1     The survival of mice was not affected by complement deficiency.

2                           The association of complement deficiency and lupus has been known for over

3  the recognized autoimmune manifestations of complement deficiency and then describes new data derive

4  B and T lymphocytes, with the proposal that complement deficiency causes incomplete maintenance of p

5 the early pathogenesis of prion diseases and complement deficiency could result in decreased PrP(Sc)

6 rgeted to the transplanted heart and, unlike complement deficiency, did not affect immunity to infect

7 tissue damage, whereas inherited or acquired complement deficiencies highly favor the development of

8 omplement activation in Cfh(+/-) mice versus complement deficiency in Cfh(-/-) animals.

9 ection or creation of hybrid cell lines with complementing deficiencies in expression of HLA class II

10 h as macrophage activation, interferon-1 and complement deficiency may contribute to atherogenesis.

11  second hypothesis, generated primarily from complement deficiencies, relates to an impairment in the

12                           On the other hand, complement deficiency strikingly reduced the incidence a

13                               In contrast to complement deficiency, targeted complement inhibition wi

14  By using various serum sources with defined complement deficiencies, we demonstrate that, under phys

15                      Using mice with various complement deficiencies, we demonstrated that the intrap

16 vide insight into the genetic association of complement deficiency with susceptibility to systemic lu