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1  mechanical hyperalgesia and inflammation in collagen-induced arthritis.
2 h atRA suppressed progression of established collagen-induced arthritis.
3 odels of arthritis: Ab-induced arthritis and collagen-induced arthritis.
4 ining of joint tissue derived from mice with collagen-induced arthritis.
5 ts in a mouse model of autoimmune arthritis, collagen-induced arthritis.
6 llular and humoral immunity and the onset of collagen-induced arthritis.
7 re resistant to LPS-induced septic shock and collagen-induced arthritis.
8  and caspase-1(-/-) mice were susceptible to collagen-induced arthritis.
9 c T cells at the site of inflammation during collagen-induced arthritis.
10 ental autoimmune encephalomyelitis (EAE) and collagen-induced arthritis.
11 c (Tg) mice expressing HLA-DRB1*0401 develop collagen-induced arthritis.
12 ed animals in a systemic model of RA disease collagen-induced arthritis.
13 as attenuated disease in MRL/lpr mice and in collagen-induced arthritis.
14 Salmonella-CFA/I can protect DBA/1 mice from collagen-induced arthritis.
15 d its impact on the clinical model of bovine collagen-induced arthritis.
16 a T cells have also been found to exacerbate collagen-induced arthritis.
17 ptor family has a role in the progression of collagen-induced arthritis.
18 reduced the clinical and histologic signs of collagen-induced arthritis.
19 mmune mice, including T cells from mice with collagen-induced arthritis.
20  for B cells during the elicitation phase of collagen-induced arthritis.
21 of experimental models of arthritis, such as collagen-induced arthritis.
22 use anti-mouse CD20 mAbs in a mouse model of collagen-induced arthritis.
23 ively correlated with paw swelling in murine collagen-induced arthritis.
24 optosis in thymocytes and pre-disposition to collagen-induced arthritis.
25  reduce the incidence and severity of murine collagen-induced arthritis.
26 efficacy in some animal models, most notably collagen-induced arthritis.
27 tic effect of sST2-Fc in the murine model of collagen-induced arthritis.
28 ansgenic mice that display susceptibility to collagen-induced arthritis.
29  of arthritis and in its ability to suppress collagen-induced arthritis.
30  therapeutic evaluation in a murine model of collagen-induced arthritis.
31 rotective, whereas loss of IL-12 exacerbates collagen-induced arthritis.
32 nase 9 on the development and progression of collagen-induced arthritis.
33 ice susceptible to porcine and human type II collagen-induced arthritis.
34 xperimental autoimmune encephalomyelitis and collagen-induced arthritis.
35 ad the capacity to prevent and/or ameliorate collagen-induced arthritis.
36 tage and IgG2a Ab's at the effector phase of collagen-induced arthritis.
37 pared following induction of monoclonal anti-collagen-induced arthritis.
38 are normally susceptible to the induction of collagen-induced arthritis.
39 d particles inhibited development of type II collagen-induced arthritis.
40  combined anti-TNF-alpha/anti-CD4 therapy in collagen-induced arthritis.
41 lays a more prominent role than IL-1alpha in collagen-induced arthritis.
42 o strongly alleviated disease development in collagen-induced arthritis.
43 exposure-dependent fashion in a rat model of collagen-induced arthritis.
44  immunocytokine F8-IL4 in the mouse model of collagen-induced arthritis.
45 ria significantly aggravated the severity of collagen-induced arthritis.
46 8-IL4 was able to cure mice with established collagen-induced arthritis.
47 on of arthritis in the experimental model of collagen-induced arthritis.
48 hase that contributes to the pathogenesis of collagen-induced arthritis, a disease model of human rhe
49 act of TNF inhibition on IL-17 production in collagen-induced arthritis, a model of RA.
50  (EGFR), reduces the severity of established collagen-induced arthritis, a mouse model of RA, and tha
51 e the contribution of NK cells to regulating collagen-induced arthritis, a well-characterized preclin
52 60(p216) but not Qa-1-Qdm strongly inhibited collagen-induced arthritis, an animal model of human rhe
53 close analog, SNX-4414, was evaluated in rat collagen-induced arthritis and adjuvant-induced arthriti
54                                           In collagen-induced arthritis and Ag-induced arthritis, alt
55 s of T15-NAb also efficiently inhibited both collagen-induced arthritis and anti-collagen II Ab-media
56 gen-specific CD4(+) T cell expansion in both collagen-induced arthritis and autoimmune diabetes mouse
57 BA/1 background were completely resistant to collagen-induced arthritis and exhibited absence of join
58 rity of the inflammatory autoimmune diseases collagen-induced arthritis and experimental autoimmune e
59 imal models of inflammatory diseases such as collagen-induced arthritis and experimental autoimmune e
60 the inflamed joint in arthritis in mice with collagen-induced arthritis and humans with RA.
61  in experimental allergic encephalomyelitis, collagen-induced arthritis and inflammatory bowel diseas
62 ved dendritic cells and in vivo in models of collagen-induced arthritis and inflammatory bowel diseas
63      miR-34a-deficient mice are resistant to collagen-induced arthritis and interaction of DCs and T
64 mit the incidence of arthritis in a model of collagen-induced arthritis and joint inflammation.
65 ese data indicate that ICOS is essential for collagen-induced arthritis and may suggest novel means f
66 d and activated in the synovium of mice with collagen-induced arthritis and patients with RA.
67 re increased in the lymph nodes of mice with collagen-induced arthritis and SLE.
68 ed up-regulation, mice developed more severe collagen-induced arthritis and spontaneous glomerular im
69  of disease severity and early onset in both collagen-induced arthritis and spontaneous lupus nephrit
70 IL-deficient mice are also hypersensitive to collagen-induced arthritis and streptozotocin-induced di
71 itis and its neutralization inhibited murine collagen-induced arthritis and suppressed IFN-gamma and
72 n that is up-regulated in the early stage of collagen-induced arthritis and that exacerbates arthriti
73  exhibited significantly reduced severity of collagen-induced arthritis and this was accompanied by a
74      PGRN-deficient mice were susceptible to collagen-induced arthritis, and administration of PGRN r
75 essed LPS-induced TNF production, alleviated collagen-induced arthritis, and blocked gout formation i
76                 In contact hypersensitivity, collagen-induced arthritis, and inflammation induced by
77 odent models of polyarthritis: rat and mouse collagen-induced arthritis, and mouse collagen antibody-
78 sitivity model, suppress the onset on murine collagen-induced arthritis, and reduce the severity of e
79 mmune disease: systemic lupus erythematosus, collagen-induced arthritis, and serum-transfer arthritis
80 ted intravenously into mice with established collagen-induced arthritis, and the effects on leukocyte
81 essfully but had increased susceptibility to collagen-induced arthritis, and the levels of huTLR8 cor
82 osomes was able suppress the onset of murine collagen-induced arthritis as well as reduce severity of
83 iency of mPGES-1 inhibits the development of collagen-induced arthritis, at least in part, by blockin
84  their ability to prevent the development of collagen induced arthritis, both the engineered DR1-CII-
85 luorescence in arthritic joints of mice with collagen-induced arthritis by both noninvasive fluoresce
86 I vaccination of DBA/1 mice protects against collagen-induced arthritis by stimulating TGF-beta- and
87  of EC144 blocked disease development in rat collagen-induced arthritis by suppressing the inflammato
88  that IL-4 DCs exert a therapeutic effect on collagen-induced arthritis by targeting IL-17.
89 s study was to investigate the regulation of collagen-induced arthritis by tryptophan catabolism medi
90 iator for PGRN-mediated anti-inflammation in collagen-induced arthritis by using PGRN and IL-10 genet
91 models of autoimmune disease; in particular, collagen induced arthritis (CIA) and experimental autoim
92                                              Collagen induced arthritis (CIA) is an animal model of R
93 acious (ED(50) < or = 0.01 mg/kg) in the rat collagen induced arthritis (CIA) model.
94  efficacious (ED 50 = 0.05 mg/kg) in the rat collagen induced arthritis (CIA) model.
95 s marked changes in the preclinical phase of collagen induced arthritis (CIA).
96 y modified DC to suppress established murine collagen-induced arthritis (CIA) after i.v. delivery.
97 nd growth of bony spurs was performed in rat collagen-induced arthritis (CIA) and adjuvant-induced ar
98 level was dramatically elevated during mouse collagen-induced arthritis (CIA) and blocking TWEAK by a
99  complementary experimental RA mouse models, collagen-induced arthritis (CIA) and collagen antibody-i
100 letely halts and reverses the development of collagen-induced arthritis (CIA) and discover cellular a
101  SSRIs, fluoxetine and citalopram, in murine collagen-induced arthritis (CIA) and in a human ex vivo
102 n vivo mechanism of action was determined in collagen-induced arthritis (CIA) and local joint TLR5 li
103                                              Collagen-induced arthritis (CIA) and proteoglycan-induce
104 3 receptor alpha-chain, developed attenuated collagen-induced arthritis (CIA) and reduced ex vivo col
105  the development of pain in the rat model of collagen-induced arthritis (CIA) and to evaluate the con
106 her mucosal administration of EtxB can block collagen-induced arthritis (CIA) and to investigate the
107 perimental autoimmune encephalomyelitis, and collagen-induced arthritis (CIA) are associated with typ
108 actor Ag I (CFA/I) fimbriae protects against collagen-induced arthritis (CIA) by eliciting two regula
109  keyhole limpet hemocyanin (KLH) study and a collagen-induced arthritis (CIA) disease model of rheuma
110 s in RA, NK cells in the joints of mice with collagen-induced arthritis (CIA) express RANKL.
111 dy to test the hypothesis that resistance to collagen-induced arthritis (CIA) in C57BL/6 (B6) mice is
112 he objective of these studies was to examine collagen-induced arthritis (CIA) in C57BL/6 mice transge
113  stress protein, BiP, prevented induction of collagen-induced arthritis (CIA) in HLA-DRB*0101+/+ (HLA
114         Adjuvant-induced arthritis (AIA) and collagen-induced arthritis (CIA) in Lewis rats were used
115 he IL-15R on the prevention and treatment of collagen-induced arthritis (CIA) in mice and probed the
116 of a soluble ligand of CD200R in established collagen-induced arthritis (CIA) in mice and to analyze
117                        We report herein that collagen-induced arthritis (CIA) in mice is inhibited by
118  A major difference between RA in humans and collagen-induced arthritis (CIA) in mice is the lack of
119                                              Collagen-induced arthritis (CIA) in mice is widely used
120 r interaction precipitated susceptibility to collagen-induced arthritis (CIA) in mice normally resist
121 te the significance of these autoantibodies, collagen-induced arthritis (CIA) in mice was used to est
122  inhibitor BMS-345541 is efficacious against collagen-induced arthritis (CIA) in mice.
123                                              Collagen-induced arthritis (CIA) is a model of inflammat
124                                              Collagen-induced arthritis (CIA) is a polygenic model of
125                          The murine model of collagen-induced arthritis (CIA) is an established disea
126                                          Rat collagen-induced arthritis (CIA) is an experimental mode
127             MSCs transplantation (MSCT) into collagen-induced arthritis (CIA) mice prevented arthriti
128 ll depletion was further studied in a murine collagen-induced arthritis (CIA) model and a respiratory
129                                          The collagen-induced arthritis (CIA) model and DR-1 transgen
130                  Oral efficacy in the murine collagen-induced arthritis (CIA) model for rheumatoid ar
131 s (PADs), are known to develop in the murine collagen-induced arthritis (CIA) model of inflammatory a
132 asts and pathological bone loss in the mouse collagen-induced arthritis (CIA) model of RA.
133                                         In a collagen-induced arthritis (CIA) model, neovascularizati
134       In this study we show, using the DBA/1 collagen-induced arthritis (CIA) model, that conjugation
135                                    Using the collagen-induced arthritis (CIA) model, we have studied
136                                       In the collagen-induced arthritis (CIA) model, while ectopic ex
137 nti-arthritic effect of engineered MSCs in a collagen-induced arthritis (CIA) model.
138 o evaluate MKK-3 and MKK-6 deficiency in the collagen-induced arthritis (CIA) model.
139 n vitro and to control disease severity in a collagen-induced arthritis (CIA) model.
140 itic joints of TSG-6 transgenic mice, in the collagen-induced arthritis (CIA) model.
141 ro, and joint inflammation and damage in the collagen-induced arthritis (CIA) model.
142  the role of CRP in a mouse model of RA, the collagen-induced arthritis (CIA) model.
143 sing delayed-type hypersensitivity (DTH) and collagen-induced arthritis (CIA) models in mice.
144                                          The collagen-induced arthritis (CIA) mouse model is the most
145 r (uPA) reduces arthritis progression in the collagen-induced arthritis (CIA) mouse model to an exten
146                                    We used a collagen-induced arthritis (CIA) mouse model to study th
147 ) and the development of bone erosion in the collagen-induced arthritis (CIA) mouse.
148 of wild type (WT) versus CRIg(-/-) mice with collagen-induced arthritis (CIA) or K/BxN serum transfer
149 nvadosomal structures by synovial cells from collagen-induced arthritis (CIA) rats and RA patients.
150                                              Collagen-induced arthritis (CIA) represents an animal mo
151                                              Collagen-induced arthritis (CIA) represents an animal mo
152  of the MEK/ERK MAP kinase pathway in murine collagen-induced arthritis (CIA) using the selective MEK
153                                      Passive collagen-induced arthritis (CIA) was induced in Jnk2(-/-
154                                              Collagen-induced arthritis (CIA) was induced in male DBA
155                            In these studies, collagen-induced arthritis (CIA) was induced in P-select
156 t and to identify their mechanism of action, collagen-induced arthritis (CIA) was therapeutically tre
157          This study in mice with established collagen-induced arthritis (CIA) was undertaken to deter
158                 All pathological features of collagen-induced arthritis (CIA) were significantly redu
159 h carrageenan-induced acute inflammation and collagen-induced arthritis (CIA) were used to assess eff
160 the ability of these cells to control murine collagen-induced arthritis (CIA), a model for rheumatoid
161                 Although the pathogenesis of collagen-induced arthritis (CIA), a model of rheumatoid
162 lator of inflammation was examined in murine collagen-induced arthritis (CIA), a model of rheumatoid
163 e such product, ES-62, is protective against collagen-induced arthritis (CIA), a model of rheumatoid
164  inflammation, contribute to the progress of collagen-induced arthritis (CIA), a mouse model for RA.
165 toring and quantifying joint inflammation in collagen-induced arthritis (CIA), a mouse model for RA.
166 ene knockout (KO) mice to assess its role in collagen-induced arthritis (CIA), a mouse model of human
167  arthritis shows any similarities with RA or collagen-induced arthritis (CIA), a mouse model of RA.
168 mmunoregulatory protein for the treatment of collagen-induced arthritis (CIA), a mouse model of rheum
169 ets, Vgamma1(+) and Vgamma4(+) cells, during collagen-induced arthritis (CIA), a mouse model that sha
170 eptibility of CD73-deficient C57BL/6 mice to collagen-induced arthritis (CIA), a well-established mou
171 table for use in DBA/1LacJ mice that develop collagen-induced arthritis (CIA), an animal model of hum
172 and evaluated their therapeutic potential in collagen-induced arthritis (CIA), an animal model of RA.
173 markedly increased during the acute stage of collagen-induced arthritis (CIA), an animal model of rhe
174 elayed the onset and reduced the severity of collagen-induced arthritis (CIA), and reduced paw tissue
175 immune responses, led to early resolution of collagen-induced arthritis (CIA), and reduced spontaneou
176                                           In collagen-induced arthritis (CIA), preventive and therape
177                            Upon induction of collagen-induced arthritis (CIA), tau-tg mice displayed
178                                 In mice with collagen-induced arthritis (CIA), the disease-modulating
179  We assessed pain behavior and mechanisms in collagen-induced arthritis (CIA), the model of preclinic
180              When transferred into mice with collagen-induced arthritis (CIA), this T cell line speci
181 cient in IL-1Ra (IL-1Ra(-/-)) were bred with collagen-induced arthritis (CIA)-susceptible DBA/1 mice
182  a therapeutic agent was evaluated in murine collagen-induced arthritis (CIA).
183 cally delivered into the ankles of mice with collagen-induced arthritis (CIA).
184 matory joint disease in mice challenged with collagen-induced arthritis (CIA).
185 was down-modulated during the progression of collagen-induced arthritis (CIA).
186  in the chronic inflammatory arthritis model collagen-induced arthritis (CIA).
187 is adaptor is required for the initiation of collagen-induced arthritis (CIA).
188 g an anti-mouse CD70 Ab in a murine model of collagen-induced arthritis (CIA).
189    Administration of misoprostol exacerbated collagen-induced arthritis (CIA).
190 motif (FibgammaDelta5), were challenged with collagen-induced arthritis (CIA).
191  in a model of rheumatoid arthritis, namely, collagen-induced arthritis (CIA).
192 imatinib potently prevents and treats murine collagen-induced arthritis (CIA).
193 emonstrated that p53 is protective in murine collagen-induced arthritis (CIA).
194 J mice to study the role of CD44 directly in collagen-induced arthritis (CIA).
195 8.CD4(-/-) mice were resistant to developing collagen-induced arthritis (CIA).
196 with bovine type II collagen (CII) to elicit collagen-induced arthritis (CIA).
197 uated in cultured Jurkat cells and in murine collagen-induced arthritis (CIA).
198 ion, including rheumatoid arthritis (RA) and collagen-induced arthritis (CIA).
199 anti-inflammatory proteins that can suppress collagen-induced arthritis (CIA).
200 omyelitis, diabetes, and, in our laboratory, collagen-induced arthritis (CIA).
201  used to locate CRIg expression in mice with collagen-induced arthritis (CIA).
202 testing in an in vivo model of inflammation, collagen-induced arthritis (CIA).
203 destructive arthritis in the murine model of collagen-induced arthritis (CIA).
204 collagen and yet have a reduced incidence of collagen-induced arthritis (CIA).
205 using a mouse model of autoimmune arthritis, collagen-induced arthritis (CIA).
206 ed natural Treg (nTreg) cells on established collagen-induced arthritis (CIA).
207 ed in human rheumatoid arthritis (RA) and in collagen-induced arthritis (CIA).
208 response gene with CII elicits an arthritis (collagen-induced arthritis [CIA]) that resembles rheumat
209 nificantly reduced incidence and severity of collagen-induced arthritis compared with wild-type (WT)
210                                           In collagen-induced arthritis, deficiency of endogenous Anx
211   In addition, 31 demonstrated efficacy in a collagen-induced arthritis disease model in rats.
212 nic mice developed more severe and sustained collagen-induced arthritis due to the enhanced Th1 respo
213                  We demonstrated that murine collagen-induced arthritis follows a similar sequential
214 3, ASC, and caspase-1 in the pathogenesis of collagen-induced arthritis have not been characterized.
215 arthritis, IFN-gamma is required, whereas in collagen-induced arthritis, IL-17 is necessary for devel
216 , HNK stabilized the severity of symptomatic collagen-induced arthritis in both CD40-LMP1 transgenic
217 elayed the onset and reduced the severity of collagen-induced arthritis in DBA/1 mice by induction of
218 n DBA/1-TCR-beta transgenic mice, as well as collagen-induced arthritis in DBA/1 mice, both animal mo
219 vivo in an autoimmune disease model, namely, collagen-induced arthritis in DBA/1 mice.
220 e autoimmune inflammatory process of chronic collagen-induced arthritis in DBA/1-TCR-beta transgenic
221      We showed that this peptide ameliorates collagen-induced arthritis in DBA/1J mice and protects a
222 i-inflammation could be unveiled by studying collagen-induced arthritis in Gal-1(-/-) mice.
223  protein LTbetaR-Ig blocked the induction of collagen-induced arthritis in mice and adjuvant arthriti
224 nistration of ERG240 reduces the severity of collagen-induced arthritis in mice and crescentic glomer
225 eptide significantly reduced the severity of collagen-induced arthritis in mice by reducing levels of
226 25 in vivo down-regulates the progression of collagen-induced arthritis in mice via targeting of the
227 8 kinase activities and clinical symptoms of collagen-induced arthritis in mice were all diminished.
228 itis in the models of adjuvant arthritis and collagen-induced arthritis in rats and mice, respectivel
229               Susceptibility to experimental collagen-induced arthritis in rodents is dependent on MH
230 on of DRB1*0402 did not lead to induction of collagen-induced arthritis in transgenic mice.
231    We studied serotonin's involvement during collagen-induced arthritis in wild-type and Tph1(-/-) mi
232 ne models of chronic inflammation, including collagen-induced arthritis, inflammatory bowel disease,
233                                              Collagen-induced arthritis is a commonly accepted model
234                 Chronic relapsing homologous collagen-induced arthritis is a valuable model for ident
235                                              Collagen-induced arthritis is a well-validated, but stra
236 4) enhanced Th17 expansion, and in mice with collagen-induced arthritis it facilitated disease onset,
237 ecule on T lymphocytes, CD40(+) T cells from collagen-induced arthritis mice were examined in paralle
238 so potent in treating established disease in collagen-induced arthritis mice with beneficial effects
239 eutic action of tolerogenic DCs in a type II collagen-induced arthritis model and to investigate thei
240  rat and dog and was found to be active in a collagen-induced arthritis model in rats.
241 ing of GLPG0634 in a therapeutic set-up in a collagen-induced arthritis model in rodents resulted in
242 n vivo, because inhibition of C5orf30 in the collagen-induced arthritis model markedly accentuated jo
243 odel of systemic lupus erythematosus and the collagen-induced arthritis model of rheumatoid arthritis
244                           We used the murine collagen-induced arthritis model to characterize the imm
245  cells to the inflammatory site, we used the collagen-induced arthritis model to determine the freque
246 mice, but not DBA/1J (the strain used in the collagen-induced arthritis model) or DBA/2J mice.
247 fficacy in adjuvant-induced arthritis model, collagen-induced arthritis model, and allergic asthma mo
248                                   In a mouse collagen-induced arthritis model, EC144 also suppressed
249                            In a prophylactic collagen-induced arthritis model, it exhibited an anti-i
250                                  In a murine collagen-induced arthritis model, JNJ-54271074 dose-depe
251 xin-1 increased skewing in Th1 cells; in the collagen-induced arthritis model, treatment of mice with
252                               In the type II collagen-induced arthritis model, we observed a signific
253 ion and cartilage destruction in this murine collagen-induced arthritis model.
254 m 50 to 80% and is milder than the classical collagen-induced arthritis model.
255 role of ICOS in rheumatoid arthritis using a collagen-induced arthritis model.
256 /PD assays and highly efficacious in a mouse collagen-induced arthritis model.
257                  Efficacy of GLPG0634 in the collagen-induced arthritis models was comparable to the
258  Here, we examine the therapeutic effects in collagen-induced arthritis of the second generation PAD
259                                In a model of collagen-induced arthritis on the C57BL/6 background, th
260 e, IL-35 inhibited clinical manifestation of collagen-induced arthritis or could cease further diseas
261 ntigens emulsified in adjuvant oils, such as collagen-induced arthritis or experimental autoimmune en
262 of the disease (as appears to be the case in collagen-induced arthritis) or T cells are required for
263                                 In mice with collagen-induced arthritis, orally administered PCI-3276
264                              Remarkably, the collagen-induced arthritis phenotype of Ccr2-/- mice mim
265 kine analysis shows that mice protected from collagen-induced arthritis produce lower amounts of Th1
266 n up to 1 month before the clinical onset of collagen-induced arthritis protected mice from severe jo
267 -AIA in DA.F344 monocongenic rats containing collagen-induced arthritis QTLs.
268           Treatment of mice with established collagen-induced arthritis reduced the severity of arthr
269 s biomarkers in RA, enhance tissue damage in collagen-induced arthritis (see the related article begi
270  Crucially, ES-62 was also found to suppress collagen-induced arthritis severity and progression when
271 tained anti-inflammatory activity in vivo in collagen induced arthritis studies in mouse but had redu
272                                              Collagen induced arthritis studies in mouse have demonst
273                       In paws taken from the collagen-induced arthritis study, the bones of vehicle-t
274  DBA/2J) to investigate the genetic basis of collagen-induced arthritis susceptibility.
275   This motif is similar to one recognized by collagen-induced arthritis-susceptible HLA-DR1- and HLA-
276                                Using a novel collagen-induced arthritis synovial window model, we dem
277 hat Fas-deficient mice developed less-severe collagen-induced arthritis than did control mice.
278       When administered in vivo to mice with collagen-induced arthritis, the SE ligand significantly
279 es are a prerequisite for the development of collagen-induced arthritis, their presence is insufficie
280 ivo studies were conducted in a rat model of collagen-induced arthritis to evaluate the therapeutic p
281              The effect of IDO deficiency on collagen-induced arthritis was assessed in vivo by admin
282 signal, a TCR-transgenic (Tg) mouse model of collagen-induced arthritis was developed.
283 ed disease, the efficacy of rh-FcgammaRIA in collagen-induced arthritis was evaluated.
284                                              Collagen-induced arthritis was induced in mice hypomorph
285                  In addition, we report that collagen-induced arthritis was not affected by the funct
286           While analyzing gene expression in collagen-induced arthritis, we discovered that a poorly
287 termine the role CD28 costimulation plays in collagen-induced arthritis, we have generated DQ8 transg
288                                In a model of collagen-induced arthritis, we use S100A8/S100A9 imaging
289 ation, late phase cutaneous anaphylaxis, and collagen-induced arthritis were aggravated, in contrast
290 T cell activation and its ability to prevent collagen-induced arthritis were analyzed.
291           Penetrance, onset, and severity of collagen-induced arthritis were recorded in DUSP-1+/+ an
292                The incidence and severity of collagen-induced arthritis were significantly reduced an
293                                    Mice with collagen-induced arthritis were treated with depleting a
294                        Male DBA/1J mice with collagen-induced arthritis were treated with etanercept.
295  models of delayed-type hypersensitivity and collagen-induced arthritis were used.
296 er inflammation of the knee joints following collagen-induced arthritis, when compared with control m
297 *0401 gene contributes to the development of collagen-induced arthritis, whereas DRB1*0402 prevents t
298 ays a dominant role in the susceptibility to collagen-induced arthritis, whereas FcgammaRIIb on B cel
299 ent development of inflammation in mice with collagen-induced arthritis, whereas sorted CD25+CD39+CD4
300                   DQ8.CD28(-/-) mice develop collagen-induced arthritis with delayed onset and less s

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