ライフサイエンスコーパス: collapsibility

1 y affect more than just upper-airway anatomy/collapsibility.

2 ut has a minimal effect on pharyngeal airway collapsibility.

3 efulness to non-REM sleep and reduces airway collapsibility.

4 uring sleep and thereby decreases pharyngeal collapsibility.

5 ctility and assessment of inferior vena cava collapsibility.

6 Ggg activity and inspiratory flow and airway collapsibility; (3) reflex increases in flow (peak flow

7 pnea rely on both more favorable anatomy and collapsibility and enhanced upper-airway dilator muscle

8 iate analysis, baseline passive upper-airway collapsibility and loop gain were independent predictors

9 four phenotypic traits (upper-airway anatomy/collapsibility and muscle function, loop gain, and arous

10 ese patients often have air trapping, airway collapsibility, and a high degree of methacholine hyperr

11 gest that co-activation decreases pharyngeal collapsibility but does not dilate the pharyngeal airway

12 es upper airway muscles, alters upper airway collapsibility by a mechanism similar to tracheal or ton

13 t two basic mechanisms by which upper airway collapsibility can be altered.

14 gative epiglottic pressure, and upper airway collapsibility during passive and active conditions were

15 properties of the upper airway determine its collapsibility during periods of muscle hypotonia.

16 erapeutic devices and agents on upper airway collapsibility during sleep.

17 - 10 ms (n.s. vs. awake)) and greater airway collapsibility during the applied pressures (P = 0.043 v

18 We found that Pcrit rose (collapsibility increased, p < 0.001) and RN fell (p = 0.

19 nterval 0.65-0.89) or the inferior vena cava collapsibility index (area under the curve 0.66; 95% con

20 e (R = 0.58), whereas the inferior vena cava collapsibility index and the internal jugular vein aspec

21 The collapsibility index expressed in percentage equaled the

22 The collapsibility index of the inferior vena cava during a

23 To investigate whether the collapsibility index of the inferior vena cava recorded

24 We measured stroke volume index and collapsibility index of the inferior vena cava under a d

25 th a significantly higher inferior vena cava collapsibility index on day 0 than nonacidotic patients

26 venous pressure than the inferior vena cava collapsibility index or the internal jugular vein aspect

27 stic analysis, the area under curve for that collapsibility index was 0.89 (95% CI, 0.82-0.97).

28 Inferior vena cava collapsibility index was not an independent predictor of

29 of the inferior vena cava with inspiration (collapsibility index) by ultrasound.

30 ke volume index, and high inferior vena cava collapsibility index, which improved with subsequent rea

31 nder the logistics model, called "absence of collapsibility," is noted in motivating VanderWeele and

32 Pharyngeal anatomy/collapsibility, loop gain (LG), upper-airway muscle resp

33 ctive sleep apnea (OSA), abnormal pharyngeal collapsibility may be offset by increased mechanoreflex-

34 l min(-1); P < 0.05) and improved pharyngeal collapsibility (mean +/- s.d. 3.4 +/- 1.4 l min(-1) vs.

35 rovides important information on both airway collapsibility (mechanics) and ventilatory control, we c

36 To determine the factors that influence the collapsibility of the hypotonic airway, the critical pre

37 The collapsibility of the isolated UA was examined in older

38 A concomitant increase in the rigidity and collapsibility of the mixed MLF was observed.

39 emodynamic profiles: fluid loading (index of collapsibility of the superior vena cava>/=36%), inotrop

40 al area change<45% without relevant index of collapsibility of the superior vena cava), or increased

41 order would vary depending on the underlying collapsibility of the upper airway.

42 gnized as major determinants of the size and collapsibility of the upper airway.

43 Pathological collapsibility of the upper airways, caused by many diff

44 Crows that had learned about the mechanism (collapsibility) of the platform without the use of stone

45 associated with decreased upper airway (UA) collapsibility (p < 0.05), unchanged maximum flow, and i

46 erotonin(2A/2C) receptor agonist improves UA collapsibility predominantly, but not exclusively, via s

47 here was a smaller decrease (p < 0.05) in UA collapsibility that was also associated with increased u

48 Upper airway collapsibility (UAC) is increased in children with sleep

49 iven by improvements in upper-airway anatomy/collapsibility under passive (1.9 +/- 0.7 vs. 4.7 +/- 0.

50 st that OA therapy improves the upper-airway collapsibility under passive and active conditions.

51 d not significantly alter pharyngeal anatomy/collapsibility, upper-airway gain, or arousal threshold.

52 Upper airway collapsibility was also reduced with desipramine compare

53 Pharyngeal collapsibility was quantified as the ventilation at CPAP

54 in 'CPAP pressure drops': pharyngeal anatomy/collapsibility was quantified as the ventilation at CPAP