ライフサイエンスコーパス: cold agglutinin

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1 exclusive use of the VH 4-34 gene segment in cold agglutinins.

2 -three percent of pretransplant patients had cold agglutinins.

3 europathy, ophthalmoplegia, IgM paraprotein, cold agglutinins and disialosyl antibodies.

4 In conclusion, cold agglutinins are common in liver transplant patients

5 ne hemolytic anemia in which autoantibodies (cold agglutinins) bind to red blood cells (RBCs) at low

6 utoantibodies from a number of patients with cold agglutinin (CA) disease include both IgM hexamers a

7 Essentially all cold agglutinins (CA) with red blood cell I/i specificit

8 prevents induction of in vitro hemolysis by cold agglutinins (CA).

9 ing renal transplantation in the presence of cold agglutinins can cause graft malfunction.

10 Primary chronic cold agglutinin disease (CAD) is a well-defined clinicop

11 One such disorder is cold agglutinin disease (CAD), an autoimmune hemolytic a

12 In cold agglutinin disease (CAD), immunoglobulin M autoanti

13 nation fludarabine and rituximab for chronic cold agglutinin disease (CAD).

14 tified 89 patients from our institution with cold agglutinin disease from 1970 through 2012.

15 Cold agglutinin disease is a rare and poorly understood

16 ns, IgM red blood cell autoantibodies, cause cold agglutinin disease with hemolysis and microvascular

17 en) on erythrocytes and B lymphocytes, cause cold agglutinin disease, and are carried by 5% of naive

18 us, immune thrombocytopenic purpura, chronic cold agglutinin disease, IgM-mediated neuropathies and m

19 valuation and treatment improves outcomes in cold agglutinin disease.

20 l neuropathy, amyloidosis, cryoglobulinemia, cold-agglutinin disease, or evidence of disease transfor

21 l neuropathy, amyloidosis, cryoglobulinemia, cold-agglutinin disease, or transformed disease should b

22 However, the presence of cold agglutinins does not impact on outcome after liver

23 eventing CP activation, TNT003 also prevents cold agglutinin-driven generation of anaphylatoxins.

24 nation of the crystal structure of an anti-I cold agglutinin has revealed a hydrophobic patch in FR1

25 Cold agglutinins, IgM red blood cell autoantibodies, cau

26 ured the pretransplant presence and titer of cold agglutinins in 327 primary liver allograft recipien

27 The IgM antibodies were also cold agglutinins in 50% of cases.

28 fference between recipients with and without cold agglutinins in usage of blood products, postoperati

29 ent samples and showed that TNT003 prevented cold agglutinin-mediated deposition of complement opsoni

30 rine protease C1s, on CP activity induced by cold agglutinins on human RBCs.

31 However, the impact of cold agglutinins on the outcome of liver transplantation

32 M(low) IgD+ IGHV4-34+ B cells and removal of cold agglutinin self-reactivity by hypermutation, often

33 Therapy of cold agglutinin syndrome often is unsatisfactory.

34 mune hemolytic anemia of warm antibody type, cold agglutinin syndrome, paroxysmal cold hemoglobinuria

35 The monoclonal IgM cold agglutinins that bind to the I/i carbohydrate Ags o

36 Cold agglutinins were more common in patients with viral

37 to coat human red blood cells, sensitized by cold agglutinins, with C4 and C3.

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