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1 r 2 (DDR2) results from its interaction with collagen type II.
2 ing and expression of the chondrocyte marker collagen type II.
3 not to another arthritis-related Ag, bovine collagen type II.
4 hondrocyte-specific enhancer in the gene for collagen type II.
5 developing cartilage is preceded by that of collagen type II.
6 type II and 12 had IgG binding to denatured collagen type II.
7 ature of IgG binding to native and denatured collagen type II.
8 y immunization with either bovine or chicken collagen type II.
9 mRNA encoding chondrocyte markers including collagen type II, aggrecan, and Pln was elevated in 10T1
11 here that the mouse Col2alpha1-Cre promoter (collagen, type II, alpha 1) is active not only in chondr
13 stimulation of annexin V channel activity by collagen (types II and X) binding can explain how MV are
14 th RA, 12 patients had IgG binding to native collagen type II and 12 had IgG binding to denatured col
15 ly distributed chondrocytes in a matrix with collagen type II and aggrecan and had significantly grea
17 re, thickness, density, chondrocyte numbers, collagen type II and aggrecan, and mechanical properties
18 e two major components of the cartilage ECM, collagen type II and aggrecan, as in vivo substrates for
19 cellular matrix, and laminin-alpha1 enhanced collagen type II and reduced collagen type I expression
22 ces have been identified in the homotrimeric collagen types II and III, however, it is unclear how VW
24 of 0.3 M MgCl(2) and immunogold labeling for collagen types II and IX were analyzed by transmission e
25 he mechanism of covalent interaction between collagen types II and IX, we present experimental eviden
26 linated filaggrin, fibrinogen, vimentin, and collagen type II), and studied its effects on arthritic
27 sitive proliferating chondroprogenitors, and collagen type II- and type X-expressing chondrocytes.
31 lesions and decreased biomarkers of type II collagen (type II collagen neoepitope) and aggrecan (pep
33 nnective tissue growth factor (CTGF), type I collagen, type II collagen, TGFbeta receptor 1 (TGFbetaR
34 89.8% LacZ(+) cells in peripheral blood and collagen type II costaining with LcZ revealed an average
35 e II fibrillar architecture and indicate the collagen type II cross-link organization, which is cruci
36 /Re) embryos showed high levels of Sox-9 and collagen type II expression and lack of development of h
38 hat may add cohesion to a weakened, existing collagen type II fibril network as part of a chondrocyte
39 ce of autoantibodies to native and denatured collagen type II has been reported in some patients with
40 IA was induced in DBA/1 mice by injection of collagen type II in complete Freund adjuvant, and cell s
41 ides and the overall D-periodic structure of collagen type II in native tissues, data that were furth
45 nts the majority of IgG binding to denatured collagen type II is caused by macromolecular complexes f
46 r chondrocytic markers, such as aggrecan and collagen types II, IX, X, and XI, and by Alcian blue sta
47 , wet weights, wet weight GAG fractions, and collagen type II levels were obtained in bioreactor-grow
49 C-MS/MS assay has been developed to quantify collagen type II neoepitope peptides as biomarkers of co
50 analyses showed that cells produced abundant collagen type-II on type-II scaffolds and collagen type-
51 ering via their beta(1) integrin receptor to collagen type II or chondroadherin was profoundly and ra
52 RTL construct covalently linked with bovine collagen type II peptide (bCII257-270) suitable for use
53 The assay was used to measure the levels of collagen type II peptides in the urine of both clinical
57 ed in the oxidation of amine groups and, for collagen type II, the specific decrease in Pyd cross-lin
58 .6 +/- 22.0% of the IgG binding to denatured collagen type II was in the excluded protein fractions.
60 was shown that a 45-mer peptide fragment of collagen type II with five hydroxyprolines (OH) can be s
62 PRP treated cartilage, with chondromodulin, collagen types II/X downregulated, deiodinase II and net
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