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1 nd distal (25 of 28 [89%] vs 24 of 28 [86%]) common bile duct.
2 li syndrome, and 30% had an isolated dilated common bile duct.
3 ry obstruction in mice with ligations of the common bile duct.
4 liver injury was induced by ligation of the common bile duct.
5 17%) of these cases there were stones in the common bile duct, 40 patients were randomised to LECBD a
8 s filled with a silicone polymer through the common bile duct and each liver lobe embedded in Bioplas
12 ediated gene transfer, 2) obstruction of the common bile duct, and 3) intravenous infusions of tauroc
13 vasion of duodenum, ampulla of Vater, and/or common bile duct, and an additional tumor invaded the po
15 respectively; P > .39 for both readers), in common bile duct area (20.7 vs 21.5 mm(2), for reader 1
16 ations (unintended wounds or injuries to the common bile duct, bowel, blood vessel(s), or other organ
18 43%), ampullary cancer (n = 70; 11%), distal common bile duct cancer (n = 65; 10%), duodenal cancer (
20 opancreatography (ERCP), sphincterotomy, and common bile duct (CBD) clearance followed by laparoscopi
22 olangiography (IOC) may decrease the risk of common bile duct (CBD) injury during cholecystectomy by
25 AIMS: Algorithms for diagnosis of malignant common bile duct (CBD) stenoses are complex and lack acc
26 creatography (ERCP) can result in failure of common bile duct (CBD) stone removal and pancreatitis.
27 n 67% of patients with intermediate risk for common bile duct (CBD) stones require therapeutic interv
28 tis is often associated with the presence of common bile duct (CBD) stones that may require endoscopi
29 Twenty-seven of these patients (64%) had common bile duct (CBD) stones, which were cleared with a
30 amination findings suggesting a stone in the common bile duct (CBD), but these factors are not highly
33 re was no significant difference in the mean common bile duct diameter (4.1 vs 4.3 mm for reader 1 an
35 tment reduced the relative kidney weight and common bile duct dilation and downregulated renal expres
36 rarenal defects in this murine model include common bile duct dilation, intrahepatic biliary duct cys
40 rocedures, including splenectomy (0.7% MIS), common bile duct exploration (24.9% MIS), gastrostomy (2
41 ic cholecystectomy (ERCP+LC) vs laparoscopic common bile duct exploration with laparoscopic cholecyst
42 ake the place of invasive surgery, including common bile duct exploration, thereby decreasing the pat
43 e Cox multivariate regression analysis, only common bile duct frozen section biopsy specimen showing
44 f bile duct proliferation by ligation of the common bile duct had no effect on the expression of thes
50 c cholecystectomy, the rate of injury to the common bile duct increased to 0.5%, and injuries were mo
51 pic cholecystectomy appears to have a higher common bile duct injury rate and a lower mortality rate.
58 m isolated hepatocytes of livers of sham and common bile duct-ligated (CBDL) animals showed a signifi
59 lestasis, male Sprague-Dawley rats underwent common bile duct ligation (BDL) for 14 days and were tre
60 ow that the cholestatic phenotype induced by common bile duct ligation (BDL) is reduced in mice genet
63 rocholate cotransporting polypeptide (Ntcp), common bile duct ligation (BDL) was performed in pregnan
65 nscription were assessed in rat livers after common bile duct ligation (CBDL) from 1-7 days, and taur
68 effects of endotoxin, ethinylestradiol, and common bile duct ligation (CBDL) on Mrp2 protein, messen
75 ein ligation; and 1-, 2-, 3-, 4-, and 5-week common bile duct ligation animals by Northern, Western a
76 yme inhibition with tin protoporphyrin IX in common bile duct ligation animals was used to define eff
77 ntravascular monocytes/macrophages in 3-week common bile duct ligation animals, whereas pulmonary mic
78 and regulation of hepatic endothelin 1 after common bile duct ligation are not fully characterized.
79 ycoprotein levels increased severalfold with common bile duct ligation but were unchanged with either
80 experimental hepatopulmonary syndrome after common bile duct ligation by stimulating pulmonary endot
82 isolated from liver and kidney 14 days after common bile duct ligation in rats and assessed by RNA pr
83 ces of hepatic endothelin 1 production after common bile duct ligation in relation to thioacetamide c
86 sma endothelin 1 levels were evaluated after common bile duct ligation or thioacetamide administratio
87 reased progressively from 3 to 5 weeks after common bile duct ligation relative to controls (5-week p
88 hosis and portal hypertension due to chronic common bile duct ligation reproduce the features of huma
90 evelopment of hepatopulmonary syndrome after common bile duct ligation, but not in thioacetamide-indu
100 This study examined rats 1 to 3 wk after common bile-duct ligation (CBDL), at which time they had
101 phology, triangular cord sign, presence of a common bile duct, liver size and echotexture, splenic ap
102 ulla (n = 24), duodenum (n = 10), and distal common bile duct (n = 3) accounting for the remainder.
103 duct changes (15 of 15 patients), and distal common bile duct narrowing (12 of 15 patients) to either
106 I vs type I; HR: 2.03, P = 0.030), nonpatent common bile duct (Ohi subtype: b, c, and d vs a; HR: 4.3
109 ental cholestasis induced by ligation of the common bile duct results in morphological and functional
112 and/or stones (P =.003, odds ratio = 1.647), common bile duct status (P =.02, odds ratio = 2.214), an
113 ed with chronic pancreatitis who have distal common bile duct stenoses (64 patients), and 3) those wi
117 ed events (HR, 2.52; 95% CI, 1.05-6.04), and common bile duct stones (HR, 11.83; 95% CI, 1.54-91).
119 ent triage resulted in the identification of common bile duct stones during preoperative ERCP in 92.3
123 concentrated on the management of difficult common bile duct stones using electrohydraulic lithotrip
125 p 1 patients underwent ERCP and clearance of common bile duct stones; group 2 patients underwent MRC;
127 velops extrahepatic choledochal cysts in the common bile duct, suggesting that this gene regulates di
129 failed, the procedure was repeated until the common bile duct was cleared of stones or an endoprosthe
130 sulated dichloromethylene diphosphonate, the common bile duct was ligated and divided; sham-operated
132 In swine, anesthesia was induced and the common bile duct was surgically cannulated with a pediat
133 opancreatoscopic views of the pancreatic and common bile ducts were generated in 16 patients by using
134 xpressed in the human gallbladder and in the common bile duct, with only minor expression observed in
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