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1 nd presented differential expression of endothelial markers compared to WT.
2 itrosoglutathione concentrations were higher in mutant mice compared to WT.
3 py, significantly decreased DKO and Mdr2(-/-) ERC viability compared to WT.
4 oryzae strains lacking NDK1 through targeted gene deletion, compared to WT.
5 in central waves (II-IV) that in some cases disappear when compared to WT.
6 te early (MIE) enhancer results in inefficient reactivation compared to WT.
7 in Src-1/-2 double-deficient (Src-1/-2(d/d) ) fetal lungs, compared to WT.
8 yoblasts demonstrated delayed and reduced fusion efficiency compared to WT.
9 lm, T2DM hearts exhibited improved contractility/relaxation compared to WT, accompanied by extensive metabolic remodellin
10 ce lung neutrophil myeloperoxidase levels in PLD2(-/-) mice compared to WT and PLD1(-/-) mice, confirming a novel role of
13 sed calcium deposition, phenotypic change and sEV secretion compared to WT CASMCs, which was associated with reduced lyso
14 ly increased oxidative phosphorylation and basal glycolysis compared to WT cells and correlated with motor dysfunction.
18 in the brains of J20-hAPP mice were substantially enhanced compared to WT controls, but this effect disappeared under no
33 tin administration during the active phase (nighttime) when compared to WT mice and treatment during the inactive phase (
36 educed synaptic vesicle pool and impaired neurotransmission compared to WT mice, while nCLCa-only mice had increased syna
37 ology we find feedforward excitatory drive is reduced in HD compared to WT mice, while recurrent inhibition also shows ph
48 in system (RAS) upregulated in the kidney of KS-tg/OVE mice compared to WT/OVE mice, suggesting a disturbed balance betwe
50 ffects (approximately 2- to 93-fold improvements) of Mut268 compared to WT using targeted deep-sequencing analyses.