ライフサイエンスコーパス: colonic carcinoma

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1 itoneal metastases from ovarian, gastric and colonic carcinoma.

2 One patient had previous surgery for colonic carcinoma.

3 er-related differences in the development of colonic carcinomas.

4 e preventive effects were evaluated in human colonic carcinoma cell line Caco-2 and neonatal rat mode

5 Using a human colonic carcinoma cell line that has many duodenal chara

6 In the human HT29Cl16E colonic carcinoma cell line, induction of goblet cell di

7 and progression, we generated novel isogenic colonic carcinoma cell lines that exhibit highly signifi

8 ed undifferentiated and differentiated human colonic carcinoma cell lines to determine if this parasi

9 ification of 87% to 35-fold in 7 of 10 human colonic carcinoma cell lines.

10 and Dsc2 play opposing roles in controlling colonic carcinoma cell proliferation through differentia

11 ive apoptotic cascade induced in SW620 human colonic carcinoma cells by the protein kinase antagonist

12 in was increased in nonconfluent cultures of colonic carcinoma cells compared to confluent cells and

13 que observation that silencing CXCL12 within colonic carcinoma cells greatly enhances their metastati

14 nolayers of MDCK renal epithelial and CaCo-2 colonic carcinoma cells grown in transwell configuration

15 se data implicate the intrinsic Deltapsim of colonic carcinoma cells in determining the probability o

16 ion-dependent growth arrest and apoptosis of colonic carcinoma cells in vitro.

17 plays a role in the biological behaviour of colonic carcinoma cells induced by migratory factors suc

18 ckdown of Mastermind-like family proteins in colonic carcinoma cells results in cell death by affecti

19 Tumor formation and metastasis of colonic carcinoma cells was monitored using in vivo biol

20 Prolonged treatment of human colonic carcinoma cells with ST led to nuclear accumulat

21 In agreement, in clone A colonic carcinoma cells, epitope-tagged PMP22 forms a co

22 erated isogenic cell lines, from SW620 human colonic carcinoma cells, which exhibit significant diffe

23 cells within an in vitro population of human colonic carcinoma cells.

24 uciferase reporter gene assay in transfected colonic carcinoma cells.

25 ted miR-21 to be expressed at high levels in colonic carcinoma cells.

26 Q were effectively activated by HCT116 human colonic carcinoma cells; the activation of the former in

27 in lumenal membrane fractions isolated from colonic carcinomas compared with that detected in the no

28 Exogenous ITF also protected another human colonic carcinoma-derived cell line (HCT116) and a nontr

29 ancy (neuroendocrine metastases from primary colonic carcinoma diagnosed 22 months after initial US)

30 In transfected colonic carcinoma epithelial layers, functional Y1 and Y

31 ell migration but not proliferation of human colonic carcinoma HT29 or immortalized mouse colonic YAM

32 The development of colonic carcinoma is associated with the mutation of a s

33 Colonic carcinomas may manifest as plaquelike, polypoid,

34 Pulmonary GW-39 human colonic carcinoma microcolonies were induced in athymic

35 ng gastrointestinal malignancy (particularly colonic carcinoma), short bowel syndrome (where factors

36 cinomas (HNPCCa) and 57 sporadic right-sided colonic carcinomas (SRSCCa) were evaluated.

37 ness for human epithelial cells derived from colonic carcinoma (T84 cells).

38 These results suggest that in developing colonic carcinomas, the early effects of cyclooxygenase-

39 mpare with 80% retention of CD44 intron 9 in colonic carcinoma tissue mRNA and confirm that multiple

40 onal mucosa, sporadic adenomas, and sporadic colonic carcinomas were studied as controls.

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