ライフサイエンスコーパス: colorectal neoplasm

1 ndent risk of developing CVD, T2DM, CKD, and colorectal neoplasms.

2 and 1,699 had multiple adenomas or advanced colorectal neoplasms.

3 nomas >/=5 mm, multiple adenomas or advanced colorectal neoplasms.

4 n mediating the association between diet and colorectal neoplasms.

5 odifiable, preneoplastic risk biomarkers for colorectal neoplasms.

6 ND1 A870G polymorphism may increase risk for colorectal neoplasms.

7 cid to prostaglandins and is up-regulated in colorectal neoplasms.

8 non that has previously been demonstrated in colorectal neoplasms.

9 ts a new approach for the early detection of colorectal neoplasms.

10 tyrosine kinase inhibitors for treatment of colorectal neoplasms.

11 that the above recommendations apply only to colorectal neoplasms.

12 ry pattern is associated with higher risk of colorectal neoplasms.

13 sensitivity and specificity for detection of colorectal neoplasms 10 mm and larger.

14 owel habits (7 patients, 64%), screening for colorectal neoplasm (3 pts, 27%) and lower gastrointesti

15 Colorectal neoplasms (adenomatous polyps) missed at OC b

16 The risk of colorectal neoplasms among siblings of patients with adv

17 mutations in stool samples for detection of colorectal neoplasms and compared this test with other a

18 ntributed to the elevated risk of breast and colorectal neoplasms and possibly ocular melanoma.

19 Our study shows that FAS is expressed in all colorectal neoplasms and there is a concomitant increase

20 vitamin D3 as chemopreventive agents against colorectal neoplasms, and CaR, VDR, CYP27B1, and CYP24A1

21 In contrast, approximately 15-20% of colorectal neoplasms arise through a distinct genetic pa

22 Regular use of aspirin reduces the risk of a colorectal neoplasm, but the mechanism by which aspirin

23 is a new-generation technique for detecting colorectal neoplasms by using volumetric CT data combine

24 The development of colorectal neoplasms can be characterized by an ordered

25 Colorectal neoplasms [colorectal cancer (CRC) and colore

26 Persons with no prior history of colorectal neoplasms completed comprehensive questionnai

27 Nonpolypoid colorectal neoplasms containing carcinoma were smaller i

28 t and indicate that risk of serrated pathway colorectal neoplasms could be reduced with lifestyle cha

29 We assessed colorectal neoplasm detection by a next-generation sDNA

30 The overall rates of colorectal neoplasm detection by quartiles of cecal inse

31 ion on the impact of cecal insertion time on colorectal neoplasm detection is limited.

32 association between cecal insertion time and colorectal neoplasm detection rate in colonoscopy screen

33 Although the vast majority of colorectal neoplasms develop as a consequence of somatic

34 Three different macroscopic subtypes of colorectal neoplasms display distinct carcinogenetic pat

35 associated with the development of advanced colorectal neoplasms during surveillance.

36 Both early and late colorectal neoplasms exhibit defective expression of sev

37 expression of the inducible COX-2 isoform in colorectal neoplasms from patients with hereditary nonpo

38 rocyclic amine (HCA) exposure in the risk of colorectal neoplasm has been suggested but not yet estab

39 The role of DPC4 inactivation in colorectal neoplasms has not been fully characterized.

40 t blood, has a low sensitivity for detecting colorectal neoplasms in asymptomatic patients at average

41 ound to be associated with a reduced risk of colorectal neoplasms in observational studies.

42 showing the existence of flat and depressed colorectal neoplasms in Western countries.

43 a suggest that the enhanced level of CD44 in colorectal neoplasms is asynchronous with cell replicati

44 iation of type 2 diabetes mellitus (DM) with colorectal neoplasms is contradictory, and African Ameri

45 A detailed case study on breast neoplasms, colorectal neoplasms, lung neoplasms, and 32 other disea

46 The appreciation that colorectal neoplasms may present as flat or depressed le

47 ns when screening for cancer, and many early colorectal neoplasms may therefore be missed.

48 There is concern that flat or depressed colorectal neoplasms might be easily missed during a col

49 e limited on the significance of nonpolypoid colorectal neoplasms (NP-CRNs).

50 Enhanced expression of CD44 in colorectal neoplasms occurred not only in epithelial cel

51 e identified 4,249 (33.5%) participants with colorectal neoplasms, of whom 1,956 had small single ade

52 e is to review the principles for diagnosing colorectal neoplasms on double-contrast images and the s

53 Enhancement of 29 consecutive colorectal neoplasms on pre- and postcontrast CT colonog

54 not exclude the effects of secular trends in colorectal neoplasm prevalence, the observed increase wa

55 and its expression is markedly increased in colorectal neoplasms, suggesting that expression is link