ライフサイエンスコーパス: compacta

1 lase expression in the substantia nigra pars compacta.

2 the thalamus, and the substantia nigra pars compacta.

3 inergic neurons of the substantia nigra zona compacta.

4 ventral tegmental area/substantia nigra pars compacta.

5 arkably, little to the substantia nigra pars compacta.

6 and very few fibers within substantia nigra compacta.

7 inergic neurons of the substantia nigra pars compacta.

8 ne (DA) neurons in the substantia nigra pars compacta.

9 ne (DA) neurons in the substantia nigra pars compacta.

10 x, caudate-putamen and substantia nigra pars compacta.

11 inergic neurons in the substantia nigra pars compacta.

12 egmental area (VTA) or substantia nigra pars compacta.

13 substantia nigra pars reticulata and/or pars compacta.

14 ventral tegmental area and substantia nigra compacta.

15 of degeneration of the substantia nigra pars compacta.

16 inergic neurons in the substantia nigra pars compacta.

17 nd inflammation in the substantia nigra pars compacta.

18 al tegmental area, and substantia nigra zona compacta.

19 inergic neurons in the substantia nigra pars compacta.

20 ession is noted in the substantia nigra pars compacta.

21 opaminergic neurons in substantia nigra pars compacta.

22 inergic neurons in the substantia nigra pars compacta.

23 inergic neurons in the substantia nigra pars compacta.

24 lesser extent, in the substantia nigra pars compacta.

25 opamine neurons of the substantia nigra pars compacta.

26 tory tract, and within substantia nigra pars compacta.

27 egmental area, and the substantia nigra pars compacta.

28 inergic neurons of the substantia nigra pars compacta.

29 inergic neurons of the substantia nigra pars compacta.

30 opamine neurons in the substantia nigra pars compacta.

31 ral tegmental area and substantia nigra pars compacta.

32 neuron activity in the substantia nigra pars compacta.

33 opamine neurons of the substantia nigra pars compacta.

34 inergic neurons in the substantia nigra pars compacta.

35 ine neurons from mouse substantia nigra pars compacta.

36 sal GL or in the right substantia nigra pars compacta.

37 c interneurons and the substantia nigra pars compacta.

38 RRK2 expression in the substantia nigra pars compacta.

39 s. 36+/-2.6 O.D. BMP-7-treated, p<0.05; pars compacta: 29.0+/-4.9 O.D. control vs. 64.4+/-6.9 O.D. BM

40 inergic neurons in the substantia nigra pars compacta 60 days after infection.

41 opamine neurons of the substantia nigra pars compacta, a deficit in dopamine neurotransmission and th

42 of serotonin, and the substantia nigra pars compacta, a major source of dopamine, while monkeys perf

43 most numerous in the substantia nigra, pars compacta, a region badly affected in homozygous weavers;

44 inergic neurons of the substantia nigra pars compacta, a susceptible brain region in PD.

45 gmental area (VTA) and substantia nigra pars compacta activates inhibitory postsynaptic D2-receptors

46 ons of 6-OHDA into the substantia nigra pars compacta and 1 week later were tested for startle after

47 nergic (DA) neurons in substantia nigra pars compacta and age-dependent L-DOPA-sensitive motor dysfun

48 oducing neurons in the substantia nigra pars compacta and decreased striatal dopamine levels are the

49 opaminergic neurons of substantia nigra pars compacta and in the CA1/2 pyramidal cells of hippocampus

50 inergic neurons in the substantia nigra pars compacta and intracellular inclusion called Lewy bodies.

51 inergic neurons in the substantia nigra pars compacta and locus ceruleus, without Lewy body pathology

52 gmented neurons of the substantia nigra pars compacta and locus coeruleus in all four DYT1 dystonia c

53 inergic neurons of the substantia nigra pars compacta and locus coeruleus, among other regions.

54 ergic neurons from the substantia nigra pars compacta and noradrenergic neurons from the locus coerul

55 ons) in neurons of the substantia nigra pars compacta and other brain areas.

56 sion, particularly the substantia nigra pars compacta and other dopamine-synthesizing cell groups.

57 pamine (DA) neurons in substantia nigra pars compacta and restored DA levels in striatum using two di

58 in neurons within the substantia nigra pars compacta and striatum, two regions critically involved i

59 inergic neurons in the Substantia Nigra pars compacta and terminal dopamine fiber density in the stri

60 inergic neurons in the substantia nigra pars compacta and the accumulation of the protein alpha-synuc

61 ergic neurons from the substantia nigra pars compacta and the presence, in affected brain regions, of

62 icking, in vivo in the substantia nigra pars compacta and the serum of 1-methyl-4-phenyl-1,2,3,6-tetr

63 c projections from the substantia nigra pars compacta and the ventral tegmental area to the basal gan

64 neurons located in the substantia nigra pars compacta and the ventral tegmental area, which form the

65 mine by neurons in the substantia nigra pars compacta and ventral tegmental also influences basal gan

66 The substantia nigra pars compacta and ventral tegmental area contain the two larg

67 2 were more numerous in the substantia nigra compacta and ventral tegmental area in the Ts65Dn compar

68 opamine neurons in the substantia nigra pars compacta and ventral tegmental area regulate behaviours

69 ic (DA) neurons in the substantia nigra pars compacta and ventral tegmental area regulate extrapyrami

70 nses from cells in the substantia nigra pars compacta and ventral tegmental area.

71 dies of neurons in the substantia nigra pars compacta and ventral tegmental area.

72 ogous to the mammalian substantia nigra pars compacta and was renamed accordingly; a group of gamma-a

73 (PT; homologous to the substantia nigra pars compacta and/or ventral tegmental area of mammals) to th

74 aminergic neurons, A9 (substantia nigra pars compacta) and A10 (ventral tegmental area), have distinc

75 within neurons of the substantia nigra pars compacta, and dual labeling and confocal imaging confirm

76 entral tegmental area, substantia nigra pars compacta, and nucleus of the solitary tract.

77 he ventral tier of the substantia nigra pars compacta, and the lowest levels in the ventral tegmental

78 Mn accumulation in the substantia nigra pars compacta, and thus, can represent a link between Mn expo

79 was upregulated in the substantia nigra pars compacta, and trkB was elevated in the striatum in MDMA-

80 ubthalamic nucleus and substantia nigra pars compacta, and was present at lower levels in the globus

81 inergic neurons of the substantia nigra pars compacta are autonomous pacemakers.

82 inergic neurons of the substantia nigra pars compacta are defective in Parkinson's disease, but the s

83 Because the VTA and substantia nigra pars compacta are the sole sources of striatal and limbic for

84 mic nucleus (STN), and substantia nigra pars compacta, are conserved throughout vertebrate phylogeny

85 and A10 region of the substantia nigra pars compacta as well as the technical and surgical steps ass

86 ns of the adult rodent substantia nigra pars compacta, as well their development in vitro.

87 robust gliosis in the substantia nigra pars compacta associated with significant upregulation of ind

88 inergic neurons of the substantia nigra pars compacta, become transduced.

89 eactive neurons in the substantia nigra pars compacta (both in total and in subregions) were performe

90 in neurons not only in substantia nigra pars compacta but also in other extrastriatal areas of the br

91 opamine neurons in the substantia nigra pars compacta, but not in the adjacent ventral tegmental area

92 , was observed in the substantia nigra, pars compacta, but not in the ventral tegmental area.

93 eactive neurons in the substantia nigra pars compacta by 2-fold (p < 0.05) in animals lesioned with 6

94 gmental area (VTA) and substantia nigra (SN) compacta (C).

95 ne terminal damage and substantia nigra pars compacta cell loss.

96 ns to the striatum and substantia nigra pars compacta compared with PV-GPe neurons.

97 ventral tegmental area-substantia nigra pars compacta complex.

98 gic neurons within the substantia nigra pars compacta coupled with depletion of striatal dopamine.

99 inergic neurons in the substantia nigra pars compacta coupled with intracytoplasmic inclusions known

100 opamine neurons in the substantia nigra pars compacta, culminating in severe motor symptoms, includin

101 neurons of the brain's substantia nigra pars compacta die in Parkinson's disease has been an enduring

102 dopaminergic neurons in the substantia nigra compacta die via apoptosis in Parkinson's disease.

103 cordings of identified substantia nigra pars compacta dopamine neurons in R1441C LRRK2 transgenic rat

104 of torsinA mRNA in the substantia nigra pars compacta dopamine neurons, the locus ceruleus, the cereb

105 reby properly maintain substantia nigra pars compacta dopaminergic neurons and their innervation in t

106 Studying mouse substantia nigra pars compacta dopaminergic neurons both in brain slice and af

107 We show that, in rat substantia nigra pars compacta dopaminergic neurons, the voltage dependences o

108 is expressed in human substantia nigra pars compacta glia as well as tyrosine hydroxylase-positive n

109 forming an ultra-short substantia nigra pars compacta --> SNr dopamine pathway that regulates the fir

110 mine (6-OHDA) into the substantia nigra pars compacta in 10 rats.

111 urodegeneration in the substantia nigra pars compacta in aged VMAT2 LO mice.

112 e vulnerability of the substantia nigra pars compacta in PD, why the disorder is age related, and the

113 ty (TH-ir) in the substantia nigra (SN) pars compacta, in the lesioned hemisphere [31.7+/-5.2 (optica

114 inergic neurons of the substantia nigra pars compacta is a cardinal feature of Parkinson's disease (P

115 ation of DA neurons in substantia nigra pars compacta is a key neuropathological feature in Parkinson

116 inergic neurons in the substantia nigra pars compacta is the primary cause for motor symptoms observe

117 onial pterobranch hemichordate, Rhabdopleura compacta, is expressed in a dramatically different patte

118 inergic neurons of the substantia nigra pars compacta leading to abnormal activity within the basal g

119 inergic neurons in the substantia nigra pars compacta, leading to nigrostriatal degeneration.

120 ucleus and dorsomedial substantia nigra pars compacta) mesodiencephalic dopaminergic neuron subset, u

121 haracterized by severe substantia nigra pars compacta neuronal loss.

122 kB) receptor occurs in substantia nigra pars compacta neurons and is required for neuroprotection.

123 Unilateral lesions of substantia nigra pars compacta neurons created rats with hyperexcitable trigem

124 spontaneous firing of substantia nigra pars compacta neurons, isolated from transgenic mice in which

125 dopaminergic (DAergic) substantia nigra pars compacta neurons, only select downregulation of the PHD2

126 nger tapping is a clinical correlate of pars compacta nigral degeneration.

127 characterised by the presence of severe pars-compacta nigral-cell loss, and accumulation of aggregate

128 s in culture or in the substantia nigra pars compacta of 5-wk-old mice.

129 ell body counts in the substantia nigra pars compacta of 6-OHDA- and free-3NT-treated mice (injected/

130 le DA neurons from the substantia nigra pars compacta of controls and subjects with idiopathic Parkin

131 opamine neurons of the substantia nigra pars compacta of mice following systemic administration of a

132 opamine neurons in the substantia nigra pars compacta of monkeys (Macaca mulatta) during a reaching t

133 21(ras) in vivo in the substantia nigra pars compacta of MPTP-intoxicated mice.

134 of glial cells in the substantia nigra pars compacta of Parkinson's disease.

135 been identified in the substantia nigra pars compacta of patients with sporadic PD.

136 s activated within the substantia nigra pars compacta of PD patients and MPTP-intoxicated mice.

137 so up-regulated in the substantia nigra pars compacta of post-mortem PD brains as compared with age-m

138 so demonstrated in the substantia nigra pars compacta of post-mortem PD brains.

139 gic neurons within the substantia nigra pars compacta of the midbrain.

140 rya and primary dendrites, while in the pars compacta of the PPN (PPNc) axosomatic synapses were rare

141 neration of dopaminergic neurons in the zona compacta of the substantia nigra.

142 ositive neurons in the substantia nigra pars compacta of wild-type mice.

143 sitive neurons in the substantial nigra pars compacta one week after the first dose of MPTP.

144 d in TH-ir soma within substantia nigra pars compacta, or in TH-ir striatal terminals.

145 opamine neurons of the substantia nigra pars compacta, other regions of the midbrain, and also the hi

146 in injured IL-6 (+/+) and IL-6 (-/-) SN pars compacta (pc), microgliosis was severely compromised in

147 amygdalopallii pars basalis (PoAb) and pars compacta (PoAc), the nucleus taeniae amgygdalae (TnA) an

148 inergic neurons in the substantia nigra pars compacta portion of the brain.

149 inergic neurons of the substantia nigra pars compacta preferentially terminate in patch-like regions

150 leasing neurons of the substantia nigra pars compacta produce an extraordinarily dense and expansive

151 (DA) neurons in their substantia nigra pars compacta, progressing to bilateral degeneration of the n

152 lis, r. subcuneiformis, r. peduncularis pars compacta, r. subcoeruleus pars alpha, raphe obscurus, ra

153 inergic neurons in the substantia nigra pars compacta region of the brain.

154 the ventral horn of the spinal cord and the compacta region of the substantia nigra.

155 inergic neurons in the substantia nigra pars compacta, reproducing an important pathological feature

156 Neuronal counts in the substantia nigra pars compacta revealed that neuronal loss in the parkin cases

157 ne hydroxylase mRNA in substantia nigra pars compacta (SC) (93%; P<0.05) compared with control rats.

158 l proliferation in the substantia nigra pars compacta (SN(C)) with a time-dependent adoption of a neu

159 inergic neurons in the substantia nigra pars compacta (SN) leads to debilitating motor dysfunction.

160 on was observed in the substantia nigra pars compacta (SN), ventral tegmental area (VTA) and retrorub

161 n-stained cells in the substantia nigra pars compacta (SN-PC)].

162 inergic neurons in the substantia nigra pars compacta (SNc) and consequent depletion of striatal dopa

163 ks the majority of the substantia nigra pars compacta (SNc) and experiences striatal denervation.

164 ine hydroxylase (TH) in the substantia nigra compacta (SNc) and in two subdivisions of the ventral te

165 nergic neurones in the substantia nigra pars compacta (SNc) and may contribute to excitotoxic cell de

166 ([DA](o)) between the substantia nigra pars compacta (SNc) and striatum.

167 n between the midbrain substantia nigra pars compacta (SNc) and the CeA.

168 opamine neurons of the substantia nigra pars compacta (SNc) and the importance of protein aggregation

169 inergic neurons in the substantia nigra pars compacta (SNc) and the presence of intracytoplasmatic in

170 amine neurons from the substantia nigra pars compacta (SNc) and ventral tegmental area (VTA) are invo

171 dopamine centers, the substantia nigra pars compacta (SNc) and ventral tegmental area (VTA), densely

172 f RET in the maintenance of substantia nigra compacta (SNC) and ventral tegmental area (VTA), we bred

173 n the ipsilateral MZI, substantia nigra zona compacta (SNC) and ventral tegmental area (VTA).

174 opamine neurons in the substantia nigra pars compacta (SNC) and ventral tegmental area (VTA).

175 ns that project to the substantia nigra pars compacta (SNc) are activated by a visual CS for food.

176 gmental area (VTA) and substantia nigra pars compacta (SNc) are not significantly modulated by anesth

177 gmental area (VTA) and substantia nigra pars compacta (SNC) contain dopamine neurons intermixed with

178 gmental area (VTA) and substantia nigra pars compacta (SNc) convey distinct signals.

179 Substantia nigra pars compacta (SNc) dopamine neurons and their targets are in

180 have established that substantia nigra pars compacta (SNc) dopamine neurons are a key node in the ci

181 underlying the loss of substantia nigra pars compacta (SNc) dopaminergic neurons in Parkinson's disea

182 Burst spiking in substantia nigra pars compacta (SNc) dopaminergic neurons is a key signaling e

183 increased activity of substantia nigra pars compacta (SNc) dopaminergic neurons, elevated baseline e

184 epend on the firing of substantia nigra pars compacta (SNc) dopaminergic neurons, we identified and c

185 ogous to the mammalian substantia nigra pars compacta (SNc) evokes increasing activation of MLR cells

186 inergic neurons of the substantia nigra pars compacta (SNc) exhibit functional heterogeneity that lik

187 ic (DA) neurons of the substantia nigra pars compacta (SNc) govern movements requires a detailed know

188 ons (TH-IR) within the substantia nigra pars compacta (SNc) in both young and aged animals.

189 tal area (VTA) and the substantia nigra pars compacta (SNc) mDA neurons.

190 receptor in regulating substantia nigra pars compacta (SNc) neuron physiology in both mice and rats.

191 ordings were made from substantia nigra pars compacta (SNC) neurons in horizontal brain slice prepara

192 s differs from that in substantia nigra pars compacta (SNc) neurons, where subthreshold calcium curre

193 much stronger in monkey than in rat SN pars compacta (SNc) neurons, whereas a moderate level of mGlu

194 drites of dopaminergic substantia nigra pars compacta (SNc) neurons.

195 eterogeneity among the substantia nigra pars compacta (SNc) neurons.

196 ositive neurons in the substantia nigra pars compacta (SNc) of C57bl/6J mice following MPTP administr

197 rAAV2/5 vectors in the substantia nigra pars compacta (SNc) on one side of the brain; the other side

198 as seen in neighboring substantia nigra pars compacta (SNC) or substantia nigra pars reticulata.

199 ave been implicated in substantia nigra pars compacta (SNc) pathology in Parkinson's disease.

200 nt dopamine neurons in substantia nigra pars compacta (SNc) play such roles.

201 The substantia nigra pars compacta (SNc) projects specifically into the rostral mi

202 aminergic cells in the substantia nigra pars compacta (SNc) respond immediately to unexpected conditi

203 ne (DA) release in the substantia nigra pars compacta (SNc) shows a limited dependence on extracellul

204 neurons, including the substantia nigra pars compacta (SNc) subpopulation that preferentially degener

205 e neuron in the monkey substantia nigra pars compacta (SNc) that retains past learned reward values s

206 viral vector into the substantia nigra pars compacta (SNc) to investigate its influence on nigrostri

207 gmental area (VTA) and substantia nigra pars compacta (SNc) to that of axonal dopamine release in the

208 atal pathway, from the substantia nigra pars compacta (SNc) to the dorsal striatum, and on related fu

209 tions of rAAV into the substantia nigra pars compacta (SNc) transduced both dopaminergic and non-dopa

210 low or Fluorogold into substantia nigra pars compacta (SNc) were combined with larger injections of T

211 neuron numbers in the substantia nigra pars compacta (SNC) were estimated using stereological method

212 ne (DA) neurons of the substantia nigra pars compacta (SNc) were found to exhibit sustained responses

213 olliculus (SC), to the substantia nigra pars compacta (SNc) where direct synaptic contacts are made w

214 inergic neurons in the substantia nigra pars compacta (SNc), but not in ventral tegmental area or sub

215 stantia nigra pars reticulata (SNr) and pars compacta (SNc), but not the thalamus.

216 rst established in the substantia nigra pars compacta (SNc), but their validity in the VTA is uncerta

217 inergic neurons of the substantia nigra pars compacta (SNc), in addition to many other regions, inclu

218 suggested that, in the substantia nigra pars compacta (SNc), the pacemaking relies more on Ca(2+) cha

219 he retrorubral field (RRF), substantia nigra compacta (SNc), ventral tegmental area (VTA), and ventro

220 to dopamine neurons in substantia nigra pars compacta (SNc), whereas matrix neurons receive sensorimo

221 ine neurons within the substantia nigra pars compacta (SNc).

222 inergic neurons in the substantia nigra pars compacta (SNc).

223 ell bodies in the substantia nigra (SN) pars compacta (SNc).

224 ne (DA) neurons in the substantia nigra pars compacta (SNc).

225 t in DA neurons of the substantia nigra pars compacta (SNc).

226 nergic neurones of the substantia nigra pars compacta (SNc).

227 ites of neurons in the substantia nigra pars compacta (SNc).

228 egmental area (VTA) and the substantia nigra compacta (SNc).

229 al tegmental area (VTA) and substantia nigra compacta (SNc).

230 ssed GFRalpha-1 in the substantia nigra pars compacta (SNC).

231 ine neurons within the substantia nigra pars compacta (SNc).

232 inergic neurons in the substantia nigra pars compacta (SNc).

233 inergic neurons of the substantia nigra pars compacta (SNc).

234 gmental area (VTA) and substantia nigra pars compacta (SNc).

235 inergic neurons in the substantia nigra pars compacta (SNc).

236 or, a homologue of the substantia nigra pars compacta (SNc)/ventral tegmental area (VTA) is present i

237 inergic neurons of the substantia nigra pars compacta (SNpc) against 6-OHDA and MPTP.

238 nergic neurons of the substantia nigra, pars compacta (SNpc) akin to what is observed in Parkinson di

239 ic (DA) neurons in the substantia nigra pars compacta (SNpc) and of noradrenergic neurons in the locu

240 ntly diminished in the substantia nigra pars compacta (SNpc) and striatum, regions most affected in h

241 ese neurons in the rat substantia nigra pars compacta (SNpc) and ventral tegmental area (VTA), and co

242 opamine neurons in the substantia nigra pars compacta (SNpc) and widespread aggregates of the protein

243 ters, compromising the substantia nigra pars compacta (SNpc) and, later, the cerebral cortex.

244 opamine neurons in the substantia nigra pars compacta (SNpc) are greatly needed to effectively change

245 inergic neurons in the substantia nigra pars compacta (SNpc) as seen in Parkinson's disease.

246 opamine neurons of the substantia nigra pars compacta (SNpc) by regulating the magnitude of the first

247 (THir) neurons in the substantia nigra pars compacta (SNpc) compared with saline treatment.

248 e hydroxylase-immunoreactive (TH-ir) SN pars compacta (SNpc) DA neurons are immunoreactive for estrog

249 DA) neurons within the substantia nigra pars compacta (SNpc) display a differential vulnerability to

250 generation of midbrain substantia nigra pars compacta (SNpc) dopaminergic (DA) neurons contributes to

251 acterized by a loss of substantia nigra pars compacta (SNpc) dopaminergic (DA) neurons, and can be mo

252 ns of the ventral tier substantia nigra pars compacta (SNpc) failed to demonstrate a preponderance of

253 inergic neurons of the substantia nigra pars compacta (SNpc) of human PD patients.

254 neration occurs in the substantia nigra pars compacta (SNpc) of patients with Parkinson's disease and

255 has been shown in the substantia nigra pars compacta (SNpc) of PD models when there has been a decre

256 sfer of RGS10 into the substantia nigra pars compacta (SNpc) of rats reduced microgliosis and protect

257 's disease (PD), in the substantia nigra par compacta (SNpc) of the brain in a PD mouse model.

258 neurons at the ventral substantia nigra pars compacta (SNpc) preferentially degenerate in Parkinson's

259 inergic neurons in the substantia nigra pars compacta (SNpc) undergo natural cell death during develo

260 s containing CR in the substantia nigra pars compacta (SNpc) were relatively spared compared to those

261 Here, we show that the substantia nigra pars compacta (SNpc), a brain region where dopamine (DA) cell

262 opaminergic neurons in substantia nigra pars compacta (SNpc), and there was no loss of dopaminergic n

263 ore TH-ir cells in the substantia nigra pars compacta (SNpc), but this difference was significant onl

264 degeneration of DNs in substantia nigra pars compacta (SNpc), decreased striatal dopamine levels and

265 neurons located in the substantia nigra pars compacta (SNpc), expression of monoamines and indolamine

266 ensity loss within the substantia nigra pars compacta (SNpc), locus coeruleus, and ventral tegmental

267 g GDF5 into either the substantia nigra pars compacta (SNpc), the lateral ventricle (LV) or the stria

268 aminergic cells in the substantia nigra pars compacta (SNpc), ventral tegmental area (VTA), and retro

269 nergic (DA) neurons in substantia nigra pars compacta (SNpc).

270 inergic neurons of the substantia nigra pars compacta (SNpc).

271 ic (DA) neurons in the substantia nigra pars compacta (SNpc).

272 on was detected in the substantia nigra pars compacta, striatum, hippocampus, or selected regions of

273 ccumulation within the substantia nigra pars compacta, suggesting that nigrostriatal dopamine dysfunc

274 ccumulation within the substantia nigra pars compacta, suppression of microglial activation, and incr

275 evel, dorsal raphe terminals in the PPT pars compacta synapse mainly with dendrites and axosomatic co

276 the BG, and dopaminergic neurons of the pars compacta that modulate thalamic excitability.

277 inergic neurons in the substantia nigra pars compacta, the exact mechanism involved is still poorly u

278 In the substantia nigra pars compacta, there was a 50-90% loss of tyrosine hydroxylas

279 allidus, thalamus, and substantia nigra pars compacta to various environmental or genetic insults.

280 a (nucleus tegmenti pedunculo-pontinus, pars compacta, TPc), and the locus coeruleus in the brainstem

281 and a 42% reduction in substantia nigra pars compacta tyrosine hydroxylase-positive neurons at 8 week

282 ociated disorders, the substantia nigra pars compacta undergoes degeneration, but the mechanism of ce

283 taining neurons in the substantia nigra pars compacta use pacemaking mechanisms common to neurons not

284 ps of neurons, such as substantia nigra pars compacta, ventral tegmental area and retrorubal field, t

285 pus and dentate gyrus, substantia nigra pars compacta, ventral tegmental area, geniculate nucleus and

286 the SN allowing readers to distinguish pars compacta ventralis and dorsalis from pars reticulata.

287 rminals in the ventral substantia nigra pars compacta (vSNc) or to the ventral tegmental area (VTA),

288 the nucleus tegmenti pedunculopontinus pars compacta was recognized as homologous to the mammalian s

289 ongly expressed in the substantia nigra pars compacta, was present at lower levels in the striatum, g

290 TRPM2 and TRPM4mRNA in substantia nigra pars compacta.We propose that ICAN is selectively activated d

291 opamine neurons in the substantia nigra pars compacta were immunonegative.

292 taining neurons in the substantia nigra pars compacta were located within the calbindin-rich zone, wh

293 ommissural putamen and substantia nigra pars compacta were processed for tyrosine hydroxylase and dop

294 aminergic cells in the substantia nigra pars compacta were unaffected by METH or haloperidol alone or

295 dorsolaterally in the substantia nigra pars compacta, whereas neurons inhibited by the stimuli were

296 ic (DA) neurons of the substantia nigra pars compacta, which are preferentially lost in human Parkins

297 e DYT1 gene within the substantia nigra pars compacta, which provides dopaminergic innervation to the

298 ify mDA neurons of the substantia nigra pars compacta while the late Shh and Gli1 lineages maintain t

299 inergic neurons in the substantia nigra pars compacta, with more than 40% of these neurons lost by ag

300 d neuron counts in the substantia nigra pars compacta, yet striatal medium spiny neuron dendritic spi