ライフサイエンスコーパス: collateral damage

1 are at the crux of these toxic effects, or "collateral damage".

2 endent apoptosis of individual cells without collateral damage.

3 f lymphocytes, while minimizing the risk for collateral damage.

4 a thermally confined manner without causing collateral damage.

5 that downregulate hyperinflammation to avoid collateral damage.

6 g structures, thereby minimizing the risk of collateral damage.

7 he penetration force is important to prevent collateral damage.

8 ng cellular functions and protecting against collateral damage.

9 a key role of the immune system, but without collateral damage.

10 he efficacy of cancer therapy while limiting collateral damage.

11 ollagen vibrational modes results in minimal collateral damage.

12 human corneoscleral rim tissues, with little collateral damage.

13 ined volumes of soft tissue with very little collateral damage.

14 hs showed similar features, i.e., 2 zones of collateral damage, a zone generally < 10 mm of extensive

15 sed for transfection, but causes significant collateral damage and a high rate of cell death, especia

16 h danger signals, are needed to protect from collateral damage and are of therapeutic interest.

17 am to prevent excessive inflammation, repair collateral damage, and restore tissue homeostasis, and f

18 of off-target cleavage events and potential collateral damage are still lacking.

19 t a major role of tolerance is in minimizing collateral damage associated with inflammation.

20 Lysis of bystanders is viewed as acceptable "collateral" damage, but the persistent presence of activ

21 that protects normal tissues from excessive collateral damage by overactive immune cells and their p

22 by enabling an antimicrobial defense without collateral damage by the adaptive immune system.

23 oduced by effector Th1 cells helps limit the collateral damage caused by exaggerated inflammation.

24 fibrin deposition protects host tissue from collateral damage caused by the immune system as it comb

25 mpaction, aggregates blood cells, and causes collateral damage due to leukocyte activation.

26 hus the host can benefit from suppression of collateral damage during parasite infection and from red

27 ) hepatectomy (eHx) was modified to minimize collateral damage; effects were compared with those of s

28 injury in vivo as well as greatly decreased collateral damage from inflammation.

29 that these broad events are a consequence of collateral damage from targeting single loci.

30 rance of pathogenic organisms while limiting collateral damage from the host inflammatory response, k

31 mpts to explain the wavelength-dependence of collateral damage have invoked a wavelength-dependent lo

32 To prevent detrimental collateral damage, IL-1beta release is tightly controlle

33 anding the observed wavelength-dependence of collateral damage in mid-infrared laser ablation.

34 lation resulted in decreased cellularity and collateral damage in the tissue during viral infection.

35 , when it is drawn into battle, it can cause collateral damage in tissues.

36 in the protection of cells and tissues from collateral damage induced by inflammatory responses.

37 The collateral damage induced on healthy tissues during radi

38 cts in the Arab world are much more than the collateral damage inflicted on civilians, infrastructure

39 Such hepatic collateral damage may be a general consequence of expand

40 exerts on an individual's own health via the collateral damage of the drug on bacteria that normally

41 T cells while simultaneously minimizing the collateral damage of their potentially lethal actions an

42 ether IL-10 is also involved in limiting the collateral damage of vigorous T cell responses, has not

43 in the liver, making this a pure example of "collateral damage" of the liver.

44 fects harmful to hermaphrodites appear to be collateral damage rather than the goal.

45 o a more complete explanation of the reduced collateral damage resulting from infrared laser irradiat

46 r, recent studies have shed new light on the collateral damage they impart on the indigenous host-ass

47 n overly exuberant immune response can cause collateral damage through immune effectors and because o

48 and/or infected cells while also minimizing collateral damage to adjacent noninfected tissues.

49 t unfolding activity is prevented from doing collateral damage to cellular proteins are not well unde

50 f the hippocampus (HIPP) or hippocampus plus collateral damage to extrahippocampal structures (HCX) w

51 . cancer cells or parasites) without causing collateral damage to healthy or to host cells is complic

52 contribute to the inactivation of pathogens, collateral damage to host proteins can also occur and ha

53 acterium tuberculosis in vivo but also cause collateral damage to host tissues.

54 e efficiency of targeted killing and prevent collateral damage to neighboring healthy cells.

55 ate tissue with negligible heat transfer and collateral damage to neighboring tissue.

56 and often severe side effects as a result of collateral damage to normal cells.

57 is specifically in cancer cells with minimal collateral damage to normal cells.

58 or removal of damaged neurons, but can cause collateral damage to normal neurons located close to def

59 nciple, yet it is accompanied by significant collateral damage to normal tissue and unwanted side eff

60 ruction of pathogens may result in excessive collateral damage to normal tissues, and the failure to

61 ted to infect only myeloma cells to minimize collateral damage to normal tissues: viral binding to it

62 effective chemotherapy treatments with less collateral damage to ocular tissues and may allow reduce

63 ced peripheral neuropathy (CIPN) arises from collateral damage to peripheral afferent sensory neurons

64 djunctive neuroprotective therapy may reduce collateral damage to photoreceptors and improve visual o

65 ole target of FALI and provide evidence that collateral damage to proximal proteins occurs following

66 lled death of cells that occurs with minimal collateral damage to surrounding cells or tissue during

67 wed for unprecedented precision with minimal collateral damage to surrounding tissues.

68 ness of the A1 pulley release and detect any collateral damage to the A2 pulley, interdigital nerves,

69 es were dissected by laser axotomy, avoiding collateral damage to the adjacent dendrite and the forma

70 while minimizing their potential to inflict collateral damage to the adjacent lung tissue and indica

71 Causing collateral damage to the B-cell genome during CSR and SH

72 o tune the immune response, thereby limiting collateral damage to the host and the risk for sepsis.

73 mune system controls inflammation and limits collateral damage to the host during acute bacterial inf

74 d downregulates Th1 responses that may cause collateral damage to the host.

75 as the eradication of pathogens with minimal collateral damage to the host.

76 ective antimicrobial protection with minimal collateral damage to the host.

77 , to prevent excessive inflammation to limit collateral damage to the host.

78 nt antimicrobials, however, exert tremendous collateral damage to the human microbiome through overus

79 ive papers emerged this past year concerning collateral damage to the liver in extrahepatic infection

80 lance of initiating immunity without causing collateral damage to the lungs because of an exaggerated

81 lusively within the tumors, and there was no collateral damage to the neighboring hepatic parenchyma.

82 ffects pathologic vessels, it can also cause collateral damage to the overlying retina.

83 s and can treat microvessels without causing collateral damage to the surrounding tissue.

84 Collateral damage to the vagal nerve and the upper gastr

85 In contrast, mice with collateral damage to the ventromedial hypothalamus and p

86 these nasty invaders while also constraining collateral damage to vital tissue.

87 this beneficial activity comes at a cost of collateral damage when the immune system overreacts to i