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1 nd distal (25 of 28 [89%] vs 24 of 28 [86%]) common bile duct.
2 li syndrome, and 30% had an isolated dilated common bile duct.
3 ry obstruction in mice with ligations of the common bile duct.
4  liver injury was induced by ligation of the common bile duct.
5 17%) of these cases there were stones in the common bile duct, 40 patients were randomised to LECBD a
6 95%) and shape (70% and 100%), and an absent common bile duct (93% and 92%).
7                                          The common bile duct and common hepatic duct were adequately
8 s filled with a silicone polymer through the common bile duct and each liver lobe embedded in Bioplas
9 the pancreatic head presented regularly with common bile duct and gastric outlet obstruction.
10           Although the parasite loads in the common bile duct and large intestine were not significan
11                                          The common bile duct and main portal vein were maintained wi
12 ediated gene transfer, 2) obstruction of the common bile duct, and 3) intravenous infusions of tauroc
13 vasion of duodenum, ampulla of Vater, and/or common bile duct, and an additional tumor invaded the po
14 ns were generated along the pancreatic duct, common bile duct, and major mesenteric vessels.
15  respectively; P > .39 for both readers), in common bile duct area (20.7 vs 21.5 mm(2), for reader 1
16 ations (unintended wounds or injuries to the common bile duct, bowel, blood vessel(s), or other organ
17 uality parameters in the pancreatic duct and common bile duct by using a five-point scale.
18 43%), ampullary cancer (n = 70; 11%), distal common bile duct cancer (n = 65; 10%), duodenal cancer (
19 duct combines with hepatic ducts to form the common bile duct (CBD) and continues along the CBD.
20 opancreatography (ERCP), sphincterotomy, and common bile duct (CBD) clearance followed by laparoscopi
21                             The treatment of common bile duct (CBD) disorders, such as biliary atresi
22 olangiography (IOC) may decrease the risk of common bile duct (CBD) injury during cholecystectomy by
23                                              Common bile duct (CBD) injury during cholecystectomy is
24 The phenotype of T cells associated with the common bile duct (CBD) is unknown.
25  AIMS: Algorithms for diagnosis of malignant common bile duct (CBD) stenoses are complex and lack acc
26 creatography (ERCP) can result in failure of common bile duct (CBD) stone removal and pancreatitis.
27 n 67% of patients with intermediate risk for common bile duct (CBD) stones require therapeutic interv
28 tis is often associated with the presence of common bile duct (CBD) stones that may require endoscopi
29     Twenty-seven of these patients (64%) had common bile duct (CBD) stones, which were cleared with a
30 amination findings suggesting a stone in the common bile duct (CBD), but these factors are not highly
31 rigin, namely, the duodenum, ampulla, distal common bile duct (CBD), or head of the pancreas.
32  vein to the microvascular blood flow in the common bile duct (CBD).
33 re was no significant difference in the mean common bile duct diameter (4.1 vs 4.3 mm for reader 1 an
34             Two independent readers recorded common bile duct diameter and area on axial CT images.
35 tment reduced the relative kidney weight and common bile duct dilation and downregulated renal expres
36 rarenal defects in this murine model include common bile duct dilation, intrahepatic biliary duct cys
37 ncluding pancreatic cysts, splenomegaly, and common bile duct dilation.
38                   RNA sequencing of NOD.Abd3 common bile duct early in disease demonstrates upregulat
39                                        Donor common bile duct excised at implantation showed preserva
40 rocedures, including splenectomy (0.7% MIS), common bile duct exploration (24.9% MIS), gastrostomy (2
41 ic cholecystectomy (ERCP+LC) vs laparoscopic common bile duct exploration with laparoscopic cholecyst
42 ake the place of invasive surgery, including common bile duct exploration, thereby decreasing the pat
43 e Cox multivariate regression analysis, only common bile duct frozen section biopsy specimen showing
44 f bile duct proliferation by ligation of the common bile duct had no effect on the expression of thes
45              ERCP showed stricture of distal common bile duct in 12 patients, irregular narrowing of
46                           Obstruction of the common bile duct in a variety of clinical settings leads
47                              Ligation of the common bile duct in mice provides an excellent model in
48 lock fibrogenesis induced by ligation of the common bile duct in rats.
49              The management of stones in the common bile duct in the laparoscopic era is controversia
50 c cholecystectomy, the rate of injury to the common bile duct increased to 0.5%, and injuries were mo
51 pic cholecystectomy appears to have a higher common bile duct injury rate and a lower mortality rate.
52                          Except for a higher common bile duct injury rate, laparoscopic cholecystecto
53                                              Common bile duct injury was defined by a second surgical
54                                              Common bile duct injury was found in 2380 (0.39%) of 613
55 e of the neonate in which the hepatic and/or common bile duct is obliterated or interrupted.
56                                   The cystic-common bile duct junction was visualized before Calot tr
57 aphy (ERCP), laparoscopic exploration of the common bile duct (LECBD), or postoperative ERCP.
58 m isolated hepatocytes of livers of sham and common bile duct-ligated (CBDL) animals showed a signifi
59 lestasis, male Sprague-Dawley rats underwent common bile duct ligation (BDL) for 14 days and were tre
60 ow that the cholestatic phenotype induced by common bile duct ligation (BDL) is reduced in mice genet
61                                              Common bile duct ligation (BDL) or feeding of a novel bi
62  In this study, Spraque-Dawley rats received common bile duct ligation (BDL) to induce cirrhosis.
63 rocholate cotransporting polypeptide (Ntcp), common bile duct ligation (BDL) was performed in pregnan
64 d the role of SHP in liver damage induced by common bile duct ligation (BDL).
65 nscription were assessed in rat livers after common bile duct ligation (CBDL) from 1-7 days, and taur
66                                    Three-day common bile duct ligation (CBDL) induced renal tubular e
67                                       In the common bile duct ligation (CBDL) model, endothelin-1 (ET
68  effects of endotoxin, ethinylestradiol, and common bile duct ligation (CBDL) on Mrp2 protein, messen
69                       To address this issue, common bile duct ligation (CBDL) was performed in wild-t
70             Cirrhosis was induced in rats by common bile duct ligation (CBDL), and they were compared
71             In a rat model of HPS induced by common bile duct ligation (CBDL), but not thioacetamide
72 imental hepatopulmonary syndrome (HPS) after common bile duct ligation (CBDL).
73 imental hepatopulmonary syndrome (HPS) after common bile duct ligation (CBDL).
74 idneys from rats with cirrhosis secondary to common bile duct ligation (CBDL).
75 ein ligation; and 1-, 2-, 3-, 4-, and 5-week common bile duct ligation animals by Northern, Western a
76 yme inhibition with tin protoporphyrin IX in common bile duct ligation animals was used to define eff
77 ntravascular monocytes/macrophages in 3-week common bile duct ligation animals, whereas pulmonary mic
78 and regulation of hepatic endothelin 1 after common bile duct ligation are not fully characterized.
79 ycoprotein levels increased severalfold with common bile duct ligation but were unchanged with either
80  experimental hepatopulmonary syndrome after common bile duct ligation by stimulating pulmonary endot
81                  Cholestasis was obtained by common bile duct ligation in mice.
82 isolated from liver and kidney 14 days after common bile duct ligation in rats and assessed by RNA pr
83 ces of hepatic endothelin 1 production after common bile duct ligation in relation to thioacetamide c
84                                              Common bile duct ligation in the rat is a model of the h
85                                    Following common bile duct ligation or left hepatic bile duct liga
86 sma endothelin 1 levels were evaluated after common bile duct ligation or thioacetamide administratio
87 reased progressively from 3 to 5 weeks after common bile duct ligation relative to controls (5-week p
88 hosis and portal hypertension due to chronic common bile duct ligation reproduce the features of huma
89                Male Sprague-dawley rats with common bile duct ligation were killed after 48 and 72 ho
90 evelopment of hepatopulmonary syndrome after common bile duct ligation, but not in thioacetamide-indu
91                               The effects of common bile duct ligation, endotoxin, and ethinylestradi
92                                        After common bile duct ligation, serum bile salts initially ro
93                     Spraque-Dawley rats with common bile duct ligation-induced cirrhosis or sham oper
94 mRNA levels, similar to that observed in rat common bile duct ligation.
95 s in a rat model of cholestasis secondary to common bile duct ligation.
96 also induced cholestasis in mouse livers via common bile duct ligation.
97 epatopulmonary syndrome developed only after common bile duct ligation.
98 noxide production have also been found after common bile duct ligation.
99 rats with chronic liver failure secondary to common bile duct ligation.
100     This study examined rats 1 to 3 wk after common bile-duct ligation (CBDL), at which time they had
101 phology, triangular cord sign, presence of a common bile duct, liver size and echotexture, splenic ap
102 ulla (n = 24), duodenum (n = 10), and distal common bile duct (n = 3) accounting for the remainder.
103 duct changes (15 of 15 patients), and distal common bile duct narrowing (12 of 15 patients) to either
104 angiocytes undergo adaptive regulation after common bile duct obstruction in the rat.
105 ECBD is as effective as ERCP in clearing the common bile duct of stones.
106 I vs type I; HR: 2.03, P = 0.030), nonpatent common bile duct (Ohi subtype: b, c, and d vs a; HR: 4.3
107  patients had either open exploration of the common bile duct or postoperative ERCP.
108  decreased with increasing distance from the common bile duct (P-trend < 0.001).
109 ental cholestasis induced by ligation of the common bile duct results in morphological and functional
110                         For reader 1, distal common bile duct scores were significantly higher with B
111 pearance, (d) pericholecystic fluid, and (e) common bile duct size and/or choledocholithiasis.
112 and/or stones (P =.003, odds ratio = 1.647), common bile duct status (P =.02, odds ratio = 2.214), an
113 ed with chronic pancreatitis who have distal common bile duct stenoses (64 patients), and 3) those wi
114 laparoscopic (10) approach, or endoscopy for common bile duct stones (2).
115 RCP) exam; even prior images had evidence of common bile duct stones (CBDS).
116                     The optimal strategy for common bile duct stones (CBDSs) encountered during chole
117 ed events (HR, 2.52; 95% CI, 1.05-6.04), and common bile duct stones (HR, 11.83; 95% CI, 1.54-91).
118                                              Common bile duct stones are unusual in children, occurri
119 ent triage resulted in the identification of common bile duct stones during preoperative ERCP in 92.3
120               Unenhanced helical CT depicted common bile duct stones in 15 of 17 patients found to ha
121 ncreatography in the evaluation of suspected common bile duct stones is discussed.
122                                  Unsuspected common bile duct stones occurred in six patients (1.4%).
123  concentrated on the management of difficult common bile duct stones using electrohydraulic lithotrip
124 , and an accuracy of 94% in the diagnosis of common bile duct stones.
125 p 1 patients underwent ERCP and clearance of common bile duct stones; group 2 patients underwent MRC;
126           At follow-up, CT abnormalities and common bile duct strictures resolved after steroid thera
127 velops extrahepatic choledochal cysts in the common bile duct, suggesting that this gene regulates di
128 nto the duodenum through the cystic duct and common bile duct system.
129 failed, the procedure was repeated until the common bile duct was cleared of stones or an endoprosthe
130 sulated dichloromethylene diphosphonate, the common bile duct was ligated and divided; sham-operated
131                In our literature review, the common bile duct was most commonly involved (56%).
132     In swine, anesthesia was induced and the common bile duct was surgically cannulated with a pediat
133 opancreatoscopic views of the pancreatic and common bile ducts were generated in 16 patients by using
134 xpressed in the human gallbladder and in the common bile duct, with only minor expression observed in

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