コーパス検索結果 (１語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 d into a liver undergoing chronic damage and compensatory proliferation.
2 es did not undergo spontaneous cell death or compensatory proliferation.
3 ugh removal of aberrant or damaged cells and compensatory proliferation.
4 tissue regeneration, transdetermination and compensatory proliferation.
5 ession, PUMA-dependent hepatocyte death, and compensatory proliferation.
6 ogated PUMA induction, hepatocyte death, and compensatory proliferation.
7 ial cells and thus lead to apoptosis-induced compensatory proliferation.
8 ppears to be critical for both apoptosis and compensatory proliferation.
9 esis by inhibiting EGFR-dependent hepatocyte compensatory proliferation.
10 he activation of Hedgehog (Hh) signaling for compensatory proliferation.
11 ate the depleted crypt in a process known as compensatory proliferation.
12 ion through increased nutritional uptake and compensatory proliferation.
13 ptotic cells activate signaling cascades for compensatory proliferation.
14 to normal-looking adult wings as a result of compensatory proliferation.
15 We discovered that Hippo signaling regulates compensatory proliferation after extensive cell death in
16 o DEN-induced injury was sufficient to block compensatory proliferation and annihilate development of
17 ently in mature pancreatic cell types, since compensatory proliferation and possible mTORC2 activatio
18 y controlled in tissue regeneration to allow compensatory proliferation and prevent the intrinsic onc
19 xposure blocked prolonged JNK activation and compensatory proliferation and prevented excessive DEN-i
21 n abundant miRNA in the beta-cell, regulates compensatory proliferation and secretion during insulin
22 cancer in TGR5(-/-) mice, hepatocyte death, compensatory proliferation, and gene expression of certa
23 cyte and HSC apoptosis, resulting in reduced compensatory proliferation, and reduced replication stre
24 omotes chemical hepatocarcinogenesis through compensatory proliferation, and suggest apoptotic induce
25 umor development, suggesting that control of compensatory proliferation by high levels of p21 is crit
26 induced hepatocyte death and cytokine-driven compensatory proliferation, disruption of JNK1 abrogated
27 ep mice by inducing hepatocyte apoptosis and compensatory proliferation during early phases of tumori
29 utive emergence of a fatty liver, apoptosis, compensatory proliferation, fibrosis, and cirrhosis that
30 r data support the hypothesis that increased compensatory proliferation following continued tissue da
31 es JNK-dependent apoptosis and JNK-dependent compensatory proliferation following radiation injury.
32 r targets of hepatocyte death in relation to compensatory proliferation have not been fully character
33 different caspases trigger distinct forms of compensatory proliferation in an apparent nonapoptotic f
34 lial injury-induced colitis showing impaired compensatory proliferation in crypts and extensive ulcer
36 ute to tissue homeostasis by promoting local compensatory proliferation in response to cell death.
37 nes, which enhance cell survival and trigger compensatory proliferation in response to tissue injury.
38 In response to the induction of apoptosis, compensatory proliferation increased in the small intest
39 mice displayed spontaneous hepatocyte death, compensatory proliferation, inflammatory cell infiltrati
40 or grp mutants, suggesting that the need for compensatory proliferation is greater for checkpoint mut
41 egulatory networks controlling inflammation, compensatory proliferation, morphology and cell migratio
42 at increased hepatocyte death and associated compensatory proliferation observed in DEN-injured ERRal
43 t the potential for functionally significant compensatory proliferation of beta cells is retained in
45 evealed a new role for Drosophila p53 in the compensatory proliferation of cells that are needed to r
46 newing capacity of hepatocytes, resulting in compensatory proliferation of DDB1-expressing hepatocyte
47 pendently, directed hepatic fibrosis and the compensatory proliferation of hepatocytes and biliary ce
48 ndent RIPK1 phosphorylation in LPCs inhibits compensatory proliferation of hepatocytes and intrahepat
49 ORC1 by rapamycin effectively attenuates the compensatory proliferation of hepatocytes in CAMK2gamma(
50 is well recognized that lymphopenia induces compensatory proliferation of immune cells, generally te
52 dant-generating pollutants causes injury and compensatory proliferation of lung epithelium, but the s
53 ticellular organisms, apoptotic cells induce compensatory proliferation of neighboring cells to maint
54 as identified a link between dying cells and compensatory proliferation of neighbouring survivor cell
57 d hepatocyte death, giving rise to augmented compensatory proliferation of surviving hepatocytes.
59 ts importance, the mechanistic basis of such compensatory proliferation remains poorly understood.
60 larity complex influences both a physiologic compensatory proliferation response after irradiation in
62 rtance of JNK1-mediated hepatocyte death and compensatory proliferation, these results strongly sugge
63 hila wing imaginal disc, dying cells trigger compensatory proliferation through secretion of the mito
64 optosis, unaffected epithelial cells undergo compensatory proliferation to maintain the integrity of
65 mediated by JNK signaling, which also drives compensatory proliferation to maintain tissue integrity
66 elial progenitor cells to undergo sufficient compensatory proliferation to rescue the deficit in prog
68 (TLR) ligands, as well as growth signals for compensatory proliferation, would also be key factors in
WebLSDに未収録の専門用語（用法）は "新規対訳" から投稿できます。