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1 us is a leading cause of both nosocomial and community-acquired infection.
2 versus 16%; P < 0.001) than in patients with community-acquired infection.
3 hypervirulent strains associated with severe community-acquired infections.
4 g our ability to treat routine hospital- and community-acquired infections.
5 thogen and a leading cause of nosocomial and community-acquired infections.
6 ria, and its presence in both nosocomial and community-acquired infections.
7 us, which is a major cause of nosocomial and community-acquired infections.
8 vant pathogens responsible for hospital- and community-acquired infections.
9 timicrobial resistance, especially regarding community-acquired infections.
10 d to distinguish health-care-associated from community-acquired infections.
11 he number of methicillin-resistant S. aureus community-acquired infections.
12 or causative agent of numerous hospital- and community-acquired infections.
13 us is a major cause of severe nosocomial and community-acquired infections.
14 lity, have traditionally been categorized as community-acquired infections.
15 ureus is responsible for many nosocomial and community-acquired infections.
16 increasing number of serious nosocomial and community-acquired infections.
17 weeks to months, considerably faster than in community-acquired infections.
18 the etiological agent of many hospital- and community-acquired infections.
19 ial resistance in both hospital-acquired and community-acquired infections.
20 nce of nosocomial transmission compared with community-acquired infection?
21 reas in the smaller group of inpatients with community-acquired infections, 12 capsid and 9 polymeras
22 with shorter times to antimicrobial therapy: community-acquired infections (-53 min; p < 0.001) and h
23 17 of 31; P = 0.05); rates were similar for community-acquired infections (80% [124 of 156] versus 8
24 nclude a predisposition to opportunistic and community-acquired infections, an altered response to va
25 ains have spread throughout hospitals, while community acquired infections and other sources ensure a
26 charges) for nosocomial-acquired infections, community-acquired infections, and sepsis, respectively.
27 ra days) for nosocomial-acquired infections, community-acquired infections, and sepsis, respectively.
28 deaths) for nosocomial-acquired infections, community-acquired infections, and sepsis, respectively.
31 Of the bacteria typically responsible for community-acquired infections, clarithromycin-resistant
32 us aureus is a major cause of nosocomial and community-acquired infections for which a vaccine is gre
34 ic characteristics have been responsible for community-acquired infections in France and Switzerland.
35 he magnitude and term of morbidity of acute, community-acquired infections in immune competent patien
36 Staphylococcus aureus, is a major source of community-acquired infections in the USA, Canada, and Eu
37 anges to antibiotic treatment guidelines for community-acquired infections of the upper and lower res
38 infection (OR, 2.752 [95% CI, 1.100-6.886]), community-acquired infection (OR, 10.432 [95% CI, 3.623-
39 rmpA (OR, 17.398 [95% CI, 4.224-71.668]) and community-acquired infection (OR, 6.844 [95% CI, 1.905-2
41 ntibiotic-resistant bacteria responsible for community-acquired infections such as Salmonella spp, Ca
42 e its being a leading cause of nosocomal and community-acquired infections, surprisingly little is kn
43 ll death rate was 13%, ranging from 5% after community-acquired infections to 25% after infections ac
44 th Pneumocystis by cohabitation, to resemble community-acquired infection, underwent lung assessments
46 in an inpatient stay are considered to have community-acquired infection, while those developing sym
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