1 dissociation rate constant (k(2)) is dramatically increased
compared with WT.
2 in the abundance of other cell-wall-related monosaccharides
compared with WT.
3 me/total volume and trabecular number in Col6alpha2-KO mice
compared with WT.
4 f2 activity is absent in the retina of REDD1-deficient mice
compared with WT.
5 We observed delayed wood morphogenesis in eki
compared with WT,
along with a more mechanically elastic camb
6 es revealed hypermethylation during leaf senescence in dml3
compared with WT,
and 20 556 differentially methylated region
7 Compared with WT animals, mice deficient in IL-17D experience
8 zation of paraoxonase 1 (PON1) activity (mumol/min/mg) when
compared with WT apoA-I and comparable PON1 activation/stabil
9 led strikingly similar changes across several lipid classes
compared with WT bacteria.
10 of NF-kappaB-dependent inflammatory mediators in mCAT BMDMs
compared with WT BMDMs.
11 rived BRAF variants, has a higher dimerization potential as
compared with WT BRAF.
12 duces nitric-oxide production and increases eNOS decoupling
compared with WT calmodulin.
13 cells produced higher amounts of proinflammatory cytokines
compared with WT CAR T cells.
14 ient CD4(+) T cells express higher levels of IFN-gamma mRNA
compared with WT CD4(+) T cells.
15 ous and larger clusters in the presence of zymosan in vitro
compared with WT cells, and the effect was also LTB4- and BLT
16 es (29% higher), and maximal respiratory rates (37% higher)
compared with WT cells.
17 ol6alpha2-KO cells expressed higher levels of TNFalpha mRNA
compared with WT cells.
18 bited reduced body weight, adiposity, and hepatic steatosis
compared with WT controls.
19 mitochondria and fatty acid oxidation in RYR1 mutants when
compared with WT controls.
20 oss of chemoconvulsant-induced increases in sIPSC responses
compared with WT controls.
21 Compared with WT counterparts, IFN-gammaR-deficient PLP-CD8 w
22 expression of mutant murine GABA(A)Rs is severely impaired
compared with WT,
due to retention in the endoplasmic reticul
23 d previously that monomeric GH-C53S has reduced bioactivity
compared with WT GH (GH-WT) because of its decreased ability
24 ional KI GlyRs that were rather insensitive to ethanol when
compared with WT GlyRs.
25 Additionally, when
compared with WT hESCs, cardiac differentiation from ARID1A (
26 nation, locomotor activity, or conditioned place preference
compared with WT littermate controls.
27 evels of proinflammatory cytokines within the neural tissue
compared with WT littermates.
28 otential was significantly reduced in homozygous del10 mice
compared with WT littermates.
29 Young's modulus, indicative of greater cellular stiffness,
compared with WT mESCs.
30 mmation, focal neutrophil infiltrates were increased in CGD
compared with WT mice and associated with higher LTB4 levels.
31 As
compared with WT mice, Clec2d(-/-) mice exhibited reduced pro
32 Compared with WT mice, cyclophilin D-knockout littermates did
33 lasts, which revealed that mutant mice had more osteoclasts
compared with WT mice, indicating that the primary effect of
34 Compared with WT mice, Pd1-/- mice exhibited increased baseli
35 In hWtEPOR
compared with WT mice, scotopic a-wave amplitudes were reduce
36 As
compared with WT mice, we show that the activity of IL-13 is
37 nd P2Y13 was significantly decreased in MECs from TSP1(-/-)
compared with WT mice, whereas several extracellular matrix a
38 in the first days of the drinking in the dark protocol, as
compared with WT mice.
39 s, VIP, and purinergic receptors are decreased in TSP1(-/-)
compared with WT mice.
40 ve crush, CnB(scko) mice have slower degeneration of myelin
compared with WT mice.
41 utrophilic alveolitis, and greater lung inflammation/injury
compared with WT mice.
42 nsgenic mice lacking GDE3 displayed doubling of LPI content
compared with WT mice.
43 lthough contraction of MECs from TSP1(-/-) mice was reduced
compared with WT mice.
44 ated disease course in cortactin gene-deficient female mice
compared with WT mice.
45 IP and UTP was significantly smaller in MECs from TSP1(-/-)
compared with WT mice.
46 rdiac function, structure, and baseline electrical activity
compared with WT (
n=10).
47 ion at 90% repolarization and after 10 nmol/L isoproterenol
compared with WT (
n=7; P<0.05).
48 n was reduced in AECIIs from Tmprss2/Tmprss4-deficient mice
compared with WT or Tmprss2-deficient mice, indicating that m
49 s of both sexes revealed a dramatic reduction in brain size
compared with WT (
Pclo(wt/wt) ) animals, attributed to a decr
50 reby enhance affinity with the effector-binding site in Ras
compared with WT RBD.