ライフサイエンスコーパス: conditioned place preference

1 reduced hind-limb sensitisation and induced conditioned place preference.

2 nucleus accumbens (NAc) and cocaine-induced conditioned place preference.

3 ersistent spine gain correlated with cocaine conditioned place preference.

4 intake and block the development of alcohol-conditioned place preference.

5 dminister cocaine, and it fails to produce a conditioned place preference.

6 f synaptic plasticity in the VTA and cocaine conditioned place preference.

7 Mzeta on the behavioral responses by cocaine conditioned place preference.

8 tor activation, behavioral sensitization, or conditioned place preference.

9 signaling (mice), suppressed opioid-induced conditioned place preference.

10 ibitory peptide in the VTA disrupted cocaine conditioned place preference.

11 rexpressing G9a in the NAc decreases cocaine-conditioned place preference.

12 tenuated cue conditioning, but also enhanced conditioned place preference.

13 ut not drug-induced reinstatement of cocaine conditioned place preference.

14 ant path that was found to underlie nicotine-conditioned place preference.

15 ionally modulates amphetamine (AMPH)-induced conditioned place preference.

16 einstatement of extinguished cocaine-induced conditioned place preference.

17 the nicotine-induced synaptic plasticity and conditioned place preference.

18 rmal morphine reward behavior as measured by conditioned place preference.

19 n diminishes morphine reward, as measured by conditioned place preference.

20 layed significantly less hyperlocomotion and conditioned place preference.

21 nd more persistent memory of cocaine-induced conditioned place preference.

22 d for morphine-induced reward as measured by conditioned place preference.

23 but not the ventral tegmental area, induced conditioned place preference.

24 show deficits in FS-induced reinstatement of conditioned place preference.

25 ts of morphine as measured by acquisition of conditioned place preference.

26 ut not the shell or ventral pallidum induced conditioned place preference.

27 o influence feeding, locomotor activity, and conditioned place preference.

28 eport that zebrafish exhibit cocaine-induced conditioned place preference.

29 with footshock to reinstate cocaine-induced conditioned place preference.

30 dose produced a nonsignificant trend towards conditioned place preference.

31 diminished behavioral reward, as measured by conditioned place preference.

32 egions of the VS and trained to the morphine conditioned place preference.

33 HT1A autoreceptors are necessary for cocaine conditioned place preference.

34 ne, blocked reinstatement of morphine-evoked conditioned place preference.

35 ssion exclusively in the NAc reduced cocaine conditioned place preference.

36 e, however, showed unaltered cocaine-induced conditioned place preference.

37 into the posterior shell of NAS established conditioned place preferences.

38 DAT and mice without 5-HTT establish cocaine-conditioned place preferences.

39 orces instrumental behaviour and establishes conditioned place preferences.

40 the development of cocaine sensitization and conditioned place preference, a measure of cocaine rewar

41 rom the injury with local anesthetic elicits conditioned place preference, activates ventral tegmenta

42 of these projections affects behavior using conditioned place preference and a task in which mice le

43 e exhibit enhanced cocaine sensitization and conditioned place preference and an increase in Alk expr

44 rol pretreatments attenuated cocaine-induced conditioned place preference and blocked the cocaine-ind

45 with (+/-)-BayK-8644 (BayK) enhanced cocaine conditioned place preference and cocaine psychomotor act

46 ular dopamine in the NAc, as well as cocaine conditioned place preference and cocaine self-administra

47 ssociated virus and Cre lines during cocaine conditioned place preference and cocaine-induced locomot

48 as4 in the NAc significantly reduced cocaine conditioned place preference and delayed learning of the

49 Conditioned place preference and locomotor sensitization

50 bited METH self-administration, METH-induced conditioned place preference and METH- or cue-induced re

51 emonstrate that the genotypic differences in conditioned place preference and passive avoidance learn

52 bachol induce reward; such injections induce conditioned place preference and rats learn quickly to s

53 We used conditioned place preference and self-administration par

54 mpared with their wild-type controls in both conditioned place preference and sensitization behaviors

55 ned protocol which successfully induced both conditioned place preference and sensitization simultane

56 ions selectively impaired the acquisition of conditioned place preference and the use of spatial info

57 sed eating behavior and also caused positive conditioned place preferences and increased positive hed

58 rain-stimulation reward, (2) cocaine-induced conditioned place preference, and (3) cocaine-triggered

59 ) dose-dependently attenuate cocaine-induced conditioned place preference, and (3) dose-dependently a

60 inistration and the expression of an ethanol conditioned place preference, and abolished stress-induc

61 or amphetamine (3 mg/kg), cocaine (20 mg/kg) conditioned place preference, and active avoidance learn

62 conditioned taste aversion, ethanol-induced conditioned place preference, and ethanol self-administr

63 tor 4 (TLR4) in opioid analgesia, tolerance, conditioned place preference, and self-administration.

64 ocomotor activity, behavioral sensitization, conditioned place preference, and striatal dopamine rele

65 mbens, the ability of cocaine to establish a conditioned place preference, and the ability of cocaine

66 its involvement in behavioral sensitization, conditioned place-preference, and self-administration of

67 s relevant to addiction: locomotor activity, conditioned place preference, anxiety, discrimination, a

68 king MCH1R exhibit decreased cocaine-induced conditioned place preference, as well as cocaine sensiti

69 instatement of cocaine-seeking behavior in a conditioned place preference assay.

70 ndence, and showed no aversive effect in the conditioned place preference assay.

71 We performed conditioned place preference assays.

72 It also blocks expression of cocaine-induced conditioned place preference at a dose (1)/(300) that of

73 caffeine also enhanced the development of a conditioned place preference at a sub-threshold dose of

74 r of dendritic protein synthesis, in cocaine conditioned place preference, behavioral sensitization,

75 both cocaine-induced locomotor behavior and conditioned place preference, but had no effect on stres

76 e had opposite effects on the acquisition of conditioned place preference by significantly enhancing

77 duces an impairment of extinction of cocaine-conditioned place preference (cocaine-CPP) independent o

78 nucleus accumbens suppresses cocaine-induced conditioned place preference (CPP) acquisition in mice.

79 We show that COC-conditioned place preference (CPP) activates ERK, CREB,

80 r blocked the acquisition of cocaine-induced conditioned place preference (CPP) and activation of tra

81 Little is known about effects of FR on drug-conditioned place preference (CPP) and brain regional me

82 CaMKII activity in the VTA affected cocaine conditioned place preference (CPP) and cocaine-evoked sy

83 r preference for morphine was examined using conditioned place preference (CPP) and drug-induced rein

84 Conditioned place preference (CPP) and in vivo microdial

85 d the role of VP dopamine in cocaine-induced conditioned place preference (CPP) and locomotor activat

86 e ventral pallidum (VP) in the expression of conditioned place preference (CPP) and motor adaptations

87 mesolimbic EX4 on two models of food reward: conditioned place preference (CPP) and progressive ratio

88 y in the hippocampus, their role in morphine conditioned place preference (CPP) and reinstatement rem

89 y attenuated cocaine-primed reinstatement of conditioned place preference (CPP) and relapse of cocain

90 Conditioned place preference (CPP) and saccharin (0.2% w

91 nduced reinstatement of drug seeking in both conditioned place preference (CPP) and self-administrati

92 methylphenidate would alter morphine-induced conditioned place preference (CPP) and sucrose-reinforce

93 ine pairing with environmental cues (ie, the conditioned place preference (CPP) apparatus) triggers a

94 In a conditioned place preference (CPP) assay, we observed th

95 attenuates the intensity of cocaine-induced conditioned place preference (CPP) behaviors in female r

96 y shown strain differences in heroin-induced conditioned place preference (CPP) between C57BL/6J (C57

97 nd caudate putamen (CPu) in morphine-induced conditioned place preference (CPP) by real-time reverse

98 Utilizing a rat paradigm of conditioned place preference (CPP) combined with ankle m

99 and spatial location cues were studied in 6 conditioned place preference (CPP) experiments with etha

100 ch predicts future reinstatement of morphine conditioned place preference (CPP) following a priming d

101 Female rats exhibit a conditioned place preference (CPP) for a context paired

102 phine-induced locomotor sensitization or for conditioned place preference (CPP) for a morphine- or a

103 ch there was no opportunity to consume food (conditioned place preference (CPP) for an environment pr

104 in LH orexin neurons varied in proportion to conditioned place preference (CPP) for morphine, cocaine

105 ncountered and, subsequently, will display a conditioned place preference (CPP) for that environment.

106 paired with environmental cues establishes a conditioned place preference (CPP) for that environment.

107 preoptic area (mPOA) on the expression of a conditioned place preference (CPP) for vaginocervical st

108 also found that the magnitude of amphetamine-conditioned place preference (CPP) in behaving rats corr

109 ch adrenal hormones regulate cocaine-induced conditioned place preference (CPP) in either sex.

110 extinction, but not acquisition, of cocaine conditioned place preference (CPP) in male mice increase

111 e find that nicotine produced dose-dependent conditioned place preference (CPP) in mice.

112 did not alter acquisition of ethanol-induced conditioned place preference (CPP) in mice.

113 hibited the acquisition of oxycodone-induced conditioned place preference (CPP) in rats.

114 at amphetamine (AMPH) conditioning induced a conditioned place preference (CPP) in sexually naive (SN

115 mmunohistochemistry after cocaine (10 mg/kg) conditioned place preference (CPP) in Sprague Dawley rat

116 in vitro and analysis of an animal model of conditioned place preference (CPP) in vivo, we investiga

117 This protocol describes conditioned place preference (CPP) in zebrafish followin

118 compare the expression and persistence of a conditioned place preference (CPP) induced by a relative

119 Conditioned place preference (CPP) is a behavioral assay

120 Reinstatement of previously extinguished conditioned place preference (CPP) is precipitated by st

121 eral amygdala memory in the consolidation of conditioned place preference (CPP) memory.

122 Using a cocaine conditioned place preference (CPP) model, we demonstrate

123 the equipment and methods used to establish conditioned place preference (CPP) or aversion (CPA).

124 MPH) motivated behavior was examined using a conditioned place preference (CPP) paradigm and was show

125 We used a conditioned place preference (CPP) paradigm to determine

126 In addition, we employed the conditioned place preference (CPP) paradigm to evaluate

127 We used a standard 4 day conditioned place preference (CPP) paradigm using 20mg/k

128 otine and cocaine reward-like effects in the conditioned place preference (CPP) paradigm, using pharm

129 Using conditioned place preference (CPP) paradigm, we observed

130 (AMPH) as the unconditioned stimulus in the conditioned place preference (CPP) paradigm.

131 aired contextual cue memory assessed using a conditioned place preference (CPP) paradigm.

132 red with cocaine experience assessed using a conditioned place preference (CPP) paradigm.

133 moderate doses of cocaine when tested in the conditioned place preference (CPP) procedure and also bl

134 Previous work using the conditioned place preference (CPP) procedure implicates

135 Using a conditioned place preference (CPP) procedure, we found t

136 pocretin neurons in mice following a cocaine-conditioned place preference (CPP) protocol.

137 Using a conditioned place preference (CPP) reinstatement procedu

138 58 has been shown to block expression of the conditioned place preference (CPP) response to cocaine i

139 efore or immediately after a cocaine-induced conditioned place preference (CPP) retrieval trial, beta

140 which food-reward behavior, assessed using a conditioned place preference (CPP) task, is monitored in

141 of the amygdala impair acquisition of a food conditioned place preference (CPP) task.

142 rats were subsequently trained in a cocaine conditioned place preference (CPP) task.

143 ysis, behavioral activity assessments, and a conditioned place preference (CPP) test were used to inv

144 istered ethanol (EtOH) were examined using a conditioned place preference (CPP) test.

145 of U50488 15 min before cocaine blocked the conditioned place preference (CPP) to cocaine, but only

146 still self-administer cocaine and/or display conditioned place preference (CPP) to cocaine, which led

147 the acquisition, but not the expression, of conditioned place preference (CPP) to cocaine.

148 This treatment enhanced conditioned place preference (CPP) to morphine (2 x 10 m

149 eated rats, MDMA-treated rats did not form a conditioned place preference (CPP) to sex.

150 Finally, we show that cocaine conditioned place preference (CPP) training (15 mg/kg; f

151 Conditioned place preference (CPP) was conducted in a 2-

152 In the present study, conditioned place preference (CPP) was induced with high

153 Conditioned place preference (CPP) was produced by 10 mi

154 Cocaine-induced locomotor sensitization and conditioned place preference (CPP) were attenuated in tP

155 nstrated by reinstatement of the behavior of conditioned place preference (CPP) with sub-threshold pr

156 ly regulates extinction of a cocaine-induced conditioned place preference (CPP), a task that requires

157 resent studies examined the effects of E2 on conditioned place preference (CPP), and E2 levels produc

158 s quinine, exhibited greater ethanol-induced conditioned place preference (CPP), and showed reduced e

159 to VTA exhibit Fos activation with morphine conditioned place preference (CPP), and whether these ce

160 on cocaine-induced behavioral sensitization, conditioned place preference (CPP), cue- and cocaine pri

161 ce, using both fear conditioning and cocaine-conditioned place preference (CPP), during acquisition a

162 reward measured by the paradigm of unbiased conditioned place preference (CPP), focusing on GABAergi

163 cocaine-induced locomotor sensitization and conditioned place preference (CPP), mice receiving SB203

164 s, conditioned motor sensitization (CMS) and conditioned place preference (CPP), to ascertain whether

165 Using reinstatement of conditioned place preference (CPP), we determined whethe

166 ith drug administration can be studied using conditioned place preference (CPP).

167 ppocampus that mediate extinction of cocaine conditioned place preference (CPP).

168 ined the specific role of the DHC in cocaine conditioned place preference (CPP).

169 s (NAc) in the expression of ethanol-induced conditioned place preference (CPP).

170 the rewarding effects of AMPH as measured by conditioned place preference (CPP).

171 ase the reward value of food, as assessed by conditioned place preference (CPP).

172 the effects of forced swim stress on cocaine-conditioned place preference (CPP).

173 PFC) is necessary for acquisition of cocaine-conditioned place preference (CPP).

174 lating effect of cocaine and cocaine-induced conditioned place preference (CPP).

175 uder as reinforcement for the development of conditioned place preference (CPP).

176 od of 7 d, males were tested for amphetamine conditioned place preference (CPP).

177 a (VTA), and reinstates extinguished cocaine-conditioned place preference (CPP).

178 of NAc silent synapse maturation in cocaine-conditioned place preference (CPP).

179 aine (10 mg/kg, s.c.) induced locomotion and conditioned place preference (CPP).

180 id not differ in their expression of cocaine-conditioned place preference (CPP).

181 injections of AMPH and blocked AMPH-induced conditioned place preference (CPP).

182 was assessed using evoked sensory stimuli or conditioned place preference (CPP).

183 littermates were tested for nicotine-induced conditioned place preference (CPP); voluntary oral nicot

184 d that NE was necessary for morphine-induced conditioned place preference (CPP; a measure of reward)

185 ous nerve block to elicit pain relief (i.e., conditioned place preference, CPP), revealing the presen

186 In contrast, conditioned place preference demonstrated an inverse cor

187 nforcement schedule, but did not affect food conditioned place preference expression.

188 Conditioned place preference, extinction and reinstateme

189 Conditioned place preference following hormone treatment

190 , as sexually experienced males did not form conditioned place preference for 0.5 mg/kg morphine.

191 creased 5-HT6 receptors in iMSNs facilitated conditioned place preference for a low dose of cocaine.

192 n arm avoidance on the elevated plus-maze or conditioned place preference for cocaine, although a lin

193 ct recognition, object location recognition, conditioned place preference for cocaine, or motor learn

194 cocaine-induced locomotor sensitization and conditioned place preference for cocaine.

195 how and high-fat diet intake, meal patterns, conditioned place preference for high-fat food, cue-indu

196 We observed a reduction of conditioned place preference for low doses of the opioid

197 amphetamine reward, indicated by sensitized conditioned place preference for low-dose (0.5 mg/kg) am

198 l behavior over repeated mating sessions, or conditioned place preference for mating.

199 st, dopamine-deficient mice display a robust conditioned place preference for morphine when given eit

200 -administer less nicotine and show decreased conditioned place preference for nicotine compared with

201 o exhibit either behavioral sensitization or conditioned place preference; however, it seems that sen

202 ucleus accumbens by itself sufficed to drive conditioned place preference in freely moving mice.

203 Also, morphine was ineffective in inducing conditioned place preference in GRK5 knockout mice, wher

204 tionship between locomotor sensitization and conditioned place preference in individual animals.

205 Here we used conditioned place preference in mice to examine the prec

206 mber, indicating morphine induced comparable conditioned place preference in ppENK (+/+) and ppENK (-

207 leus accumbens reduced expression of cocaine-conditioned place preference in Prkcz(-/-) mice.

208 acilitate the extinction of morphine-induced conditioned place preference in rats.

209 ly paired with cocaine, nor did they exhibit conditioned place preference in response to cocaine.

210 Each strain displays cocaine-conditioned place preference in this major mouse model f

211 Morphine produced hyperlocomotion and conditioned place preference in wild-type mice, whereas

212 cal brain stimulation, and (3) can establish conditioned place preferences in laboratory animals, sug

213 chloride and cocaine methiodide to establish conditioned place preferences in rats with self-administ

214 ing neurons in freely behaving mice promoted conditioned place preference, indicating that such activ

215 an exacerbated psychomotor sensitization and conditioned place preference induced by low doses of coc

216 assessing drug reward, while methylphenidate-conditioned place preference is also maintained in DAT k

217 the role of enkephalins in morphine-induced conditioned place preference, locomotor sensitization, a

218 We found that an established morphine conditioned place preference (mCPP) was persistently dis

219 ne, and alcohol, and is also observed in the conditioned place preference model in rats and mice.

220 nges in dopamine release associated with the conditioned place preference model of drug craving.

221 Conditioned place preference (n = 112) and context-induc

222 ockdown failed to influence cocaine-elicited conditioned place preferences, nor did it produce consis

223 that stress-induced potentiation of cocaine conditioned place preference occurred by a similar mecha

224 ciated with an increase in NAcc dopamine and conditioned place preference only under certain testing

225 orphanin FQ (0.1-100 nmol) failed to produce conditioned place preference or aversion, but a pronounc

226 tivity in these assays and did not produce a conditioned place preference or aversion, but elicited C

227 GAT211 did not induce conditioned place preference or aversion.

228 velopment of analgesic tolerance but not for conditioned place preference or behavioral sensitization

229 effects, acute antinociceptive tolerance, or conditioned-place preference or aversion.

230 ute administration, KO mice were impaired in conditioned place preference, oral nicotine intake and m

231 Furthermore, in the conditioned place preference paradigm with 10 mg/kg morp

232 Using an unbiased conditioned place preference paradigm with rats, we exam

233 ding effects of morphine, as measured in the conditioned place preference paradigm, were substantiall

234 attenuation of alcohol reward, measured in a conditioned place preference paradigm.

235 the reinforcing properties of morphine in a conditioned place preference paradigm.

236 gative form of CREB) in the VTA and, using a conditioned place-preference paradigm, found that CREB a

237 solidation of extinction in fear and cocaine conditioned place preference paradigms.

238 ects of ketamine in the drug discrimination, conditioned place preference, pre-pulse inhibition and o

239 nuated the rewarding effects of cocaine in a conditioned place preference procedure but did not affec

240 We used a novel conditioned place preference procedure to show that opto

241 An unbiased conditioned place preference procedure was used to deter

242 In the conditioned place preference procedure, nicotine was suf

243 n both periadolescent and adult mice using a conditioned place preference procedure.

244 Here, we used a conditioned place-preference procedure to investigate th

245 In the present study, we use a conditioned place preference/reinstatement paradigm in m

246 ns of heroin, or saline, in the setting of a conditioned place preference study.

247 ng ASIC1A in the mouse NAc increased cocaine-conditioned place preference, suggesting an unexpected r

248 creases their development of cocaine-induced conditioned place preference, suggesting reduced sensiti

249 ccurred in the extinction of cocaine-induced conditioned place preference, suggesting that the observ

250 enhance the ability of morphine to establish conditioned place preferences, suggesting that altered G

251 the rewarding properties of morphine in the conditioned place preference test were greater in the be

252 Using the hot-plate and conditioned place preference test, we investigated opioi

253 cial interactions, assessed using a socially conditioned place preference test.

254 exes, and reduced ongoing pain assessed by a conditioned place preference test.

255 cocaine reward/reinforcement, as measured by conditioned place-preference testing.

256 initial cocaine exposures as well as robust conditioned place preference to a lower dose of cocaine,

257 In these studies, we used conditioned place preference to assess the activity of N

258 Furthermore, mTOR deletion attenuated conditioned place preference to cocaine and cocaine-indu

259 mice also have a decreased ability to form a conditioned place preference to cocaine.

260 We used conditioned place preference to concomitantly determine

261 shifted the dose-response curve for cocaine-conditioned place preference to the left, indicating alt

262 ith olfactory-dependent fear conditioning or conditioned place preference using acetophenone.

263 vity to cocaine (0, 2.5, 5, 10, or 20 mg/kg) conditioned place preference was assessed.

264 Conditioned place preference was established by EM-1 inj

265 A conditioned place preference was evident in controls and

266 rence in GRK5 knockout mice, whereas cocaine conditioned place preference was retained.

267 Conditioned place preference was used to assess the epig

268 algesia, tolerance, physical dependence, and conditioned place preference, we used mice having target

269 Carbachol produced conditioned place preferences when injected into the pos

270 that p11 knockout mice have enhanced cocaine conditioned place preference, which is reproduced by the

271 tinction of a previously established cocaine-conditioned place preference, while simultaneously enhan