q10 biosynthesis pathway, suggesting that SRNS
17 omparison of the rate of reduction of TQ and coenzyme Q10 by NQO1 showed that TQ is reduced more effi
18           They provide further evidence that coenzyme Q10 can exert neuroprotective effects that migh
19 econd step of the final reaction sequence of Coenzyme Q10 (CoQ) biosynthesis.
20                            We focused on the coenzyme Q10 (CoQ10) biosynthesis gene Coq2, the silenci
21 DCK3, which has been shown to participate in coenzyme Q10 (CoQ10) biosynthesis.
22  investigated whether oral administration of coenzyme Q10 (CoQ10) could attenuate 1-methyl-4-phenyl-1
23                                      Primary coenzyme Q10 (CoQ10) deficiencies are rare, clinically h
24    The authors performed sequencing of known Coenzyme Q10 (CoQ10) deficiency genes in 22 patients wit
25 irst described in 1989, our understanding of coenzyme Q10 (CoQ10) deficiency is only now coming of ag
26                                   The use of coenzyme Q10 (CoQ10) has been increasing rapidly during
27                                              Coenzyme Q10 (CoQ10) has shown a protective effect in ne
28                                              Coenzyme Q10 (CoQ10) may represent a safe therapeutic op
29     Here we report the important role of the coenzyme Q10 (CoQ10) on the activity of caspase-2 upstre
30 ence available for oral supplementation with coenzyme Q10 (CoQ10) to improve the tolerability of canc
31                                              Coenzyme Q10 (CoQ10) was encapsulated successfully in a
32 evented the adverse effects of alcohol while coenzyme Q10 (CoQ10) was not very effective against alco
33 stnatal supplementation with the antioxidant coenzyme Q10 (CoQ10) would prevent this programmed pheno
34                                              Coenzyme Q10 (CoQ10), an antioxidant and mitochondrial c
35                                              Coenzyme Q10 (CoQ10), an antioxidant that supports mitoc
36 he effect of long-term (6 mo) treatment with coenzyme Q10 (CoQ10), an endogenous antioxidant.
37 olet, has been used for the determination of coenzyme Q10 (CoQ10).
38 fied starch (OSA-ST) was used to encapsulate coenzyme Q10 (CoQ10).
39 termediate in the synthesis of ubiquinone or coenzyme Q10 (CoQ10).
40 pe B inhibitors (selegiline and rasagiline), coenzyme Q10, creatine, and exercise in early Parkinson'
41 are due to defects in nuclear DNA, including coenzyme Q10 deficiency and mutations in genes controlli
42                                              Coenzyme Q10 deficiency appears to be a relatively commo
43 eficiency, and one patient was found to have coenzyme Q10 deficiency due to compound heterozygous mut
44 are due to defects in nuclear DNA, including coenzyme Q10 deficiency, and mutations in genes that con
45                                              Coenzyme Q10 does not affect ejection fraction, peak oxy
46                                    Levels of coenzyme Q10 in lymphoblastoid cells and brain tissue we
47 and antioxidant levels (alpha-tocopherol and coenzyme Q10) in brain PM during aging.
48                                 Feeding with coenzyme Q10 increased cerebral cortex concentrations in
49 ough the mean (+/-SD) serum concentration of coenzyme Q10 increased from 0.95+/-0.62 microg/mL to 2.2
50 ese results show that oral administration of coenzyme Q10 increases both brain and brain mitochondria
51                                              Coenzyme Q10 is an essential cofactor of the electron tr
52                                              Coenzyme Q10 is commonly used to treat congestive heart
53 vidence for managing myopathic patients with coenzyme Q10 is not conclusive.
54                                              Coenzyme Q10 is the electron acceptor for complex I and
55 cronutrients involved in cardiac metabolism: coenzyme Q10, l-carnitine, thiamine, and amino acids, in
56      Patients with ADCK4 mutations had lower coenzyme Q10 levels, and coenzyme Q10 supplementation am
57                       Oral administration of coenzyme Q10 markedly attenuated striatal lesions produc
58  We found that oral administration of either coenzyme Q10 or remacemide significantly extended surviv
59 ductase 1, NADH-ferrocyanide reductase, NADH-coenzyme Q10 reductase, and NADH-cytochrome c reductase)
60                      These data suggest that coenzyme Q10-related forms of SRNS and hearing loss can
61       In 12-month-old rats administration of coenzyme Q10 resulted in significant increases in cerebr
62 mutations had lower coenzyme Q10 levels, and coenzyme Q10 supplementation ameliorated renal disease i
63  Therapeutic trials of exercise training and coenzyme Q10 supplementation should continue to be offer
64 lls lacking MFN2 can be partially rescued by coenzyme Q10 supplementation, which suggests a possible
65               Some bioactive substances like coenzyme Q10, taurine, glutamine, creatine, creatinine,
66 enzoquinone (coenzyme Q1) as a surrogate for coenzyme Q10, the cofactor of this enzyme.
67 sed apoptosis that was partially reversed by coenzyme Q10 treatment.
68 ects of an antioxidant mixture of vitamin A, coenzyme Q10, vitamin C, and vitamin E were evaluated fo
69 A was more effective than ascorbic acid, and Coenzyme Q10 was more effective than vitamin E acetate.
70                                              Coenzyme Q10 was safe and well tolerated in this populat
71                   We found that the level of coenzyme Q10 was significantly lower in mitochondria fro
72 decanoic acid) and lipophilic nutraceutical (Coenzyme Q10) was investigated using a rat feeding study

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