ライフサイエンスコーパス: cognition

1 y, and solve day-to-day problems (ie, normal cognition).

2 gnosis, and 13.9% with a diagnosis of intact cognition.

3 captivated by this link between language and cognition.

4 elated to both lower and higher level social cognition.

5 r such interventions synergistically enhance cognition.

6 sychological test battery was used to assess cognition.

7 rapeutic efficacy of OT for enhancing social cognition.

8 ence showed that vitamin E had no benefit on cognition.

9 ble in advancing our understanding of higher cognition.

10 other involves areas related to higher order cognition.

11 de reasoning and problem solving, and social cognition.

12 nformation transmission during sensation and cognition.

13 c plasticity underlying learning, memory and cognition.

14 discharge coordination is crucial to spatial cognition.

15 in processes underlying rapid perception and cognition.

16 oad role of fluid intelligence in successful cognition.

17 ding brain development, reward, emotion, and cognition.

18 maternal vitamin B12 status and offspring's cognition.

19 ogress in uncovering the structure of social cognition.

20 establish treatments targeting in particular cognition.

21 1% (95% CI 15%-27%) of the overall effect on cognition.

22 caused and reinforced by maladaptive social cognition.

23 lia activity disrupt both motor function and cognition.

24 comorbidities and other factors that affect cognition.

25 ve and negative symptoms as well as impaired cognition.

26 evelopment, plasticity, neural survival, and cognition.

27 ect and impaired integration of emotion with cognition.

28 ve functions like decision making and social cognition.

29 d implications for perception, behavior, and cognition.

30 te in vivo activity in preclinical models of cognition.

31 ential role of HPA axis genetic variation in cognition.

32 C degeneration act synergistically to impair cognition.

33 l manipulations to test and refine models of cognition.

34 hippocampal calbindin expression and impair cognition.

35 ating over time and some reverting to normal cognition.

36 microlevels of lithium, may influence human cognition.

37 neuronal circuits that mediate awareness and cognition.

38 es of the hippocampus have distinct roles in cognition.

39 patients compared with subjects with normal cognition.

40 ion in synaptic transmission, plasticity and cognition.

41 events, neuronal processing, and potentially cognition.

42 perspective on human rhythmic perception and cognition.

43 in basic activities of daily living or with cognition.

44 T: Working memory (WM) is a key component of cognition.

45 leterious effects of anxiety on higher-order cognition.

46 etal cortex of 39 human subjects with intact cognition.

47 literacy and adherence while controlling for cognition.

48 f implicit representations of possibility in cognition.

49 spective on a nutrition-driven modulation of cognition.

50 ural and functional connections that support cognition.

51 nderstanding of how vascular disease affects cognition.

52 = 5.7 x 10-5) were associated with residual cognition.

53 efined as an acute disorder of attention and cognition.

54 accumulation, preserving neuronal health and cognition.

55 bidity, including ambulation, toileting, and cognition.

56 sex-specific effects, and few examined child cognition.

57 rainment, and subtle connections with social cognition.

58 rest associated with value, self, and social cognitions.

59 increasing startle response and agoraphobic cognitions.

60 l should algorithms try to approximate human cognition?

61 erience sampling methodology to monitor mood/cognitions (10 times a day for 6 days) and calculated af

62 Examination revealed normal cognition (29 of 30 points on the Mini-Mental State Exam

63 e processed by a general cortical network of cognition, a network essential for conscious experiences

64 multi-component brain responsivity supports cognition across the adult lifespan, and the maintenance

65 on and activity were important predictors of cognition across the entire sample of depressed subjects

66 tive seropositivity to various microbes) and cognition/AD.

67 ident parkinsonism per SD decrease in global cognition, adjusted for age, sex, and study subcohort.

68 gher and lower SBP levels in the decrease in cognition; adjusted differences between the group with S

69 , diet, alcohol consumption, and smoking-and cognition, adjusting for demographic factors and chronic

70 Primary outcomes were cognition (Alzheimer's Disease Assessment Scale-cognitiv

71 fish consumption is associated with improved cognition among children, but the mediating pathways hav

72 ese differences may extend to differences in cognition among individuals.

73 bias, 6 enrolled healthy adults with normal cognition and 5 enrolled adults with MCI.

74 High plasma NFL correlated with poor cognition and AD-related atrophy (at baseline and longit

75 We studied the influence of cognition and APOE epsilon4 on the global and regional a

76 thout dementia and to study the influence of cognition and APOE epsilon4 status on this association.

77 , a key mediator in the regulation of social cognition and behavior, accumulates in cerebrospinal flu

78 coherent memory representations that support cognition and behavior.

79 fects of localized white matter pathology on cognition and behavior.

80 l circuits in the brain that are involved in cognition and behaviors, by using radiotracers that dete

81 mission and their function impacts learning, cognition and behaviour.

82 brain regions showing an association between cognition and both 18F-AV-1451 uptake and grey matter vo

83 e brain expansion and the evolution of human cognition and culture remain contentious despite extensi

84 Behavioral phenotypes related to cognition and depression were assessed at 15 and 24 mont

85 ciation of these discordant SBP targets with cognition and differences by race have not been systemat

86 the human cortex might be relevant to human cognition and disease.

87 m due to numerous associations with auditory cognition and dysfunction.

88 the neurobiological systems supporting human cognition and emerging computational systems capable of

89 ted to instead reduce reliance on individual cognition and exploration.

90 y endpoints of disease progression measuring cognition and function and hippocampal atrophy were obse

91 es of integrative brain functions supporting cognition and goal-directed behaviors, but electrophysio

92 naptic integrity that was linked to improved cognition and in an aggressive model of AD.

93 lead to poor functional outcome via impaired cognition and increased negative symptoms.

94 ors could disrupt neuronal processing during cognition and is relevant to the design of cognitive enh

95 new way to explore the white-matter role in cognition and its disturbances in neuropsychiatric disor

96 days of treatment we observed enhancement of cognition and learning behaviour.

97 ur developmental domains with 12 indicators: cognition and learning, social and communication, emotio

98 opercular network, which has a broad role in cognition and learning.

99 ine symptoms were related to improvements in cognition and left thalamic volume preservation followin

100 functional role of the prefrontal cortex in cognition and memory?

101 tter volume reductions in areas that control cognition and mood functions, even if such losses are ap

102 neuromodulators can have related effects on cognition and perception.

103 t of distributed thalamo-frontal circuits in cognition and point to the MD as a potential therapeutic

104 he association of neuRopsychiatric symptoms, cognition and qUality of life in ParkinSon disease); SP0

105 underline the importance of fully assessing cognition and quality of life in progressive multiple sc

106 lved in episodic memory, spatial navigation, cognition and stress responsiveness.

107 tive impairment (MCI) from those with normal cognition and those with Alzheimer disease (AD).

108 ns of synchrony to specific aspects of human cognition and to further develop the therapeutic manipul

109 what is known about the structure of social cognition and to suggest how further progress can be mad

110 ere, we investigated to what extent physical cognition and two domains of social cognition of dogs ha

111 nal accounts of hippocampal contributions to cognition, and a unique role for the human subiculum in

112 sideline assessment tool measuring symptoms, cognition, and balance in preadolescent children.

113 mmatory signaling in the brain affects mood, cognition, and behavior and is linked with the etiology

114 anatomy and function in the study of aging, cognition, and decision-making.SIGNIFICANCE STATEMENT Ag

115 al role in developing theory of mind, social cognition, and early relationships.

116 essing external sensory information, reward, cognition, and executive functions.

117 , including brain functions such as mood and cognition, and influence many neurological and psychiatr

118 that enhancing cognitive reserve may benefit cognition, and maintenance of cognitive health may be su

119 ns between iron exposures and mood, emotion, cognition, and memory; animal studies to determine epige

120 thy volunteers tACS can modulate perception, cognition, and motor function but the underlying neural

121 al capacities in activities of daily living, cognition, and physical performance were worse than thei

122 22q11DS to ND in comorbidities, functioning, cognition, and psychosis features across the full range

123 wn from psychology, neuroeconomics, embodied cognition, and social neuroscience.

124 otential benefits for reduction in delirium, cognition, and symptomatic stroke merit larger trials wi

125 minants might not sufficiently enhance child cognition, and that programmes addressing socioenvironme

126 The evolution of cognition, and thus the effect of culture on cognitive e

127 cess, is a fundamental organization of human cognition, and we tested this hypothesis using fMRI task

128 STATEMENT Working memory plays a key role in cognition, and working memory is impaired in several neu

129 al comparison processes are central to human cognition; and (2) intuitive physical knowledge is captu

130 Deficits in cognition are a core feature of many psychiatric conditi

131 Gender differences in social cognition are a long discussed issue, in particular thos

132 Visual dysfunction and poor cognition are highly prevalent among older adults; howev

133 ems, important in both synaptic function and cognition, are abnormal in psychiatric illnesses such as

134 Prior research on modal cognition asks how humans explicitly and deliberatively

135 Theories of creative cognition assert that the quantity of automatic associat

136 Poor global cognition at baseline was associated with a higher hazar

137 ests a broad impact of communication mode on cognition at large, beyond language processing.

138 f the pharmacological effects of nicotine on cognition, attention, and affective states.

139 ll to moderate effects were found for global cognition, attention, working memory, learning, and memo

140 ent [b] = -0.50 [0.15], P < .001) and global cognition (b = -0.27 [0.10], P = .009) at 15 months.

141 roach to identify genes influencing residual cognition, based on our prior observation that independe

142 ith accelerated decline in cognition (global cognition: beta=-0.029, P=0.003), which was similar afte

143 Recent experiments have shown that visual cognition blends current input with that from the recent

144 features, CSF biomarkers of neuronal injury, cognition, brain structure, and metabolism.

145 ive deterioration, others lower the level of cognition but exert relatively stable effects over time.

146 tion not only for numerical and mathematical cognition, but also for decision making.

147 28k is critical for hippocampal function and cognition, but its expression is markedly decreased in v

148 tween brain areas responsible for vision and cognition, but neurophysiological evidence for the invol

149 cortex (LPFC) is essential for higher-level cognition, but the nature of its interactions in support

150 es about how neural properties important for cognition can arise in a circuit and make clear experime

151 To directly test whether cognition can override fixation suppression, we alternat

152 ablished correlations among measures of EAP, cognition, clinical symptoms, and functional outcome.

153 brain, and support frameworks like embodied cognition, common coding, and sensorimotor contingency t

154 propose that, conceptually, the principle of cognition could amount to the existence of appropriate r

155 peutic approaches to the treatment of social cognition deficits, which are often observed in patients

156 yed impairments in peripheral metabolism and cognition, deficits in hippocampal-dependent spatial lea

157 nitive processes underlies the maturation of cognition during adolescence.

158 dge not only of technology but also of human cognition, emotion, and behavior.

159 oposed as a promising therapeutic target for cognition enhancement though the development of new anta

160 Loss of cognition even after survival is the salient feature of

161 remained robust after adjusting by baseline cognition except for dual-task gait cost when dichotomiz

162 rted idea is increasing genetic influence on cognition for children raised in higher socioeconomic st

163 18% of participants reverted to normal cognition from PD-MCI.

164 an impaired sweet-taste perception system on cognition functions were examined by analyzing synaptic

165 n was associated with accelerated decline in cognition (global cognition: beta=-0.029, P=0.003), whic

166 idence suggests a role for the cerebellum in cognition, granule cells are known to encode only sensor

167 a that synchronous neural activity underlies cognition has driven an extensive body of research in hu

168 The complex nature of human cognition has resulted in cognitive genomics lagging beh

169 individuals on demographics, comorbidities, cognition, hippocampal volume, and small-vessel disease

170 neurological deficits (most commonly memory/cognition impairment), hemiparesis, and visual defects.

171 Cognition improved in the FMT, but not the SOC, group.

172 zinon, malathion, and oxydemeton-methyl) and cognition in 7-year-old children.

173 We examined immune activation and cognition in a cohort of HIV-infected and uninfected wom

174 imaging biomarkers that were associated with cognition in active male professional fighters.

175 ld reveal imaging biomarkers associated with cognition in active professional fighters.

176 ynaptic plasticity and hippocampal-dependent cognition in aged mice.

177 tegic and global cerebral tissue injury, and cognition in carotid artery disease (CAD).

178 ffects of n-3 PUFAs on clinical symptoms and cognition in children and adolescent with ADHD; and (2)

179 mer's disease (AD) affect brain function and cognition in healthy adult samples may help to identify

180 associations between the gut microbiota and cognition in human infants.

181 Perception and cognition in infants have been traditionally investigate

182 e M1-M2 macrophage polarization and improved cognition in mild cognitive impairment patients on omega

183 ) is a safe and efficacious intervention for cognition in older adults.

184 nd self-reported visual impairment (VI) with cognition in older US adults.

185 eneral and domain-specific accounts of human cognition in order to situate attempts to identify gener

186 hanges in white matter microstructure impact cognition in part by disturbing the ability of neural as

187 easurement and treatment research to improve cognition in schizophrenia (MATRICS) consensus cognitive

188 s factor analysis on the Brief Assessment of Cognition in Schizophrenia battery yielded a single fact

189 Future efforts to improve cognition in schizophrenia by enhancing alpha7 nAChR fun

190 easurement and Treatment Research to Improve Cognition in Schizophrenia Consensus Cognitive Battery (

191 on reasoning and problem solving, and social cognition in schizophrenia patients.

192 ative Symptom Scale, the Brief Assessment of Cognition in Schizophrenia, and the Calgary Depression S

193 l unfolding of the link between language and cognition in the first year of life.

194 for PRMT8 in regulating neuron function and cognition in the mammalian brain.

195 ficantly deteriorated neurite morphology and cognition in these mice.

196 the evolution of all forms of human cultural cognition, including language.

197 n of human neural activity underlying social cognition, including negative emotion processing; howeve

198 Cognition indicates the probability of parkinsonism over

199 g and their further development for clinical cognition indications.

200 Overall cognition, individual cognitive domains, psychosocial fu

201 ke et al. to introduce key elements of human cognition into deep neural networks and future artificia

202 The brain pathways for spatial and temporal cognition involve overlapping and interacting systems th

203 Here, we used an operational test of cognition involving timing and behavioral inhibition and

204 Social cognition is a topic of enormous interest and much resea

205 read to neocortical regions and worsening of cognition is associated with beta-amyloid (Abeta).

206 n childhood violence victimization and later cognition is largely noncausal, in contrast to conventio

207 An important aspect of animal perception and cognition is learning to recognize relationships between

208 Cognition is one of the most flexible tools enabling ada

209 n between domain-general and domain-specific cognition is unsubstantiated.

210 longitudinal interactions between vision and cognition is warranted.

211 While GluA1 is important for cognition, it is not clear what the role of GluA1 is in

212 By tracing the emergence of a language-cognition link in infancy, this article reveals a dynami

213 directional interplay between physiology and cognition, LIP has served as fertile ground for developi

214 lly rich descriptions of changes in mood and cognition, loss of interest and anhedonia and emphasized

215 We also argue that causal cognition may be more general than social learning, whic

216 n the iCST and TAU groups in the outcomes of cognition (mean difference [MD] = -0.55, 95% CI -2.00-0.

217 95% CrI -48 to 32; high certainty evidence), cognition measured by short-term attention (-0.23 points

218 It defines foundational limits on cognition, memory, and behavior.

219 s evaluated via annual assessments of global cognition, memory, and executive function.

220 motor processing, affective and nonaffective cognition, mental health and personality, physical healt

221 ffect on height (low certainty evidence) and cognition (moderate certainty evidence).

222 computational role of stress in behavior and cognition more broadly.

223 eached consensus for the following outcomes: cognition, muscle and/or nerve function, physical functi

224 eling to examine the associations among EAP, cognition, negative symptoms, and functional outcome.

225 igh fat diet (HFD) on amyloid deposition and cognition of 12-months old APP23 mice, and correlated th

226 ognitive training for improving learning and cognition of children with learning disabilities.

227 physical cognition and two domains of social cognition of dogs have been affected by domestication an

228 nteracting persons robustly modulates social cognition of the perceiver.

229 Normative models of human cognition often appeal to Bayesian filtering, which prov

230 ography, gait speed over 4.57 m (15 ft), and cognition on the Mini-Mental State Examination and Trail

231 xposure was not associated with worse global cognition or executive function at 3 or 12 months in mod

232 r cognitive protection in adults with normal cognition or MCI.

233 cognitive protection in persons with normal cognition or MCI.

234 line, MCI, or dementia in adults with normal cognition or MCI.

235 red breathing was not associated with global cognition or memory.

236 ent dementia outcomes for adults with normal cognition or mild cognitive impairment (MCI).

237 e was no evidence that iCST has an effect on cognition or QoL for people with dementia.

238 Thus, cognition-or brain power-can be viewed as the zero-emiss

239 d for NNSS domains of ambulation (p=0.0622), cognition (p=0.0040) and speech (p=0.0423).

240 nexpected events interrupt action and impact cognition, partly at least, by recruiting this global su

241 The strength of this brain-to-cognition pathway in different white matter tracts was s

242 brain activation maps (n = 108) encompassing cognition, perception, action, and emotion behavioral do

243 gn linguistics and developmental research in cognition presented by Goldin-Meadow & Brentari (G-M&B)

244 howed worse cognition than females on social cognition, processing speed, verbal learning and visual

245 les on reasoning and problem solving, social cognition, processing speed, working memory, verbal lear

246 d anxiety phenotype based on the Agoraphobia Cognition Questionnaire (ACQ) in a sample of 1370 health

247 fusivity showed the closest correlation with cognition (R(2) = 0.311, P < .001) and promising diagnos

248 ep could be a candidate mediator of the fish-cognition relationship.

249 We also agree that human cognition relies heavily on structured relational repres

250 hanisms underlying these negative effects on cognition remain to be elucidated.

251 ive/negative classification, most effects on cognition remained.

252 ive performance, the effect of GHRD on human cognition remains unknown.

253 rgued that this salient feature of our moral cognition represents a profound puzzle for evolutionary

254 Primate cognition requires interaction processing.

255 They contribute to cognition, reward, mood, and nociception and are implica

256 valuated for their association with residual cognition: RNA levels of both UNC5C (estimated effect =

257 entions) on growth, educational achievement, cognition, school attendance, quality of life, and adver

258 of playing football with a reduced composite cognition score (-0.04 reduction in cognition vs all con

259 bal fluency, and word learning) and a global cognition score was derived from principal component ana

260 Median global cognition scores were similar in patients with surgery/a

261 comorbidities, baseline disability, baseline cognition, severity of illness, delirium, coma, sepsis,

262 during development and adulthood in shaping cognition, sociability, emotional behavior and stress su

263 balance in PFC pyramidal neurons as well as cognition, social interaction, and anxiety.

264 arent report of symptom number and severity, cognition (Standardized Assessment of Concussion-child v

265 portant component in the evolution of social cognition such as empathy.

266 stinctly human-signals in the brain: acts of cognition such as thoughts, intentions and memories.

267 e of the vmPFC in multiple aspects of social cognition, such as facial emotion recognition, theory-of

268 ocioenvironmental determinants with improved cognition suggests present reproductive, maternal, neona

269 ted significantly with subsequent decline in cognition (symbol-digit modality test r=-0.374, p<0.0001

270 er imitation in the general context of motor cognition, taking examples from both typical and atypica

271 so, male schizophrenia patients showed worse cognition than females on social cognition, processing s

272 wed that healthy controls exemplified better cognition than patients in both genders in all examined

273 urnish a more expansive account of numerical cognition that remedies these shortcomings.

274 on (eg, aerobic, strength, balance, and even cognition) that are particularly relevant for older pati

275 tary uses the dynamic of identity-protective cognition to pose a friendly challenge to Jussim (2012).

276 In older adults with normal cognition, training improves cognitive performance in th

277 isease, mild cognitive impairment, or normal cognition underwent T1-weighted MRI and florbetapir imag

278 We assessed cognition using a cognitive battery.

279 his study examined the gender differences of cognition using the measurement and treatment research t

280 omposite cognition score (-0.04 reduction in cognition vs all controls; 97.5% CI, -0.14 to 0.05) or a

281 Cognition was assessed using the Modified Mini-Mental St

282 Cognition was impaired in 22q11DS, but it did not correl

283 Cognition was measured by the Letter Number Span test an

284 ation data (n = 648), we found that residual cognition was related to differential DNA methylation of

285 METHODS AND "Residual cognition" was quantified by regressing out the effects

286 To identify genes influencing residual cognition, we leveraged neuropathological, genetic, epig

287 ild cognitive impairment (aMCI) or normative cognition, we measured AD-associated neurodegeneration (

288 ormation processing in nearly all domains of cognition, we sought to reconcile these competing accoun

289 ought to explain their effects on action and cognition, we suggest a unifying theory.

290 In considering how culture shapes cognition, we suggest that a process-level model of cogn

291 The changes in cognition were linked to an increase in Abeta and interf

292 No differences in spatial cognition were observed, but blasted rats as a whole exh

293 or the synthesis score and global and memory cognition were similar when stratified by 25(OH)D tertil

294 Multiple measures of victimization and cognition were used, and comparisons were made of cognit

295 This system is shown to be capable of basic cognition, which is illustrated numerically and with mus

296 at baseline correlated with baseline global cognition, while a similar trend was observed in the rig

297 redictive of poor technique, whereas age and cognition with poor lung function distinguished those wi

298 These findings provide new evidence linking cognition with WM via the reliance of neural synchroniza

299 gaging general rather than modality-specific cognition, with separate pathways that involved or bypas

300 Enhanced cognition without reduction of plaque pathology in paral