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1 hepatitis C virus + serology, donor age and cold ischemic time.
2 ric transplantation and cases with prolonged cold ischemic time.
3 t on portal venous lactate concentration and cold ischemic time.
4 point of retrieval, and so does not include cold ischemic time.
5 diatric transplants and cases with prolonged cold ischemic times.
6 transplants (1.556, P<0.01), with prolonged cold ischemic time (1.097, P=0.03), for black recipients
9 rm ischemia time greater than 30 minutes and cold ischemic time also occurred over the same period.
10 0009), anastomotic time (P=0.0012), combined cold ischemic time and anastomotic time (P=0.00018), and
11 riate analyses accounting for the effects of cold ischemic time and donor age, Treg suppressive funct
12 s associated with first transplants, shorter cold ischemic time and operative time, and less intraope
14 e of edema, donor/recipient HLA-DR mismatch, cold ischemic time, and donor age were independently ass
20 phics, blood product use, primary diagnosis, cold ischemic time, and surgeon were similar between the
22 aphics, primary diagnosis, surgeon, warm and cold ischemic times, and blood product use were recorded
24 quency of poor ex vivo perfusion, had longer cold ischemic times, and were transplanted into older re
25 after circulatory death livers with extended cold ischemic time at 10 degrees C demonstrates superior
28 l, 149 renal transplants were performed with cold ischemic times (CI) greater than 16 hr (UW 87, HTK
29 median 30% versus 35%, P < 0.001) but longer cold ischemic time (CIT) (median 21.0 h versus 18.6 h, P
30 We also examined the relationship between cold ischemic time (CIT) and likelihood of surgical comp
37 of pulsatile perfusion (PP) across different cold ischemic times (CIT) within different donor groups
38 The heart transplant was completed first (cold ischemic time [CIT]: 131 minutes), followed by the
39 of diabetes, body mass index, waiting time, cold ischemic time, delayed graft function, and coronary
40 , but not long-term, graft survival, whereas cold ischemic time did not have statistically significan
41 tivariate logistic regression accounting for cold ischemic time, donor age, previous transplant, and
42 nes the association between transit time and cold ischemic time for adult, deceased kidney transplant
43 erence between the two groups with regard to cold ischemic time for organ storage, donor age, recipie
45 egionally and shared nationally, livers with cold ischemic time >12 hours, livers from hepatitis C vi
48 y was to examine whether older donor age and cold ischemic time interact to produce inferior allograf
50 y mass index <35, non-status 1 registration, cold ischemic time <8 hours, and either hepatocellular c
51 ause they are typically young and have short cold ischemic times, may be advantageous for HCV-infecte
52 DPI (median 30% vs. 35%, p<0.001) but longer cold ischemic time (median 21.0 hours vs. 18.6, p<0.001)
53 42.0%, P < .001), despite a slightly longer cold ischemic time (median: 14.8 vs 14.1 hours, P < .001
54 schemia/reperfusion injury by prolonging the cold ischemic time of the allograft did not affect the s
55 FEV1 achieved, age and gender of the donor, cold ischemic time of the graft, and matching of CMV ser
57 rshall's solution was associated with longer cold ischemic time, older donors, kidney-only donors, do
59 cytomegalovirus (CMV) status or blood type, cold ischemic times, or the incidence of outflow obstruc
62 erative transfusion requirements (P=0.0001), cold ischemic time (P<0.0001), use of roux-en-Y biliary
63 e of death (P=0.0053), donor age (P=0.0017), cold ischemic time (P=0.0009), anastomotic time (P=0.001
64 c artery thrombosis (P=0.0018) and prolonged cold ischemic time (P=0.034), were independent risk fact
65 of type (DCD vs DBD), donor age, steatosis, cold ischemic time, peak aspartate transaminase, day 5 b