ライフサイエンスコーパス: colitic

1 he neutrophil chemokine, KC was decreased in colitic A(2B)AR(-/-) mice.

2 lammatory response to luminally administered colitic agents in the face of intact systemic immune res

3 ation, 25.4 years) resected 70 polyps (60 in colitic and 10 in noncolitic mucosa).

4 In colitic and inflamed diverticular mucosa p27 was express

5 Both colitic and noncolitic T5KO exhibited transiently unstab

6 ring both the dextran sodium sulfate-induced colitic and the recovery phase.

7 opria mononuclear cells (LPMC) isolated from colitic animals blocked OVA IFN-gamma/IL-17 responses in

8 Adrenals of colitic animals were enlarged and hypervascularized.

9 ases in both IFN-gamma and TNF-alpha mRNA in colitic animals, independent of genotype.

10 consistency when administered to chronically colitic animals.

11 purified from the mesenteric lymph nodes of colitic BM-->tg epsilon26 mice were adoptively transferr

12 Serum Ab responses of colitic C3H/HeJBir and noncolitic parental C3H/HeJ mice

13 acterial-antigen-activated CD4+ T cells from colitic C3H/HeJBir but not from control C3H/HeJ mice int

14 omain with integrin alpha9, overexpressed on colitic CD103(-) DCs, suppressed the inflammatory potent

15 -1 blocked recruitment of lymphocytes to the colitic colon, and more importantly, these Abs significa

16 f BM-derived vascular smooth muscle cells in colitic compared with noninflamed regions.

17 amed areas, with striking upregulation under colitic conditions that correlated with increased expres

18 is of colonic inflammatory disease using the colitic cotton-top tamarin, an animal model of human ulc

19 Chronically colitic cotton-top tamarins were given either a cross-re

20 el of colitis revealed a correlation between colitic disease severity and tissue ITGB1 expression (R(

21 ly provide a protective adaptation to murine colitic disease.

22 ophages in recipient mice abrogated the anti-colitic effect of HdBc(7(d)).

23 imal Mlh1 promoter in hypoxic YAMC cells and colitic Gialpha2-/- crypts.

24 uced by several cytokines present within the colitic gut, including IL-22 and IFN-gamma.

25 udies with the DNBS model, revealed the anti-colitic HdBc(7(d)) was within the follicular B cell popu

26 Treatment of infected colitic IL-10 KO mice with anti-IL-12 p40 resulted in ma

27 Colonization of colitic IL-10-deficient mice with H. polygyrus suppresse

28 s and SIECs as well as CECs from healthy and colitic IL-2(-/-) mice to present antigen to T cells.

29 sodium sulfate exposure abolished the excess colitic inflammation and reduced colonic IL-5 and eosino

30 The effects of chemically blocking PI3K in colitic interleukin-10(-/-) mice were examined.

31 Colitic lesions are much more severe in C3H/HeJBir (C3H)

32 significantly smaller subset of T cells from colitic lesions expresses this ligand (24 +/- 2%).

33 ma and interleukin-10 from H. bilis-infected colitic mdr1a-/- mice.

34 of NF-kappaB p65 in lamina propria cells of colitic mice and in activated macrophages.

35 IL-1alpha was detected in the stool of colitic mice before IL-1beta.

36 Multiple strains of colitic mice had elevated serum anti-flagellin IgG2a res

37 icantly more alpha-V particles accumulate in colitic mice relative to (i) control mice (i.e., selecti

38 on of EGCG to dextran sodium sulfate-induced colitic mice significantly reduced the colonic myelopero

39 els and apoptosis were reduced in IL-10(-/-) colitic mice treated with neutralizing TNF mAb and the i

40 cremaster muscle microvessels of normal and colitic mice using a light/dye injury model.

41 roducing, bacterial-reactive CD4+ T cells in colitic mice was 1 out of 2,000 compared to 1 out of 20,

42 from the blood, the MLNs, and the spleen of colitic mice.

43 njury, and blunted thrombus formation in DSS colitic mice.

44 e monitored in untreated and muTF-Ab-treated colitic mice.

45 ke growth factor 1 expression was reduced in colitic mice.

46 A was similarly induced in WT and STAT6(-/-) colitic mice; however, we observed increased mRNA expres

47 serum insulin-like growth factor 1 levels in colitic mice; this preceded improvements in weight gain

48 he gut epithelium were a striking feature of colitic microbiota.

49 T cells were selectively accumulated in the colitic microenvironment and associated colon carcinoma.

50 ine the immune reactivity of a spontaneously colitic mouse strain, C3H/HeJBir, to epithelial, food, a

51 splasia (2), or carcinoma (1) in polyps from colitic mucosa.

52 xenograft formation will be useful to survey colitic patients at risk of developing cancer.

53 ifying potential tumor-initiating cells from colitic patients, suggesting that sphere and/or xenograf

54 n II into wild-type mice mimicked the severe colitic phenotype of RenTgMK mice, and treatment with lo

55 we assessed the putative generation of anti-colitic regulatory B cells following H. diminuta infecti

56 tibility to, and clinical manifestations of, colitic spondylarthritis.

57 es of AS associated with IBD (referred to as colitic spondylarthritis; comprising 78 with CD and 119

58 ble microbiotas, with lasting differences in colitic T5KO, while their noncolitic siblings stabilized

59 e, conditioned media from colonic tissues of colitic Th17 cell recipients induced IFN-gamma productio

60 osis of IECs occurred in the lower crypts of colitic tissue from humans and mice.

61 Serine proteinase levels increased in colitic tissue, with major species of 23 kd, 30 kd, and

62 rigins of sporadic colon cancer, the primary colitic tissues did not express any histologic evidence

63 ated inflammatory cytokine production in the colitic tissues.

64 imary FOXP3(+) T cells in blood, tumors, and colitic tissues.

65 MMP-2 and MMP-9 activation, was elevated in colitic tissues.

66 Colitic Tyk2(-/-) mice have less p-STAT3 in colon tissue

67 et genes is reduced in IECs from healthy and colitic Tyk2(-/-) mice.

68 igand (alpha-V) exhibit enhanced adhesion to colitic vasculature.

69 Activated protein C (APC), administered to colitic WT mice immediately prior to photoactivation, al

70 sed p-STAT6(+) IL-13-producing NKT cells, in colitic WT mice.