ライフサイエンスコーパス: complement-fixing

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1 elong predominantly to the IgG class and are complement fixing.

2 activity using flow cytometry assays (FACS), complement-fixing ability, and CDC activity of a panel o

3 nce of systemic delayed hypersensitivity and complement-fixing Abs in this deviant response probably

4 ypersensitivity responses and a reduction in complement-fixing Abs.

5 By contrast, complement-fixing alloantibody levels were reduced.

6 oliferative and cytotoxic responses and both complement fixing and immunoglobulin (Ig)G alloantibody

7 kit that measures C1q binding to distinguish complement fixing and nonfixing antibodies, studies show

8 sial infection is postulated to require both complement-fixing and opsonizing antibodies in addition

9 eloped more serum anti-AChR Ab, and had more complement-fixing anti-AChR IgG2a and 2b and less IgG1 t

10 is a second protective mechanism provided by complement-fixing anti-B5 IgG.

11 were reduced after intravaginal injection of complement-fixing anti-Thy-1.2 and GK 1.5 anti-CD4 antib

12 ing, suggesting the presence of low affinity complement-fixing antibacterial Abs in certain preparati

13 on has been hyperacute rejection mediated by complement fixing antibodies directed against galactose

14 C5b-9, C4d, activated C1, and C5a) and major complement-fixing antibodies (Luminex).

15 delayed-type hypersensitivity responses and complement-fixing antibodies on subsequent challenge wit

16 In contrast, complement-fixing antibodies to merozoites did not decli

17 Complement-fixing antibodies to STm develop by 2 years o

18 ata suggest that Pfs230 is a major target of complement-fixing antibodies which may be important for

19 s in anamnestic production of nonprotective, complement-fixing antibodies, immune complex deposition

20 nd H-DNA recipients developed high titers of complement-fixing antibodies.

21 P < .01) and those with maximum coccidioidal complement fixing antibody titers >/=1:128 (median, 174

22 treated with atRal and then incubated with a complement-fixing antibody followed by stimulation with

23 s mechanism may place individuals exposed to complement-fixing bacteria at risk for autoimmunity.

24 1.4%) of 58 patients whose anti-coccidioidal complement-fixing (CF) antibodies were detectable and in

25 hown to react with patient anti-Coccidioides complement-fixing (CF) antibody and is a valuable aid in

26 Detection of anti-Coccidioides complement-fixing (CF) antibody is a valuable diagnostic

27 s of CP activation, such as capillary C4d or complement-fixing donor-specific antibodies (DSA).

28 cells led to robust cytotoxicity mediated by complement-fixing DSAs and phagocytic cells.

29 with CDC, the current standard in detecting complement-fixing HLA antibodies.

30 Complement-fixing HLA class II antibodies were found les

31 ilar, but unexpectedly, the concentration of complement fixing IgG(1) antibodies was lower in a group

32 nd substantial intra-articular deposition of complement-fixing IgG Abs.

33 gamma-/- mice does not suffice to reduce all complement-fixing IgG subclasses, perhaps because as in

34 end-plate damage initiated by the binding of complement-fixing IgG to end-plate AChRs.

35 ure-activated DCs, induce B cells to produce complement-fixing IgG1 and IgG3 Abs.

36 edominance of vascular deposition of the non-complement-fixing IgG2c isotype and a concomitant decrea

37 Infusion of complement-fixing IgG3 mAbs resulted in either hyperacut

38 h idiopathic membranous nephropathy have non-complement-fixing IgG4 autoantibodies to the phospholipa

39 neutralize VACV unless the antibodies are of complement-fixing isotypes and complement is present.

40 , nonneutralizing, non-fusion-inhibiting but complement-fixing MAb, B1, provided no significant prote

41 mechanism of xenograft rejection mediated by complement-fixing natural antibodies recognizing alpha(1

42 bstantially diminish renal injury induced by complement-fixing nephrotoxic Ab.

43 d unchanged, implying normal N-glycation and complement-fixing potential, and antibody binding affini

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