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1 trasonographically in the distal 1 cm of the common carotid artery.
2 ents a distant athero-protected UF site, the common carotid artery.
3 n injection followed by ligation of the left common carotid artery.
4 animals underwent balloon angioplasty of the common carotid artery.
5 were subjected to balloon angioplasty of the common carotid artery.
6 monitored with a cannula placed in the left common carotid artery.
7 cipation in response to ligation of the left common carotid artery.
8 he external jugular vein is connected to the common carotid artery.
9 al assessments of the far wall of the distal common carotid artery.
10 otein E knockout mice subjected to injury of common carotid artery.
11 nt dye, after balloon angioplasty of the rat common carotid artery.
12 order via a diagnostic catheter located in a common carotid artery.
13 rmed after endothelial denudation of the rat common carotid artery.
14 gonads underwent balloon injury to the right common carotid artery.
15 ere subjected to balloon injury of the right common carotid artery.
16 rrected velocity was measured with US in the common carotid artery.
17 bility of plasma lipids and stiffness of the common carotid artery.
18 as elicited with a hydraulic occluder on the common carotid artery.
19 Carotid artery IMT was measured in the left common carotid artery.
20 nonocclusive silastic collar around the left common carotid artery.
21 tal grey matter and B-mode ultrasound of the common carotid artery.
22 low-disturbed left and the undisturbed right common carotid artery.
23 an posterior-wall CIMT of the left and right common carotid arteries.
24 y compared with its levels in uninjured left common carotid arteries.
25 ared by injection of epoxy resin through the common carotid arteries.
26 of the right middle cerebral artery and both common carotid arteries.
27 eased in the left and increased in the right common carotid arteries.
29 ression of the intima-media thickness of the common carotid artery, 0.032 vs. 0.046 mm; P=0.01; and p
31 e report that inward remodeling of the mouse common carotid artery after ligation of the ipsilateral
32 ernal iliac arteries but not the brachial or common carotid arteries and not correlated significantly
33 sed to assess the thickness of the bilateral common carotid arteries and the mean maximal IMT of 12 s
34 -Evans rats by a bilateral occlusion of both common carotid arteries and the right middle cerebral ar
35 focal ischemia was induced by occluding both common carotid arteries and the right middle cerebral ar
36 s compared with its levels in uninjured left common carotid artery and both CsA and GFPVIVIT negated
38 balloon withdrawal injury (BWI) of the left common carotid artery and continued on this drug regimen
39 beginning before balloon injury of the right common carotid artery and continuing for 14 days afterwa
40 hind limb CTA were anastomosed to recipient common carotid artery and external jugular vein without
41 Intima-media thickness of the walls of the common carotid artery and internal carotid artery may ad
42 ischemia by permanent ligation of the right common carotid artery and temporary occlusion of the rig
43 ng, and the distal descending aorta, and the common carotid artery and the brachial artery were asses
44 sured the mean intima-media thickness of the common carotid artery and the maximum intima-media thick
45 We bilaterally and transiently occluded the common carotid artery and then examined the molecular an
46 p115 RhoGEF tyrosine phosphorylation in rat common carotid arteries, and siRNA-mediated down-regulat
48 AChT-immunoreactive cells in segments of the common carotid artery, aorta, and pulmonary artery appea
49 ial chemoreceptors have been isolated to the common carotid artery, aorta, and pulmonary artery of tu
50 of the intima-medial thickness (IMT) of the common carotid artery, as an indicator of atherosclerosi
51 rnal carotid arteries were ligated, reducing common carotid artery blood flow from 8.0+/-0.6 to 0.5+/
52 t cases, paraganglioma is located around the common carotid artery, but may also be located within th
53 arotid sites; changes in maximum CIMT of the common carotid artery, carotid bulb, and internal caroti
54 Carotid IMT was measured at 3 locations (common carotid artery, carotid bulb, internal carotid ar
57 nd increased intimal-medial thickness of the common carotid artery (CCA IMT), a purported index of at
58 individuals aged 30 to 70 years, we measured common carotid artery (CCA) and carotid bulb IMT, and al
59 ndent association of whole-grain intake with common carotid artery (CCA) and internal carotid artery
60 maximal intima-media thickness (IMT) of the common carotid artery (CCA) and the internal carotid art
64 Obesity has been associated with increased common carotid artery (CCA) intima-media thickness (IMT)
66 e application of a microvascular clip to the common carotid artery (CCA) might decrease variability o
69 1.25 m/sec, internal carotid artery (ICA) to common carotid artery (CCA) peak-systolic velocity ratio
70 sized that variant geometric patterns of the common carotid artery (CCA) predict the incidence of car
72 sed Doppler ultrasound waveforms of the ECA, common carotid artery (CCA), and internal carotid artery
73 Intima-media thickness (IMT) measures of the common carotid artery (CCA), internal carotid artery (IC
74 ltrasonography and wave form analysis of the common carotid artery (CCA), with its conversion to the
77 media thickness of the right far wall of the common carotid artery (CCA-IMT) and carotid artery bifur
80 nced magnetic resonance (MR) imaging in both common carotid arteries (CCAs) and in one internal carot
82 particles, and intima-media thickness of the common carotid artery (CIMT) in elderly men residing in
84 lic strain rate were regressed on IMT of the common carotid artery defined by ultrasound, with adjust
85 Sprague-Dawley rats were subjected to left common carotid artery denudation, after which arteries w
86 ll measures of C-IMT and the interadventitia common carotid artery diameter (ICCAD) were associated w
89 n intima-media thickness of the right distal common carotid artery far wall in computer image process
90 mode ultrasound quantification of the distal common carotid artery far wall intima-media thickness (I
91 e, the annual progression rate of the distal common carotid artery far wall intima-media thickness de
92 rial end point was the rate of change in the common carotid artery far-wall intima-media thickness (I
93 schemia was induced by ligation of the right common carotid artery followed by 2 hrs of hypoxia (8% o
94 to formation of an occlusive thrombus in the common carotid artery following experimentally induced e
97 idative stress was induced by occluding both common carotid arteries for 90 min, followed by reperfus
101 cles by Sir Astley Cooper on ligature of the common carotid artery for aneurysm, John Abernethy on mi
102 ivascular application to balloon-injured rat common carotid arteries, halofuginone versus vehicle (n=
103 d I/M ratio seem preferable to estimation of common carotid artery IMT in imaging cardiovascular risk
104 morphisms was undertaken with 6 internal and common carotid artery IMT phenotypes using an additive m
105 trasound (22 MHz) to estimate the individual common carotid artery IMTs in 55 women at PE diagnosis a
106 t B-mode ultrasonography of the internal and common carotid arteries in 1994-1996 and again in 1998-2
107 2 expression was also increased in the right common carotid artery in a time-dependent manner after b
110 Compliance was significantly lower in the common carotid artery in symptomatic and asymptomatic wo
116 +1 serving; 200-250 g/d) intakes slows 12-mo common carotid artery intima media thickness (CCA IMT) p
117 associated with a significantly thicker mean common carotid artery intima, thinner media, and higher
121 (LAMP-LD) and evaluated the association with common carotid artery intima-media thickness (cCIMT) usi
123 herosclerosis, as measured by progression of common carotid artery intima-media thickness (cIMT), in
125 tween years since bilateral oophorectomy and common carotid artery intima-media thickness (IMT).
129 cation is strongly inversely associated with common carotid artery intima-media thickness in the Athe
130 Europeans (fully-adjusted P=0.004) and with common carotid artery intima-media thickness in the Fram
131 difference in the rates of change in either common carotid artery intima-media thickness or coronary
134 ography, and B-mode ultrasonography measured common carotid artery intima-medial thickness (IMT) in 5
136 naive (inversely) cells were associated with common carotid artery intimal media thickness (CC IMT) i
137 s progression of atherosclerosis measured by common carotid artery intimal medial thickness (IMT).
139 sion of intima-media thickness in the distal common carotid artery is a useful surrogate end point fo
141 of arterial remodeling in which flow in the common carotid artery is interrupted by ligation of the
142 arotid artery reduced blood flow in the left common carotid artery (LC) compared with sham and contra
143 ht common carotid artery (RCCA) and the left common carotid artery (LCCA), and fasting lipid profile
145 y 4 (P4) SD rats were subjected to bilateral common carotid artery ligation (BCAL), followed by 8% ox
146 -ischemia (HI) damage in adult mice by right common carotid artery ligation followed by hypoxia.
148 preconditioning ischemic insult (unilateral common carotid artery ligation) to 12- to 14-week-old mi
149 natal day 7 rat pups were subjected to right common carotid artery ligation, followed by 8% oxygen in
150 (P4) SD rat pups were subjected to bilateral common carotid artery ligation, followed by exposure to
151 Regions of interest were drawn within the common carotid artery lumen and immediately adjacent to
152 cerebral ischemia induced by middle cerebral/common carotid arteries occlusion (MCA/CCAo) induced up-
153 bal cerebral ischaemia in mouse is bilateral common carotid artery occlusion (BCCAo) and the neuropat
154 poperfusion and VaD was induced by bilateral common carotid artery occlusion (BCCAO) in adult male Sp
157 ischemia was induced by 10 min of bilateral common carotid artery occlusion and hypotension in rats.
158 re in adult mice that consists of unilateral common carotid artery occlusion and hypoxia with tightly
159 y-six fasted Wistar rats underwent bilateral common carotid artery occlusion and severe hypotension (
160 tized gerbils by a 5-min period of bilateral common carotid artery occlusion followed by reperfusion
162 re subjected to global ischemia by bilateral common carotid artery occlusion under controlled ventila
164 and Fpr2/3(-/-)) were subjected to bilateral common carotid artery occlusion, followed by reperfusion
165 bal ischemic injury was induced by bilateral common carotid artery occlusion, whereas severe focal st
173 ric chloride injury of the midportion of the common carotid arteries of apoE(-/-) mice (n=22) induced
174 e in vivo efficacy of the 12-LO Rz, the left common carotid arteries of rats were injured with a ball
175 m donor WT mice that had been anastamosed to common carotid arteries of recipient WT mice (WT:WT) exh
176 rterial remodeling in which flow in the left common carotid artery of FVB mice was interrupted by lig
179 ssion of intima-media thickness (IMT) of the common carotid arteries over 18 months was determined ul
183 stenosis; two additional parameters, ICA-to-common carotid artery PSV ratio and ICA end-diastolic ve
184 on, sex, and systolic blood pressure for the common carotid artery (r2=0.17); age, race, and systolic
185 ), intima-media thickness (IMT) of the right common carotid artery (RCCA) and the left common carotid
187 rotid for 14 days to lower blood flow in the common carotid artery reduced the lumen diameter of caro
188 UDP end labeling (TUNEL) in the injured rat common carotid artery show that TUNEL-positive cells in
189 38 (95% CI, -0.0064 to -0.0013) mm/y for the common carotid artery sites (P<.001), -0.0040 (95% CI, -
190 mean CIMT for the rosuvastatin group for the common carotid artery sites was 0.0004 (95% CI, -0.0011
192 le C57Bl/6J mice were subjected to bilateral common carotid artery stenosis or a sham operation and f
193 ty liver and intimal medial thickness of the common carotid arteries, subcutaneous adipose tissue, an
194 nternal carotid artery is anastomosed to the common carotid artery, this obviates the need for patch
197 aphy and CT angiography findings of the left common carotid artery trifurcation were described with i
198 emia as follows: permanent ligation of right common carotid artery under halothane anesthesia, 2-h re
199 s embolic stroke propensity through the left common carotid artery using an idealized aortic arch mod
200 The distance from the bifurcation at which common carotid artery velocity is measured should be sta
201 he PSV in the ICA to that in the ipsilateral common carotid artery (VICA/VCCA) were compared with the
202 The intima-media thickness (IMT) of the common carotid arteries was measured ultrasonographicall
203 se in the mean intima-media thickness of the common carotid artery was 1.13 (95% CI, 1.02 to 1.24), w
206 ntricle for I.C.V. injection of drugs, and a common carotid artery was catheterized to measure mean a
210 nd and arterial applanation tonometry on the common carotid artery) was lower (P:<0.05) in middle-age
213 ve rats, the middle cerebral artery and both common carotid arteries were occluded for 4.9+/-0.13 h (
215 l thickness (IMT) and plaque height from the common carotid arteries were used as noninvasive markers
216 y stenosis and intima-media thickness of the common carotid artery were assessed by using duplex ultr
217 ene (eNOS) did not remodel their ipsilateral common carotid arteries whereas wild-type mice did.
218 f endovascular balloon occlusion of the left common carotid artery, which created an aneurysm at the
219 trasonic flow probe and meter applied to the common carotid artery while cerebral blood flow was meas
221 ayed in cultured endothelial cells and mouse common carotid artery with or without OGD treatment.
222 ypoxia, induced by permanent ligation of the common carotid artery with transient hypoxia, resulted i
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