ライフサイエンスコーパス: compensatory change

1 hages and induce disease pathology through a compensatory change.

2 reased for the BK current, consistent with a compensatory change.

3 n-functional intermediate state prior to the compensatory change.

4 retained the D441-to-arginine change and no compensatory changes.

5 sion, or recombination, or a series of rapid compensatory changes.

6 t appear to further affect the noradrenergic compensatory changes.

7 lenced images in combat controls may reflect compensatory changes after trauma exposure that are not

8 mporal lobe epilepsy and provide evidence of compensatory changes after trauma to the hippocampal for

9 ehavioral remediation may be associated with compensatory changes anchored in L.FFG, which reflect at

10 orted in other species, such as rodents, but compensatory changes appear to prevent these deleterious

11 These compensatory changes are driven by Akt-mediated negative

12 ar interest are the roles of development and compensatory changes, as well as other factors, such as

13 However, the compensatory changes associated with vitamin A deprivati

14 ERC is often assumed to result from compensatory changes at interaction interfaces (i.e., in

15 e kinetics in H9 cells, contains second-site compensatory changes at MA amino acids 73 (E-->K) and 82

16 Whereas we found no evidence of compensatory changes at the mRNA level of wounded knocko

17 d toward control values driven by reciprocal compensatory changes at two joints.

18 etGC2, and GCAP-1 expression did not undergo compensatory changes, but the absence of GCAP2 affected

19 The data of patient compensatory changes conformed to the following equation

20 The observation that compensatory changes do not involve regeneration of N-li

21 for Cnr1 during development and suggest that compensatory changes during development may mitigate the

22 stand why G88R was favored by the virus as a compensatory change for the NLS loss and resultant repli

23 s for disrupting viral functions or impeding compensatory changes for vaccine escape or drug resistan

24 We find that the fixation of compensatory changes has caused the regulation of gene e

25 quences and second-site mutations leading to compensatory changes have been shown in mosaic individua

26 iferation in crypts of Lieberkuhn, without a compensatory change in basal apoptosis and produces a dr

27 ediate and folded states, but in all cases a compensatory change in entropy results in a small net fr

28 This is probably due to a compensatory change in gait or loading regime, which red

29 There is little or no compensatory change in other proteins or structures rela

30 ing of synaptic transmission, expressed as a compensatory change in quantal size following chronic ac

31 hat Pum-mediated change in para results in a compensatory change in Shal.

32 ow that chronic activity blockade triggers a compensatory change in the abundance of GLR-1, a Caenorh

33 ry lymphoid tissue architecture, we examined compensatory changes in ADAM17 and TNF-alpha in ADAM10(B

34 ver from Mpc1 hypomorphic embryos identified compensatory changes in amino acid and lipid metabolism.

35 ral occlusion during infancy, which leads to compensatory changes in auditory spatial tuning that ten

36 Thus, compensatory changes in B-class MADS box gene duplicate

37 PNP, and GDP, respectively), associated with compensatory changes in binding entropy and enthalpy.

38 No compensatory changes in Ca2+ regulatory protein expressi

39 hepsin activity in living cells, documenting compensatory changes in cathepsin-deficient cells, and C

40 These findings suggest that compensatory changes in cellular excitability, rather th

41 ates in the epithelial cytoplasm, initiating compensatory changes in cellular gene expression.

42 These findings show that there are dynamic, compensatory changes in cerebral activation during verba

43 ns increased as a function of intake despite compensatory changes in cholesterol and bile acid synthe

44 diated by enhanced CRF receptor signaling or compensatory changes in CRF receptor density within thes

45 Here we show that compensatory changes in demographic rates are buffering

46 partially denervated striatum without active compensatory changes in dopamine uptake or release.

47 s, leading to seizures, neuronal damage, and compensatory changes in EAAT3 and neuropeptide Y.

48 er ondansetron treatment was not achieved by compensatory changes in eating behaviour such as by a sm

49 ondrial metabolism that feed back and induce compensatory changes in electron transport.

50 tent alterations in network activity trigger compensatory changes in excitation and inhibition that r

51 in calcium influx, and translates this into compensatory changes in excitatory quantal amplitude.

52 Compensatory changes in expression levels of several bil

53 There was no evidence for compensatory changes in expression of either family memb

54 rine global knock-out approach may result in compensatory changes in expression of other membrane pro

55 tion reduced the amount of PP1cdelta with no compensatory changes in expression of other MYPT1 family

56 esponse to Clcn3 gene deletion, there may be compensatory changes in expression of other proteins tha

57 ether changes in dietary salt intake lead to compensatory changes in expression of the guanylin signa

58 MET activation appears to undergo long-term compensatory changes in expression that may be a hallmar

59 nsensitive to ischaemia, probably because of compensatory changes in extracellular potassium ions.

60 ates that dietary restriction elicits robust compensatory changes in food consumption.

61 These compensatory changes in functional brain activity were p

62 These findings suggest that compensatory changes in G alpha q expression occur in mi

63 We observed compensatory changes in G protein accumulation following

64 Additional compensatory changes in Gag permitted efficient virus re

65 tk alter B cell subpopulations and may cause compensatory changes in gene expression, we used B cells

66 although anti-NMDAR antibodies do not induce compensatory changes in glutamate receptor gene expressi

67 Furthermore, no compensatory changes in HMG-14b or histone protein level

68 ons up or down of frataxin expression caused compensatory changes in HSC20 expression inversely, as e

69 such that a loss of hepatic CPR would cause compensatory changes in intestinal P450 expression and c

70 For these mice, no effective compensatory changes in ion channel gene expression were

71 These compensatory changes in islet vascularization may influe

72 NMR relaxation indicates compensatory changes in loop fluctuations upon binding,

73 Both appear to produce compensatory changes in microtubule assembly that counte

74 Compensatory changes in mRNA expression of GPR120 in GPR

75 l heterogeneity of this receptor and also of compensatory changes in mu-, delta- or kappa-receptors i

76 hat the lack of RLC phosphorylation promotes compensatory changes in MyBP-C and TnI phosphorylation,

77 with selection of reversions and second-site compensatory changes in Nef.

78 Neuroadaptation theories predict that compensatory changes in neurochemical systems that are a

79 sis and biophysical properties, and possible compensatory changes in other channels that contribute t

80 rsistent activity of variant channels, or to compensatory changes in other conductance(s) in response

81 rned by the nature of the death stimulus and compensatory changes in other forms of autophagy.

82 the possibility of two different patterns of compensatory changes in other ion-translocating transpor

83 e and body weight remained normal because of compensatory changes in other meal parameters.

84 clean and are apparently not accompanied by compensatory changes in other muscarinic receptors.

85 IV-1 restriction, in some cases depending on compensatory changes in other nearby charged residues.

86 ctable xyloglucan does not cause significant compensatory changes in other polysaccharides, although

87 have led to the hypothesis that correlated, compensatory changes in particular parameters can at lea

88 on emission tomography study, we demonstrate compensatory changes in PD in another midbrain dopamine

89 ckbone atomic rms shifts (<0.5 A) because of compensatory changes in phi and psi backbone torsion ang

90 rom 50% to 150% normal accompanied by marked compensatory changes in plasma angiotensin II and renin

91 decreased PLC-alpha mRNA that may represent compensatory changes in PLC expression.

92 surprisingly survive into adulthood without compensatory changes in protein expression levels.

93 stresses, each of which results in specific compensatory changes in protein expression that can be a

94 Compensatory changes in protein levels of SERCA1, TRP an

95 sponds to oxygen level changes by undergoing compensatory changes in reduced electron carrier levels.

96 onal and expression studies did not indicate compensatory changes in relevant transporters.

97 Several of these are likely to be compensatory changes in response to Purkinje cell injury

98 g LC noradrenergic neurons in PD demonstrate compensatory changes in response to the neuronal loss, a

99 phenotypically normal, and have no apparent compensatory changes in ROCK2.

100 nstem, and hypothalamus, possibly reflecting compensatory changes in serotonergic innervation precedi

101 alpha*q expression are maintained in part by compensatory changes in signal transduction and other pa

102 ensing changes in tubular flow and eliciting compensatory changes in single nephron GFR (SNGFR).

103 Compensatory changes in SPAK/OSR1-independent phosphoryl

104 ce show an absence of tonic currents without compensatory changes in spontaneous IPSCs (sIPSCs), sEPS

105 nal activity (over a period of days) trigger compensatory changes in synaptic function that seem to c

106 Homeostatic plasticity is characterized by compensatory changes in synaptic strength and intrinsic

107 l activity is reduced over a period of days, compensatory changes in synaptic strength and/or cellula

108 in a network is perturbed for hours to days, compensatory changes in synaptic strength are triggered

109 n a physiologically functional range through compensatory changes in synaptic strength or intrinsic c

110 soleus burst amplitude and may have induced compensatory changes in the activity of other muscles.

111 eceptor alpha1 subunit expression can induce compensatory changes in the affected areas.

112 channels, however, may be complicated by the compensatory changes in the animals in response to the t

113 -Tsr* suppression most likely occurs through compensatory changes in the conformation or dynamics of

114 networks, indicating frequent coevolutionary/compensatory changes in the context of protein structure

115 n addition to determining the effects of the compensatory changes in the context of the original muta

116 No compensatory changes in the distribution of group-III mG

117 Evidence for compensatory changes in the evolved mechanism sets the s

118 port metabolon is likely one of the multiple compensatory changes in the exocrine parotid gland of Nh

119 pact on F-actin levels and did not result in compensatory changes in the expression of endogenous ADF

120 igh levels of CaMKII-Asp286 have reversible, compensatory changes in the expression of genes associat

121 ese alterations could partly be explained by compensatory changes in the expression of matrix-related

122 Compensatory changes in the expression of mRNA for other

123 ciency in Bcl-x(L) expression did not induce compensatory changes in the expression of other Bcl-2 pr

124 n in the hBCRP mice and the absence of major compensatory changes in the expression of other genes in

125 tion of TRPC6 protein without any detectable compensatory changes in the expression of other TRPC cha

126 mmatory cell recruitment was associated with compensatory changes in the expression profiles of CCL2,

127 d with an internal exon can be suppressed by compensatory changes in the first two positions of a con

128 erving the deprived sense, but also produces compensatory changes in the functionality of other senso

129 cells and are not accompanied by detectable compensatory changes in the level of expression of other

130 L-Myc deficiency by other Myc oncoproteins, compensatory changes in the levels of c- and/or N-myc tr

131 confirmed gene ablation and demonstrated no compensatory changes in the levels of other FABP mRNA in

132 of morphogenic scaling that have focused on compensatory changes in the morphogen gradient itself.

133 se data suggest that previously unrecognized compensatory changes in the nigrostriatal system occur i

134 Compensatory changes in the number of inhibitory and exc

135 ither improved nor degraded, suggesting that compensatory changes in the outputs of the spinal circui

136 ciated with these structural alterations are compensatory changes in the physiology and ultrastructur

137 ocytosis were followed in less than 0.7 s by compensatory changes in the rate of endocytosis.

138 evelopmental resources to one trait produces compensatory changes in the relative sizes of other trai

139 However, compensatory changes in the serotonergic system that are

140 in iput1 mutants is complicated by the vast compensatory changes in the sphingolipidome; however, ou

141 in young-adult mice results in wide-ranging compensatory changes in the structure and dynamics of th

142 ion of preproenkephalin mRNA expression, and compensatory changes in the synthesis and metabolism of

143 nd that target specificity can be changed by compensatory changes in the target site and the donor in

144 t not position 1, of the VSV peptide induced compensatory changes in the TCR in both the amino acid r

145 Compensatory changes in the two parameters were observed

146 ese changes may be related to lesion-induced compensatory changes in the use of the non-impaired (ips

147 Dynamic compensatory changes in this network and interconnected

148 Moreover, microarray analyses revealed no compensatory changes in transcripts encoding ion channel

149 lear how the 5' intron finds the 3' introns, compensatory changes in U1 snRNA rescue trans-splicing o

150 3-Int3 3' splice site mutants by introducing compensatory changes in U1 snRNA.

151 dation of the mutant tubulin does not elicit compensatory changes in wild-type tubulin synthesis or a

152 In addition, compensatory changes independent of striatal dopamine ha

153 tadpole to metamorphose, it may result from compensatory changes initiated by de novo growth of axon

154 These data demonstrate unexpected and compensatory changes involving the TM in the NHP model o

155 These data suggest that short- and long-term compensatory changes maintain dopaminergic control over

156 lts of the present study indicate that early compensatory changes may be taking place within the neur

157 can diminish viral replicative fitness, but compensatory changes may explain the limited impact of n

158 tructural integrity, and suggest sites where compensatory changes may stabilize the mutant structure.

159 Compensatory changes occur in the structure of the activ

160 ntral alpha7 expression, it is possible that compensatory changes occur that confound the results obt

161 of 5-lipoxygenase metabolites was not due to compensatory changes of 5-lipoxygenase or 5-lipoxygenase

162 restoration of KYNA levels, suggesting that compensatory changes or ontogenetic expression of anothe

163 d neuropathological changes and suggest that compensatory changes or ontogenic expression of another

164 utations in SIV Gag p11C was coincident with compensatory changes outside of the epitope.

165 to paradoxical functional facilitation, with compensatory changes particularly in the right posterior

166 base-pairing potential of the pseudoknot by compensatory changes restores telomerase activity to ess

167 nformative, they can be problematic, because compensatory changes sometimes occur during development.

168 rturbed directly and the neural basis of the compensatory changes studied in detail.

169 ess of interspecies hybrids suggest that the compensatory change that makes a CPD fit usually occurs

170 inhibition leads to previously unanticipated compensatory changes that affect cytoplasmic Ca(2+) home

171 at limits further ATP depletion and promotes compensatory changes that maintain cellular ATP levels.

172 Sdc1-/- mecs show compensatory changes that maintain the number of HS chai

173 mice, we evaluated hepatic transporters for compensatory changes that might circumvent the profound

174 g-term absence of NHE1 does not elicit major compensatory changes that might negate the cardioprotect

175 c lack of NPY during development may lead to compensatory changes that normalize regulation of food i

176 es have aimed at differentiating the initial compensatory changes that occur within the heart with ag

177 an be explained by the presence of secondary compensatory changes that render these mutations phenoty

178 portant for its regulatory function and that compensatory changes that restored base pairing partiall

179 Disruption of TAR reduced processing, while compensatory changes that restored the RNA structure als

180 perturbation and triggering several distinct compensatory changes that should help to recover and mai

181 these same sites, rather than by second-site compensatory changes, the more frequently observed mecha

182 Because of these and other compensatory changes, the population growth rates of sou

183 nal recovery is due to a more complex set of compensatory changes throughout the spinal network.

184 In fact, this decline may reflect a compensatory change to help reverse the decline of angio

185 constraint on epitope sequences that require compensatory changes to go to fixation.

186 in this ratio between groups, indicative of compensatory changes to keep the cholesterol/phospholipi

187 e; their population is dynamic and undergoes compensatory changes to maintain euglycemia.

188 dependent upon T3 or MCT8 and there were no compensatory changes to promote T3 uptake in a T3-deplet

189 The introduction of compensatory changes to restore base pairing potential l

190 This insensitivity derives from compensatory changes to the pattern of local and long-ra

191 The most common compensatory change was the acquisition of thymidines im

192 When U-box mutants did replicate detectably, compensatory changes were consistently observed in the v

193 When these compensatory changes were eliminated by a 20-hour fast,

194 ence to interfere with ribozyme binding, and compensatory changes were generated in the ribozyme reco

195 No compensatory changes were seen in the expression of othe

196 These lesions appear to lead to compensatory changes, with N/OFQ mRNA levels approximate

197 Because compensatory changes within species may obscure differen