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1 to unravel the pathways leading to improved collateral circulation.
2 eral extent for use in investigations of the collateral circulation.
3 the best technique to quantify the degree of collateral circulation.
4 ients with extensive versus poorly developed collateral circulation.
5 se of the possible interruption of important collateral circulation.
6 of the luminal diameter, and the absence of collateral circulation.
7 w-up, which may be related to development of collateral circulation.
8 bility, especially in patients with coronary collateral circulation.
9 esis in rat hearts, which have little native collateral circulation.
10 ial arterial occlusion, and moderate-to-good collateral circulation.
11 n test occlusion (BTO) is used to assess the collateral circulation and cerebrovascular reserve in pa
14 icrovessels resulting in the re-formation of collateral circulation associated with enhanced neuronal
15 eatment remains undefined, and the status of collateral circulation at presentation may be a key feat
19 infarct core, salvageable brain tissue, and collateral circulation in patients suspected of acute st
20 underlines the importance of being aware of collateral circulation in patients with chronic aortoili
21 ostatin) related to the presence of coronary collateral circulation in patients with significant coro
26 ng cardiomyocytes, suggesting that increased collateral circulation may provide an important source o
28 In an unusual situation of bleeding from collateral circulation near the pseudocyst wall during p
29 Patients with a large infarct core or poor collateral circulation on computed tomography (CT) and C
31 he level of the subclavian vein and abnormal collateral circulation over the chest wall consistent wi
34 , a small infarct core, and moderate-to-good collateral circulation, rapid endovascular treatment imp
35 ew microvessels and leads to re-formation of collateral circulation, reperfusion and better recovery.
37 enous imaging-based biomarkers with grade of collateral circulation, the ischaemic penumbra and clini
39 s ischemic brain injury in SHR by increasing collateral circulation to the ischemic region and allowi
40 tenotic plaques are more likely to stimulate collateral circulation to the post-stenotic segment, pla
42 lumped parameter model of the human coronary collateral circulation was used to simulate normal and a
43 who received nitroglycerin and had coronary collateral circulation were more likely to exhibit impro
44 he vexing dilemma that an effective coronary collateral circulation, which is determined primarily by
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