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1 yos occurred between 14.5 and 16.5 days post-coitus.
2 pathogens and foreign male Ags introduced at coitus.
3 , vaginal coitus with use of a condom, or no coitus.
4 response at the female genital mucosa after coitus.
5 d epithelium and deeper stromal tissue after coitus.
6 radrenergic areas at 1 h, but not 2 h, after coitus.
7 ioallantoic fusion and die by 10.5 days post coitus.
8 ccur without coitus or with condom-protected coitus.
9 cells were also abundant and increased after coitus.
12 ent, allowing for silent transmission during coitus and contributing to high seroprevalence of HSV-2
15 ryo expresses RDH1 as early as 7.0 days post-coitus, and expression is especially intense within the
16 Fbw7-null mice die around 10.5 days post coitus because of a combination of deficiencies in hemat
20 ylinder stage at approximately 6.5 days post coitus (dpc), but development was arrested before 7.5 dp
21 and embryonic tissues 7.5 to 11.5 days after coitus (dpc), resolved an SP in each, and demonstrated t
23 jects reporting deep kissing with or without coitus had the same higher risk of infection than those
25 erated within 4 hr of sexual stimulation and coitus in female mice; prolactin-triggered oviductal flu
26 may influence cervical immune function after coitus in women, and potentially be a determinant of fer
28 t the participation of ACh in the control of coitus-induced ovulation may include activation of alpha
30 ctic acid content and acidity for CVM during coitus may contribute to both vaginal and penile protect
32 show severe defects at 3.5 d.p.c. (days post-coitus) morula/blastocyst stage and lethality before 8.5
37 n 48 h later, 12 h after unprotected vaginal coitus, vaginal coitus with use of a condom, or no coitu
39 pendent growth restriction at 15.5 days post-coitus while Egfr(Wa5) heterozygous placentas are only s
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