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1 mptomatic carrier state similar to bacterial commensalism.
2 nisms other than altering lipid A to support commensalism.
3  acquiring branched-chain amino acids during commensalism.
4 ear to be a determinant for C. jejuni during commensalism.
5 ic host cells to promote pathogenesis and/or commensalism.
6 cues the inability of mutants to compete for commensalism.
7 uestion of how these molecules contribute to commensalism.
8 ion to the changing intestinal milieu during commensalism.
9 ition (10% of cases) to host-parasite (77%), commensalism (12%), and mutualism (1%).
10  is likely to play an important role in both commensalism and dissemination to cause invasive disease
11  cause phenotypes to shift from mutualism to commensalism and even parasitism.
12                                  During both commensalism and infection, it must match the immunologi
13 tation, and cell wall remodelling, influence commensalism and infection.
14 tinct genetic programs to transition between commensalism and invasive pathogenesis.
15   Our findings indicate that many aspects of commensalism and pathogenicity are intertwined and that
16 ral mutation that alters the balance between commensalism and pathogenicity.
17 ing that the protein is associated with both commensalism and the pathogenesis of disease.
18 tory IgA immunity, the regulation of mucosal commensalism, and defense of the barrier against enterop
19 t and its intestinal microflora that lead to commensalism are unclear.
20 omal cell interaction in itself is a form of commensalism, because it has been demonstrated that thes
21  enforcing mucosal tolerance and maintaining commensalism by promoting intestinal Treg cell formation
22                        In addition, frequent commensalism generates CD25(hi) (Tregs) which modulate m
23                                              Commensalism implies a hitchhiking role for viruses-self
24 tion strategies C. jejuni employs to achieve commensalism in a natural host.
25 cterial requirements necessary for promoting commensalism in a vertebrate host.
26 EtN by EptC is key to its ability to promote commensalism in an avian host and to survive in the mamm
27                                   To promote commensalism in animals and disease in humans, Campyloba
28                 This mechanism occurs during commensalism in poultry to alter the colonization capaci
29                          During infection or commensalism, induction of IL-10 by B. fragilis is criti
30                                              Commensalism is critical to a healthy Th1/Th2 cell balan
31 fer from those employed for symbiosis during commensalism is unknown.
32 ng in the states of symbiosis, colonization, commensalism, latency, and disease.
33 ains of CiaI were analysed for importance in commensalism or invasion.
34 mutualism is likely in P-limited systems and commensalism or parasitism is likely in N-limited system
35  relationships encompass obligate mutualism, commensalism, parasitism, and pathogenicity.
36 d by wild animals, are most likely shaped by commensalism related to human activities.
37                                   Successful commensalism requires dampening of the inflammatory resp
38              Phoresy is a widespread form of commensalism that facilitates dispersal of one species t
39 tic mechanism leading to the transition from commensalism to a pathogenic lifestyle.
40 sh and their worms can shift from parasitism/commensalism to mutualism as crayfish age.
41               Evidently, the transition from commensalism to opportunism in S. aureus does not requir
42  review, we will discuss the transition from commensalism to pathogenesis, the key players of the fun
43 obligatory endosymbiosis and from restricted commensalism to semi-parasitism, with the specialisation
44 investigate E. faecalis factors required for commensalism, we identified E. faecalis genes that are u
45     To understand how the bacterium promotes commensalism, we used signature-tagged transposon mutage
46 coordinated Treg-IgA response is to maintain commensalism with the microbiota.
47  microflora and the immune system to promote commensalism within the gut.

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