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1 g networks are specifically dysfunctional in conduct disorder.
2 hildren (ages 6 to 12) with ADHD but without conduct disorder.
3 ion to comparison subjects and patients with conduct disorder.
4 ng MAO-A contributes to the genetic risk for conduct disorder.
5 y but not of monoamine oxidase A on risk for conduct disorder.
6 tly associated with offspring depression and conduct disorder.
7 e/dependence, adult antisocial behavior, and conduct disorder.
8 e behavior satisfied the DSM-IV criteria for conduct disorder.
9 disorder independent of its association with conduct disorder.
10 (ADHD) typically occurred first, followed by conduct disorder.
11 often preceded by childhood-onset aggressive conduct disorder.
12 primarily confined to those with persistent conduct disorder.
13 years and who met the DSM-III-R criteria for conduct disorder.
14 of mothers that is of etiologic significance conduct disorder.
15 ance use disorders than in the patients with conduct disorder.
16 th conduct disorder than in patients without conduct disorder.
17 r (ADHD), oppositional defiant disorder, and conduct disorder.
18 orders include depression, anxiety, ADHD and conduct disorder.
19 ol was also more pronounced in patients with conduct disorder.
20 n the basis of violence and did not focus on conduct disorder.
21 lent schizophrenia patients with and without conduct disorder.
22 apine in violent schizophrenia patients with conduct disorder.
23 lenging problem, especially in patients with conduct disorder.
24 zophrenia, bipolar disorder, depression, and conduct disorder.
25 specially enriched in children with comorbid conduct disorder.
26 order relative to ADHD case subjects without conduct disorder.
27 ernal smoking during pregnancy and offspring conduct disorder.
28 jects and relative to those without comorbid conduct disorder.
29 13.3 appeared to be associated with comorbid conduct disorder.
30 on deficit hyperactivity disorder (ADHD) and conduct disorder.
31 e of adolescent-onset as well as early-onset conduct disorder.
32 tigating brain structure in both subtypes of conduct disorder.
33 major diagnostic classification systems for conduct disorders.
34 .5% vs 10.9%; OR, 0.6 [95% CI, 0.3-0.9]) and conduct disorder (0.3% vs 2.9%; OR, 0.1 [95% CI, 0.0-0.4
35 (10-15) for disruptive, impulse-control, and conduct disorders; 11% (9-13) for depressive disorders;
36 brain activation of 13 boys with noncomorbid conduct disorder, 20 with noncomorbid ADHD, and 20 norma
37 1% +/- 1.0%), tic disorders (4.2% +/- 0.9%), conduct disorder (3.3% +/- 0.6%), oppositional defiant d
38 sm, self-esteem, and early-onset anxiety and conduct disorder), 3) late adolescence (educational atta
39 .2% vs 1.9%; OR, 3.4 [95% CI, 1.6-7.4]), and conduct disorder (6.4% vs 2.1%; OR, 3.1 [95% CI, 1.7-5.8
40 al defiant disorder (97.3 [0.66]; P = .007), conduct disorder (97.1 [0.82]; P = .02), substance use d
43 g association between affective disorder and conduct disorder; adolescents with mania had much higher
44 ts of four parental externalizing disorders (conduct disorder, adult antisocial behavior, alcohol dep
46 that alcohol and drug dependence, childhood conduct disorder, adult antisocial behavior, and disinhi
47 od disorders, substance abuse disorders, and conduct disorder, age at the birth of the first child, a
48 cific vulnerabilities were also detected for conduct disorder, alcohol dependence, and drug dependenc
49 occurring major depression, suicide attempt, conduct disorder, alcohol dependence, nicotine dependenc
51 treatment for aggression in inpatients with conduct disorder, although its use is associated with ad
52 ith increased rates of depression, PTSD, and conduct disorder among boys and reduced rates of depress
54 ontrast, independent of its association with conduct disorder, an ADHD diagnosis did not significantl
55 embership in high-symptom trajectories of 1) conduct disorder and 2) hyperactivity throughout childho
58 that are associated with the development of conduct disorder and affect the quality of parenting.
61 showed little association with AAS use, but conduct disorder and body-image concerns showed strong a
63 ention in children and adolescents with pure conduct disorder and children and adolescents with pure
65 nalizing problems at age 11, and had greater conduct disorder and excessive motor activity at age 17.
67 a meaningful clinical entity independent of conduct disorder and highlight the extremely detrimental
69 association between prenatal stress and both conduct disorder and hyperactivity, after adjusting for
71 arly-onset (N=36) or adolescent-onset (N=27) conduct disorder and in healthy comparison subjects (N=2
72 magnetic resonance imaging (fMRI) studies in conduct disorder and in oppositional defiant disorder ha
73 has been little previous distinction between conduct disorder and ODD in studies of genetic and envir
74 hs with disruptive behavior disorders (DBD) (conduct disorder and oppositional defiant disorder) have
75 ith disruptive behavior disorders, including conduct disorder and oppositional defiant disorder, show
76 antly later than the onset of either ADHD or conduct disorder and significantly later than the first
77 th decreased syndrome consequences, comorbid conduct disorder and social phobia, higher interpersonal
79 The authors examined the association between conduct disorder and substance use disorders in adolesce
81 tween medication-naive adolescents with pure conduct disorder and those with pure ADHD to identify bi
82 mbic motivation networks in individuals with conduct disorder and, in contrast, ventrolateral fronto-
83 irritability, oppositional defiant behavior, conduct disorders and attention deficit hyperactivity di
85 iours influence the development of childhood conduct disorders and that behavioural family interventi
86 EBF was associated with fewer than average conduct disorders and weakly associated with improved co
87 rders, antisocial personality disorder (from conduct disorder), and anxiety disorders, although this
88 or disorders (oppositional defiant disorder, conduct disorder), and substance use disorders (alcohol
89 r depressive episode, 146 [22.9%] of 638 for conduct disorder, and 102 [15.9%] of 641 for alcohol dep
90 or depressive episode, 43 [27.0%] of 159 for conduct disorder, and 41 [25.6%] of 160 for alcohol depe
92 ve disorder, post-traumatic stress disorder, conduct disorder, and attention-deficit hyperactivity di
93 djusted odds ratios for depression, anxiety, conduct disorder, and autism were 1.81 (95% CI, 1.33-2.4
96 d to assess the independent effects of ADHD, conduct disorder, and gender on current substance use, f
97 ntion deficit hyperactivity disorder (ADHD), conduct disorder, and gender to substance abuse were stu
99 umatic stress disorder, depressive disorder, conduct disorder, and neurodevelopmental disorder) limit
100 at less risk for comorbid major depression, conduct disorder, and oppositional defiant disorder than
101 s (attention deficit hyperactivity disorder, conduct disorder, and oppositional defiant disorder).
102 , aggression, oppositional defiant disorder, conduct disorder, and psychopathy than their normally he
103 Comorbid oppositional defiant disorder, conduct disorder, and substance use disorder increased t
104 ividuals with oppositional defiant disorder, conduct disorder, and substance use disorder, ADHD remai
106 en attention-deficit/hyperactivity disorder, conduct disorders, and antisocial personality disorders
107 about the causes of antisocial behaviour and conduct disorders, and now there are effective intervent
108 ity disorder, oppositional defiant disorder, conduct disorder, antisocial personality disorder, alcoh
109 traits that culminate in later diagnoses of conduct disorder, antisocial personality disorder, and p
110 of attention deficit hyperactivity disorder, conduct disorder, antisocial personality, or substance u
111 A subgroup of children and adolescents with conduct disorder are characterized by severe and persist
112 on deficit hyperactivity disorder (ADHD) and conduct disorder are often comorbid and overlap clinical
113 t conduct disorder, whereas adolescent-onset conduct disorder arises as a result of social mimicry of
114 sorder, intermittent explosive disorder, and conduct disorder, as assessed post hoc with a validated
115 t-risk group of ninth-graders diagnosed with conduct disorder, as well as self-reported delinquency.
116 (e.g., gambling, number of sexual partners, conduct disorder at 7 years, and parental socioeconomic
117 ive disorder, oppositional defiant disorder, conduct disorder, attention deficit hyperactivity disord
118 ncreased risk for psychopathology, including conduct disorder, attention deficit hyperactivity disord
119 ings were not explained by symptom levels of conduct disorder, attention-deficit hyperactivity disord
120 n remained clearly observable when childhood conduct disorder, attention-deficit/hyperactivity disord
121 milestones, disorders of the acoustic nerve, conduct disorders, attention-deficit/hyperactivity disor
122 ry for hyperactivity (B = 0.46, p < .05) and conduct disorder (B = 0.88, p < .01), respectively.
124 o have increased risk of substance abuse and conduct disorder but not other disorders, increased use
125 ymptoms of oppositional defiant disorder and conduct disorder can be validly applied to preschoolers.
126 included male sex, tobacco use, depression, conduct disorder, cannabis use, having peers exhibiting
127 Little is known about the prevalence of Conduct Disorder (CD) and symptoms of CD in high risk ps
128 on-Deficit/Hyperactivity Disorder (ADHD) and conduct disorder (CD) exemplify top-down dysregulation c
130 tional magnetic resonance imaging studies of conduct disorder (CD) have mostly been limited to males.
137 of the etiologic influences contributing to conduct disorder (CD) symptoms as defined in the DSM.
138 ntion deficit hyperactivity disorder (ADHD), conduct disorder (CD), antisocial personality disorder (
139 inattention and hyperactivity-impulsivity), conduct disorder (CD), depression/dysthymia, and general
142 n deficit/hyperactivity disorders (ADHD) and conduct disorders (CD) between non-Hispanic White and no
144 r time or relates to comorbid disorders (eg, conduct disorder [CD], oppositional defiant disorder [OD
148 ty, such as oppositional defiant disorder or conduct disorder, criminality, accidents, and substance
150 l variables specifically related to parental conduct disorder (d = 0.13; 95% confidence interval, 0.0
151 logical evidence suggests that children with conduct disorder demonstrate more prominent motivational
152 ty of comorbid psychiatric disorders such as conduct disorder, depression, mania, anxiety, and learni
155 DSM-IV symptoms of oppositional defiant and conduct disorders distinguish referred from nonreferred
156 me diagnoses of major depression, dysthymia, conduct disorder, drug abuse, and cigarette smoking.
157 eater impact in men: childhood sexual abuse, conduct disorder, drug abuse, prior history of major dep
158 emotions" specifier within the diagnosis of conduct disorder (DSM-5) and of conduct-dissocial disord
159 dysfunction demonstrated among children with conduct disorder during sustained attention, support the
161 were more strongly associated with risk for conduct disorder, early and heavy alcohol use, and low e
162 t youth, generally had a lower prevalence of conduct disorder (eg, age 6-12 years: first-generation i
163 attention-deficit/hyperactivity disorder and conduct disorder (eg, HR, 2.27 [95% CI, 1.74-2.96] for m
164 s of antisocial behavior and rates of DSM-IV conduct disorder, even after the authors controlled for
165 each childhood disruptive disorder, but only conduct disorder exhibited a significant shared environm
169 cely studied, while ODD (in combination with conduct disorder) has been associated with structural ab
170 ing behavior (aggression, hyperactivity, and conduct disorder) has been increasingly viewed as a publ
172 l health (high externalizing, high childhood conduct disorder, high fear somaticism), neurocognitive
175 als and 431 female individuals) but not with conduct disorder in a substance-dependent sample (950 ma
176 t that attention to preschool depression and conduct disorder in addition to maternal history of depr
181 DSM-based oppositional defiant disorder and conduct disorder in preschool children, and outline an a
182 symptoms of DSM-IV oppositional defiant and conduct disorders in 2.5-5.5-year-old children who were
183 ls were adjusted for conduct disorder, since conduct disorder is a potent predictor of subsequent sub
187 e DSM-IV's textual claim that a diagnosis of conduct disorder is valid only when symptoms are due to
188 esult in lower violence; in patients without conduct disorder, it was three times more likely to do s
189 y-adolescence (self-esteem, social deviance, conduct disorder), late-adolescence (education, personal
190 rates of depression or anxiety, frequency of conduct disorders, lifetime drug use, or health care uti
191 mbination of parent and youth informants for conduct disorder, major depression, generalized anxiety
192 mbination of parent and youth informants for conduct disorder,major depression, generalized anxiety d
193 sk of substance abuse among adolescents with conduct disorder may be primarily confined to those with
194 r disorder (oppositional defiant disorder or conduct disorder) met the specific criteria for explosiv
196 patients, ages 9-16 years, with noncomorbid conduct disorder (N=14) and noncomorbid ADHD, combined h
197 iant disorder alone (N=643) or with comorbid conduct disorder (N=262) and a psychiatric comparison gr
199 anxiety disorders, oppositional defiant and conduct disorders (ODD/CD), and attention deficit/hypera
200 gnificantly more likely to have a child with conduct disorder (odds ratio, 4.4; P = .001) than mother
201 ender differences in the effects of ADHD and conduct disorder on substance use and abuse, although th
203 oamine oxidase A activity increased risk for conduct disorder only in the presence of an adverse chil
204 ng attention-deficit/hyperactivity disorder, conduct disorder, oppositional defiant disorder, antisoc
206 12 had callous-unemotional traits and either conduct disorder or oppositional defiant disorder, 12 ha
207 ty disorder (OR, 5.19 [95% CI, 2.01-13.38]), conduct disorder (OR, 2.03 [95% CI, 1.03-3.99]), and mar
209 xisting substance use disorder is suggested; conduct disorder patients without a coexisting substance
212 er levels of impulsivity among patients with conduct disorder, personality disorders, substance use d
214 , and oppositional defiant disorder (but not conduct disorder) predicted later anxiety disorders and
215 ty) and externalizing (ie, hyperactivity and conduct disorder) problem behaviors, positive behaviors
217 attention deficit hyperactivity disorder and conduct disorder), psychosis-spectrum symptoms, and fear
218 e Measure DSM-IV symptoms of childhood-onset conduct disorder rated by trained interviewers using a s
220 eripheral nervous system and special senses, conduct disorder, receptive language disorder, chronic s
222 om conducting ordered regions through poorly conducting disordered regions, consistent with what has
223 nificantly higher in ADHD case subjects with conduct disorder relative to ADHD case subjects without
224 r scores in ADHD case subjects with comorbid conduct disorder relative to comparison subjects and rel
225 tronger association between maltreatment and conduct disorder severity in patients carrying the low M
227 ext of the DSM-IV states that a diagnosis of conduct disorder should be made only if symptoms are cau
228 th both early- and adolescent-onset forms of conduct disorder show impaired emotional learning and ab
231 reward condition, patients with noncomorbid conduct disorder showed disorder-specific underactivatio
233 disorder (ADHD), including comorbidity with conduct disorder, showing frontal-striatal dysfunction.
236 omparisons between healthy subjects and each conduct disorder subgroup revealed lower amygdala volume
238 inal and family-based research suggests that conduct disorder, substance misuse, and ADHD involve bot
239 eritable, age- and sex-normed indexes of DV, conduct disorder symptoms (CDS), and a composite index o
241 tly higher rates of oppositional defiant and conduct disorder symptoms than nonreferred children.
243 Although level of maltreatment and lifetime conduct disorder symptoms were significantly correlated,
244 ity ADHD symptoms, oppositional defiant, and conduct disorder symptoms) and ADHD medication treatment
245 r-rated), maternal smoking during pregnancy, conduct disorder symptoms, and family adversity were ass
247 ete trios had more hyperactive-impulsive and conduct disorder symptoms, lower IQ, and lower socioecon
250 bstantially more pronounced in patients with conduct disorder than in patients without conduct disord
251 e/dependence, adult antisocial behavior, and conduct disorder; the second, on major depression, gener
252 This study investigated the progression from conduct disorder to antisocial personality disorder amon
254 Disruptive behavior diagnoses, particularly conduct disorder, typically precede the initiation of us
258 patterns of psychopathology with and without conduct disorder were at risk of later criminality.
259 ional defiant disorder youth with or without conduct disorder were found to have significantly higher
260 fiant disorder alone and those with comorbid conduct disorder were seen primarily in rates of mood di
262 line/valine homozygotes had more symptoms of conduct disorder, were more aggressive, and were more li
263 ticularly those with a history of adolescent conduct disorder, were more likely to be depressed in yo
264 critical role in the etiology of early-onset conduct disorder, whereas adolescent-onset conduct disor
265 sttraumatic stress disorder had histories of conduct disorder, whereas those with other anxiety disor
266 U) traits designate a subgroup of youth with conduct disorders who have unique causal processes under