ライフサイエンスコーパス: conditioned response

1 elevance to behaviour (initiate versus reset conditioned responses).

2 it is their integration that results in the conditioned response.

3 hat produced weak sensitization instead of a conditioned response.

4 togenetically in parallel with the eye-blink conditioned response.

5 ere perception of the CS in the absence of a conditioned response.

6 neuron synapses, underlying production of a conditioned response.

7 ereby limit the development of the eye-blink conditioned response.

8 blocked learning but not performance of the conditioned response.

9 lesions of either structure disrupted the HR conditioned response.

10 te the ability of a paired cue to elicit its conditioned response.

11 xcitability after initial acquisition of the conditioned response.

12 the BLA is necessary for the expression of a conditioned response.

13 ys, paired VNS accelerated extinction of the conditioned response.

14 capable of acquiring the ability to evoke a conditioned response.

15 red throughout the experiment as an index of conditioned responses.

16 auditory and visual signals involved in fear-conditioned responses.

17 eraction between pharmacological effects and conditioned responses.

18 e cerebellar cortex before the appearance of conditioned responses.

19 d thereby limit the development of eye-blink conditioned responses.

20 storage and expression of naturally acquired conditioned responses.

21 ffects on the amplitude, area, or latency of conditioned responses.

22 s, cerebellar patients failed to acquire new conditioned responses.

23 ns suggests laterality of reinforcing and/or conditioned responses.

24 ibution to the performance or acquisition of conditioned responses.

25 in or vehicle resulted in the acquisition of conditioned responses.

26 equired for the storage of naturally learned conditioned responses.

27 ense tremors and impaired the acquisition of conditioned responses.

28 ignificantly between trials with and without conditioned responses.

29 e cerebellum and allow for the expression of conditioned responses.

30 hese forms of plasticity interact to produce conditioned responses.

31 ent mutants did not develop drug-paired, cue-conditioned responses.

32 h the cerebellum to mediate adaptively timed conditioned responses.

33 elivery of AMPA receptors and acquisition of conditioned responses.

34 t cerebellar circuitry during acquisition of conditioned responses.

35 audal VP during the performance of ST and GT conditioned responses.

36 atient groups showed impaired acquisition of conditioned responses.

37 s well-recognized role in the acquisition of conditioned responses.

38 um that are necessary for acquisition of the conditioned response: (1) long-term depression (LTD) at

39 ed differentially on S+ trials (mean percent conditioned responses=66) versus S- trials (30).

40 d in the early stages of conditioning during conditioned response acquisition, whereas a cytosolic fo

41 ear conditioning increased generalization of conditioned response across tone frequencies, whereas PV

42 ons from very slow learning (exhibited < 30% conditioned responses after 15 training sessions) or fro

43 disconnected, failed to acquire second-order conditioned responses (although they did acquire second-

44 asticity in the cerebellar cortex influences conditioned response amplitude and timing, whereas plast

45 cterization of a training procedure in which conditioned response amplitude is brought under experime

46 ditioned stimulus (US) is made contingent on conditioned response amplitude: the US is delivered for

47 both cases, the reflex comes to resemble the conditioned response and follows some of the same behavi

48 cquisition, retention, and generalization of conditioned responses and accounts for the effects of se

49 cerebellum interact functionally since both conditioned responses and conditioned hippocampal pyrami

50 BAergic and glutamatergic components, encode conditioned responses and control compulsive reward-seek

51 renia group (n = 55) had significantly fewer conditioned responses and longer onset latencies than ag

52 etween the expectation for the drug effects (conditioned responses) and the blunted pharmacological e

53 aking; (2) misinterpreting the evidence; (3) conditioned response; and (4) missed cues.

54 r lesions who cannot acquire new classically conditioned responses are able to retain and express con

55 Conditioned responses are completely, but reversibly, ab

56 Conditioned responses are learned and generated by the c

57 aimed at inhibiting dopamine increases from conditioned responses are likely to be therapeutically b

58 strongly support the general hypothesis that conditioned responses are the result of strengthening of

59 hich are critical for the acquisition of new conditioned responses, are not essential for the storage

60 caine-associated stimuli likely represents a conditioned response as opposed to a generalized stimulu

61 study individual variation in acquisition of conditioned responses as a possible risk factor for drug

62 isual threat are probably naturally acquired conditioned responses because they extinguish in normal

63 tween-group differences in the percentage of conditioned responses between both groups.

64 mg kg(-1) propranolol delayed acquisition of conditioned responses but all groups were able to learn

65 0 mg kg-1 propranolol delayed acquisition of conditioned responses but all groups were able to learn

66 s influence the initial expression of reward-conditioned responses but that their contribution to per

67 t it was related to the strength of previous conditioned responses, but also that predictive compared

68 ssociated with cocaine use produces a strong conditioned response characterised by autonomic hyperaro

69 odulation of extinction of addiction-related conditioned responses, consider possible limitations of

70 imulus is inhibited during the expression of conditioned responses-consistent with the inhibitory pro

71 fic environment, contextual cues can control conditioned responses, context-specific sensitization, a

72 ing, whereas population codes and behavioral conditioned responses continued to develop during subseq

73 CS-EBS pairings yielded conditioned response (CR) acquisition, in which Groups T

74 nals during training sessions should prevent conditioned response (CR) acquisition.

75 KHA and WKHT rats exhibited similar rates of conditioned response (CR) acquisition.

76 examined the influence of learning the trace conditioned response (CR) after having acquired the CR d

77 Reinstatement--the return of an extinguished conditioned response (CR) after reexposure to the uncond

78 Discrimination of the eyeblink conditioned response (CR) between conditioned stimuli (C

79 facilitation of activity correlated with the conditioned response (CR) developed in the IPN and PN du

80 Retention of the eyeblink conditioned response (CR) during both tests was highest

81 in behaving animals, we demonstrate that the conditioned response (CR) expression and timing are comp

82 on, lesions disrupted accuracy of timing the conditioned response (CR) for both delay and trace CSs:

83 Because robust developmental differences in conditioned response (CR) generation and CR latency meas

84 In Pavlovian eyeblink conditioning, the conditioned response (CR) is highly lateralized to the e

85 ISI, thus permitting the characterization of conditioned response (CR) learning at one ISI and the ex

86 Animals with MD lesions acquired the EB conditioned response (CR) more slowly than sham-lesioned

87 The conditioned response (CR) of approaching the site of foo

88 but the KO mice showed a significantly lower conditioned response (CR) percentage than the WT mice in

89 haping procedures on lever-press autoshaping conditioned response (CR) performance and 3H-8-OH-DPAT-l

90 Eyeblink conditioned response (CR) timing was assessed in adult a

91 was seen in the hippocampus on trials when a conditioned response (CR) was executed.

92 Acquisition of the EB conditioned response (CR) was faster and reached a highe

93 hout the US (reactivation), and the freezing conditioned response (CR) was measured.

94 interstimulus intervals (ISIs), manifests a conditioned response (CR) with 2 distinctive peaks that

95 important component to the generation of the conditioned response (CR), a number of studies have sugg

96 essary for acquisition and expression of the conditioned response (CR), but loci of long-term memory

97 After extinction of the conditioned response (CR), half of the rats received pre

98 erm retention of the standard delay eyeblink conditioned response (CR).

99 with the US results in the emergence of the conditioned response (CR; eyeblink after CS presentation

100 Preterm subjects acquired significantly less conditioned responses (CR) compared to controls with slo

101 in rats, variation in the form of Pavlovian conditioned responses (CRs) and associated dopamine acti

102 nd an unconditioned stimulus did not acquire conditioned responses (CRs) and did not demonstrate any

103 HRs exhibited faster acquisition of eyeblink conditioned responses (CRs) and displayed mistimed (earl

104 re assessed during conditioning sessions for conditioned responses (CRs) and on separate test days fo

105 lesions on eyeblink (EB) and heart rate (HR) conditioned responses (CRs) in both delay and trace cond

106 tem plays a modulatory role in the timing of conditioned responses (CRs) in eyeblink classical condit

107 The percentage and amplitude of eye-blink conditioned responses (CRs) increased as a function of a

108 The performance of conditioned responses (CRs) is diminished when trained s

109 Rabbits acquire conditioned responses (CRs) normally with bilateral remo

110 conditioning sessions, but decreased it when conditioned responses (CRs) reached asymptotic values.

111 ts with amnesia produced significantly fewer conditioned responses (CRs) than did control participant

112 Concomitant heart rate (HR) conditioned responses (CRs) were also recorded.

113 Conditioned responses (CRs) were determined from the ele

114 Conditioned responses (CRs) were determined from the evo

115 of cerebellar deep nuclei abolished eyeblink conditioned responses (CRs) when the CS was either a ton

116 ompany drug administration may come to evoke conditioned responses (CRs), and these CRs may be the ba

117 jects during phase 2 prevented extinction of conditioned responses (CRs), shown by initial high CR fr

118 e receptors in the IO produces extinction of conditioned responses (CRs), suggesting that it blocks t

119 ission in the IN altered the time profile of conditioned responses (CRs), suggesting that the main fu

120 iven paired training acquired high levels of conditioned responses (CRs), which occurred in both eyel

121 hey reached a criterion of 10 consecutive EB conditioned responses (CRs).

122 ing task to a criterion of 10 consecutive EB conditioned responses (CRs).

123 intervals of 0 and 250 ms yielded well-timed conditioned responses (CRs); intervals of 500 ms or more

124 subjects revealed reduced activation for the conditioned response (CS+ > CS-) in the left inferior fr

125 ngs indicate that VTA glutamate release is a conditioned response dependent on an associative process

126 new and the extinction of previously learned conditioned responses depends on a similar set of cerebe

127 which emphasizes extinction deficits of fear-conditioned responses, does not fully consider the role

128 shock conditioning improved retention of the conditioned response during the subsequent 2-day period.

129 anisomycin does not block the expression of conditioned responses during conditioning or in well-tra

130 Muscimol infusions blocked the expression of conditioned responses during phase 2.

131 functioning was evaluated with the number of conditioned responses during the 6 blocks of EBCC and 1

132 Such conditioned responses (e.g. drug seeking) can be diminis

133 nicline would escalate if it also diminished conditioned responses elicited by alcohol-predictive cue

134 examined the effect of DCS on extinction of conditioned responses elicited by cues paired with admin

135 Eyeblink-conditioned responses established with pontine stimulati

136 t have plasticity such that the valence of a conditioned response evoked by their reactivation can be

137 rd and omission contingencies on 2 Pavlovian conditioned responses evoked by a visual conditioned sti

138 lved in response timing and other aspects of conditioned response execution.

139 ude of conditioning, indexed by differential conditioned response expression (conditioned SCR to CS+

140 stablished in the cerebellar cortex, whereas conditioned response expression begins later as plastici

141 s, subjects were trained to robust levels of conditioned response expression using a shorter ISI.

142 situs nucleus is necessary or permissive for conditioned response expression.

143 s the percentage difference between test and conditioned response for all intervals and was described

144 Descriptions of conditioned response generation in Hermissenda stipulate

145 change depending on the extent to which the conditioned response has been extinguished.

146 Activity related to the expression of conditioned responses, however, cannot be excluded.

147 tion was correlated across subjects with the conditioned response in both acquisition and early extin

148 ed effects on the rate of acquisition of the conditioned response in male vs. female rats.

149 the stressor facilitated acquisition of the conditioned response in males, whereas exposure to the s

150 aily 80-trial sessions to a criterion of 80% conditioned responses in a session.

151 aily 80 trial sessions to a criterion of 80% conditioned responses in a session.

152 bbits to reach a behavioral criterion of 60% conditioned responses in an 80 trial session.

153 t to play a modulating role in the timing of conditioned responses in classical trace conditioning.

154 ts with cerebellar lesions failed to acquire conditioned responses in four consecutive training sessi

155 Drug-associated cues elicit conditioned responses in human drug users, and are thoug

156 these inputs may be involved in driving the conditioned responses in LH orexin neurons.

157 c mice and control littermates showed larger conditioned responses in mutant mice that were associate

158 Furthermore, whereas onset conditioned responses in the control group remained elev

159 comparison subjects showed similar rates of conditioned responses in the delay paradigm, but patient

160 radigm, but patients showed reduced rates of conditioned responses in the trace paradigm.

161 The percentage and amplitude of eye-blink conditioned responses increased as a function of postnat

162 leus with muscimol infusions abolished these conditioned responses, indicating that cerebellar involv

163 drug infusions suppressed the expression of conditioned responses, indicating that the protein synth

164 , such as a conditioning context, to evoke a conditioned response is diminished.

165 r learning that is maintained even after the conditioned response is well learned.

166 her, the data demonstrate that extinction of conditioned responses is a dynamic process in which the

167 simulations the adaptive timing displayed by conditioned responses is mediated by two factors: (1) di

168 Although bees exposed to acute doses showed conditioned responses less frequently than controls, we

169 interstimulus interval, involve decreases in conditioned response magnitude and likelihood as well as

170 dicate that the development of the eye-blink conditioned response may depend on the development of st

171 st that the thalamus, a region involved with conditioned responses, may mediate the enhancement of th

172 re acquired, or about the specificity of the conditioned response modalities.

173 The conditioned responses of male and female Japanese quail

174 Conditioned responses of place cells were gated by their

175 nt inhibition (LI) refers to the decrease in conditioned response produced by the repeated nonrein-fo

176 fting to a new ISI had negligible effects on conditioned response rates in both groups.

177 on on Day 1, men showed significantly larger conditioned responses relative to women; early cycle and

178 reward association responses; however, their conditioned response robustness was drastically blunted.

179 ck and only if that lick were also part of a conditioned response sequence initiated earlier, consist

180 h GABA(A) antagonists produces short-latency conditioned responses (SLRs).

181 KTER is a unique type of naturally acquired conditioned response system which is maintained by avers

182 or exhibited significantly less retention of conditioned responses than rabbits injected with vehicle

183 those infused with the vehicle emitted more conditioned responses than unstressed males.

184 used with vehicle and stressed emitted fewer conditioned responses than unstressed vehicle controls.

185 llar activation with expectation may reflect conditioned responses that are not linked to conscious r

186 the morphological changes in the NAc encode conditioned responses that are sensitive to extinction a

187 ells, some auditory cortex cells showed late conditioned responses that seemed to anticipate the unco

188 formed between drug-associated cues and the conditioned responses they elicit.

189 onditioning, rats often acquire 2 classes of conditioned responses: those whose form is determined by

190 that the hippocampus is not specialized for conditioned response timing, but rather is a general-pur

191 demonstrated impaired learning and abnormal conditioned response timing.

192 whereas fear-potentiated startle reflects a conditioned response to a fear-eliciting stimulus.

193 ed extinction subjects showed an increase in conditioned response to stimuli from the previous day, b

194 stimulus retards subsequent acquisition of a conditioned response to that stimulus.

195 t affect either the initial acquisition of a conditioned response to the light CS in the first traini

196 bellar deep nuclei from inhibition, allowing conditioned responses to be elicited via the red nucleus

197 opamine release may also mediate some of the conditioned responses to cocaine cues.

198 and the LC, is mediated in part by Pavlovian conditioned responses to cues that predict ethanol admin

199 ion displays a reduced ability to extinguish conditioned responses to drug-associated stimuli.

200 ly reported), but also in updating Pavlovian-conditioned responses to morphine-associated stimuli.

201 lthy nondependent volunteers readily acquire conditioned responses to neutral stimuli paired with a d

202 e-dependent individuals, possibly reflecting conditioned responses to noningestive sources of infecti

203 conditioned response to cocaine itself and a conditioned response triggered by cocaine-predictive cue

204 g Pavlovian conditioning the expression of a conditioned response typically serves as evidence that a

205 ent from the mean size for trials in which a conditioned response was absent.

206 The conditioned response was measured as the fear-potentiati

207 However, the amplitude of the conditioned response was notably reduced in the tetracai

208 d AP5 infusions for the first time after the conditioned response was well learned showed temporary a

209 nent at the first time the maximum number of conditioned responses was achieved.

210 e discrimination phase, acquisition of the 2 conditioned responses was not significantly different; h

211 Given the relatively long latency of conditioned responses we observed in PL (approximately 1

212 uronal activity correlated with the eyeblink conditioned response were evident in the cerebellum befo

213 In subjects who received cerebellar cTBS, conditioned responses were fewer and their onsets were e

214 blink responses as early as P12, and by P18, conditioned responses were fully developed in all animal

215 econds after the modulation of ILD, and such conditioned responses were influenced by the modulation

216 Test and conditioned responses were recorded from flexor carpi ra

217 s observed regardless of the expression of a conditioned response when mice were trained using an unp

218 decrease in the amplitude and frequency of a conditioned response when the conditioned stimulus that

219 dizocilpine had no effect on acquisition of conditioned responses when given alone.

220 sensory cortex and to support trace-eyeblink conditioned responses when paired with corneal airpuff u

221 MDA receptor antagonist had little effect on conditioned responses when these same rabbits were infus

222 e climbing fibres prevents extinction of the conditioned response, whereas blocking excitatory input

223 or response whose form resembles that of the conditioned response which develops after the stimuli ar

224 her explicit cues or contexts, evoke anxiety conditioned responses, which are dissociable from fear r

225 S-US presentations led to the development of conditioned responses, which showed extinction following

226 l lesions diminished the magnitude of the HR-conditioned response without affecting the cardiac-orien