1 he same or different genes by performing the
complementation test.
2 d in the evolution of this trait using a QTL
complementation test.
3 In a
complementation test and by genetic mapping we have show
4 Complementation test and gene expression analysis reveal
5 one of the candidate genes as Rj4 using both
complementation tests and CRISPR/Cas9-based gene knockou
6 Complementation tests and linkage analysis reveal that t
7 ws the mutation to be used experimentally in
complementation tests and other genetic crosses and it m
8 Complementation tests and recombinational mapping sugges
9 ted based on data gathered from both genetic
complementation tests and RFLP analysis.
10 e in E. coli NM81 (delta nhaA) in a separate
complementation test,
and an orfD with no significant si
11 Genetic mapping,
complementation tests,
and genomic sequencing show that
12 Among 34 other flathead group members
complementation tests are incomplete, but at least six u
13 omosome carrying wild-type Polq and a failed
complementation test between chaos1 and a Polq-disrupted
14 Complementation testing between jal and a Gata3 null all
15 Complementation tests between mtl and dreher and between
16 Complementation tests between rad23-1 and swi10-154 muta
17 Recent studies highlight how
complementation tests between species can bridge the gap
18 Complementation tests between spp1 and spp3 revealed the
19 Complementation tests between the original orpk mutation
20 Complementation tests between the smi60E mutant and seve
21 On the basis of
complementation tests between transgenic and Os/+ mice,
22 In the Saccharomyces genus, interspecies
complementation tests can be used to evaluate functional
23 Thus, interspecies
complementation tests can be used to identify (1) mutati
24 Complementation tests confirmed that the lack of growth
25 Complementation tests,
cytological mapping, P-element mo
26 y comparison to published data and by direct
complementation tests,
finding both dominant and recessi
27 i cysS deletion strain permitted a stringent
complementation test;
growth could be supported only by
28 n in the Sema-5c background and quantitative
complementation tests identified epistatic interactions
29 of melanophores, and previous interspecific
complementation tests identified fms as a potential cont
30 Finally, interspecific
complementation tests identify a candidate genetic pathw
31 A color
complementation test in bacteria confirmed that the papa
32 tant was complemented by wild-type At5g32470
Complementation tests in Escherichia coli and enzyme ass
33 Complementation tests in interspecies hybrids identified
34 Complementation tests in the polyprenol-deficient yeast
35 f resequencing efforts and that quantitative
complementation tests in yeast can be used to evaluate v
36 Linkage analysis and genetic-
complementation testing indicate that lor is an allele o
37 The
complementation tests indicated that a large portion (am
38 The results from these
complementation tests indicated that all six genes locat
39 Complementation tests indicated that the 8 lines contain
40 Furthermore, a cross-species
complementation test indicates that Os-POLLUX can restor
41 While the
complementation test is highly effective and accurate in
42 ystematically characterized before, inter se
complementation testing of newly recovered mutants was c
43 We used
complementation tests of deficiencies to fine map these
44 Quantitative
complementation tests of mutations at these coregulated
45 map these QTL to 12 chromosomal regions and
complementation tests of mutations to identify 13 positi
46 Complementation tests of these strains plus three previo
47 Quantitative
complementation tests performed for 45 positional candid
48 there are a number of instances in which the
complementation test provides misleading answers, either
49 Furthermore,
complementation test results suggest that phosphorylatio
50 Genetic
complementation tests reveal functions for ABC(RNAi) tra
51 Complementation tests reveal that 5B10 represents a new
52 A stringent genetic
complementation test revealed that when the mutant prote
53 A
complementation test revealed the RPE65 knockout and tvr
54 Allelism and
complementation tests revealed that both pf mutants were
55 Genetic
complementation tests revealed that the insertion mutant
56 Functional
complementation tests revealed that the S344D mutation b
57 Complementation tests revealed that this mutation is an
58 Complementation tests show that alanine substitution at
59 In silico analyses and genetic
complementation tests showed that entA locates next to a
60 Genetic
complementation tests showed that the cop8 and fus4 muta
61 Complementation tests showed that the helicase-dead muta
62 binding GCAP, remained high, and functional
complementation tests showed that the RetGC1 active site
63 ries of markerless gene knockout mutants and
complementation tests,
specific combinations of pauABCD
64 Lethal phase analysis and
complementation testing suggest that the three isoforms
65 severely disturbed and no longer active in a
complementation test,
suggesting that the residues are i
66 arf1Delta allele and defined seven genes by
complementation tests (
SWA1-7 for synthetically lethal w
67 using primary fibroblasts, including a virus
complementation test,
takes 1 week to complete.
68 dney disease (jcpk), led us to undertake the
complementation testing that confirmed T(2;10)67Gso and
69 We present
complementation tests that confirm the allocation of Eco
70 verse genetic approaches and a cross-species
complementation test to characterize the function of the
71 minimal heart enhancer and used a cis-trans
complementation test to show that Ets1/2 can interact wi
72 Subsequent quantitative
complementation tests to all available mutations of posi
73 o the lower arm of chromosome I and found by
complementation tests to be allelic to the salicylic aci
74 QTL mapping followed by quantitative
complementation tests to deficiencies and mutations is a
75 candidate loci, and results of quantitative
complementation tests to mutations at candidate loci aff
76 t on chromosome 3 into at least six QTL, and
complementation tests to mutations identified four candi
77 Complementation tests to mutations show that Ddc is a po
78 etic screens, CloudMap contains an in silico
complementation testing tool that allows users to rapidl
79 Complementation tests using a second allele of Gli3 (Gli
80 ctivity on negatively supercoiled DNA, while
complementation tests using mutants implicated in the in
81 Classic genetic
complementation tests using the nonautoimmmune 129/SvJ m
82 rpretation of abp1-5 phenotypes for which no
complementation test was conducted.
83 rsion of FliI, which was fully functional in
complementation tests,
was purified to homogeneity.
84 Through linkage mapping and
complementation testing,
we identified the mutation as a
85 cross multiple resistant strains and genetic
complementation tests,
we associated a nonsynonymous mut
86 Using quantitative
complementation tests,
we demonstrate that this QTL is a
87 By applying genetic
complementation tests,
we estimate that about two-thirds
88 Complementation tests were also performed with plasmids
89 ities of the promoters identified by genetic
complementation tests were quantitatively measured by tr
90 Genomic resequencing and
complementation tests were used to identify the causal g
91 k2; hence, both in vivo and in vitro genetic
complementation tests were used to prove that this Col3a
92 permitted partial restoration of motility in
complementation tests,
whereas the truncation did not.
93 Complementation testing with an engineered null allele o
94 Mapping and
complementation testing with candidate mutations reveale
95 Complementation testing with para null mutations strongl
96 root-hair phenotypes were characterized and
complementation testing with uncloned root-hair genes wa
97 Complementation tests with E. coli mutants established t
98 Growth and
complementation tests with insertion mutants, combined w
99 Complementation tests with partially and completely defe
100 Quantitative
complementation tests with piggyBac-tagged mutants for 2
101 ring a folate cofactor, retained activity in
complementation tests with tetrahydropterin-depleted E.
102 Complementation tests with the leaner mutant allele (Cac
103 Complementation tests with various long hypocotyl (hy) m