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1 ponds to the effect of 6 years aging on this cognitive domain.
2 egree of change from 12 to 72 months for any cognitive domain.
3 gicity did not contribute to variance in any cognitive domain.
4 entia (fvFTD) may reflect impairment in this cognitive domain.
5 veraged to compute composite scores for each cognitive domain.
6    Most participants had at least 1 impaired cognitive domain.
7  working memory, a related but non-numerical cognitive domain.
8 a for cognitive function improvement in each cognitive domain.
9 memory (P = .03), and visuospatial (P = .02) cognitive domains.
10 of cortical networks that influence multiple cognitive domains.
11 s, with consequences for sensory, motor, and cognitive domains.
12 iated with postoperative changes in specific cognitive domains.
13  performance tests in common that assessed 5 cognitive domains.
14 n and to explore specificity with respect to cognitive domains.
15  with decreased executive function and other cognitive domains.
16  could be fruitfully applied to a variety of cognitive domains.
17 reate a measure of global cognition and five cognitive domains.
18  which includes five measures assessing four cognitive domains.
19 ntal factors to influence specific executive cognitive domains.
20 ral brain damage and lower scores in various cognitive domains.
21 guage function but relative sparing of other cognitive domains.
22  as better function in multiple higher-order cognitive domains.
23 of brain regions supporting memory and other cognitive domains.
24 endence of age-related deficits in different cognitive domains.
25 -function relationships within other complex cognitive domains.
26 ional outcomes, including motor, sensory and cognitive domains.
27 egions may relate differentially to distinct cognitive domains.
28 nt formal testing of performance in multiple cognitive domains.
29 ariance in other developmental disorders and cognitive domains.
30  of the variation in performance in specific cognitive domains.
31 duced FA) was negatively correlated with all cognitive domains.
32 an accurately discriminate between different cognitive domains.
33 itive measures representing at least 2 other cognitive domains.
34  and third, to determine the most vulnerable cognitive domains.
35 ilar cognitive trajectories across different cognitive domains.
36 vocal, and few studies have examined several cognitive domains.
37 ates of cognitive decline across a number of cognitive domains.
38 bjects, LLD patients performed poorer in all cognitive domains.
39 on but may have synergistic effects on other cognitive domains.
40 d standardized testing may measure different cognitive domains.
41 chological test battery assessing a range of cognitive domains.
42 s the shared variation in performance across cognitive domains.
43 gical tests measuring performance in several cognitive domains.
44 ments showed progressive decline in multiple cognitive domains.
45 hy ageing has disparate effects on different cognitive domains.
46 lain differential ageing trajectories across cognitive domains.
47 l subregions have specialized roles in other cognitive domains.
48  are also associated with low scores in most cognitive domains.
49 ognition in multiple species and in multiple cognitive domains.
50 ubset completed standard NP assessment for 8 cognitive domains.
51 y unstable response patterns across multiple cognitive domains.
52 acute antidepressant treatment on a range of cognitive domains.
53  respectively) completed 12 tests covering 6 cognitive domains.
54 f atrophy influence decline across different cognitive domains.
55 ate summary measures of global cognition and cognitive domains.
56 n Aphasia Battery-Revised and tests of other cognitive domains.
57 omputerized CogState battery that assessed 5 cognitive domains.
58 e transient cognitive impairment in multiple cognitive domains.
59 etworks widely implicated in supporting many cognitive domains.
60 ogical measure and performance on other MCCB cognitive domains.
61 tion and accelerated decline across multiple cognitive domains.
62 , resulting in more rapid decline in several cognitive domains.
63 ATCV-1 proteins and had decreases in certain cognitive domains.
64  with performance decrements across multiple cognitive domains.
65 D, affecting sensorimotor, as well as higher cognitive, domains.
66 14.6% of children had low ECDI scores in the cognitive domain, 26.2% had low socioemotional scores, a
67 ecline by multiple indicators in two or more cognitive domains, a significantly higher rate than in t
68                               In analyses of cognitive domains, AD pathology (p = 0.006), PHFtau tang
69                    Careful separation of the cognitive domains affected by nicotinic stimulation has
70 better performance in the executive function cognitive domain after 36 months in apolipoprotein E (AP
71 adverse associations of tibia lead in 3 of 7 cognitive domains after adjustment for age, sex, race/et
72 on (P < 0.001), decline in all five specific cognitive domains (all P-values < 0.001), and to fluctua
73 as associated with widespread effects across cognitive domains, although these additional association
74 rment following systemic therapy in selected cognitive domains among some older patients with cancer.
75 t predictors were in the motor, imaging, and cognitive domains: an increase of one SD in total motor
76 hould attempt to use multiple tests for each cognitive domain and feature populations from ethnocultu
77  critically reviewed and tabulated by study, cognitive domain and follow-up period, with particular e
78 een diet scores and cognitive change in each cognitive domain and for the global score.
79 ing 19 neuropsychological tests from which 5 cognitive domain and global summary scores were derived,
80 n association appeared to exist depending on cognitive domain and SiN target or masker assessed.
81 bserved in 79% MCI with deficits in multiple cognitive domains and 31% amnesic MCI.
82 e used to construct composite scores for six cognitive domains and a global cognitive score.
83 than nonanthracycline regimens on particular cognitive domains and brain network connections.
84                    We also assessed multiple cognitive domains and compared findings with outcomes at
85 the contribution of the striatum across many cognitive domains and contexts.
86 seemingly disparate findings across multiple cognitive domains and could provide a unified account of
87 ole of self-care confidence between specific cognitive domains and heart failure self-care.
88  hearing loss to impaired performance across cognitive domains and increased risk for dementia diagno
89 dard deviation below normative means in >/=2 cognitive domains and neurocognitive symptoms).
90 tients with type 2 diabetes mellitus but the cognitive domains and patient groups most affected have
91 tistical approach is applicable across other cognitive domains and provides a key to understanding ti
92 ser portions of covariance between the other cognitive domains and schizophrenia were also found to b
93 tween men and women and how it varies across cognitive domains and subjective dimensions has not been
94 episodic and working memory but not in other cognitive domains), and it was elevated in those who dev
95 le HbA1c measurement at baseline, 1 test per cognitive domain, and potential geographic confounding o
96 rformed between 18F-AV-1451 binding and each cognitive domain, and we used the Biological Parametric
97 which mazes should be utilized to test these cognitive domains, and control measures to be used.
98 ever, efficacy varies across populations and cognitive domains, and little is known about the efficac
99 T is efficacious on global cognition, select cognitive domains, and psychosocial functioning in peopl
100           We used only one test to represent cognitive domains, and though a prototypical one, we nev
101                  Mild impairment in multiple cognitive domains appears to represent a more advanced d
102 ease and magnetic resonance variables, which cognitive domains are most vulnerable, the influence of
103 regions predicting individual differences in cognitive domains are predominantly located in regions o
104  z scores were calculated and clustered into cognitive domains as follows: episodic and working memor
105 gimens may have nonspecific effects on other cognitive domains as well as certain patient reported ou
106 s of BD is unclear, but many of the affected cognitive domains, as well as neuroanatomical abnormalit
107 in largely unknown for a variety of critical cognitive domains, as, for example, risky decision makin
108 r of genomic copies at the 16p11.2 locus and cognitive domains assessed in 62 deletion carriers, 44 d
109  association between LDL-C variability and 4 cognitive domains at month 30 in 4428 participants of PR
110 PFC interneuron-driven gamma oscillations in cognitive domains at the core of schizophrenia.
111 al tests were categorized according to eight cognitive domains: attention, executive functioning, inf
112 s clinical remission outcomes, on a range of cognitive domains: attention, response inhibition, execu
113 he PFC specifically produces deficits in the cognitive domain, but does not give rise to PFC-dependen
114 itative differences are apparent across many cognitive domains, but are especially obvious in episodi
115 nces contributed substantially to all of the cognitive domains, but intelligence and working memory w
116 ted for overall cognitive function and eight cognitive domains by calculating standardized mean diffe
117 ent studies indicate that impairments in two cognitive domains characterize the cognitive abnormaliti
118 ody pathology to dementia, global cognition, cognitive domains, cognitive decline and fluctuations in
119 n or performance in any of the five assessed cognitive domains, compared with the non-hallucinators.
120 estigate how atrophy patterns and non-memory cognitive domains contribute to memory impairment.
121                    Impairment in one or more cognitive domains could have important effects in the da
122                 Correlations showed that all cognitive domains declined with age.
123 a for amnestic MCI: 87 had memory plus other cognitive domain deficits and 21 had pure memory deficit
124 ely impaired patients with memory plus other cognitive domain deficits, rather than those with pure a
125 atial navigation and long-term memory, major cognitive domains dependent on hippocampal function, hav
126 cored lower than the healthy controls on all cognitive domains (eg, intelligence quotient [IQ], 76 [s
127 estimate -0.10 [SE 0.04], p=0.0096) and four cognitive domains (episodic memory -0.10 [0.04], p=0.017
128 on of language but relative sparing of other cognitive domains, especially memory.
129 g performance, as two potentially vulnerable cognitive domains essential in everyday life functioning
130  included measures of global cognition and 2 cognitive domains (executive functioning and memory), as
131 y fractional anisotropy (FA) and three broad cognitive domains (fluid intelligence, processing speed,
132 functions but relative preservation of other cognitive domains for at least the first two years of di
133 nt change in cognitive performance for eight cognitive domains from baseline to 12- and 72-month foll
134 ression model with a latent process for each cognitive domain: global cognition (Mini Mental State Ex
135          The CNVs do not all affect the same cognitive domains, hence the cognitive deficits that dri
136 trong association with impairment in several cognitive domains highlights the clinical relevance of w
137  the number of microbleeds and the number of cognitive domains impaired (r = 0.44, P = 0.03).
138  decline after 36 months in the visuospatial cognitive domain in APOE varepsilon4 allele non-carriers
139 ese limitations, we assessed a wide range of cognitive domains in a large sample (N=100) of 14- to 16
140 l learning is one of the most widely studied cognitive domains in neuroscience.
141 of interest (ROIs), is associated with these cognitive domains in normal adults and adults with mild
142 extent of impairment in social and nonsocial cognitive domains in patients with bipolar disorder comp
143 te matter injury to dysfunction in different cognitive domains in patients with multiple sclerosis ha
144 a, 10 tests were selected to represent seven cognitive domains in the MATRICS Consensus Cognitive Bat
145 t that a weakened link between emotional and cognitive domains in the psychopathic brain may combine
146 ementia; lower and more rapid decline in all cognitive domains including episodic memory and fluctuat
147 y childhood onset of dysfunction in multiple cognitive domains including socio-emotional function, sp
148 ng frequently demonstrates deficits in other cognitive domains, including language.
149 formance in a wide variety of linguistic and cognitive domains, including morphological agreements su
150 ubsequent decrease in performance in several cognitive domains, including ones involving recognition
151  a reduction of one SD in Stroop word score (cognitive domain) increased risk by 2.32 times (1.88-2.8
152 he findings into the major categories of the cognitive domains investigated, considering sample chara
153   We conclude that performance in the social-cognitive domain is equally impaired in male and female
154 nal compensation is well documented in other cognitive domains, it is presently unclear whether it ca
155  owing to different methods to assess SDB or cognitive domains, making it difficult to draw conclusio
156                 Data suggest that the social cognitive domain may be an important target for linking
157 asures as well as measures of mood, specific cognitive domains (memory, attention, and language), mot
158        In this study we investigate four key cognitive domains (mentalizing and emotion perception, e
159                                          The cognitive domains most frequently affected were attentio
160                                  Identifying cognitive domains most vulnerable to decline should guid
161 ssociated with lower performance in specific cognitive domains, most notably perceptual speed, separa
162                     These differences across cognitive domains need to be further investigated, by co
163 ect pre-morbid individual differences in the cognitive domain of interest.
164                                          The cognitive domain of spelling does not take place at the
165 ovided measures of accuracy and speed on the cognitive domains of abstraction and mental flexibility;
166 B score (R=0.52, P=0.033), as did individual cognitive domains of attention/vigilance, verbal learnin
167  is common but may not preferentially impact cognitive domains of episodic memory, attention, working
168  and examined each biomarker's relation with cognitive domains of episodic memory, executive function
169 tients had significantly lower scores in the cognitive domains of nonverbal intelligence, nonverbal m
170 ns jointly influencing schizophrenia and the cognitive domains of reaction time and verbal-numerical
171         Importantly, performance in specific cognitive domains of the task did not correlate with dif
172                                For the other cognitive domains, only verbal abilities differed betwee
173 mins had no significant effect on individual cognitive domains or global cognitive function or on cog
174 ngle supramodal mechanism common to multiple cognitive domains or modality-specific mechanisms idiosy
175 hought to be involved in processing in these cognitive domains, or involve possible compensatory proc
176  successful test performance across multiple cognitive domains (p < 0.05).
177                        However, the specific cognitive domains particularly vulnerable to inflammator
178 ded correlations with measures from multiple cognitive domains, particularly declarative and working
179 odules predominantly coactivated by specific cognitive domains (perception, action, and emotion, resp
180              However, in specific non-social cognitive domains, performance within ASC depends on sex
181                             Performance in 4 cognitive domains (perseveration, working memory, fluid
182                                        These cognitive domains pertain to memory and executive functi
183         These results suggest that these two cognitive domains play different roles in bipolar disord
184 n unadjusted analyses, lower scores on all 3 cognitive domains predicted poorer medication adherence
185                 When adjusting for the other cognitive domains, processing speed at age 8 (odds ratio
186                Overall cognition, individual cognitive domains, psychosocial function, and activities
187 tests assessing inhibition and set-shifting, cognitive domains related to freezing.
188 nks/day) showed superior performance in many cognitive domains relative to abstainers.
189 t contribute to disruption of this important cognitive domain remain unclear.
190 ch rats and mice are comparable in these two cognitive domains remains unclear.
191 ence, but persistence or recovery across all cognitive domains remains underresearched.
192 Regarding relative impairment across the two cognitive domains, results revealed a significant group-
193 sociation between PARKIN mutation status and cognitive domain scores and UPDRS-III scores.
194 ciations among salivary cortisol metrics and cognitive domain scores in an urban adult population.
195                                              Cognitive domain scores were calculated from executive,
196 notype was found to be associated with worse cognitive domain scores, with evidence of associations i
197 d the longitudinal relationships between key cognitive domains, selected a priori based on their asso
198  0.23) (11 studies, n=858) nor each of eight cognitive domains (SMDs=-0.03 to 0.11) (n=367-940) in th
199 od to promote skill learning across multiple cognitive domains, spanning executive functions, working
200 r CMBs was associated with impairment in all cognitive domains such as attention, language, visuospat
201 s that these structures are involved in more cognitive domains such as language processing.
202 is model and its application to other social cognitive domains such as Theory of Mind are discussed.
203                                Impairment in cognitive domains such as working memory and behavioral
204 ognitive function was impaired in 4 of the 6 cognitive domains tested (median global T-scores: 50.8 v
205 andard deviations better (p = 0.006) on this cognitive domain than those exceeding all risk factor gu
206 eported greater cognitive dysfunction in all cognitive domains than both norms and the comparison can
207             Working memory represents a core cognitive domain that is impaired in schizophrenia for w
208 gration is reorganized due to expertise in a cognitive domain that largely involves audiovisual integ
209 atients (20-25%), particularly in vulnerable cognitive domains that include memory.
210 emonstrated that repeated losses in the same cognitive domain, that is, successive changes from bette
211                                For component cognitive domains, the association with (pooled) SiN per
212 ggested that rTPJ is implicated in divergent cognitive domains, the demonstration of an involvement i
213 eeping with findings in other behavioral and cognitive domains, the response to an increase in presum
214 e administration and treatment confirmation, cognitive domains to be tested and which mazes should be
215 > .05), but the contribution of the specific cognitive domains to coding task performance differed si
216 mittee evaluated more than 90 tests in seven cognitive domains to identify the 36 most promising meas
217 ation of information processing in different cognitive domains to reach the capacity-limited consciou
218 er treatment (mean duration: 2.3 y) in the 4 cognitive-domain trials (1340 individuals); Mini-Mental
219 ed homocysteine concentrations by 28% in the cognitive-domain trials but had no significant effects o
220     The present study sought to characterise cognitive domains underlying a large test battery and fo
221     Changes in global cognition and specific cognitive domains up to 1 year post-TAVR were evaluated.
222           NP effects were most pronounced in cognitive domains vulnerable to MS: IFNbeta-1a had a sig
223  impairment in memory and at least one other cognitive domain was associated with greatest risk for p
224 de in any brain region and impairment in any cognitive domain was identified.
225        In the former, impairment in a single cognitive domain was observed in 40%, with the same numb
226 ognitive monitoring can operate on different cognitive domains, we ignore whether it involves a singl
227 , but those with impairment in more than one cognitive domain were more likely to progress or remain
228           Performance deficits in particular cognitive domains were associated with disproportionate
229 udy of dementia-free individuals, only a few cognitive domains were associated with established LOAD
230                                     Multiple cognitive domains were evaluated at midday and late afte
231 osite score (primary outcome) and individual cognitive domains were evaluated.
232 verse associations between tHcy and multiple cognitive domains were observed for individuals aged 60
233 variables from clinical, psychophysical, and cognitive domains were related to change in symptoms usi
234 t effect on self-care maintenance, the other cognitive domains were unrelated to self-care.
235 arison subjects on both social and nonsocial cognitive domains, whereas both groups differed from sch
236 s associated with lower function in multiple cognitive domains, whereas HS without TDP-43 did not hav
237 hildhood social class was significant in all cognitive domains, whereas within-pair estimates were at
238 identified performance deficits in all three cognitive domains, which were associated with regionally
239 was associated with lower performance in all cognitive domains, while higher estimated verbal intelli
240  in the LTI(+) vs the LTI(-) group in 6 of 7 cognitive domains with statistical significance reached
241 ger associated with performance in the other cognitive domains with the exception of visual learning.
242 thin brain regions known to subserve these 2 cognitive domains, with Met allele carriers having small
243 d mice allows for the assessment of multiple cognitive domains within the same testing environment.
244 Met allele carriers vs Val homozygotes) on 5 cognitive domain z scores and magnetic resonance imaging
245 SANS) total scores and change in the average cognitive domain z scores.

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