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1 cognitive therapy (thus, 71% did not accept cognitive therapy).
2 f a mood disorder were more likely to accept cognitive therapy.
3 more rapid remission than augmentation with cognitive therapy.
4 ide if necessary; others received individual cognitive therapy.
5 and comparable overall outcomes to standard cognitive therapy.
6 ssion rates were 46% for medication, 40% for cognitive therapy.
7 iversity, where medications were superior to cognitive therapy.
8 s to treat pharmacologically, but respond to cognitive therapies.
9 .61 to -10.23]; 11 trials and 287 patients), cognitive therapy (-13.36 [-18.40 to -8.21]; six trials
10 herapy for PTSD, 3 months of standard weekly cognitive therapy, 3 months of weekly emotion-focused su
11 eks, response rates in medications (50%) and cognitive therapy (43%) groups were both superior to the
12 e, < or =9) were significantly greater after cognitive therapy (58%) and phenelzine (58%) than after
16 changes in the brain have been observed with cognitive therapy and certain forms of meditation and le
17 reating pain and depression in FMS by adding cognitive therapy and coping skills components to a comp
18 f the conceptual and practical components of cognitive therapy and highlights some of the empirical e
19 of the depression-specific psychotherapies, cognitive therapy and interpersonal therapy, is reviewed
20 The authors compared the effectiveness of cognitive therapy and pharmacotherapy as second-step str
23 versus any augmentation strategy (including cognitive therapy), and a medication switch strategy onl
24 d all treatment strategies, 3% accepted only cognitive therapy, and 26% accepted cognitive therapy (t
25 step treatments: cognitive therapy versus no cognitive therapy, any switch strategy versus any augmen
27 These findings support the efficacy of meta-cognitive therapy as a viable psychosocial intervention.
29 augmentation was more rapidly effective than cognitive therapy augmentation of citalopram, whereas sw
33 psychotherapy for atypical depression or of cognitive therapy compared with a monoamine oxidase inhi
34 low-up evaluation, patients who had received cognitive therapy continued to improve, while those in t
37 risk of relapse/recurrence after acute phase cognitive therapy (CT), a continuation phase model of th
41 to second-step treatment, those who received cognitive therapy (either alone or in combination with c
42 ivational interviewing, relaxation training, cognitive therapy, exposure, problem-solving skills trai
43 8 (50%) of 197 participants were assigned to cognitive therapy for command hallucinations + treatment
44 ther people were assigned in a 1: 1 ratio to cognitive therapy for command hallucinations + treatment
45 e voices compared with 22 (28%) of 79 in the cognitive therapy for command hallucinations + treatment
46 and efficacy of a 7-day intensive version of cognitive therapy for PTSD and to investigate whether co
49 ) were randomly allocated to 7-day intensive cognitive therapy for PTSD, 3 months of standard weekly
50 rious adverse events: two in patients in the cognitive therapy group (one attempted overdose and one
51 ly greater proportion of members of the meta-cognitive therapy group demonstrated improvement compare
52 ed the efficacy of a 12-week manualized meta-cognitive therapy group intervention designed to enhance
53 total scores were consistently lower in the cognitive therapy group than in the treatment as usual g
54 cognitive therapy group, 77% of the standard cognitive therapy group, 43% of the supportive therapy g
55 atment/wait assessment, 73% of the intensive cognitive therapy group, 77% of the standard cognitive t
57 therapy for PTSD and to investigate whether cognitive therapy has specific treatment effects by comp
60 a critical window during which prophylactic cognitive therapy may benefit people at risk of schizoph
62 or insomnia, especially stimulus control and cognitive therapy, may be a helpful adjunct to treatment
64 ought to determine whether mindfulness-based cognitive therapy (MBCT) reduces distress in men with ad
65 g (n = 85) were randomly assigned to receive cognitive therapy (n = 28), a self-help booklet based on
67 ion [N=56] or buspirone [N=61]) or switch to cognitive therapy (N=36) or another antidepressant (N=86
68 ts to either augmentation of citalopram with cognitive therapy (N=65) or medication (N=117; either su
69 the cognitive theory of psychopathology and cognitive therapy of specific psychiatric disorders was
72 who consented to random assignment to either cognitive therapy or alternative pharmacologic strategie
73 ized, controlled trial comparing acute-phase cognitive therapy or clinical management plus either phe
74 otherwise randomly assigned to receive meta-cognitive therapy or supportive psychotherapy in a group
77 psychiatric severity and the superiority of cognitive therapy plus GDC compared with supportive-expr
78 counseling plus group drug counseling (GDC), cognitive therapy plus GDC, supportive-expressive therap
80 als were randomly assigned to receive either cognitive therapy plus treatment as usual (n=37), or tre
81 muted block sizes of four or six, to receive cognitive therapy plus treatment as usual, or treatment
93 -i incorporated at least 3 of the following: cognitive therapy, stimulus control, sleep restriction,
94 there is only 1 placebo-controlled trial of cognitive therapy, this trial fills a gap in the literat
95 ted only cognitive therapy, and 26% accepted cognitive therapy (thus, 71% did not accept cognitive th
97 The findings have implications for using cognitive therapy to reduce defeatist attitudes that may
99 ity of the following second-step treatments: cognitive therapy versus no cognitive therapy, any switc
100 entation of citalopram, whereas switching to cognitive therapy was better tolerated than switching to
105 and general anxiety, intensive and standard cognitive therapy were superior to supportive therapy.
106 s by comparing intensive and standard weekly cognitive therapy with an equally credible alternative t
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