ライフサイエンスコーパス: companion paper

1 new genes: Gpr65, Plzp, Toso, and Cd5l (in a companion paper).

2 ) and food as unconditional stimulus (US) [ (companion paper)].

3 as based on studies described in a preceding companion paper.

4 article is part of a Special Issue entitled Companion Paper.

5 sional cellular microscopy, described in the companion paper.

6 ta-analysis papers are discussed in the next companion paper.

7 comparison of arms A and B is described in a companion paper.

8 mer's Disease Genetic Consortium (ADGC) in a companion paper.

9 comparison of arms A and C is described in a companion paper.

10 hes, EMG sites and motor units reported in a companion paper.

11 ctivity of KatG is addressed here and in our companion paper.

12 investigated and discussed in the following companion paper.

13 nd high-hroughput AMS measurement are in the companion paper.

14 e algorithms and parameters discussed in the companion paper.

15 for the preparation of AHA is presented in a companion paper.

16 of the DURS2 phenotype, as described in the companion paper.

17 n alternative route to AHA is presented in a companion paper.

18 of the selected sequences is described in a companion paper.

19 d in vitro, and the data are reported in the companion paper.

20 entatively identified as cytokeratin 18 in a companion paper.

21 ucture of wild-type HPPK as described in the companion paper.

22 ribution of ejecta blocks are discussed in a companion paper.

23 ies of the 'ponds' are discussed in a second companion paper.

24 and propriospinal input was examined in the companion paper.

25 chromatography (IC), which is discussed in a companion paper.

26 brovascular risk factors is the subject of a companion paper.

27 g the forward and backward potential sweeps (companion paper 1), the semiempirical treatment here, wh

28 agenic activity results are presented in our companion paper.1 The results show that ELCR values for

29 in complex with both enzymes described in a companion paper, (34) provides a rationale for the obser

30 We have presented in a companion paper a suppressor-based electrodialytic buffe

31 This outcome is evaluated further in a companion paper appearing later in this journal.

32 o be a source of confusion.Dual Perspectives Companion Paper: Are the Neural Correlates of Consciousn

33 In a companion paper both mutants are shown to be deficient i

34 A companion paper by McClintock, printed and bound back-to

35 A companion paper comes to similar conclusions on the basi

36 Our companion paper describes the performance of the model f

37 A companion paper describes the thalamic connections of CM

38 The companion paper () describes two neurophysiological corr

39 A companion paper discusses several other utilitarian attr

40 The companion paper (DOI: 10.1021/jm100060b) describes how t

41 Evidence is provided in a companion paper for an IL-7-associated molecular complex

42 ype method for amino acids, developed in the companion paper in this issue of the Journal, are used.

43 A companion paper in this issue provides details on demogr

44 of aging-associated variables reported in a companion paper in this issue) and targeted replication

45 free and DNA-bound forms is presented in the companion paper in this issue.

46 xperiments complement those presented in the companion paper in which binding and protonation of CH3-

47 lyse new data, presented in more detail in a companion paper, in which BrdU/IdU cell-labelling experi

48 matic studies reported in this article and a companion paper of theoretical calculations of the nanog

49 ng therapies in the future.Dual Perspectives Companion Paper: Parkinson's Disease Is Not Simply a Pri

50 As reported in a companion paper (Piperno DR, et al., in this issue of PN

51 ence that Slit is the midline Robo ligand; a companion paper presents biochemical evidence that Slit

52 This is the second of two companion papers proposing priority problems for researc

53 The companion paper provided evidence that patch-controlled

54 ut the etiopathology of PD.Dual Perspectives Companion Paper: Prying into the Prion Hypothesis for Pa

55 This work is accompanied by a companion paper (Pt.2/2).

56 lation of gene expression was described in a companion paper published in this journal, using flux-ba

57 (Companion papers report on the plasma, magnetic field, p

58 f our genetic humanization approach, and the companion paper reports that the humoral immune systems

59 promising research avenues.Dual Perspectives Companion Paper: Should a Few Null Findings Falsify Pref

60 and an antibody-stabilized active structure (companion paper) shows how binding events at both the ex

61 er results, some of which are presented in a companion paper, suggest that DspA acts as a global regu

62 een GluR6Q and GluR2Q(flip), reported in the companion paper, suggests that at a glutamate concentrat

63 urinary signals that can be quantified by a companion paper test.

64 We showed in the companion paper that a member of the cadherin family (ze

65 p and a YAC-based physical map reported in a companion paper, the fundamental maps required for mouse

66 In a companion paper, the method is applied to data from the

67 In two companion papers, the model developed herein has been ap

68 consistent with bioaccumulation results in a companion paper (this issue) observed for amphipods.

69 consistent with bioaccumulation results in a companion paper (this issue) using a microbial bioreport

70 In a companion paper (this issue), we reported that repeated

71 chimeric mice with deletion of Tssk1 and 2 (companion paper) this centriolar kinase/substrate pair i

72 and the gp32 monomer-binding results of the companion paper to propose a detailed model for how gp32

73 In the companion paper to this work, we described development o

74 A companion paper uses pre-explosion images to rule out lu

75 In a companion paper we demonstrate that the C-rich strand of

76 In the companion paper we reported that Bacillus subtilis requi

77 In a companion paper we use growth data from lake trout (Salv

78 In these two companion papers we introduce a new approach to the anal

79 As we describe in the companion paper, we attempted to make mice that more eff

80 In a companion paper, we demonstrate that mutations at a diff

81 In an accompanying companion paper, we demonstrate the applicability of our

82 In the companion paper, we demonstrate the utility of F(n)()Ys

83 In a companion paper, we identified a novel CK2 site adjacent

84 In a companion paper, we provided indirect evidence that cell

85 In a companion paper, we reported a data integration methodol

86 In a companion paper, we reported that the goldfish oculomoto

87 In a companion paper, we show that spontaneous release from t

88 In a companion paper, we show that these defects can be mimic

89 In a companion paper, we study the contributions of linker hi

90 In the companion paper, we used the virtual space (VS) techniqu

91 In this first of two companion papers, we analyze the natural sources of lumi

92 In these two companion papers, we introduce a new approach to the ana

93 In two companion papers, we present a list of problems nominate

94 ysical properties (with the exception of the companion paper where Tapia et al. demonstrate a direct