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1 and ileostomists (subjects without an intact colon).
2 colon; r = 0.82, P = .006 for the descending colon).
3 easurements of 5-HT overflow from the entire colon.
4 e and flat low-grade dysplasia (fLGD) in the colon.
5 ending colon, hepatic flexure, or transverse colon.
6 rs to Enterobacteriaceae in the lumen of the colon.
7 myenteric cell loss observed in the proximal colon.
8 tonicity on the absorption of 2-PMPA in the colon.
9 doscopically, at the esophagus, stomach, and colon.
10 testine and by dextran sulfate sodium in the colon.
11 in the polyp and iodine in the lumen of the colon.
12 nitrergic myenteric neurons in the proximal colon.
13 -HT is an important pro-kinetic agent in the colon.
14 ging epithelial cells in small intestine and colon.
15 ellular, and morphologic properties of human colon.
16 uggesting a direct effect of salt within the colon.
17 s of Cajal that are reduced in the nNOS(-/-) colon.
18 ng both the proximal small intestine and the colon.
19 sue from the duodenum and ileum, but not the colon, 300 mmol/L glucose potently stimulated GLP-1 rele
23 years with histologically confirmed primary colon adenocarcinoma diagnosed between 1998 and 2007.
24 Expression of miR-487b-3p is decreased in colon adenocarcinomas and inversely correlates with GRM3
26 enteric neurons were reduced in the proximal colon after 9 and 12 weeks of WD and this was also assoc
27 continuity without stoma; and group 3, other colon anatomies), and disease features (with inflammator
28 e-convolute variable cellular composition of colon and adipose tissue samples, highlighting one use o
29 ssion analysis in i) normal colon cells, ii) colon and breast cancer cell lines and iii) cancer stem-
31 mpaired healing of superficial wounds in the colon and impaired mucosal innate immune responses again
32 In addition, depletion of SMYD5 in human colon and lung cancer cells results in increased tumor g
34 costs between elective open and laparoscopic colon and rectal cancer resection in a daily practice mu
35 was more pronounced for cancer of the distal colon and rectum and for physicians with higher PDRs.
36 l, bronchus, and lung cancer; 2484476 due to colon and rectum cancer; 1573593 due to breast cancer; 1
37 pharynx; other pharynx; esophageal; stomach; colon and rectum; liver; gallbladder and biliary; pancre
38 eptor (VDR) enhanced Claudin-2 expression in colon and that bile salt receptors VDR and Takeda G-prot
39 mmatory pathways activated in human inflamed colon and TNF-alpha-treated cells (false discovery rate
40 and nucleosome occupancy in cell lines, and colon and tumor samples, and by benchmarking the method
47 ic, DU145 prostate, HeLa cervical and CaCo-2 colon, as well as normal human MCF10A mammary epithelial
52 malignancies in men and women in SEER (lung, colon, breast, and prostate cancers), we found no signif
53 ures to samples collected from patients with colon, breast, or ovarian cancer and cell lines harborin
54 ion from (13) C-labelled fibres in the human colon by measurement of (13) C-labelled SCFA concentrati
55 The C statistics for colorectal (C = 0.607), colon (C = 0.603), and rectal (C = 0.639) cancer were si
57 ting data on overall survival for left-sided colon cancer (LCC) compared with right-sided colon cance
61 rectal cancer cases: 690 cases with proximal colon cancer and 690 cases with distal colorectal cancer
63 potent tumour growth inhibition in melanoma, colon cancer and human papilloma virus-E6/E7 tumour mode
64 CCDC66 expression was elevated in polyps and colon cancer and was associated with poor prognosis.
71 sal cytosolic Ca(2+) concentration of HCT116 colon cancer cell line and modified the cytosolic Ca(2+)
72 consisting of four defined derivatives of a colon cancer cell line that resulted from consecutive ep
74 carcinoma proliferation, a) in vitro against colon cancer cell lines and b) in vivo on tumor growth i
75 (PKG2) to activate forkhead box O (FoxO) in colon cancer cells and in the colon epithelium of mice.
76 d the metastatic dissemination capability of colon cancer cells HT29, including the migration and inv
77 JMJD2B enhanced subcutaneous tumor growth of colon cancer cells in a p53-dependent manner, and geneti
79 ium butyrate-induced differentiation of HT29 colon cancer cells is associated with a reduced CD133 ex
81 hanced cancer cell killing in cultured human colon cancer cells, but also improved antitumor activity
82 beta-catenin, inhibited the proliferation of colon cancer cells, repressed colon CSCs and prevented x
88 f exosomes is affected by differentiation of colon cancer cells; exosomes might be used by differenti
89 had no colonoscopy, had an increased risk of colon cancer compared with those without both diverticul
91 fusion and sepsis were associated with worse colon cancer disease-specific survival [(+)transfusion:
93 To investigate the role that NOX1 plays in colon cancer growth, we used shRNA to decrease NOX1 expr
94 to-alanine) mutant in either HEK293 cells or colon cancer HT29 cells showed dramatically reduced NF-k
97 18,186 patients with resected stage I to III colon cancer in the National Cancer Data Base (2004-2012
98 ers in different locations of the gut, where colon cancer is primarily driven by inflammation and the
105 stering coefficient was 0.50 in the proximal colon cancer network and 0.30 in the distal colorectal c
108 somal miRNA was isolated from 50 early-stage colon cancer patients and 50 matched healthy volunteers.
112 ereditary mixed polyposis syndrome is a rare colon cancer predisposition syndrome caused by a duplica
113 ehensively characterized a cellular model of colon cancer progression consisting of four defined deri
118 ly associated with overall CRC, but proximal colon cancer risk was higher in the proinflammatory-chan
119 We constructed a tissue microarray of 999 colon cancer specimens from patients who underwent surgi
120 .5 mum for phosphorus and platinum in HCT116 colon cancer spheroids upon treatment with the clinicall
124 prove cisplatin efficacy in the treatment of colon cancer through the creation of orally administered
127 alysis, different miR expression patterns in colon cancer versus non tumour cells using the previousl
128 es beta-catenin signaling and cell growth in colon cancer via binding RXRalpha, which provide new str
131 s of overall CRC, colon cancer, and proximal colon cancer were higher in the highest quintile compare
134 ancer Registry to identify all patients with colon cancer who underwent resection between January 1,
136 atients had normal findings, one patient had colon cancer with a hepatic metastasis, one patient had
138 in patients undergoing elective surgery for colon cancer without mechanical bowel preparation (n = 1
140 atients with high-risk stage II or stage III colon cancer, adjuvant chemotherapy with fluoropyrimidin
141 ng nonusers of NSAIDs, risks of overall CRC, colon cancer, and proximal colon cancer were higher in t
143 OM/DSS mice, a model of IBD and inflammatory colon cancer, augmented DSS-induced weight loss and incr
144 condition associated with increased risk for colon cancer, but its role in the development of colorec
145 seases such as Crohn's disease, colitis, and colon cancer, but mechanistic insights into these proces
147 iets are associated with an elevated risk of colon cancer, particularly for proximal colon cancer and
149 rious Slco2a1 genotypes in a murine model of colon cancer, the adenomatous polyposis (APC) mutant (Ap
150 nown to exhibit tumor suppressor activity in colon cancer, the mechanism of which is not understood.
151 lied to the dynamic (18)F-FDG measurement of colon cancer, the proposed algorithm accurately identifi
153 analysis of 999 patients with a diagnosis of colon cancer, we associated statin with reduced risk of
154 ouse systems to engineer and study recurrent colon cancer-associated EIF3E-RSPO2 and PTPRK-RSPO3 chro
175 only diagnosed cancers in the United States (colon cancer: R = 0.61; P < .001; lung cancer: R = 0.73;
176 d for the survival of primary and metastatic colon cancers and that oncogenic K-RAS activates TGF-bet
177 ntaining the proliferative phenotype of some colon cancers and the potential of NOX1 as a therapeutic
179 , prospectively collected set of 278 primary colon cancers spanning diverse tumor stages and clinical
184 nvestigated the preventive effects of KJT on colon carcinogenesis using the azoxymethane (AOM)-induce
192 e show that PTEN deletion in HCT116 and DLD1 colon carcinoma cells leads to suppression of CHK1 and C
195 When injected i.v. in mice bearing CT26 colon carcinoma or B16 melanoma, the 4PD nanoparticles p
196 comparatively investigated their effects on colon carcinoma proliferation, a) in vitro against colon
198 re, berberine suppresses the growth of human colon carcinoma xenograft in nude mice in an RXRalpha-de
199 es, exerts growth inhibitory effects against colon carcinoma, suggesting a nutraceutical potential in
201 show that USP39 is up-regulated in lung and colon carcinomas and its expression correlates with KRAS
202 a stability expression analysis in i) normal colon cells, ii) colon and breast cancer cell lines and
203 iffer between a diseased area of the sigmoid colon chronically affected by diverticulitis and adjacen
206 of gastric (MKN-45P), ovarian (SKOV-3), and colon (CT-26) origin, and that peritoneal tumors in mice
208 uited fewer neutrophils and monocytes to the colon during peak infection, which correlated with incre
212 ts identify a novel signaling pathway in the colon epithelium, where FoxO tumor suppressors could pro
213 regarding the overall population, nor in the colon (FFT: 23% vs LFT: 19%, P = 0.636) or rectal (FFT:
214 FT group and 133 in LFT group, including 106 colon (FFT: n = 52 and LFT: n = 54) and 157 rectal cance
221 ing framework, we show how water flow in the colon, in concert with other physiological factors, dete
222 with SBS and inflammatory bowel diseases had colon-in-continuity (10.5% [n = 2/19]), whereas most pat
223 with SBS and vascular or other diseases had colon-in-continuity (84.4% [n = 27/32] and 67.6% [n = 23
224 up 1, jejunostomy/ileostomy; group 2, >/=50% colon-in-continuity without stoma; and group 3, other co
225 ients with IBD (n = 62) and patients without colon inflammation (controls, n = 23) were analyzed by q
226 ges revealed that hgd40 and anti-TNF reduced colon inflammation over 3 days; hgd40 reduced colitis in
228 se and increased escape of bacteria from the colon into the lymphatic system in a dextran sodium sulf
230 lial cells (IECs) in the small intestine and colon is required for enteric IFN-lambda antiviral activ
231 octreotide in cancers from prostate, breast, colon, kidney, thyroid, and lymphoid tissues as well as
232 We analyzed a group of patients with large colon lesions to identify factors associated with SMIC,
233 cellular matrix (ECM) constructed by AKAP12+ colon mesenchymal cells (CMCs) generated M2 macrophages
234 acterize structural and metabolic changes in colon mucosa associated with WD and predisposition to co
236 f mosaicism in the APC gene in patients with colon neoplasms not associated with any other genetic va
237 alpha expression were examined in the distal colon of 3, 12, 18 and 24-month old mice and faecal outp
238 mouse bowel and transplanted into the distal colon of 3- to 4-week-old wild-type recipient mice.
241 CC1 (and gammaENaC) protein abundance in the colon of the Nedd4L knock-out animals was increased, ind
248 distinguish patients with UC from those with colon-only CD based on increased mucosal expression of g
250 220-450, with mucosal ulcers in the ileum or colon, or both, and a Crohn's Disease Endoscopic Index o
252 We used 3-dimensional small intestine and colon organoids, along with RNA-Seq and gene ontology me
253 logy to delete key DNA repair genes in human colon organoids, followed by delayed subcloning and whol
254 lammatory effects; it reduces development of colon polyps in mouse models of colorectal cancer (CRC).
255 ensity (r = 0.88, P = .033 for the ascending colon; r = 0.82, P = .006 for the descending colon).
256 r show that transplantation of ENSC into the colon rescues impaired colonic motility with formation o
258 larization, atrophy of intestinal villus and colon-resident lymphoid follicle, and degeneration and a
260 uggest that lumen and mucosa in the proximal colon should be conceptualized not as stratified compart
261 litis-associated dysplasia in the descending colon showed good correlation with normalized (19)F sign
264 CD103 expression, promotes expression of the colon-specific trafficking molecule GPR15, and inhibits
271 flow cytometry analysis of paraffin-embedded colon tissue, we detected aneuploidy in 15 of 37 samples
273 reaction and immunofluorescence analyses of colon tissues from patients with Crohn's disease (n = 61
274 Levels of DHA-derived epoxides are lower in colon tissues from patients with UC than healthy and res
275 tumor necrosis factor (Tnf) mRNA for 6 days; colon tissues were collected and analyzed histologically
278 st Tnf mRNA reduced protein levels of TNF in colon tissues, compared with mice with colitis given siR
279 , IL33, and thymic stromal lymphopoietin) by colon tissues, which activated type 2 innate lymphoid ce
284 emokine CXCL1 gene was highly upregulated in colon tumor epithelium in a HIF-2alpha-dependent manner.
285 an hour in a large volume of mouse xenograft colon tumor, and 3) determine the impact of the HIFU/nan
286 key mucosal barrier components in regulating colon tumorigenesis and cancer progression remains uncle
289 of tumor-infiltrating lymphocytes in primary colon tumors and liver metastases have improved outcomes
290 novel regulator of neutrophil recruitment to colon tumors and that it is essential in shaping the pro
293 er initially defined on endothelial cells in colon tumors that was discovered recently to be upregula
294 However, after stratification by histology (colon vs. rectum) we found that rs1525489 was associated
295 hrough cGMP has therapeutic potential in the colon, where it has been implicated in the suppression o
297 murine intestinal tissue, predominantly the colon, which persisted after pathogen clearance and irre
298 nificant spasmolytic activity (on rat distal colon), with PhPeITC being almost 100 times more potent
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