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1  understanding of the physiological bases of communication disorders.
2 rtise, and implications for the treatment of communication disorders.
3 th autism spectrum disorder (ASD) and social communication disorders.
4 terpreting clinico-anatomical correlation in communication disorders.
5  2q36.3 that segregates with early childhood communication disorders and WMH in 15 unrelated families
6  age-related hearing loss, is the number one communication disorder, and one of the top three chronic
7 ditory feedback, and genes underlying social communication disorders, and (b) contributions of songbi
8 normal social behavior, repetitive behavior, communication disorders, and seizures.
9      One genetic factor was common to Social Communication Disorder Checklist measures across develop
10  at 8, 11, 14 and 17 years) using the Social Communication Disorder Checklist.
11 ies above a clinical threshold on the Social Communication Disorders Checklist was strongly associate
12 previously identified separate dimensions of communication disorder in schizophrenia vary differently
13 ised and Diagnostic Interview for Social and Communication Disorders in phase 3.
14   Mutations in the FOXP2 gene cause a severe communication disorder involving speech deficits (develo
15 the National Institute of Deafness and other Communication Disorders (NIDCD/VA).
16  Stuttering is a common and sometimes severe communication disorder, of unknown primary etiology, tha
17 ns of the human FOXP2 gene cause a monogenic communication disorder, primarily characterized by diffi
18 l alterations that encompass emotion-related communication disorders, reduced emotional arousal, init
19 ene mutations to behavior in mouse models of communication disorders, such as autism [1].

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