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1 mechanical hyperalgesia and inflammation in collagen-induced arthritis.
2 h atRA suppressed progression of established collagen-induced arthritis.
3 odels of arthritis: Ab-induced arthritis and collagen-induced arthritis.
4 ining of joint tissue derived from mice with collagen-induced arthritis.
5 ts in a mouse model of autoimmune arthritis, collagen-induced arthritis.
6 llular and humoral immunity and the onset of collagen-induced arthritis.
7 re resistant to LPS-induced septic shock and collagen-induced arthritis.
8 and caspase-1(-/-) mice were susceptible to collagen-induced arthritis.
9 c T cells at the site of inflammation during collagen-induced arthritis.
10 ental autoimmune encephalomyelitis (EAE) and collagen-induced arthritis.
11 c (Tg) mice expressing HLA-DRB1*0401 develop collagen-induced arthritis.
12 ed animals in a systemic model of RA disease collagen-induced arthritis.
13 as attenuated disease in MRL/lpr mice and in collagen-induced arthritis.
14 Salmonella-CFA/I can protect DBA/1 mice from collagen-induced arthritis.
15 d its impact on the clinical model of bovine collagen-induced arthritis.
16 a T cells have also been found to exacerbate collagen-induced arthritis.
17 ptor family has a role in the progression of collagen-induced arthritis.
18 reduced the clinical and histologic signs of collagen-induced arthritis.
19 mmune mice, including T cells from mice with collagen-induced arthritis.
20 for B cells during the elicitation phase of collagen-induced arthritis.
21 of experimental models of arthritis, such as collagen-induced arthritis.
22 use anti-mouse CD20 mAbs in a mouse model of collagen-induced arthritis.
23 ively correlated with paw swelling in murine collagen-induced arthritis.
24 optosis in thymocytes and pre-disposition to collagen-induced arthritis.
25 reduce the incidence and severity of murine collagen-induced arthritis.
26 efficacy in some animal models, most notably collagen-induced arthritis.
27 tic effect of sST2-Fc in the murine model of collagen-induced arthritis.
28 ansgenic mice that display susceptibility to collagen-induced arthritis.
29 of arthritis and in its ability to suppress collagen-induced arthritis.
30 therapeutic evaluation in a murine model of collagen-induced arthritis.
31 rotective, whereas loss of IL-12 exacerbates collagen-induced arthritis.
32 nase 9 on the development and progression of collagen-induced arthritis.
33 ice susceptible to porcine and human type II collagen-induced arthritis.
34 xperimental autoimmune encephalomyelitis and collagen-induced arthritis.
35 ad the capacity to prevent and/or ameliorate collagen-induced arthritis.
36 tage and IgG2a Ab's at the effector phase of collagen-induced arthritis.
37 pared following induction of monoclonal anti-collagen-induced arthritis.
38 are normally susceptible to the induction of collagen-induced arthritis.
39 d particles inhibited development of type II collagen-induced arthritis.
40 combined anti-TNF-alpha/anti-CD4 therapy in collagen-induced arthritis.
41 lays a more prominent role than IL-1alpha in collagen-induced arthritis.
42 o strongly alleviated disease development in collagen-induced arthritis.
43 exposure-dependent fashion in a rat model of collagen-induced arthritis.
44 immunocytokine F8-IL4 in the mouse model of collagen-induced arthritis.
45 ria significantly aggravated the severity of collagen-induced arthritis.
46 8-IL4 was able to cure mice with established collagen-induced arthritis.
47 on of arthritis in the experimental model of collagen-induced arthritis.
48 hase that contributes to the pathogenesis of collagen-induced arthritis, a disease model of human rhe
50 (EGFR), reduces the severity of established collagen-induced arthritis, a mouse model of RA, and tha
51 e the contribution of NK cells to regulating collagen-induced arthritis, a well-characterized preclin
52 60(p216) but not Qa-1-Qdm strongly inhibited collagen-induced arthritis, an animal model of human rhe
53 close analog, SNX-4414, was evaluated in rat collagen-induced arthritis and adjuvant-induced arthriti
55 s of T15-NAb also efficiently inhibited both collagen-induced arthritis and anti-collagen II Ab-media
56 gen-specific CD4(+) T cell expansion in both collagen-induced arthritis and autoimmune diabetes mouse
57 BA/1 background were completely resistant to collagen-induced arthritis and exhibited absence of join
58 rity of the inflammatory autoimmune diseases collagen-induced arthritis and experimental autoimmune e
59 imal models of inflammatory diseases such as collagen-induced arthritis and experimental autoimmune e
61 in experimental allergic encephalomyelitis, collagen-induced arthritis and inflammatory bowel diseas
62 ved dendritic cells and in vivo in models of collagen-induced arthritis and inflammatory bowel diseas
65 ese data indicate that ICOS is essential for collagen-induced arthritis and may suggest novel means f
68 ed up-regulation, mice developed more severe collagen-induced arthritis and spontaneous glomerular im
69 of disease severity and early onset in both collagen-induced arthritis and spontaneous lupus nephrit
70 IL-deficient mice are also hypersensitive to collagen-induced arthritis and streptozotocin-induced di
71 itis and its neutralization inhibited murine collagen-induced arthritis and suppressed IFN-gamma and
72 n that is up-regulated in the early stage of collagen-induced arthritis and that exacerbates arthriti
73 exhibited significantly reduced severity of collagen-induced arthritis and this was accompanied by a
75 essed LPS-induced TNF production, alleviated collagen-induced arthritis, and blocked gout formation i
77 odent models of polyarthritis: rat and mouse collagen-induced arthritis, and mouse collagen antibody-
78 sitivity model, suppress the onset on murine collagen-induced arthritis, and reduce the severity of e
79 mmune disease: systemic lupus erythematosus, collagen-induced arthritis, and serum-transfer arthritis
80 ted intravenously into mice with established collagen-induced arthritis, and the effects on leukocyte
81 essfully but had increased susceptibility to collagen-induced arthritis, and the levels of huTLR8 cor
82 osomes was able suppress the onset of murine collagen-induced arthritis as well as reduce severity of
83 iency of mPGES-1 inhibits the development of collagen-induced arthritis, at least in part, by blockin
84 their ability to prevent the development of collagen induced arthritis, both the engineered DR1-CII-
85 luorescence in arthritic joints of mice with collagen-induced arthritis by both noninvasive fluoresce
86 I vaccination of DBA/1 mice protects against collagen-induced arthritis by stimulating TGF-beta- and
87 of EC144 blocked disease development in rat collagen-induced arthritis by suppressing the inflammato
89 s study was to investigate the regulation of collagen-induced arthritis by tryptophan catabolism medi
90 iator for PGRN-mediated anti-inflammation in collagen-induced arthritis by using PGRN and IL-10 genet
91 models of autoimmune disease; in particular, collagen induced arthritis (CIA) and experimental autoim
96 y modified DC to suppress established murine collagen-induced arthritis (CIA) after i.v. delivery.
97 nd growth of bony spurs was performed in rat collagen-induced arthritis (CIA) and adjuvant-induced ar
98 level was dramatically elevated during mouse collagen-induced arthritis (CIA) and blocking TWEAK by a
99 complementary experimental RA mouse models, collagen-induced arthritis (CIA) and collagen antibody-i
100 letely halts and reverses the development of collagen-induced arthritis (CIA) and discover cellular a
101 SSRIs, fluoxetine and citalopram, in murine collagen-induced arthritis (CIA) and in a human ex vivo
102 n vivo mechanism of action was determined in collagen-induced arthritis (CIA) and local joint TLR5 li
104 3 receptor alpha-chain, developed attenuated collagen-induced arthritis (CIA) and reduced ex vivo col
105 the development of pain in the rat model of collagen-induced arthritis (CIA) and to evaluate the con
106 her mucosal administration of EtxB can block collagen-induced arthritis (CIA) and to investigate the
107 perimental autoimmune encephalomyelitis, and collagen-induced arthritis (CIA) are associated with typ
108 actor Ag I (CFA/I) fimbriae protects against collagen-induced arthritis (CIA) by eliciting two regula
109 keyhole limpet hemocyanin (KLH) study and a collagen-induced arthritis (CIA) disease model of rheuma
111 dy to test the hypothesis that resistance to collagen-induced arthritis (CIA) in C57BL/6 (B6) mice is
112 he objective of these studies was to examine collagen-induced arthritis (CIA) in C57BL/6 mice transge
113 stress protein, BiP, prevented induction of collagen-induced arthritis (CIA) in HLA-DRB*0101+/+ (HLA
115 he IL-15R on the prevention and treatment of collagen-induced arthritis (CIA) in mice and probed the
116 of a soluble ligand of CD200R in established collagen-induced arthritis (CIA) in mice and to analyze
118 A major difference between RA in humans and collagen-induced arthritis (CIA) in mice is the lack of
120 r interaction precipitated susceptibility to collagen-induced arthritis (CIA) in mice normally resist
121 te the significance of these autoantibodies, collagen-induced arthritis (CIA) in mice was used to est
128 ll depletion was further studied in a murine collagen-induced arthritis (CIA) model and a respiratory
131 s (PADs), are known to develop in the murine collagen-induced arthritis (CIA) model of inflammatory a
145 r (uPA) reduces arthritis progression in the collagen-induced arthritis (CIA) mouse model to an exten
148 of wild type (WT) versus CRIg(-/-) mice with collagen-induced arthritis (CIA) or K/BxN serum transfer
149 nvadosomal structures by synovial cells from collagen-induced arthritis (CIA) rats and RA patients.
152 of the MEK/ERK MAP kinase pathway in murine collagen-induced arthritis (CIA) using the selective MEK
156 t and to identify their mechanism of action, collagen-induced arthritis (CIA) was therapeutically tre
159 h carrageenan-induced acute inflammation and collagen-induced arthritis (CIA) were used to assess eff
160 the ability of these cells to control murine collagen-induced arthritis (CIA), a model for rheumatoid
162 lator of inflammation was examined in murine collagen-induced arthritis (CIA), a model of rheumatoid
163 e such product, ES-62, is protective against collagen-induced arthritis (CIA), a model of rheumatoid
164 inflammation, contribute to the progress of collagen-induced arthritis (CIA), a mouse model for RA.
165 toring and quantifying joint inflammation in collagen-induced arthritis (CIA), a mouse model for RA.
166 ene knockout (KO) mice to assess its role in collagen-induced arthritis (CIA), a mouse model of human
167 arthritis shows any similarities with RA or collagen-induced arthritis (CIA), a mouse model of RA.
168 mmunoregulatory protein for the treatment of collagen-induced arthritis (CIA), a mouse model of rheum
169 ets, Vgamma1(+) and Vgamma4(+) cells, during collagen-induced arthritis (CIA), a mouse model that sha
170 eptibility of CD73-deficient C57BL/6 mice to collagen-induced arthritis (CIA), a well-established mou
171 table for use in DBA/1LacJ mice that develop collagen-induced arthritis (CIA), an animal model of hum
172 and evaluated their therapeutic potential in collagen-induced arthritis (CIA), an animal model of RA.
173 markedly increased during the acute stage of collagen-induced arthritis (CIA), an animal model of rhe
174 elayed the onset and reduced the severity of collagen-induced arthritis (CIA), and reduced paw tissue
175 immune responses, led to early resolution of collagen-induced arthritis (CIA), and reduced spontaneou
179 We assessed pain behavior and mechanisms in collagen-induced arthritis (CIA), the model of preclinic
181 cient in IL-1Ra (IL-1Ra(-/-)) were bred with collagen-induced arthritis (CIA)-susceptible DBA/1 mice
208 response gene with CII elicits an arthritis (collagen-induced arthritis [CIA]) that resembles rheumat
209 nificantly reduced incidence and severity of collagen-induced arthritis compared with wild-type (WT)
212 nic mice developed more severe and sustained collagen-induced arthritis due to the enhanced Th1 respo
214 3, ASC, and caspase-1 in the pathogenesis of collagen-induced arthritis have not been characterized.
215 arthritis, IFN-gamma is required, whereas in collagen-induced arthritis, IL-17 is necessary for devel
216 , HNK stabilized the severity of symptomatic collagen-induced arthritis in both CD40-LMP1 transgenic
217 elayed the onset and reduced the severity of collagen-induced arthritis in DBA/1 mice by induction of
218 n DBA/1-TCR-beta transgenic mice, as well as collagen-induced arthritis in DBA/1 mice, both animal mo
220 e autoimmune inflammatory process of chronic collagen-induced arthritis in DBA/1-TCR-beta transgenic
221 We showed that this peptide ameliorates collagen-induced arthritis in DBA/1J mice and protects a
223 protein LTbetaR-Ig blocked the induction of collagen-induced arthritis in mice and adjuvant arthriti
224 nistration of ERG240 reduces the severity of collagen-induced arthritis in mice and crescentic glomer
225 eptide significantly reduced the severity of collagen-induced arthritis in mice by reducing levels of
226 25 in vivo down-regulates the progression of collagen-induced arthritis in mice via targeting of the
227 8 kinase activities and clinical symptoms of collagen-induced arthritis in mice were all diminished.
228 itis in the models of adjuvant arthritis and collagen-induced arthritis in rats and mice, respectivel
231 We studied serotonin's involvement during collagen-induced arthritis in wild-type and Tph1(-/-) mi
232 ne models of chronic inflammation, including collagen-induced arthritis, inflammatory bowel disease,
236 4) enhanced Th17 expansion, and in mice with collagen-induced arthritis it facilitated disease onset,
237 ecule on T lymphocytes, CD40(+) T cells from collagen-induced arthritis mice were examined in paralle
238 so potent in treating established disease in collagen-induced arthritis mice with beneficial effects
239 eutic action of tolerogenic DCs in a type II collagen-induced arthritis model and to investigate thei
241 ing of GLPG0634 in a therapeutic set-up in a collagen-induced arthritis model in rodents resulted in
242 n vivo, because inhibition of C5orf30 in the collagen-induced arthritis model markedly accentuated jo
243 odel of systemic lupus erythematosus and the collagen-induced arthritis model of rheumatoid arthritis
245 cells to the inflammatory site, we used the collagen-induced arthritis model to determine the freque
247 fficacy in adjuvant-induced arthritis model, collagen-induced arthritis model, and allergic asthma mo
251 xin-1 increased skewing in Th1 cells; in the collagen-induced arthritis model, treatment of mice with
258 Here, we examine the therapeutic effects in collagen-induced arthritis of the second generation PAD
260 e, IL-35 inhibited clinical manifestation of collagen-induced arthritis or could cease further diseas
261 ntigens emulsified in adjuvant oils, such as collagen-induced arthritis or experimental autoimmune en
262 of the disease (as appears to be the case in collagen-induced arthritis) or T cells are required for
265 kine analysis shows that mice protected from collagen-induced arthritis produce lower amounts of Th1
266 n up to 1 month before the clinical onset of collagen-induced arthritis protected mice from severe jo
269 s biomarkers in RA, enhance tissue damage in collagen-induced arthritis (see the related article begi
270 Crucially, ES-62 was also found to suppress collagen-induced arthritis severity and progression when
271 tained anti-inflammatory activity in vivo in collagen induced arthritis studies in mouse but had redu
275 This motif is similar to one recognized by collagen-induced arthritis-susceptible HLA-DR1- and HLA-
279 es are a prerequisite for the development of collagen-induced arthritis, their presence is insufficie
280 ivo studies were conducted in a rat model of collagen-induced arthritis to evaluate the therapeutic p
287 termine the role CD28 costimulation plays in collagen-induced arthritis, we have generated DQ8 transg
289 ation, late phase cutaneous anaphylaxis, and collagen-induced arthritis were aggravated, in contrast
296 er inflammation of the knee joints following collagen-induced arthritis, when compared with control m
297 *0401 gene contributes to the development of collagen-induced arthritis, whereas DRB1*0402 prevents t
298 ays a dominant role in the susceptibility to collagen-induced arthritis, whereas FcgammaRIIb on B cel
299 ent development of inflammation in mice with collagen-induced arthritis, whereas sorted CD25+CD39+CD4
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