ライフサイエンスコーパス: colitogenic

1 IFN-gamma and IL-17 are colitogenic.

2 and cohousing experiments revealed that the colitogenic activity of this microbiota is transferable

3 signaling in T cells that contribute to its colitogenic activity.

4 t influences bacterial populations to become colitogenic, and this colitis is communicable to genetic

5 he host immune response as a way to identify colitogenic bacteria.

6 terized by increased levels of mucolytic and colitogenic bacteria.

7 litis by regulating neutrophil infiltration, colitogenic CD4(+) T cell activation, and proinflammator

8 - or dextran sulfate sodium-induced colitis, colitogenic CD4(+) T cells (T-helper 1 and Th17 cells) a

9 ith colitis, the local release of opioids by colitogenic CD4(+) T cells led to significant reduction

10 We investigated whether colitogenic CD4(+) T cells that accumulate in the inflam

11 lamina propria, and IFN-gamma production by colitogenic CD4(+) T cells.

12 The colitogenic Cdcs1 allele impairs innate immunity to bact

13 olonic fibrosis both spontaneously and under colitogenic conditions.

14 ression of toll-like receptors, and produced colitogenic cytokines, such as IL-6 and IL-23, after act

15 5(+) population is masked by the presence of colitogenic effector cells that cannot be suppressed.

16 he Enterobacteriaceae recently identified as colitogenic in a T-bet(-/-)Rag2(-/-) ulcerative colitis

17 inflammatory roles of Treg in the context of colitogenic innate immune response during pathogenic bac

18 ow that high IgA coating uniquely identifies colitogenic intestinal bacteria in a mouse model of micr

19 e IL-23 receptor defines an IL-10-regulated, colitogenic memory CD4+ T cell subset that is poised to

20 al and environmental factors, leading to pro-colitogenic perturbations of the host-commensal relation

21 r of the intestinal microbiome transfers the colitogenic phenotype from tryptophan starved animals to

22 Supporting their colitogenic phenotype, CD161(+) Th17 cells were found in

23 into the T(H)17 subset of helper T cells and colitogenic potential, in a manner dependent on transfor

24 iginal hypothesis, limits their survival and colitogenic potential.

25 This mechanism was investigated using the colitogenic, protease-secreting enteric microbe Enteroco

26 A major C3H-derived colitogenic QTL (Cdcs1) on chromosome (Chr.) 3 contribut

27 The resistant B6 background also contributed colitogenic QTL: Cdcs4 (Chr. 8), Cdcs5 (Chr. 17, MHC), a

28 ies qualitative features of the IL-23-driven colitogenic response by negatively regulating IL-23R exp

29 ies T-bet as a key modulator of IL-23-driven colitogenic responses in the intestine and has important

30 on of Treg cells in the colon and control of colitogenic responses.

31 ated colitis in wild-type mice, confirming a colitogenic role for the endogenous RAS.

32 Lachnospiraceae) and a greater abundance of colitogenic strains (of the family Erysipelotrichaceae).

33 In conclusion, a colitogenic susceptibility QTL on Chr. 3 has been shown

34 GFP) (EGFP, enhanced GFP) was crossed to the colitogenic susceptible strain IL-10-/- and derived into

35 cell populations or whether the IL-23-driven colitogenic T cell program regulates upstream hematopoie

36 s of T cell-mediated colitis, TRPV1 promoted colitogenic T cell responses and intestinal inflammation

37 g T cell proliferation in vitro and Th1-type colitogenic T cell responses in vivo.

38 atory functions for IFN-gamma to orchestrate colitogenic T cell responses through its distinct action

39 ygyrus before reconstitution with IL-10(-/-) colitogenic T cells are protected from colitis.

40 commensal bacteria in the donor mice drives colitogenic T cells into the Ag-experienced/memory T cel

41 n whether they act to prevent the priming of colitogenic T cells or actively control these cells as p

42 In mice, colitogenic Th1 and Th17 cells promote intestinal inflam

43 ed chronic enterocolitis via an imbalance of colitogenic Th1 cells and Treg cells.

44 Taken together, these data indicate that colitogenic Th1 cells enter into the Ag-experienced pool

45 In this study, we describe the presence of colitogenic Th1 cells within the CD4(+)CD45RB(low) popul

46 As OX40 also promoted the colitogenic Th1 response, its expression on T reg cells

47 Anti-IL-23p19 induced apoptosis in colitogenic Th17 cells in vitro and in vivo.

48 itis; however, a cellular mechanism by which colitogenic Th17 immunity arises in vivo remains unclear

49 T cells impaired mucosal iTreg and enhanced colitogenic Th17 responses in mice with CD4+ T cell-indu

50 mesenteric lymph nodes (mLNs), and limiting colitogenic Th17 responses.