ライフサイエンスコーパス: colony formation

1 in, decreases sphere formation, and inhibits colony formation.

2 ults in an elevated IFN response and reduces colony formation.

3 that none of them have the ability to induce colony formation.

4 h P50-siRNA caused a significant decrease in colony formation.

5 ion, and decrease mammosphere and progenitor colony formation.

6 s with high endogenous GRHL1 levels promoted colony formation.

7 , camptothecin, or etoposide and assayed for colony formation.

8 indicated that PML is involved in erythroid colony formation.

9 arget of ROS, and enhanced proliferation and colony formation.

10 rmally bonded to control embryonic stem cell colony formation.

11 ads to increased proliferation and soft agar colony formation.

12 gnaling and increased cell proliferation and colony formation.

13 a dramatic decrease in anchorage-independent colony formation.

14 ession of miR-378 enhanced cell survival and colony formation.

15 ls inhibited cell proliferation and impaired colony formation.

16 1-positive repair foci, and cell survival by colony formation.

17 d DeltaEGFR-stimulated anchorage-independent colony formation.

18 miR-26a-induced tumor cell proliferation and colony formation.

19 ncordance between assay signal and bacterial colony formation.

20 on library for mutants defective in wrinkled colony formation.

21 ulation of cell adhesion, proliferation, and colony formation.

22 with a dramatic reduction of cell growth and colony formation.

23 inhibiting substance (MIS), which decreased colony formation.

24 ling potential pathogens without waiting for colony formation.

25 resulting in enhanced apoptosis and reduced colony formation.

26 he minimal cell number needed for successful colony formation.

27 itro elevated the low ROS level and promoted colony formation.

28 ges in morphology, which occur prior to iPSC colony formation.

29 overexpression that results in enhanced AML colony formation.

30 ryptophan repeat can support LIF-independent colony formation.

31 ll lines and reduced their proliferation and colony formation.

32 nhibited cytokine-independent megakaryocytic colony formation.

33 rregular trilineage anemia, with deficits in colony formation.

34 increased tumor cell apoptosis and inhibited colony formation.

35 for proliferation, apoptosis, migration, and colony formation.

36 n naive hESCs reduces cell proliferation and colony formation.

37 with the increased frequency of aberrant 3D colony formation.

38 rrier of Ink4a/Arf locus, thus enhancing iPS colonies formation.

39 To automatically analyse and determine iPSC colony formation, a machine learning-based classificatio

40 oundly reduced cell migration, invasion, and colony-formation abilities of cells overexpressing EMSY

41 YAP1 mutation was shown to increase the colony formation ability and invasion potential of lung

42 n and upregulation of miR-128 suppressed the colony formation ability and invasiveness of pituitary t

43 A-mediated attenuation of HSF1 abrogated the colony formation ability of both lymphoma and AML CSCs.

44 tently, TIPE1 inhibited both cell growth and colony formation ability of cultured HCC cell lines, whi

45 eased apoptosis and decrease in invasion and colony formation ability of HNSCC cell lines.

46 cells, such as increased cell proliferation, colony formation, anchorage-independent growth in soft a

47 allow accurate and unbiased determination of colony formation and anchorage independent growth over t

48 ficantly inhibited tumor cell proliferation, colony formation and anchorage-independent cell growth.

49 A549R cells resulted in increased levels of colony formation and cell migration as well as reduced a

50 This subsequently reduced the level of colony formation and cell migration facilitating ATO-ind

51 6 and IL-8 expression dramatically inhibited colony formation and cell survival in vitro and stanched

52 3 binding, contributing to the inhibition of colony formation and cell-cycle arrest.

53 cells of Meis1-deficient mice showed reduced colony formation and contained significantly fewer numbe

54 , but not Rxfp1 knockout mice as assessed by colony formation and flow cytometry.

55 ergistic enhancement of CSF induction of HPC colony formation and for HPC response to myelosuppressiv

56 short telomeres to support alveolar organoid colony formation and found that type 2 alveolar epitheli

57 We analyzed colony formation and genomic features and gene expressio

58 wn of Mer or Axl significantly reduced NSCLC colony formation and growth of subcutaneous xenografts i

59 also can be used to monitor the dynamics of colony formation and growth.

60 te that polygamain effectively inhibits PC-3 colony formation and has excellent cellular persistence

61 sis identified multiple factors that recruit colony formation and highlights novel facets of LVCP fun

62 out (PKRKO) mice has increased potential for colony formation and HSPCs are more actively proliferati

63 ssels were assayed at postoperative day 7 by colony formation and immunofluorescence.

64 expression of Y102F mutant of WASp decreases colony formation and in vivo tumor growth.

65 rols, ARC-deficient RCCs exhibited decreased colony formation and increased apoptosis in vitro.

66 of PDE10 by short hairpin RNA also inhibits colony formation and increases doubling time of colon tu

67 pression of TET1 catalytic domain suppressed colony formation and induced apoptosis of tumor cells of

68 I-CBP112 resulted in substantially impaired colony formation and induced cellular differentiation wi

69 ir proliferation and tumor growth, impairing colony formation and inducing cellular senescence.

70 R1 in prostate cancer cells markedly reduced colony formation and inhibited tumor growth in animals.

71 blocked cell proliferation, DNA replication, colony formation and invasion in SCC25 and CAL27 cells.

72 tion of ESRRA impaired anchorage-independent colony formation and invasion of OSCC cells.

73 CCE cells also showed markedly higher colony formation and invasion, typical of cancer cells.

74 ted phenotypes including cell proliferation, colony formation and invasiveness, which were restored b

75 nt mouse HSPCs and suppresses human leukemic colony formation and leukemia progression of primary hum

76 MP production, induces apoptosis, and blocks colony formation and mammary tumor growth.

77 rgize with inhibitors of Aurora B to inhibit colony formation and oncogenic transformation.

78 ity attenuates cell proliferation, soft-agar colony formation and orthotopic GBM growth in NOD/SCID m

79 3A, supports cell survival, drug resistance, colony formation and proliferation in vitro, and promote

80 ort functions, secretes factors that promote colony formation and proliferation of purified quiescent

81 G12D/+)-induced splenomegaly and spontaneous colony formation and prolongs the survival of CMML-beari

82 ibited growth factor-independent KRASG12D BM colony formation and sensitized cells to a low dose of t

83 ss than 1% of cancer cells and has increased colony formation and shorter tumor-free intervals in viv

84 CaP cells expressing Lin28 was determined by colony formation and soft agar assays.

85 tion that results in ER-stress-mediated cell-colony formation and survival, growth, and invasion, whi

86 Reexpression of DACH1 reduced NSCLC colony formation and tumor growth in vivo via p53.

87 ckdown of AIB1 decreased cell proliferation, colony formation and tumorigenesis of these CRC cells.

88 SECs, induced cell proliferation, migration, colony formation and tumorigenesis.

89 ne(R) and PKC412(R) had higher capability of colony formation and wound healing than parental cells i

90 The results of colony formation and xenograft assays showed a mutation

91 ivation, and renders sorafenib resistance in colony formation and xenograft tumor assays.

92 ted ICN1 to facilitate anchorage-independent colony formation and xenograft tumor growth with increas

93 found that E6, but not E7, recovers FA iPSC colony formation and, furthermore, that p53 inhibition i

94 e expression suppression of CSR1, suppressed colony formation, and blocked cell cycle entry to the S

95 vity against melanoma cells in cytotoxicity, colony formation, and cell invasion studies.

96 in (i) a decrease in cellular proliferation, colony formation, and cellular migration; (ii) induction

97 Alterations of cell proliferation, colony formation, and cellular senescence were evaluated

98 demonstrated superior hematopoietic growth, colony formation, and diversification compared to ALD an

99 that promote cell proliferation, anchor-free colony formation, and epithelial-mesenchymal transition

100 CC cells abrogates cell branching, invasion, colony formation, and growth in a murine xenograft model

101 signaling, transwell invasion and soft agar colony formation, and in vivo promoted lung metastasis i

102 resulted in decreased proliferation rate and colony formation, and in vivo xenograft models showed sl

103 R33b decreased MM cell viability, migration, colony formation, and increased apoptosis and sensitivit

104 ted in decreased cell proliferation, reduced colony formation, and increased apoptosis.

105 profoundly suppressed cell proliferation and colony formation, and induced cell cycle arrest accompan

106 iR-155 retarded tumor cell growth, decreased colony formation, and induced G(1)-S cell-cycle arrest i

107 -LK inhibits cell-cycle progression, reduces colony formation, and induces differentiation, suggestin

108 ulted in decreased tumor cell proliferation, colony formation, and invasion in vitro Blocking endogen

109 Crispr/Cas9 greatly promotes cell viability, colony formation, and invasion of cancer cells in vitro

110 ssed MAN2A1-FER had increased proliferation, colony formation, and invasiveness and formed larger (>2

111 oma cells enhanced tumor cell proliferation, colony formation, and invasiveness, in vitro.

112 Functionally, proliferation, colony formation, and long-term self-renewal were impair

113 uced inhibition of tumor cell proliferation, colony formation, and migration.

114 on of EphB4 promotes cellular proliferation, colony formation, and motility, while EphB4 inhibition r

115 ncident with increased cell death, decreased colony formation, and reduced tumor growth in an in vivo

116 IC50, suppresses PCa cell proliferation and colony formation, and reduces migration and invasion.

117 /progenitor cells, inhibited cell growth and colony formation, and significantly prolonged survival i

118 rix metalloproteinases (MMPs), invasiveness, colony formation, and spheroid formation.

119 and BCL2 markedly suppressed proliferation, colony formation, and survival of Ph(+) ALL cells ex viv

120 as the percentage of CD34(+) cells in cycle, colony formation, and survival, demonstrating its partic

121 an increased RBC mass, spontaneous erythroid colony formation, and the JAK2V617F mutation.

122 of EMT, the loss of TIC-mediated clonogenic colony formation, and the loss of cell motility and inva

123 TD1A inhibits colorectal cancer cell growth, colony formation, and tumor engraftment.

124 significantly reduced Myc-induced autophagy, colony formation, and tumor formation.

125 signaling and inhibited cell proliferation, colony formation, and tumor growth in subcutaneous and o

126 h tenascin C exhibited enhanced adhesion and colony formation as mediated by integrin alpha9beta1.

127 ramatically altered cell invasion potential, colony formation, as well as tumorigenesis in orthotopic

128 micals were tested for their cytotoxicity by colony formation assay in cells of different BRCA2 statu

129 In cancer research, colony formation assay is a gold standard for the invest

130 Finally, colony formation assay revealed a dramatic decrease in t

131 In colony formation assay the fucoidans from different spec

132 ly, as assessed in vitro and in vivo using a colony formation assay, a spheroid formation assay and a

133 illing, the ability of cells to survive in a colony formation assay, and proliferation rates after ra

134 n vitro and in vivo, which were confirmed by colony formation assay, transwell invasion assay, and tu

135 ntification is a promising technique for the colony formation assay.

136 -dependent hypersensitivity by hematopoietic colony formation assays and phospho-STAT5 (pSTAT5) flow

137 Soft agar colony formation assays and xenograft studies show that

138 AF WT, transformed NIH3T3 cells in soft-agar colony formation assays, increased kinase activity in vi

139 ed using mammosphere formation and soft-agar colony formation assays.

140 eatic cancer cell lines tested using MTT and colony formation assays.

141 ffects of ALK inhibition using viability and colony formation assays.

142 o radiation-induced cell death determined in colony-formation assays.

143 ed to not only increased tumor formation and colony formation but also increased tumor dispersal to s

144 significantly reduced cell proliferation and colony formation but induced tumor cell senescence.

145 nuated progenitor cell proliferation (spleen colony formation), but the sEH products 12,13-dihydroxyo

146 formation by 55%-90% while inhibiting normal colony formation by 22%-30%.

147 -cadherin stabilizer Thiazovivin resulted in colony formation by 25% to 30% of single-sorted ISCs.

148 h Nutlin-3 and Peg IFN-alpha 2a inhibited PV colony formation by 55%-90% while inhibiting normal colo

149 with knocked-down Mer demonstrated decreased colony formation by 67-87%, relative to control cell lin

150 cell-specific inhibition of MCF7 and SK-BR-3 colony formation by estrogen receptor alpha (ESR1) and (

151 al inhibition of Gln uptake blocks soft agar colony formation by Hace1(-/-) MEFs.

152 ation and invasion of cultured HCC cells and colony formation by HCC cells.

153 Colony formation by immature subsets of HPCs was greatly

154 y measuring anchorage-independent growth and colony formation by immortalized fibroblasts, we made si

155 It strongly inhibits colony formation by MM cells while sparing surrounding B

156 enous IL-33 promoted cytokine production and colony formation by primary CD34+ MPN stem/progenitor ce

157 ng this pathway can partially reduce myeloid colony formation by Rcor1-deficient erythroid progenitor

158 lows Runx1 to increase Cebpa and granulocyte colony formation by Runx1-deleted murine marrow.

159 Moreover, PCI-32765 prevents in vitro colony formation by stem-like cells from MM patients.

160 ntly reduced B16F10 melanoma growth and lung colony formation by triggering tumor apoptosis.

161 MERTK-mediated downstream signaling, reduced colony formation by up to 59%, and diminished tumor volu

162 170 not only promoted cell proliferation and colony formation by up-regulating the expression of cycl

163 und reductions in cellular proliferation and colony-formation capacities.

164 ntially induced apoptosis and eliminated the colony formation capacity of mouse lymphoma CSCs and hum

165 ith Wnt agonists increased proliferation and colony-formation capacity.

166 CDH10 promoted cell proliferation, soft-agar colony formation, cell migration and cell invasion, and

167 ated with increased levels of cell survival, colony formation, cell migration and decreased cellular

168 ecrease in medulloblastoma cell growth, cell colony formation, cell migration, invasion, and tumor sp

169 etion of SATB2 inhibited cell proliferation, colony formation, cell motility and expression of beta-c

170 d with controls showed significantly reduced colony formation, cell proliferation, induced cell cycle

171 Contrasting the reduced colony formation, cells attached on narrow patterns were

172 r in vitro hematopoietic-differentiation and colony formation (CFU assay).

173 hrombocytosis, leukocytosis, Epo-independent colony formation, characteristic bone marrow histology,

174 ve for long-term growth, as shown by reduced colony formation compared with cells expressing either c

175 -GBM astrocytes exhibited greater growth and colony formation compared with female Mes-GBM astrocytes

176 in HCT116 cells resulted in the reduction of colony formation, consistent with tumour suppressor capa

177 epletion of MLL target Ikzf2 in LSCs reduced colony formation, decreased proliferation, and increased

178 secretion, increased invasiveness, increased colony formation, decreased PTEN expression, and formati

179 nd reduced colony-forming unit-megakaryocyte colony formation driven by JAK2-V617F, but was not suffi

180 s had rapid proliferation rates and enhanced colony formation efficiencies.

181 uced in vitro proliferation rates, soft agar colony formation efficiency, and migration rates, indica

182 transformed cells and significantly reduces colony formation efficiency, in a dose-dependent manner,

183 udy showed that sesamol treatment suppressed colony formation, elicited S phase arrest during cell cy

184 into clusters induced a >15-fold increase in colony formation ex vivo and a >100-fold increase in met

185 miR-21 also led to an increase in erythroid colony formation from primary MDS bone marrow progenitor

186 However, as determined by colony formation, GMX1778 long term cytotoxicity in canc

187 nd were demonstrated to yield a high rate of colony formation (>/=85%) after removal from the array.

188 exhibit significantly reduced migration and colony formation, impaired anchorage-independent growth,

189 ng in a strong differentiation phenotype and colony formation impairment, confirming the potential of

190 f RSK2, and another RSK2 inhibitor increased colony formation implicating a role for this kinase in e

191 2 suppresses leukemia cell proliferation and colony formation in a manner dependent on WT1.

192 From colony formation in bacteria to wound healing and embryo

193 We observed a strong inhibition of colony formation in cells overexpressing these tsRNAs co

194 against SCD had decreased cell migration and colony formation in culture and reduced tumorigenicity i

195 se inhibitor, PF-562271, impaired growth and colony formation in Ewing sarcoma cell lines.

196 eral hematological cell lines, inhibition of colony formation in HEL cells, and analysis of apoptosis

197 nucleation, decreased cell proliferation and colony formation in human NSCLC cells, and reduced tumor

198 cytokine-independent growth, and/or enhanced colony formation in Jurkat T cells.

199 in multiple leukemia cell lines and reduced colony formation in leukemic, but not normal, CD34+ cell

200 knockdown of CBL enhanced cell motility and colony formation in NSCLC cells, and these activities we

201 rowth, induced differentiation, and impaired colony formation in primary AML blasts.

202 cell proliferation and anchorage-independent colony formation in primary human melanocytes.

203 ptosis, decreased invasiveness and decreased colony formation in soft agar across multiple melanoma c

204 -3 tumor cells decreased cell proliferation, colony formation in soft agar and strikingly diminished

205 TGM2 suppressed colony formation in soft agar and tumor formation in a x

206 Also IGF1 enhanced colony formation in soft agar in an alpha6beta4-dependen

207 tely reversed the increased Wnt activity and colony formation in soft agar induced by Apc siRNA treat

208 Colony formation in soft agar is the gold-standard assay

209 esistance to anti-cancer drug, invasion, and colony formation in soft agar, and in vivo tumor growth

210 d to cell hyperproliferation, enhanced tumor colony formation in soft agar, and increased xenograft t

211 icantly inhibits both cell proliferation and colony formation in soft agar, and induces apoptosis in

212 ing in decreased cell proliferation, reduced colony formation in soft agar, and induction of apoptosi

213 aling, induced apoptosis in culture, reduced colony formation in soft agar, and inhibited invasion of

214 owth conditions, however, including impaired colony formation in soft agar, spheroid formation, and x

215 t tumor cells dependent on IRS1 activity for colony formation in soft agar.

216 roliferation and migration but essential for colony formation in soft agar.

217 ion of NFAT3 at Ser259 led to a reduction of colony formation in soft agar.

218 ve mutant, suppressed cell proliferation and colony formation in soft-agar assays.

219 bit HRAS expression, metabolic activity, and colony formation in T24 cancer cells.

220 ivity versus MAO A and remarkably inhibiting colony formation in THP-1 human leukemia cells, were ass

221 n addition, overexpression of MGL suppressed colony formation in tumor cell lines and knockdown of MG

222 y reduced cell viability, proliferation, and colony formation in vitro and delayed tumor growth in vi

223 Finally, FBXO32 inhibits colony formation in vitro and primary tumor initiation a

224 cell proliferation, migration, invasion, and colony formation in vitro and to inhibit HCC tumor growt

225 lthough all SLE-derived HSPCs exhibited poor colony formation in vitro compared with controls, SLE HS

226 Tumor growth was determined with colony formation in vitro or in vivo.

227 ble transfectants suppressed cell growth and colony formation in vitro, and PTTG cell knockout also c

228 n human CD34(+) cells enhances hematopoietic colony formation in vitro, whereas HPIP knockdown leads

229 one explants significantly increased myeloid colony formation in vitro, which was blocked by G-CSF-ne

230 iminate granulocyte/macrophage and erythroid colony formation in vitro.

231 KX2-1 inhibited both AP-1 activity and tumor colony formation in vitro.

232 ppression of either ZNF148 or CTNNB1 reduced colony formation in WNT-dependent, but not WNT-independe

233 c traits, including migration, invasion, and colony formation, in soft agar with CD66(high) cells.

234 when we used a new Huh-7-derived cell line, colony formation increased approximately 70-fold.

235 e, which displayed growth arrest, lower lung colony formation index, and smaller tumor size than in c

236 n the control of HSPC cycling, survival, and colony formation induced by CXCL12.

237 of U251MG cells with Cmpd1 reduced in vitro colony formation, induced cell cycle arrest in the G2/M

238 GRHL1 levels abrogated anchorage-independent colony formation, inhibited proliferation, and retarded

239 esulted in increased cellular proliferation, colony formation, invasion, and development of a multinu

240 80A mutant Nanog reduced cell proliferation, colony formation, invasion, migration and the CIC popula

241 Phenotypic profiling reveals that 3D colony formation is heterogeneous and increased stiffnes

242 4-3-3zeta, suppressed cell proliferation and colony formation, markedly reduced activation of HER2, E

243 tly reduces glioblastoma cell proliferation, colony formation, migration and invasion.

244 t of miR-125b was analyzed in wound closure, colony formation, migration, and invasion assays in two

245 levels and leading to reduced proliferation, colony formation, migration, and invasion.

246 ound that miR-125b suppressed proliferation, colony formation, migratory, and invasive capacity of cS

247 larynx development reveals a unique type of colony formation never before described in Hydrozoa, in

248 ight required amino acids does not allow for colony formation of (p)ppGpp(0) cells when transitioning

249 A first case study describes the colony formation of a rod-shaped species on a planar sub

250 2HG levels in vitro, and efficiently blocked colony formation of AML cells from IDH1-mutated patients

251 suppressed the proliferation, invasion, and colony formation of breast cancer cells in vitro and tum

252 l cycle arrest, and attenuated migration and colony formation of breast cancer cells.

253 ition, IFN-gamma increased proliferation and colony formation of CD34(+) stem/progenitor cells from C

254 tudies showed decreased survival and reduced colony formation of Fan1(-/-) mouse embryonic fibroblast

255 657 dramatically increases proliferation and colony formation of HCC cells in vitro and induces tumor

256 Loss of TCF7L1 impaired growth and colony formation of HCT116 CRC cells and reduced tumor g

257 Depletion of JMJD1C impairs expansion and colony formation of human leukemic cell lines, with the

258 Depletion of PPP2R1A inhibited colony formation of Mad2-overexpressing HeLa cells but n

259 K578R mutant inhibited anchorage-independent colony formation of MCF7 breast cancer cell line.

260 ncing resulted in enhanced proliferation and colony formation of MEC1 and JVM3 cells, implying a role

261 roliferation, neoplastic transformation, and colony formation of mouse epidermal cells JB6 Cl41, huma

262 Depletion of Jmjd1c impairs growth and colony formation of mouse MLL-AF9 cells in vitro as well

263 ession of H1.3 decreases the growth rate and colony formation of OVCAR-3 cells.

264 ibition of FOXO3a restored proliferation and colony formation of Pdk1(-/-) melanoma cells.

265 promoted the growth, invasion, adhesion, and colony formation of prostate cancer cells at low ligand

266 th robust proliferative potential, secondary colony formation on replating, and de novo blood vessel

267 istant cells, whereas it was unable to block colony formation or cell survival, suggesting that the r

268 5-azacytidine reduced the proliferation and colony formation potential in ERBB2-positive breast canc

269 In addition, GSK2830371 suppressed the colony-formation potential of p53 wild-type NB cell line

270 itative and objective method to describe the colony formation process and the development of colony s

271 Hence, the colony formation process could be quantitatively represe

272 termining the reprogramming process and iPSC colony formation quantitatively, a mathematical model wa

273 cell cycle arrest at S phase, and decreased colony formation rate.

274 tivity to ADI-PEG 20, evidenced by decreased colony formation, reduced cell viability, and increased

275 The inhibition of colony formation, regulated by cisplatin, was more signi

276 ic devices produced a 10-fold improvement in colony formation relative to electroporation and cell-pe

277 Hormone-independent luminal progenitor colony formation required NF-kappaB, ataxia telangiectas

278 pecific 3-D microgeometries can support hESC colony formation, self-renewal, and directed differentia

279 ellular assays and primary mouse bone marrow colony formation studies.

280 osis, decreased proliferation, and decreased colony formation, suggesting that SRSF2 has oncogenic fu

281 TMEM43 deficiency significantly affects colony formation, survival of anoikis-induced cell death

282 Both diffusion inhibition zone tests and colony formation unit tests showed clear antimicrobial e

283 n for bacterial detection equal to 10(2)CFU (colony formation unit) for live bacteria detection with

284 In the absence of inhibitors, colony formation units (CFUs) per milliliter in blood fr

285 After 30 min irradiation, no colony formation was detected using TCP-C60 or TCP/TCP-C

286 he effect of batimastat on cell survival and colony formation was enhanced when sAPPalpha downregulat

287 myeloma cells, whereas normal hematopoietic colony formation was unaffected.

288 nematics of single and pairs of hESCs impact colony formation, we study their mobility characteristic

289 ncer cell lines, both cell proliferation and colony formation were decreased.

290 in vitro Conversely, migratory potential and colony formation were enhanced in PHD3-deficient cells,

291 On day 1, in ARDSp, different patterns of colony formation were found, with nonstromal cells (main

292 bilization, and both mobilization and spleen colony formation were reduced in sEH(-/-) mice.

293 f TIE2, and increase survival, invasion, and colony formation when expressed in human umbilical vein

294 ITC antagonized dsRNA-mediated inhibition of colony formation, whereas SFN enhanced the inhibition.

295 auxiliary proteins had the ability to induce colony formation, while both Tax-3 and antisense protein

296 o significantly induce apoptosis and inhibit colony formation with superior antitumor effects against

297 revealed an increased frequency of aberrant colony formation with the increased stiffness; however,

298 d NG108-15 cells inhibited proliferation and colony formation, with concomitant defects in AMP-activa

299 o decreased transwell invasion and soft agar colony formation, without affecting proliferation.

300 epithelial-to-mesenchymal transition (EMT), colony formation, xenograft-tumor growth in athymic mice