ライフサイエンスコーパス: colony-forming unit-erythroid

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1 y, in vitro burst-forming unit-erythroid and colony-forming unit-erythroid assays showed increased er

2 ficant reduction in fetal liver cellularity, colony forming unit-erythroid (CFU-E) progenitors, and a

3 sis of PML+/+ and PML-/- MEFs, and inhibited colony-forming unit erythroid (CFU-E) and CFU granulocyt

4 both Epo and stem cell factor (SCF) to form colony-forming unit erythroid (CFU-E) colonies.

5 ype burst-forming unit erythroid (BFU-e) and colony-forming unit erythroid (CFU-e) progenitors in res

6 While colony-forming unit-erythroid (CFU-E) and burst-forming

7 Both agents decreased colony-forming unit-erythroid (CFU-E) and colony-forming

8 red burst-forming unit-erythroid (BFU-E) and colony-forming unit-erythroid (CFU-E) formation compared

9 subsequent increase in the total numbers of colony-forming unit-erythroid (CFU-E) progenitors and er

10 gp55-P induce erythroid differentiation from colony-forming unit-erythroid (CFU-E) progenitors in fet

11 reased in Mll -/- cultures, while numbers of colony-forming unit-erythroid (CFU-E), colony-forming un

12 th antisense oligodeoxynucleotides inhibited colony-forming unit-erythroid (CFU-E)-derived colony gro

13 ng unit-erythroid (BFU-E), mature BFU-E, and colony-forming unit-erythroid (CFU-E).

14 whereas lenalidomide specifically increased colony-forming unit-erythroid (CFU-E).

15 ificant anemia despite an increase in marrow colony-forming units-erythroid (CFU-E).

16 w erythroid burst-forming units (BFU-Es) and colony-forming units-erythroid (CFU-Es) activities and r

17 and myeloid hematopoietic progenitor cells (colony-forming unit-erythroid [CFU-E], burst-forming uni

18 results in a loss of erythroid progenitors (colony-forming units-erythroid, CFU-E) and severe anemia

19 macrophage, burst-forming unit-erythroid, or colony-forming unit-erythroid colonies.

20 wn to significantly and specifically enhance colony-forming unit-erythroid colony formation.

21 shown that burst-forming units-erythroid and colony-forming units-erythroid from patients with polycy

22 losuppressive therapy in vivo and to enhance colony-forming unit-erythroid generation in vitro may be

23 er erythroid colonies derived from the later colony-forming unit erythroid progenitor erythropoietin

24 table with erythropoietin (Epo), because the colony-forming unit erythroid progenitors (CFU-Es) that

25 n and differentiation of committed erythroid colony-forming unit-erythroid progenitors by external si

26 -derived vector, and its capacity to support colony-forming unit-erythroid proliferation and developm

27 opoiesis at the burst-forming unit-erythroid/colony-forming unit-erythroid transition, but without af

28 entiation of burst-forming unit-erythroid to colony-forming unit-erythroid, we hypothesize that this

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