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1 lation of collagen I, and down-regulation of collagen III.
2 en III levels and the ratio of Collagen I to Collagen III.
3 f collagen III was similar to that of native collagen III.
4 r T3-SCI, the MCA had more collagen I (42%), collagen III (24%), transforming growth factor beta (47%
5 (33%, P < 0.02), collagen I (28%, P < 0.01), collagen III (34%, P < 0.01), fibronectin (48%, P < 0.01
6 renal expression of fibronectin, collagen I, collagen III, alpha-SMA, PAI-1, fibroblast-specific prot
9 ccumulation of collagen III in the liver and collagen III and collagen IV in the heart; this is induc
11 splayed a reduced deposition of interstitial collagen III and fibronectin as well as total tissue col
13 f Matrix Metalloproteinase (TIMP-3, TIMP-4), collagen-III and elastin levels were measured in whole b
14 ulation and immunohistochemical staining for collagens III and IV and attenuated glomerular and tubul
15 (total lung collagen content, peribronchial collagens III and V) and significantly less peribronchia
16 tin, beta-myosin heavy chain), and fibrosis (collagen III), and increased LV prosurvival signaling (a
18 tial infiltrates and fibrosis, deposition of collagen III, and apoptosis of tubular epithelial cells.
19 disease progression, including fibronectin, collagen III, and chemoattractants that were identified
21 rix-associated genes, including collagen VI, collagen III, and tissue inhibitor of metalloproteases-1
23 ellular matrix, we identified collagen I and collagen III as well as mixtures of collagen I/collagen
24 d postradiation accumulation of interstitial collagen III but less myocardial degeneration in hearts
28 g of 32 distinct combinations of collagen I, collagen III, collagen IV, fibronectin, and laminin.
29 extracellular matrix molecules (collagen I, collagen III, collagen IV, laminin and fibronectin) on c
31 ntracutaneous fibrillin-rich microfibril and collagen III deposition and decreased mammalian target o
33 immunoreactive inflammation area, endomysial collagen III deposition, and hind limb grip strength.
39 ptor on the activation of human platelets by collagen; (iii) generated low-GPVI mice in which the alp
40 gh affinity integrin-binding motifs in human collagen III (GROGER and GLOGEN) and a third motif (GLKG
41 methods included whole-slide digital images: collagen III immunohistochemistry and a new technique us
42 alysis, an approximately twofold increase in collagen III immunolabeling within the interstitial comp
44 in intervention reverses the accumulation of collagen III in the liver and collagen III and collagen
45 protein connective tissue growth factor and collagen III in vitro and decreased pulmonary vascular f
46 ow that AMPK activation by metformin reduced collagen III levels and the ratio of Collagen I to Colla
47 as potentially the best for clinical trials: collagen III morphometry and visual assessment of trichr
48 sion of collagen I mRNA lagged expression of collagen III mRNA and peaked at day 42 after PRK with a
51 t radiation-induced elevation of total gland collagen III mRNA was also blocked by neutralizing antib
52 n in previously published referent controls; collagen III N-terminal propeptide (5.6 [4.3-6.9] ng/mL)
53 galectin-3, matrix metalloproteinase-2, and collagen III N-terminal propeptide were measured in the
54 n content (P<0.0001), collagen I (P<0.0001), collagen III (P<0.0001), and myocyte size (P<0.0001).
55 ICP) and the amino terminal peptide from pro-collagen III (PIIINP), correlate with left atrial (LA) f
57 (WT) mice, associated with lower collagen I, collagen III, plasminogen activator inhibitor (PAI-1), a
59 -alpha2 integrin-subunit antibody and type I collagen, (iii) recombinant alpha2-I domain bound the wi
66 by trypsin, a property also seen for native collagen III, supporting a local structural relaxation o
69 chimera containing six triplets (P7-P11') of collagen III was similar to that of native collagen III.
70 ing the entire triplehelical domain of human collagen III was used to locate binding sites for the co
71 titial area stained with picrosirius red and collagen III) was significantly increased 14 d after UUO
72 onal studies suggest that the interaction of collagen III with its receptor GPR56 inhibits neural mig
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