ライフサイエンスコーパス: comatose

1 noncomatose patients who subsequently became comatose.

2 core and cumulative doses of sedatives while comatose.

3 roups based on their best examination as (1) comatose, (2) arousable (eye opening or attending toward

4 ale scores grouped into four levels denoted "comatose" (-5), "deeply sedated" (-4 to -3), "lightly se

5 Before LT, the majority of patients were comatose (76%), on hemodialysis (16%), and ICU-bound.

6 mpiric use of naloxone and flumazenil in the comatose adult patient who presents to the emergency dep

7 We examined a prospective cohort of 103 comatose adult patients who were unconscious 48 hours af

8 atients were assessed for eligibility and 98 comatose adults (Glasgow Coma Scale [GCS] score of </=8

9 Consecutive comatose adults admitted after cardiac arrest, identifie

10 Therapeutic hypothermia is recommended for comatose adults after witnessed out-of-hospital cardiac

11 ed temperature management is recommended for comatose adults and children after out-of-hospital cardi

12 Patients still comatose after a witnessed OHCA of presumed cardiac orig

13 multicenter cohort were 46 patients who were comatose after CA.

14 Patients who are comatose after cardiac arrest continue to be a challenge

15 been standard of care for patients remaining comatose after resuscitation from out-of-hospital cardia

16 dicate ICP monitoring in patients who remain comatose after resuscitation if the admission computed t

17 n-traumatic cardiac arrest patients who were comatose after return of spontaneous circulation.

18 as follows: age >/=18, nontrauma arrest, and comatose after return of spontaneous circulation.

19 than 17 years, nontrauma cardiac arrest, and comatose after return of spontaneous circulation.

20 Brains of comatose ALF mice were processed for histological and bi

21 rature-sensitive paralytic mutations in NSF (comatose) also block synaptic transmission, but over a m

22 lar synapses of Drosophila double mutant for comatose (an NSF mutant) and Kum (a SERCA mutant), and p

23 chieved spontaneous circulation but remained comatose and (except one) died within 58 hrs with multip

24 uillain-Barre syndrome, or when a patient is comatose and cardinal signs may be lacking.

25 All patients were initially comatose and had invasive brain monitoring placed.

26 between 0.4 hours (for patients who are not comatose and had no prior seizure) and 16.4 hours (comat

27 thalamus, brain stem, and cerebellum between comatose and noncomatose patients acutely after TBI.

28 However, compared between comatose and noncomatose patients, CMRglc values in the

29 se and had no prior seizure) and 16.4 hours (comatose and prior seizure).

30 ositive for delirium, 13% were classified as comatose, and 62% were delirium-free and coma-free.

31 are units who were possibly brain stem dead (comatose, apparently apnoeic with unresponsive pupils) d

32 Eighty-one percent of patients who were comatose at admission survived.

33 itated by prehospital personnel yet remained comatose at arrival to the hospital.

34 H were restricted to those patients who were comatose at hospital admission.

35 was performed in 241 of 941 (25.6%) persons comatose at hospital admission.

36 Over half of patients were comatose at presentation (53%).

37 the primary admission diagnosis and 48% were comatose at the time of cEEG.

38 ain networks had typically become nonhubs of comatose brain networks and vice versa.

39 All FHF rats became comatose by 24 hours postoperatively.

40 Prognostication studies on comatose cardiac arrest (CA) patients are limited by lac

41 ppropriate time frame for prognostication in comatose cardiac arrest survivors treated with therapeut

42 In initially comatose cardiac arrest survivors, improvements in funct

43 therapeutic hypothermia improves outcomes in comatose cardiac arrest survivors.

44 hin 6 hours after the return of circulation, comatose children older than 48 hours and younger than 1

45 Among comatose children who survived in-hospital cardiac arres

46 In comatose children who survived out-of-hospital cardiac a

47 In comatose children, with Glasgow Motor Scale score less t

48 The Drosophila comatose (comt) gene encodes an NSF homolog, dNSF1.

49 temperature-sensitive paralytic phenotype in comatose (comt) mutants.

50 temperature-sensitive Drosophila NSF mutant comatose (comt) to study the function of NSF in neurotra

51 ve cerebral malaria and 264 age-matched, non-comatose controls were followed up for a median of 495 d

52 Whilst the morphology of comatose (cts) embryos is not altered, physiological ana

53 COMATOSE (CTS) encodes a peroxisomal ATP-binding cassett

54 Here we show that the Arabidopsis COMATOSE (CTS) locus is required for this transition, an

55 Using COMATOSE (CTS), a plant representative of the ABCD famil

56 phenotype comparable with that reported for comatose (cts-2), a mutant in a peroxisomal ABC transpor

57 ns, we have undertaken a genetic analysis of comatose (dNSF-1) in Drosophila.

58 Ventilator-dependent, comatose, do not resuscitate, and female patients were e

59 tire vesicle pool can be depleted in shibire comatose double mutants, demonstrating that NSF activity

60 ors had favorable outcomes despite remaining comatose for 1 to 2 weeks postarrest.

61 Fifteen comatose (Glasgow Coma Scale score of < or = 7) adult pa

62 The incidence of survival of comatose hospital patients decreased by 29% from 1975 to

63 Comatose humans have upper airway soft tissue obstructio

64 In 1960, measurements on comatose humans with or without cardiac arrest, with or

65 umented bacterial aspiration pneumonia among comatose ICU patients with symptoms suggesting either ba

66 atus, resuscitation from cardiac arrest, and comatose immediately after resuscitation.

67 peroxisomal ATP-binding cassette transporter COMATOSE is required for the accumulation of benzoylated

68 Mutation of the COMATOSE locus in Arabidopsis results in a marked reduct

69 behavioral paralysis that normally occurs in comatose mutants, indicating NSF activity is not require

70 of mammalian NSF (G274E) and Drosophila NSF (comatose) mutants revealed an evolutionarily conserved N

71 ac mutant was identified as an enhancer of a comatose mutation's effects on general locomotion.

72 Characterization of 16 comatose mutations demonstrates that NSF mediates disass

73 Performance Category 3-5, severe disability, comatose, or death).

74 able neurologic outcome in a large cohort of comatose out-of-hospital cardiac arrest patients treated

75 The classification of the comatose patient has been greatly improved with the use

76 A comatose patient has neither wakefulness nor awareness;

77 approach for the clinical management of the comatose patient.

78 evels of the inflammatory mediator IL-6 than comatose patients (129.3 vs. 35.0 pg/mL, p=0.05).

79 One hundred ten comatose patients (aged 24-76 years) who had experienced

80 nter prospective cohort study included adult comatose patients admitted to the intensive care unit (I

81 o rapidly induce and maintain hypothermia in comatose patients after cardiac arrest and a small numbe

82 res provides unequalled prognostic value for comatose patients after cardiac arrest and enables bedsi

83 Two hundred eighty-three consecutive comatose patients after cardiac arrest.

84 measures contribute to outcome prediction of comatose patients after cardiac arrest.

85 logy assessments, or functional survival for comatose patients after out-of-hospital cardiac arrest.

86 ble, the results were abnormal for all eight comatose patients as well as for all three noncomatose p

87 Excluding evaluations of comatose patients because of lack of characteristic deli

88 Parasitized comatose patients dying of other causes are less likely

89 the utility of the neurologic examination in comatose patients from nontraumatic causes in the modern

90 Acute administration of exenatide to comatose patients in the intensive care unit after OHCA

91 Among comatose patients receiving mechanical ventilation, thos

92 Comatose patients resuscitated after out-of-hospital car

93 We randomly assigned 120 consecutive comatose patients resuscitated from OHCA in a double-bli

94 In-hospital mortality in comatose patients resuscitated from out-of-hospital card

95 postresuscitation care for resuscitated but comatose patients that includes therapeutic hypothermia

96 We studied a cohort of consecutive comatose patients treated after cardiac arrest from Janu

97 A total of 110 comatose patients who had experienced OHCA from a cardia

98 l outcomes after global ischaemia-hypoxia in comatose patients who have had cardiac arrest, and is on

99 Among 249 comatose patients who received prehospital care, 205 die

100 Among 101 comatose patients who were hospitalized, 63 died; the od

101 hin 6 hours after the return of circulation, comatose patients who were older than 2 days and younger

102 d, HIPAA-compliant retrospective study of 80 comatose patients with cardiac arrest who underwent diff

103 s safe and can provide metabolic support for comatose patients with fulminant hepatic failure for up

104 This prospective cohort study included 90 comatose patients with high-grade spontaneous subarachno

105 age should be considered in all stuporous or comatose patients with intraventricular haemorrhage and

106 study, we prospectively enrolled consecutive comatose patients with irreversible brain injury undergo

107 thesized musical instrument tones in 22 post-comatose patients with severe brain injury causing varia

108 After inclusion of 98 comatose patients, the trial was stopped early at the re

109 lography responses to auditory stimuli in 94 comatose patients, under hypothermia and after re-warmin

110 g/min, P < 0.05) were significantly lower in comatose patients.

111 meeting readiness criteria, particularly for comatose patients.

112 ement are associated with acute awakening in comatose patients.

113 global network properties were conserved in comatose patients.

114 targeted temperature management) be used in comatose post-cardiac arrest patients?

115 Of our cohort of comatose postarrest patients, 59% (29 of 49) were male,

116 wn about when to assess neurologic status in comatose, postarrest patients receiving therapeutic hypo

117 Consecutive comatose postcardiac arrest patients were prospectively

118 Quantitative DWI in comatose postcardiac arrest survivors holds promise as a

119 Outcome prediction is challenging in comatose postcardiac arrest survivors.

120 or no analgesia/sedation, in a predominantly comatose sample.

121 es, spend a considerable amount of time in a comatose state that can include time in burst suppressio

122 y in the descending aorta, and pre-operative comatose state were independent predictors, again with i

123 ltrates or cavities on chest radiograph; and comatose state, intubation, receipt of pressors, or deat

124 Pre-operative cerebral malperfusion, comatose state, peripheral malperfusion, visceral malper

125 multimodality physiological recordings in 48 comatose subarachnoid hemorrhage patients to better char

126 Twenty of 56 comatose survivors (32%) treated with hypothermia who aw

127 ute ischemic stroke, induced hypothermia for comatose survivors of cardiac arrest, and endovascular c

128 Surface cooling improves outcome in selected comatose survivors of cardiac arrest.

129 hermia (TH) attenuates reperfusion injury in comatose survivors of cardiac arrest.

130 t mild induced hypothermia be considered for comatose survivors of in-hospital cardiac arrest.

131 Among comatose survivors of OHCA, in comparison with hypotherm

132 rmia attenuates brain white matter injury in comatose survivors of out-of-hospital cardiac arrest (OH

133 It is recommended that comatose survivors of out-of-hospital cardiac arrest sho

134 Among comatose survivors of out-of-hospital cardiac arrest, in

135 peutic hypothermia and neurologic outcome in comatose survivors of out-of-hospital cardiac arrest.

136 ia induced after successful resuscitation of comatose survivors of ventricular fibrillation cardiac a

137 The study cohort included 154 comatose survivors of witnessed out-of-hospital cardiopu

138 (13.5%) were excluded because they remained comatose throughout the investigation.

139 m recorded (CAM-ICU-), and 27 (29%) remained comatose until extubation or death.

140 Patients who were comatose were less likely to receive these medications.