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1 , and gene involvement in human diseases and communication disorders.
2  D as a therapeutic option for ASD and other communication disorders.
3 eraction deficits, repetitive behaviors, and communication disorders.
4 and potential early indicators of speech and communication disorders.
5 nd future workforce working with progressive communication disorders.
6  understanding of the physiological bases of communication disorders.
7 rtise, and implications for the treatment of communication disorders.
8 th autism spectrum disorder (ASD) and social communication disorders.
9 terpreting clinico-anatomical correlation in communication disorders.
10 rapeutic solutions for addressing a range of communication disorders(22,23).
11 me measure assessing the association between communication disorders and participation in daily commu
12  US National Institute on Deafness and Other Communication Disorders and the US National Institute on
13  2q36.3 that segregates with early childhood communication disorders and WMH in 15 unrelated families
14 rder defined by cognitive impairment, social-communication disorder, and early-onset seizures.
15  age-related hearing loss, is the number one communication disorder, and one of the top three chronic
16 ditory feedback, and genes underlying social communication disorders, and (b) contributions of songbi
17 normal social behavior, repetitive behavior, communication disorders, and seizures.
18      One genetic factor was common to Social Communication Disorder Checklist measures across develop
19  at 8, 11, 14 and 17 years) using the Social Communication Disorder Checklist.
20 e assessments of the parent-rated Social and Communication Disorders Checklist (SCDC) spanning ages 7
21 ies above a clinical threshold on the Social Communication Disorders Checklist was strongly associate
22 r risk of ASD (HR, 1.20; 95% CI, 1.10-1.31), communication disorders (HR, 1.14; 95% CI, 1.02-1.28), a
23 previously identified separate dimensions of communication disorder in schizophrenia vary differently
24 ised and Diagnostic Interview for Social and Communication Disorders in phase 3.
25 y disorder (ADHD), autism spectrum disorder, communication disorders, intellectual disability, motor
26   Mutations in the FOXP2 gene cause a severe communication disorder involving speech deficits (develo
27 SD), intellectual disability, tic disorders, communication disorders, learning disorders, and any neu
28 the National Institute of Deafness and other Communication Disorders (NIDCD/VA).
29  Stuttering is a common and sometimes severe communication disorder, of unknown primary etiology, tha
30 ns of the human FOXP2 gene cause a monogenic communication disorder, primarily characterized by diffi
31 l alterations that encompass emotion-related communication disorders, reduced emotional arousal, init
32 ene mutations to behavior in mouse models of communication disorders, such as autism [1].