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1 xymethane was administered to rats to induce colonic neoplasms.
2 , colonocyte antipodal plasma membranes, and colonic neoplasms.
3       Mutations of c-K-ras occur commonly in colonic neoplasms.
4 l on a series of murine intestinal and human colonic neoplasms.
5 ht and near-infrared (NIR) images, to reveal colonic neoplasms after the intravenous administration o
6                               However, in 16 colonic neoplasms, an average of 23-fold and 29-fold inc
7 mine the prevalence and location of advanced colonic neoplasms and the risk of advanced proximal neop
8 r colon cancer, diverticular disease, benign colonic neoplasm, and ulcerative colitis/regional enteri
9 S is associated with the exophytic growth of colonic neoplasms, and that (b) some colorectal cancers
10 es, the predictive value of risk factors for colonic neoplasms, and the impact of colonoscopic findin
11  We conclude that ACF are marker lesions for colonic neoplasms, but only in the distal colon where tu
12              Before the development of human colonic neoplasms, colonic epithelial cells showed alter
13 ty, previously described in the detection of colonic neoplasms, could affect prostate cancer detectio
14   Colonoscopic screening can detect advanced colonic neoplasms in asymptomatic adults.
15 tively determined the prevalence of proximal colonic neoplasms in asymptomatic patients at average ri
16       The substantial prevalence of proximal colonic neoplasms, including advanced lesions, in asympt
17                                              Colonic neoplasms sometimes are more difficult to detect
18 mechanism for the combinatorial treatment of colonic neoplasms targeting both COX-2/PGE(2) and the EG
19 een aberrant crypt foci (ACF) and subsequent colonic neoplasms, we investigated the distribution of A
20          CDX2 heterozygous-null mice develop colonic neoplasms, which have suggested that CDX2 is a t

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