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1 8.6 kDa), and the tetrameric protein complex Concanavalin A (103 kDa).
2          Pretreatment of HEK-hCaR cells with concanavalin A (250 microg/ml) to block CaR internalizat
3 GC-5 cells were differentiated with succinyl concanavalin A (50 microg/mL) and transferred to a press
4         The two gammaGTases were resolved by concanavalin A (Con A) affinity chromatography, indicati
5 s been created using Alexa Fluor 647-labeled concanavalin A (Con A) and a fourth-generation PAMAM Ale
6 e material was derivatized with two lectins, Concanavalin A (Con A) and Aleuria aurantia lectin (AAL)
7  monitoring the specific interaction between Concanavalin A (Con A) and D-(+)-mannose.
8 ion of the electrical synapse was blocked by concanavalin A (Con A) and dynamin inhibitory peptide (D
9 e data available on the binding constants of Concanavalin A (Con A) and glycogen and Con A-mannan usi
10 bohydrate platform for the immobilization of Concanavalin A (Con A) and is capable of LPS binding mea
11 ew as the sensing probes for lectins such as concanavalin A (Con A) based on the molecular recognitio
12 eral crystal structures of the legume lectin concanavalin A (Con A) bound to closely related carbohyd
13 set of serial lectin columns consisting of a concanavalin A (Con A) column coupled to an SNA column f
14 sed to measure the adhesive strength between concanavalin A (Con A) coupled to an AFM tip and Con A r
15  of alpha-Man to the mannose-specific lectin concanavalin A (Con A) even though homogeneous beta-Lact
16 el on their surfaces was probed using lectin concanavalin A (Con A) from Canavalia ensiformis.
17                         Hepatitis induced by concanavalin A (Con A) in mice is well known to be a T-l
18                                 Injection of concanavalin A (Con A) into mice recapitulates the histo
19                                              Concanavalin A (Con A) kills procyclic (insect) forms of
20 vation and expansion were studied by using a concanavalin A (Con A) liver injury model followed by pa
21 g the direct pili-mannose binding as well as Concanavalin A (Con A) mediated lipopolysaccharides (LPS
22 ial, we have developed a nanoscale chelating Concanavalin A (Con A) monolithic capillary prepared usi
23                        These sensors utilize Concanavalin A (Con A) protein hydrogels with a 2D PC em
24 ntaining 2-D photonic crystal sensor detects Concanavalin A (Con A) through shifts in the 2-D diffrac
25 ible binding of a mobile fluorophore-labeled Concanavalin A (Con A) to immobile pendant glucose moite
26 -SNPs with fluorescein isothiocyanate (FITC)-concanavalin A (Con A) was determined using a fluorescen
27 sembled monolayer in combination with lectin concanavalin A (Con A) was used as molecular recognition
28          Conformational structure changes in concanavalin A (Con A), a legume lectin protein which is
29 achite green (MG-dextran), was conjugated to concanavalin A (Con A), an enzyme with specific affinity
30 ns including Maackia amurensis lectin (MAL), concanavalin A (Con A), and wheat germ agglutinin (WGA).
31  were cultured in the absence or presence of concanavalin A (Con A), phorbol myristate acetate (PMA)/
32          Construction of biosensors based on Concanavalin A (Con A), Sambucus nigra agglutinin type I
33  excellent selectivity to its target lectin, concanavalin A (Con A), surpassing the formerly used lin
34 lines, we identify a plant-derived compound, Concanavalin A (Con A), which differentially kills p53-n
35 ran-coated nanoparticles are aggregated with concanavalin A (Con A), which results in a significant s
36                                              Concanavalin A (Con A), which selectively blocks KAR des
37 mmune or viral hepatitis, and of mice during concanavalin A (Con A)-induced hepatitis (CIH).
38                                              Concanavalin A (Con A)-induced injury is an established
39                                      We used concanavalin A (Con A)-induced liver injury to study the
40 dhesion of the alpha-mannose-specific lectin concanavalin A (Con A).
41 ramide (alphaGalCer), anti-CD3 antibody, and concanavalin A (Con A).
42 nsity on the clustering of a model receptor, concanavalin A (Con A).
43 nd clustering of a model protein, the lectin concanavalin A (Con A).
44 ell lymphoma cells (TIB 155) stimulated with concanavalin A (Con A).
45 tive protein (CRP), transthyretin (TTR), and concanavalin A (Con A).
46 ondrial O2 consumption) in mice treated with concanavalin A (Con A).
47  UV-vis spectroscopy during its binding with concanavalin A (Con A).
48 -HIV antibody 2G12 (shortest distance 31 A), concanavalin A (ConA) (shortest distance 72 A), RCA120 (
49                                   The lectin concanavalin A (ConA) activates immune cells, resulting
50  rate, compared to wild-type (wt) mice, upon concanavalin A (ConA) administration.
51 y, including T/NKT cell hepatitis induced by concanavalin A (ConA) and alpha-galactosylceramide (alph
52 CM electrodes present a higher adsorption of Concanavalin A (ConA) and Bovine Serum Albumin (BSA) pro
53 arbohydrates to the Man/Glc-specific lectins concanavalin A (ConA) and Dioclea grandiflora lectin (DG
54 arbohydrates to the Man/Glc-specific lectins concanavalin A (ConA) and Dioclea grandiflora lectin (DG
55 binding of the carbohydrate-binding proteins concanavalin A (ConA) and jacalin to arrays composed of
56 cterized chemical inhibitors of endocytosis: concanavalin A (conA) and phenylarsine oxide (PAO).
57 UVL patients exhibited normal ATP release to Concanavalin A (ConA) and phytohemagglutinin (PHA; 190+/
58 K using the receptor internalization blocker concanavalin A (ConA) and the carboxyl terminus-truncate
59                         The jack bean lectin concanavalin A (ConA) and the Dioclea grandiflora lectin
60 d from a detailed analysis of the binding of concanavalin A (ConA) and wheat germ agglutinin (WGA) to
61 on, cells treated with ICZ increased surface concanavalin A (ConA) binding, corroborating an increase
62                                              Concanavalin A (ConA) bound to PTP1 and to the polar tub
63 ated in T cell-mediated hepatitis induced by concanavalin A (ConA) but are less extensively elevated
64                                              Concanavalin A (ConA) causes immune cell-mediated liver
65 anisms in optical glucose sensors based upon Concanavalin A (ConA) has tended to prevent the sensors'
66 mpetitive binding assays based on the lectin Concanavalin A (ConA) have displayed significant potenti
67         Competitive binding assays utilizing concanavalin A (ConA) have the potential to be the basis
68                                              Concanavalin A (ConA) inhibited phi C31 infection of S.
69 peptide reacted strongly with both lotus and concanavalin A (ConA) lectins, it bound to lotus stronge
70 robe was designed by noncovalent assembly of concanavalin A (ConA) on gold nanoparticles (AuNPs).
71 ects of T cell-mediated hepatitis induced by concanavalin A (ConA) on the regenerative response in vi
72 ls of hepatitis, including administration of concanavalin A (ConA) or alpha-galactosyl-ceramide, whic
73 in unstimulated or lipopolysaccharide (LPS), concanavalin A (ConA) or phytohemagglutinin (PHA) stimul
74          The treatment of splenic cells with concanavalin A (ConA) plus CT enhanced the production of
75 pha-D-mannopyranoside residues to the lectin concanavalin A (ConA) show increasing negative cooperati
76  cells showed enhanced responses to in vitro concanavalin A (ConA) stimulation when compared with WT
77 ent of either type of mutant with the lectin concanavalin A (ConA) that cross-links surface receptors
78    Here, we show that treatment of mice with concanavalin A (ConA) to induce liver injury triggered a
79                               The binding of Concanavalin A (ConA) to mannose-functionalized self-ass
80 l apoptosis induced by the administration of concanavalin A (ConA) to pregnant mothers.
81                                              Concanavalin A (ConA) was used as an activator of lympho
82 opaeus (gorse, furze), Triticum vulgaris and Concanavalin A (ConA) was used for probes to evaluate bi
83  coefficients that showed mannose to bind to concanavalin A (conA) with 3.7 times greater affinity th
84 e binding affinity of mannose and glucose to concanavalin A (ConA), a 106 KDa homotetramer protein, i
85 ension of macroporous hydrogel particles and concanavalin A (ConA), a glucose-specific lectin, that a
86                                Starting from concanavalin A (ConA), a mannose (Man)-binding protein,
87 tein, PZR displays a strong association with concanavalin A (ConA), a member of the plant lectin fami
88 tal model of autoimmune hepatitis induced by concanavalin A (ConA), a process involving T cell activa
89                     MSCs were activated with concanavalin A (ConA), a Toll-like receptor (TLR)-2 and
90 feration and IL-2 production induced by PHA, concanavalin A (conA), and anti-TCR MAb.
91 oteins, namely, transthyretin (TTR), avidin, concanavalin A (conA), and human serum amyloid P compone
92                     In situ competition with concanavalin A (ConA), another high-mannose specific lec
93                                     By using concanavalin A (ConA), as a recognition template, peptid
94                 This was followed by binding concanavalin A (ConA), glucose oxidase (GOx), and Au nan
95  which consists of three mixtures of lectins concanavalin A (ConA), jacalin (JAC), and wheat germ agg
96 ure induced by Fas-agonistic antibody (Jo2), concanavalin A (ConA), or D-galactosamine/lipopolysaccha
97  and lipopolysaccharide (LPS), or the lectin concanavalin A (ConA), suggesting that physiologic level
98 tested with several plant lectins, including concanavalin A (conA), Vicia villosa isolectin B4 (VVL-B
99     On the basis of the model system glucose-Concanavalin A (ConA), we explore the application of Tra
100 tosamine (GaIN)/lipopolysaccharide (LPS) and concanavalin A (ConA)-induced acute liver failure (ALF),
101 lated by natural killer T (NKT) cells during concanavalin A (ConA)-induced acute liver injury.
102 ore D-Gal/LPS-induced FH and before or after concanavalin A (ConA)-induced FH.
103 ated the effects of THC in a murine model of concanavalin A (ConA)-induced hepatitis.
104 o chronic choline-deficient diet exacerbates concanavalin A (ConA)-induced liver hepatitis, which is
105  of the molecular and cellular mechanisms of concanavalin A (ConA)-induced liver injury have provided
106 ) mice, germ-free (GF) mice are resistant to Concanavalin A (ConA)-induced liver injury.
107 y susceptible to liver destruction following concanavalin A (ConA)-induced T cell activation.
108 DTA mice) exhibited enhanced lymph node (LN) concanavalin A (ConA)-induced Th1 responses after tick i
109 ingly, NK cells mediated hypersensitivity to concanavalin A (ConA)-mediated hepatitis in GNMT(-/-) mi
110 to Fas-specific antibody or co-cultured with concanavalin A (ConA)-stimulated hepatic mononuclear cel
111                                              Concanavalin A (ConA)-stimulated peripheral blood mononu
112  increased production of interferon-gamma by concanavalin A (ConA)-stimulated spleen T cells and expr
113 d interferon-gamma (IFN-gamma) production in concanavalin A (conA)-stimulated spleen T cells, and dim
114  sensitive to apoptosis upon activation with concanavalin A (ConA).
115 from autoimmune hepatitis induced by mitogen concanavalin A (ConA).
116 P) and then with the Fas agonist Jo2 or with concanavalin A (ConA).
117 ere identified by perfusion with fluorescent concanavalin A (ConA).
118 cose- and mannose-specific binding protein - Concanavalin A (ConA).
119 chemical displacement sensor for the protein concanavalin A (ConA).
120 er injury in the lipopolysaccharide/GalN and concanavalin A (ConA)/GalN models, but not in a ConA-onl
121      Activation of CD8(+) T lymphocytes with concanavalin A (ConA)/interleukin-2 (IL-2), and activati
122         Thymocyte proliferative responses to concanavalin A + interleukin-2 were also significantly d
123                    Mannosidase digestion and concanavalin A adsorption indicate that the terminal res
124 eted cryptococcal antigens were separated by concanavalin A affinity chromatography into adherent (ma
125 tionation, diethylaminoethyl chromatography, concanavalin A affinity chromatography, and chromatofocu
126                          APS was purified on concanavalin A affinity columns to minimize the loss of
127 ctional valency of a triantennary analog for concanavalin A and D. grandiflora lectin are observed.
128 rate analogs to the Man/Glc-specific lectins concanavalin A and Dioclea grandiflora lectin was invest
129 a of infected insect cells using immobilized concanavalin A and immobilized BACE inhibitor, P10-P4' S
130   B10.BR splenic lymphocytes stimulated with concanavalin A and interleukin 2 were infected with a re
131 from broccoli flower buds was purified using concanavalin A and ion-exchange chromatography.
132                               Here, by using Concanavalin A and LC-MS analysis, we identified a novel
133                                              Concanavalin A and lectins from pea and lentil, also man
134 nt experimental hepatitis models, induced by concanavalin A and Listeria monocytogenes.
135 , T cell proliferation upon stimulation with concanavalin A and phytohemagglutinin A was only 40-50%
136 ion lymphocytes were examined in response to concanavalin A and purified protein derivative (PPD) in
137 or the ability to proliferate in response to concanavalin A and purified protein derivative (PPD) in
138 tely 100 kDa, contained sugars that bound to concanavalin A and ricin.
139  protein receptors such as the plant lectins concanavalin A and the highly toxic mistletoe Viscum alb
140 her mannose/glucose binding lectins, such as concanavalin A and the pea, lentil and Calystegia sepium
141  in C57BL/6 mice by intravenous injection of concanavalin A and then analyzed liver concentrations of
142 nti-bovine serum albumin (BSA) antibody, and concanavalin A are embedded in distinct regions of a 7.5
143 that of the wild type and did not react with concanavalin A as did wild-type LAM.
144 ives supported the integration of the lectin concanavalin A as well as the enzymes alpha1-2,3 mannosi
145                       MIPP co-localized with concanavalin A at the endoplasmic reticulum, suggesting
146 suggesting that the polymers bind the lectin concanavalin A at the same site as natural carbohydrates
147 investigated by bromelain IgE inhibition and concanavalin A binding assays using sera of cypress poll
148 n chromium-release assays against a panel of Concanavalin A blast targets.
149 ffectors were also able to specifically lyse Concanavalin A blasts isolated from H-2(d) mice (BALB/c,
150 ng BiP/Grp78 messenger RNA (mRNA) in splenic concanavalin A blasts.
151        A model of reactive lymph nodes after concanavalin A challenge served as an additional control
152 ZM-INVINH1 with the glycoprotein fraction by concanavalin A chromatogaphy suggests that ZM-INVINH1 in
153                                              Concanavalin A chromatography effectively enriched and p
154                                   The lectin concanavalin A conjugated to colloidal gold particles wa
155       Circular dichroism, mass spectrometry, concanavalin A detection, immunoblotting, enzyme-linked
156                    Blocking endocytosis with concanavalin A eliminated the accumulation of fluorescen
157 or LcL were compared with those obtained for concanavalin A i.e. lectin, which interacts with the car
158 ttenuated the suppression of the response to concanavalin A in immunized mice, providing further evid
159 nnosylated conjugated polymer (sugar-PPE) by Concanavalin A is positively dependent upon sugar-PPE co
160            Leukocyte adhesion was assayed by concanavalin A labeling.
161  were simultaneously quantified by combining concanavalin A lectin (ConA) perfusion labeling with a f
162 asured by fluorescein isothiocyanate-coupled concanavalin A lectin and acridine orange labeling.
163 res show surprisingly high affinities toward Concanavalin A lectin receptor in comparison to their ho
164 tive EC injury using the concanavalin A/anti-concanavalin A model and after ischemia/reperfusion (I/R
165 nding site separation of the sugar sites for concanavalin A of 3.6-4.3 nm was determined and a critic
166 compared with that of commercially available concanavalin A on agarose beads.
167 urement of the toxic effect of 100 microg/mL concanavalin A on macrophages and hepatocytes, but not o
168              We have immobilized the protein Concanavalin A onto a self-assembled monolayer of multiv
169 nalized graphene indicate that adsorption of Concanavalin A onto graphene is accompanied by near-comp
170  no significant difference in the binding of concanavalin A or Aleuria aurantia lectin was detected.
171 n) and this was not blocked by agents (i.e., concanavalin A or hypertonic sucrose) that inhibit D1 re
172 , and the generation of activated T cells by Concanavalin A or L-PHA was also reduced in Fng tKO mice
173    Splenic T cells were then stimulated with concanavalin A or ovalbumin in vivo or in vitro, and CD2
174 hole spleen cells following stimulation with concanavalin A or PPD.
175 d apoptosis following activation either with concanavalin A or with antibodies to CD3 and CD28 and le
176 911) and cyclic peptide (D002) reactive with concanavalin A presented in a multiple antigen peptide (
177 arger than that of the wild type, had gained concanavalin A reactivity, and that the arabinan termini
178 ration in response to phytohemagglutinin and concanavalin A remained stable or increased for the Inte
179  of bacterial growth inhibition, addition of concanavalin A results in the formation of extensive dex
180 microbalance (QCM) to show that tripod-bound Concanavalin A retains its affinity for polysaccharides
181 , and Rac-1 coprecipitates with rhodopsin on Concanavalin A Sepharose.
182 ve loss of T cells, compromised responses to concanavalin A stimulation, and absence of inflammatory
183  and interleukin (IL)-4 (Th2 cytokine) after concanavalin A stimulation.
184                    In DRG cells treated with concanavalin A the EC50 for ATPA was 341 nM.
185 oclonal antibody (M1) and for the binding of concanavalin A to a glycoprotein have been determined.
186     Flipping was assayed by using the lectin Concanavalin A to capture M5-DLOs that had been transloc
187 icrotubules that normally follows binding of concanavalin A to leukocyte cell surface receptors.
188  endocytosis of transferrin and transport of concanavalin A to the lysosome are both impaired, confir
189         This analysis implies that, although Concanavalin A tolerates the additional 6 arm GlcNAc pre
190  with BSA, and the affinity of photoattached concanavalin A toward ovalbumin was compared with that o
191 as there was no protection against CCl(4) or concanavalin A toxicity.
192 o O-linked N-acetylglucosamine or the lectin concanavalin A was detected.
193  1.5-microm diameter microsphere coated with concanavalin A was inserted though an ablated hole in th
194 oglobulin, ribonuclease A, E-cadherin 5, and concanavalin A were co-lyophilized with carbohydrates (t
195 ol) cows stimulated with the general mitogen concanavalin A were highly similar (overall r = 0.84), s
196 or all seven lectins, and similar to that of concanavalin A which is also a member of the Diocleinae
197                                   Given that Concanavalin A's tertiary structure is thought to be rel
198 unction of DR signaling in T cell-dependent (concanavalin A) and independent (lipopolysaccharide/gala
199 d targets (carbonic anhydrase, streptavidin, concanavalin A) to identify desired ligands.
200                                              Concanavalin A, a lectin selective for high mannose and
201                                              Concanavalin A, a lectin that binds glucose and mannose
202 ectin binding experiments using succinylated concanavalin A, a plant lectin with high affinity for ma
203 f oocytes with 20% sucrose and 250 microg/ml concanavalin A, agents that inhibit the endocytosis of R
204 superoxide dismutase, dimeric and tetrameric concanavalin A, and heptameric GroES and Gp31; ranging i
205 asurements were performed with cytochrome c, concanavalin A, and poly-L-lysine, biomolecules that exh
206 y from Escherichia coli lipopolysaccharides, concanavalin A, and Pseudomonas aeruginosa exotoxin A.
207 ulin and enolase), tetrameric (streptavidin, concanavalin A, and pyruvate kinase), and pentameric (C-
208 an inducible pattern on T cells activated by concanavalin A, anti-CD3 mAb in combination with anti-CD
209 l activation, as stimulation of T cells with concanavalin A, but not phorbol 12-myristate 13-acetate
210 riefly, the well-known lectin macromolecule (concanavalin A, Con A) monolayer was functionalized on 3
211            While no effect was observed with concanavalin A, cyclothiazide greatly enhanced the Glu-,
212 that treatment of OAT1-expressing cells with concanavalin A, depletion of K(+) from the cells, or tra
213              Following enzymatic hydrolysis, concanavalin A, immobilized or soluble, was added to the
214 s attenuated by blockade of endocytosis with concanavalin A, indicating a critical role for internali
215 more, a non-selective endocytosis inhibitor, concanavalin A, inhibited the internalization of wild ty
216 n in guinea pig spleen cells stimulated with concanavalin A, lipopolysaccharide (LPS), phorbol myrist
217 ion in response to the homologous immunogen, concanavalin A, or lipopolysaccharide was similar for al
218 itogen-induced lymphoproliferative activity (concanavalin A, range: 74,218 dropping to 55,880 counts
219 A, avidin, monoclonal anti-BSA antibody, and concanavalin A, respectively.
220 ctra of the amide I region of poly-l-lysine, concanavalin A, ribonuclease A, and lysozyme show cross-
221   Moreover, in response to aoHGE extracts or concanavalin A, splenocytes from ehrlichia-infected mice
222 nocytes from infected animals in response to concanavalin A, suggesting a role for NO in mediating th
223 -alpha(5)beta(1) interaction is inhibited by concanavalin A, suggesting that GT1b binds to mannose st
224 roteases and organic extraction but bound to concanavalin A, suggesting that IRI is a sulfated glycan
225               Likewise, after treatment with concanavalin A, there was no change in either desensitiz
226 n with fluorescein isothiocyanate-conjugated concanavalin A, was increased in the absence of Dp71.
227     By using a Sepharose-immobilized lectin, concanavalin A, we isolated a fraction enriched with LRV
228 . coli 2443 with a fluorescent derivative of concanavalin A, we observed a similar helical organizati
229  genes after exposure to interferon-gamma or concanavalin A, which resulted in minimal HLA-B27 up-reg
230 ned a high amount of mannose, as detected by concanavalin A, while the UT-A1 in lipid rafts was the m
231 pha-Man-(1--> 6)]-Man to bind to the lectin, Concanavalin A, with almost the same affinity as the tri
232 ncluded: (a) reduced IFN-gamma production by concanavalin A- or antigen-stimulated T cells; and (b) h
233      Downregulation of chIL-17RA occurred in concanavalin A- or lipopolysaccharide-activated splenic
234 scriptional regulation of CSF-2 and CSF-3 in concanavalin A-activated MSCs requires MT1-MMP signaling
235       GPI-IL-12-induced the proliferation of concanavalin A-activated T cells and induced IFN-gamma s
236 ene (aSMase(-/-)), we found that thymocytes, concanavalin A-activated T cells, and lipopolysaccharide
237  was also lost when they were incubated with concanavalin A-agarose beads, suggesting that the inhibi
238                         After binding to the concanavalin A-agarose beads, the inhibitor in BAL fluid
239 th a putative sequence tract RYRY that mimic concanavalin A-binding glycans.
240                                     Finally, concanavalin A-coated colloids were allowed to adsorb on
241 protease domain of MDC9, Western analysis of concanavalin A-enriched glomerular microsomal extracts d
242 ruited iNKT cells were anergic and prevented concanavalin A-induced (ConA-induced) hepatitis by speci
243 imicrobial susceptibility assay based on the concanavalin A-induced clustering of dextran-coated gold
244  that myosin phosphorylation is critical for concanavalin A-induced gathering of surface receptors.
245                                     Instead, concanavalin A-induced hepatitis was completely prevente
246 endogenous Gal-1 protected the liver against concanavalin A-induced hepatitis with the B6 genetic bac
247 henotype of these cells, reduced severity of concanavalin A-induced hepatitis, and alterations in the
248 ver regeneration, and in the murine model of concanavalin A-induced liver inflammation.
249                                   Similarly, concanavalin A-induced liver injury, where type 2 cytoki
250   These findings were confirmed in mice with concanavalin A-induced liver injury.
251 vents and treats both IL12-, IFN-gamma-, and concanavalin A-induced liver toxicity.
252 ion in A549 human lung epithelial cells, and concanavalin A-induced monocyte proliferation.
253  K12-Fc inhibited in a dose-dependent manner concanavalin A-induced proliferation, but not anti-TcRal
254 mal (BLV-negative) cows and had no effect on concanavalin A-induced proliferation.
255 ot wild type RLC almost completely abolished concanavalin A-induced receptor cap formation.
256 y, IL-22TG mice were completely resistant to concanavalin A-induced T cell hepatitis with minimal eff
257         Neutralization of CXCL10 ameliorated concanavalin A-induced tissue injury in vivo, which was
258                              The loss of the concanavalin A-induced, but not the lipopolysaccharide-i
259 characterized member of its superfamily, the Concanavalin A-like lectins/glucanases.
260 lminant hepatitis, but was without effect on concanavalin A-mediated hepatitis.
261 e, phenyl-Sepharose hydrophobic interaction, concanavalin A-Sepharose affinity and Superose 12 gel fi
262                 PG27 significantly increased concanavalin A-stimulated in vitro IL-4 production by da
263               However, neither inhibition of concanavalin A-stimulated spleen cells nor keyhole limpe
264 , trough) and for lymphocyte functions using concanavalin A-stimulated whole blood assays to measure
265 ies agrin-G3 as a functional analogue of the concanavalin A-type lectins, highlights functional simil
266 ssessed after perfusion with FITC-conjugated concanavalin A.
267  of the second through sixth generations and Concanavalin A.
268 pon aggregation of unilamellar vesicles with concanavalin A.
269 ssessed by labeling adherent leukocytes with concanavalin A.
270 d has a surface that is capped by the lectin concanavalin A.
271  which is only manifested in the presence of concanavalin A.
272 te and sodium azide, and one model biotoxin, concanavalin A.
273 th immobilized Sambucus nigra agglutinin and concanavalin A.
274 at were resistant to the cytotoxic action of concanavalin A.
275 - and bivalent ligands for the legume lectin concanavalin A.
276 10 is inducible by interferons (IFNs) and by concanavalin A.
277 after stimulation of cells with the mitogen, concanavalin A.
278 n of peripheral blood mononuclear cells with concanavalin A.
279 cation exchange column and was shown to bind concanavalin A.
280 ho-proliferative response to alloantigens or concanavalin A.
281 tic liver damage induced by the injection of concanavalin A.
282 ose-containing bath solution or treated with concanavalin A.
283 ction in response to either C. pneumoniae or concanavalin A.
284 ting the enzyme's aggregation induced by the concanavalin A.
285 d Th-2 cytokine responses to SME antigen and concanavalin A.
286 ion-induced programmed cell death induced by concanavalin A.
287  desensitization blockers, cyclothiazide and concanavalin A.
288  separate group of mice by administration of concanavalin A.
289  binding of the polymers to the plant lectin concanavalin A.
290 r induction of selective EC injury using the concanavalin A/anti-concanavalin A model and after ische
291 uired for TNFalpha-induced injury induced by concanavalin A/GalN but not by ConA alone.
292 ulin G, avidin:biotin, antibody:antigen, and concanavalin A:glycoprotein interactions are used to dem
293 ) kainate receptors, two unrelated proteins, concanavalin-A (Con-A) and postsynaptic density protein
294 interaction force between a polymer-tethered concanavalin-A protein (ConA) and a similarly tethered m
295 -induced binding to rhodopsin immobilized on concanavalin-A resin.
296          Upon transplantation into mice with concanavalin-A-induced acute liver failure and fatal met
297 s not evident after 48-hour stimulation with concanavalin-A.
298 transthyretin, avidin, bovine serum albumin, concanavalin, human serum amyloid protein, and Immunoglo
299 tional space presented by both Ubiquitin and Concanavalin; it appears that diverse conformational fam
300 tems such as human pancreatic alpha-amylase, concanavalin, Pichia pastoris lysyl oxidase, and Klebsie

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