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1  fate of donor DNA as it is processed by the competent cell.
2 ficient tumor cells while sparing checkpoint-competent cells.
3  clustering occurred only in chemotactically competent cells.
4 ule phosphorylation site) in chemotactically competent cells.
5 ls that can differentiate into mature immune-competent cells.
6 e (DHAP) concentration to levels seen in TPI-competent cells.
7 here it has ready access to activated immune competent cells.
8 tion and enhance this process in leukotriene-competent cells.
9 tions which are found infrequently in repair competent cells.
10 associated with the membranes of genetically competent cells.
11 r enzymes in whole-cell extracts from repair-competent cells.
12 process requires engraftment of donor immuno-competent cells.
13 truments and tedious steps in preparation of competent cells.
14 dered the efficient generation of clinically competent cells.
15 ells comprising therapy-resistant metastasis-competent cells.
16 l pathways impair peptidoglycan synthesis in competent cells.
17 increase chemotherapy-induced damage in BRCA-competent cells.
18 kinetics of transfer during fusion in fusion-competent cells.
19  signaling but is cleared from IFN signaling-competent cells.
20  effectively both as viable and biologically competent cells.
21 as a stop signal for the migration of immune-competent cells.
22 ring their differentiation into functionally competent cells.
23 r circadian rhythmicity in transcriptionally competent cells.
24 onal migration of transplanted remyelination-competent cells.
25 tive Vero cells but severe impairment in IFN-competent cells.
26 ltaneously increasing the frequency of IL-10-competent cells.
27 a and that the fragment must also include wg-competent cells.
28  ClpC and ClpP localize to the cell poles of competent cells.
29 population of undifferentiated, regeneration-competent cells.
30 as a recombination intermediate in wild-type competent cells.
31 uction of this noise decreases the number of competent cells.
32 d from the plasma membrane of mineralization-competent cells.
33 s are as sensitive to NDV as other caspase-8-competent cells.
34 eased radiosensitivity compared with TGFbeta competent cells.
35 within a mammalian host, yet generate mating-competent cells.
36 t caused induction of prostin-1 in PLC gamma competent cells.
37 eases in the levels of ICL and MAs in repair-competent cells 24 h after 8-MOP/UVA treatment, there wa
38               To distinguish the minority of competent cells, a translational fusion between ComK, th
39 plored the use of different Escherichia coli competent cells and fusion protein tags for the recombin
40 sponse in RIG-I and Toll-like receptor (TLR) competent cells and in mice.
41 nd only in mitochondria, both in respiratory-competent cells and in rho0 cells in which the bc1 compl
42 protein to productively replicate in the IFN-competent cells and in the cells of mosquito origin was
43 r replication was severely attenuated in IFN-competent cells and in young calves.
44 ression is restricted to the putative fusion-competent cells and is not detected in unfused founder c
45  contributing to increased sensitivity of SG-competent cells and xenografts to ER stress inducers inc
46 A and SsbB are directed to the cell poles in competent cells, and that the uptake of transforming DNA
47                    We report that in editing-competent cells BCR ligand-induced RAG mRNA expression i
48        Furthermore, in contrast to autophagy-competent cells, both proliferation and transformation i
49 ctivity were highly attenuated in interferon-competent cells but not in cells unable to establish ant
50 yl methanesulfonate (MMS) response in repair-competent cells, but it greatly amplified the MMS sensit
51    The protocol involves the transduction of competent cells by a chimeric retroviral vector containi
52  (sc) expression defines a group of neurally-competent cells called the proneural cluster (PNC).
53 nstrated that latently infected, replication-competent cells can be generated in vitro after eliminat
54      Initially we confirmed that respiratory-competent cells can be produced following incubation of
55 tion, and by increasing the pool of division-competent cells capable of anchorage-independent growth.
56  in approximately 25% of the mismatch repair-competent cell clones analyzed (cell lines HeLa and PMS2
57                                    Thus, IDO-competent cells constitute a distinctive B-lymphoid cell
58 led EFF-1 is specifically targeted to fusion-competent cell contacts via reciprocal localization to t
59                Platelets are immunologically competent cells containing cytokines such as TGF-beta1 t
60  (half-life of latently infected replication-competent cells decreased from 44 months to 1.5 months).
61 density difference between competent and non-competent cells depends on the competence-specific growt
62 ells with phenotypic attributes matching IDO-competent cells developed normally and expressed IDO in
63 he temporary effects of spCD25 on clinically competent cells did not impede their use in a safety/fea
64 ctuation in this signaling helps to sort out competent cells during successive cell-fate determinatio
65 ling out the hypothesis that neuromesodermal competent cells enable proportional regulation.
66               Moreover, DNMAML1-enriched EMT-competent cells exhibited robust upregulation of ZEBs, d
67                                Co-expression competent cells express graded levels of S- and M-opsins
68                                              Competent cells express specialized proteins that assemb
69                        Here we show that IDO-competent cells express the B-lineage commitment factor
70 ine adducts are substrates for repair by NER competent cell extracts but not XP12BE cell extracts def
71   Overall, increased glycolysis in autophagy-competent cells facilitates Ras-mediated adhesion-indepe
72 a unique gene regulatory network in germline-competent cells for PGC specification.
73 ee species have three cell types: two mating-competent cell forms (a and alpha) and the product of th
74 t cells and by the release of mineralization-competent cell fragments (matrix vesicles and apoptotic
75                                  Replication-competent cell-free extracts from other human cells were
76 to control the switch of the neuromesodermal competent cells from the epiblast to the chordoneural hi
77  the majority of vesicles in fully secretion-competent cells had no Munc18-1 associated within distan
78 hich can take up sterols aerobically in heme-competent cells have been selected.
79 table switch between non-active and transfer competent cells have long remained enigmatic.
80  reporter system and that only proliferation-competent cells have the ability to rapidly adapt ERK an
81   We present evidence that Drosophila immune-competent cells have the potential to express more than
82 ur data therefore reveals that proliferation-competent cells in dysfunctional populations show a prev
83  assay to measure the function of the immune-competent cells in PD effluent from PD patients.
84  occurs through an inductive process whereby competent cells in the post-implantation epiblast differ
85 tween cells leads to the elimination of less competent cells in the presence of more competent neighb
86 in thymic cellularity and enrichment for ILK-competent cells in the spleen and lymph nodes.
87 e, with an increase in the number of S-phase competent cells in the upper suprabasal layers, while th
88 equently induce activation of various immune-competent cells, including dendritic cells, thereby prov
89 triplex in a plasmid transfected into repair competent cells, indicating that approximately 25% of th
90 ribe Il10 illustrated that the pool of IL-10-competent cells is dominated by CD4 T cells and macropha
91 egies for generating potent, stable, CRISPRa-competent cell lines present limitations for the broad u
92                               In leukotriene-competent cells, LTC4 augmented phagocytosis to the grea
93             Upon dilution into fresh medium, competent cells maintain this appearance for about 2 h.
94 eased upon activation of a variety of immuno-competent cells may be important mediators that give ris
95  vector (pET-28a(+)) and converted it to the competent cell of BL21(DE3) to gain recombinant MAF-1 fu
96                                       In the competent cells of a comGA mutant culture, chromosomal r
97 that are activated during the development of competent cells of B. subtilis, a developmental program
98                                              Competent cells of Bacillus subtilis efficiently bind an
99 ssing and transport of transforming DNA into competent cells of Bacillus subtilis is described.
100                                        IL-10-competent cells of the B lineage are rare in vivo and, a
101                                As the immune-competent cells of the brain, microglia play an increasi
102                               Transformable (competent) cells of Bacillus subtilis are blocked in cel
103  has been traversed occurred in gap junction-competent cells only.
104 ed heterodicentric chromosome fusions in ATM-competent cells, only those mutant repeat sequences with
105 nous internal pH fluctuations in respiration-competent cells orchestrate the successive waves of prot
106 ed fewer lung metastases than syngeneic MKK4-competent cells (P = 0.0034).
107                                         IL-4-competent cells persisted in cured animals, and memory r
108 y leading to cell branching and formation of competent cell poles.
109 ly activated at a low level in all Sevenless-competent cells prior to Sevenless signaling, and this r
110            Whereas autophagic flux in OXPHOS-competent cells promoted cell survival, it was impaired
111             In contrast, the majority of non-competent cells rapidly resume growth, exhibiting chaini
112  exchanged for postimplantation factors, but competent cells remain devoid of lineage-specific transc
113                                              Competent cells resemble stationary phase cells; the maj
114                                   Chemotaxis-competent cells respond to a variety of ligands by activ
115  is a poorly understood process during which competent cells respond to inducing conditions, allowing
116  of infections with these two viruses in IFN-competent cells showed that W956IC activated NF-kappaB,
117 cilitating forward selection of regeneration-competent cell sources.
118 s recruited to sites of fusion by the fusion-competent cell-specific receptor Sns and acts as a posit
119  are a mixture of ribosome-competent and non-competent cells suggest a global regulatory mechanism in
120 sin production and cell lysis of its own non-competent cells, suggesting a possible active mechanism
121 ted an approximate 20-fold enrichment of ILK-competent cells, suggesting these cells have a competiti
122 s of the ComG proteins in DNA binding to the competent cell surface are discussed in the light of the
123 of severe inflammation by engaging signaling-competent cell surface receptors.
124 l for the binding of transforming DNA to the competent cell surface.
125 horylated in vitro by expression in the TKX1-competent cells that carry an inducible tyrosine kinase
126  self-renewal, proliferation, or survival of competent cells that it inappropriately contacts.
127                       By contrast, autophagy-competent cells that partition mitochondria asymmetrical
128 redifferentiated in vitro into metabolically competent cells that supported the replication of hepati
129                                    In repair-competent cells the 1-NP adduct induced 1.7% CpG deletio
130                        Although in silencing competent cells there was no correlation between the fra
131 eted silencing of p100/p52 sensitizes the HR-competent cells to camptothecin, while sensitization is
132 , dendritic cells (DCs) are the first immune-competent cells to encounter antigens within skin or muc
133 R3DL1-mediated NK cell inhibition of peptide-competent cells to levels seen with HLA-B*57:01.
134 ibutes were not essential for B-lymphoid IDO-competent cells to regulate T cells.
135 ion that enables the resultant gametogenesis-competent cells to respond to feminizing or masculinizin
136                                       Immune-competent cell trafficking to synovial tissue is integra
137 ficient in IL-1alpha expression or IL-1alpha-competent cells treated with IL-1alpha-specific small in
138 Restoring KCNA1 expression in transformation-competent cells triggered variation in membrane potentia
139 potent stem cells that are the only division-competent cell type outside of the reproductive system.
140 coupled to fusion receptors; in other fusion-competent cell types it may be triggered by different up
141 o increased survival or activity of division-competent cell types under anchorage-independent growth
142  protein Maf is synthesized predominantly in competent cells under the direct control of the transcri
143  in a manner that depends on AMPK, with AMPK-competent cells upregulating glycolysis and AMPK-deficie
144                            Enrichment of EMT-competent cells was more evident in the presence of p53
145            To validate the identification of competent cells, we demonstrated the coincident expressi
146 , single strands from native donor DNA enter competent cells, where they associate with an unidentifi
147 ant host hepatocytes must be replaced by AGT-competent cells, which is beyond the capacity of current
148  showed attenuated replication in interferon-competent cells while retaining the ability to infect an
149 ceptor-mediated signaling pathways in immuno-competent cells will provide a new insight for understan
150 nisms for the inhibition of cell division in competent cells with Maf acting downstream of ComGA.
151 ) and DprA stably co-localize at one pole in competent cells, with sigma(X) physically conveying DprA
152 tly, they were cleared from interferon (IFN)-competent cells without CPE development.
153 ed with enhanced viral replication in immune-competent cells, yet this enhanced replication is not fu

 
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