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1 ves intact the development of a LiCl-induced conditioned taste aversion.
2 itched from "good" to "bad" as occurs during conditioned taste aversion.
3 on corticosterone secretion, body weight, or conditioned taste aversion.
4 or memory, since they successfully expressed conditioned taste aversion.
5 rtical lesions had no impact on LiCl-induced conditioned taste aversion.
6 ecreases food intake in rats without causing conditioned taste aversion.
7 epting or avoiding taste stimuli following a conditioned taste aversion.
8 inducing agent used in laboratory studies of conditioned taste aversion.
9 sed with respect to the neural substrates of conditioned taste aversion.
10 e acquisition of latent inhibition (LI) of a conditioned taste aversion.
11 endocrine responses including development of conditioned taste aversion.
12 t due to sickness as icv TTR did not cause a conditioned taste aversion.
13 2, or 4 g/kg) aversive effects as indexed by conditioned taste aversion.
14 duce food intake, induce pica, and produce a conditioned taste aversion.
15 rs, increased feeding and the formation of a conditioned taste aversion.
16 finds as evidence that drugs of abuse induce conditioned taste aversions.
17 depletion, induces pica, and produces robust conditioned taste aversions.
20 conditioning, nociception, and extinction of conditioned taste aversion and an appetitive instrumenta
21 e is a correlate or cause of meal satiation, conditioned taste aversion and aversive brain stimulatio
22 C75 administration in two models of illness, conditioned taste aversion and need-induced sodium appet
24 ar cortex blocked behavioral expression of a conditioned taste aversion and this was evident not only
25 ion rate of conditioned fear responses and a conditioned taste aversion as well as enhanced performan
27 levels and enhanced novel taste learning and conditioned taste aversion, but not memory retrieval.
32 4 T magnetic field is sufficient to induce a conditioned taste aversion (CTA) and induce brainstem ex
33 ite following sodium depletion, and a normal conditioned taste aversion (CTA) for alanine when paired
34 basolateral complex (BLA) of the amygdala on conditioned taste aversion (CTA) in a latent inhibition
37 rsion-associated brain areas and to induce a conditioned taste aversion (CTA) in these strains is unk
38 rabrachial nucleus (PBN) failed to acquire a conditioned taste aversion (CTA) induced by lithium chlo
47 ar cortex, is widely regarded as integral to conditioned taste aversion (CTA) retention, a link that
51 nol-induced (1.5 g kg(-1) 20% ethanol, i.p.) conditioned taste aversion (CTA) to saccharin taste.
54 linergic agonist, was infused into IC before conditioned taste aversion (CTA) training with a familia
55 tently as a correlate of the expression of a conditioned taste aversion (CTA) when conditioning occur
56 Paraquat is a herbicide capable of eliciting conditioned taste aversion (CTA), a behavioral response
57 both strains readily acquired a LiCl-induced conditioned taste aversion (CTA), the suppressive effect
72 ], low drinking [STDRLO]) or ethanol-induced conditioned taste aversion (CTA; high [HTA], low [LTA]).
74 t such as lithium chloride (referred to as a conditioned taste aversion, CTA); (2) access to a very p
75 he effects of permanent forebrain lesions on conditioned taste aversions (CTAs) and conditioned odor
76 postrema (AP) lesions attenuate LiCl-induced conditioned taste aversions (CTAs) by disruption of info
77 ral role in the acquisition and retention of conditioned taste aversions (CTAs) in rodents, but large
79 nsected chorda tympani nerves (CTX) received conditioned taste aversions (CTAs) to the free fatty aci
82 acterized the acquisition of ethanol-induced conditioned taste aversion, ethanol-induced conditioned
83 ehaviours, including active place avoidance, conditioned taste aversion, fear conditioning and spatia
85 ve induces locomotor circling and leads to a conditioned taste aversion if paired with a novel taste.
86 ith a saccharin taste developed a persistent conditioned taste aversion in both preference and taste
87 ering food intake, body weight, or causing a conditioned taste aversion in mice lacking neuronal GLP1
91 vidly were trained first to reject it (i.e., conditioned taste aversion learning) and then to enjoy i
92 high EtOH withdrawal with low drinking, high conditioned taste aversion, low tolerance to EtOH-induce
94 srupts spatial memory in the hippocampus and conditioned taste aversion memory in the insular cortex.
95 nsumption with LiCl injection, by making the conditioned taste aversion more resistant to extinction.
96 instrumental training procedures and either conditioned taste aversion or satiation devaluation proc
97 e neuronal substrates of LI as assessed in a conditioned taste aversion paradigm by comparing regiona
100 reas lithium chloride, at doses that produce conditioned taste aversion, reduced metabolic rate.
101 and show a delay in extinguishing an ethanol-conditioned taste aversion, suggesting that they drink l
102 nsitivity (experiment 4) or consumption on a conditioned taste aversion test that does not elicit ant
105 pani nerve transection (CTX) acquired a LiCl-conditioned taste aversion to 0.1 M NaCl at the same rat
106 core administration of GLP-1 did not cause a conditioned taste aversion to saccharin, suggesting that
107 of an ingestate was confirmed in rats with a conditioned taste aversion to sucrose: after paired expo
109 ar cortex in acquisition and expression of a conditioned taste aversion was assessed using two differ
110 However, LiCl can induce memory given that a conditioned taste aversion was obtained for a novel tast
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