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1 houghts (obsessions) and repetitive actions (compulsions).
2 es (obsessions) and by repetitive behaviors (compulsions).
3 the percentage change of these bands during compulsion.
4 ism may underlie diverse disorders involving compulsion.
5 rtant for developing interventions to reduce compulsion.
6 d not control malaria; its use required mass compulsion.
7 -predicting stimuli, which may contribute to compulsion.
8 mma oscillations were highly correlated with compulsion.
9 ases with prominent distress when preventing compulsions.
10 gers, and the intent of ERP is to extinguish compulsions.
11 triggers but prevented from expressing their compulsions.
12 on, and 4) hoarding: hoarding obsessions and compulsions.
13 te disinhibition and apathy, and may exhibit compulsions.
14 are more specific to the phenotype than are compulsions.
15 ssions and repeating, ordering, and counting compulsions; 2) forbidden thoughts: aggression, sexual,
17 ligious, and somatic obsessions and checking compulsions, 3) cleaning: cleaning and contamination, an
18 der had a higher rate of both obsessions and compulsions, an earlier age at onset, and more comorbid
20 l real-time neurophysiological biomarkers of compulsion and warrant further assessment of the use of
21 underlying neurophysiological mechanisms of compulsion and warrants further assessment of the use of
25 reduced binge-eating-related obsessions and compulsions, and SGAs reduced symptoms of depression (MD
29 that patients know that their obsessions and compulsions are excessive, but their symptoms neverthele
30 caine, suggests that cortical-limbic induced compulsions are significantly different in their origin
32 evaluated for the presence of obsessions and compulsions by means of the Structured Clinical Intervie
34 asses: unaffected (class 1), ritual/symmetry compulsions (class 2), germ/contamination obsessions (cl
35 ted behaviors suggest that the expression of compulsions could in part involve loss of control of suc
36 ives had higher rates of both obsessions and compulsions; however, this finding is more robust for ob
39 gories used to group types of obsessions and compulsions in the Yale-Brown Obsessive Compulsive Scale
40 genic mouse model of cortical-limbic induced compulsions in which dopamine D1 receptor-expressing (D1
42 ens core (NAcore) are critical for promoting compulsion-like alcohol drinking, where rats consume alc
44 ing (rather than potentiation) of D1CT mouse compulsion-like behaviors by cocaine, suggests that cort
45 ted, the incidence of D1CT transgene-induced compulsion-like behaviors such as repeated leaping and p
47 not found in other parts of the brain during compulsion, or in the BNST during noncompulsive behavior
48 ysthymia, anxiety, panic, phobia, obsession, compulsion, posttraumatic, care management, case managem
49 ems and the presence of other obsessions and compulsions related to their hoarding, such as fear of c
50 ring, arranging, counting, writing-rewriting compulsions, repetitive writing tics) and disinhibition
51 uggest that the motor circuits stimulated in compulsions represent only a subset of the parallel circ
52 and decreased during the initiation phase of compulsion, respectively, whereas beta increased after c
53 and decreased during the initiation phase of compulsion, respectively, whereas beta increased after c
59 behaviors, according to both the Yale-Brown compulsion subscale and CGI rating of obsessive-compulsi
60 Repetitive behaviors were measured with the compulsion subscale of the Yale-Brown Obsessive Compulsi
61 oral abnormalities uniquely resembling human compulsions, such as non-aggressive biting of cagemates
62 lsive disorder (OCD) involves obsessions and compulsions that cause impairment and distress, and whic
63 is characterized by recurrent obsessions and compulsions that create distress and interfere with dail
64 cluding generalized anxiety, depression, and compulsions that preceded the birth of their children.
65 cognitive distortions: 1) dichotomania, the compulsion to perceive quantities as dichotomous even wh
67 onically relapsing disorder characterized by compulsion to seek and take drugs and has been linked to
68 ically relapsing disorder characterized by a compulsion to seek and take drugs and the development of
69 ically relapsing disorder characterized by a compulsion to seek and take drugs, the development of de
71 ical stimulation, in terms of suppression of compulsion, was significantly correlated with the percen
75 understand the mechanisms of addictions and compulsions, which involve a failure of aversive outcome
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