ライフサイエンスコーパス: conduction block

1 ropathy, are defined by the demonstration of conduction block.

2 ns, a setting which is particularly prone to conduction block.

3 asystoles, and thus enhance vulnerability to conduction block.

4 trical properties, culminating in paroxysmal conduction block.

5 ms around newly formed sites of intra-atrial conduction block.

6 areas of low voltage, double potentials and conduction block.

7 rkinson-White syndrome) and atrioventricular conduction block.

8 tations that result in atrioventricular (AV) conduction block.

9 scles, following recovery from chronic nerve conduction block.

10 l radiofrequency lesions can create lines of conduction block.

11 riability, sinus pause, and atrioventricular conduction block.

12 rocess possibly involving presynaptic axonal conduction block.

13 se patients was also associated with isthmus conduction block.

14 lting in premature activation of the CCP and conduction block.

15 e intrinsic AV conduction or intermittent AV conduction block.

16 iated with decreased conduction velocity and conduction block.

17 s ablated by achieving bidirectional isthmus conduction block.

18 istinguish slow conduction from complete CTI conduction block.

19 tients with multifocal motor neuropathy with conduction block.

20 further reduced theta', accentuating this RV conduction block.

21 those with multifocal motor neuropathy with conduction block.

22 h scar was seen in 2 specimens from PVs with conduction block.

23 spontaneous cardiac arrhythmias and complete conduction block.

24 l heterogeneity, increasing vulnerability to conduction block.

25 predisposed the heart to the development of conduction block.

26 y multifocal motor neuropathy has only motor conduction block.

27 ntractures and prominent cardiomyopathy with conduction block.

28 eentrant arrhythmias by producing reversible conduction blocks.

29 mpathetic branches, respectively, eliminated conduction blocks.

30 ions in the mild hypothermia group developed conduction block (0/6).

31 rdia (25 cases), ictal atrioventricular (AV)-conduction block (11 cases), postictal AV-conduction blo

32 V)-conduction block (11 cases), postictal AV-conduction block (2 cases), (post)ictal atrial flutter/a

33 ogressive P-R and Q-T interval prolongation, conduction blocks, 2-fold prolongation of the ventricula

34 s, (4) less frequent temporal dispersion and conduction block, (5) no sural sparing, (6) greater numb

35 donor cells, the vulnerable time window for conduction block across the host-donor interface most st

36 Epicardial mapping demonstrated complete conduction block across the lesions in all animals, with

37 ncy catheter ablation is to create a line of conduction block across this isthmus.

38 A novel criterion for lateral conduction block after catheter ablation is identificati

39 Of 28 episodes, 20 were initiated by conduction block along large PM ridges, leading to wave

40 ion may also contribute significantly to the conduction block, although the mechanisms are not unders

41 Atrioventricular conduction block and arrhythmias caused by sinoatrial no

42 n of repolarization, resulting in functional conduction block and circulating wave fronts.

43 autoantibody depletion and with recovery of conduction block and distal motor amplitude suggesting a

44 at infringed on TDR, resulting in functional conduction block and reentrant excitation.

45 ity of repolarization, and susceptibility to conduction block and reentrant PVT.

46 This could result in functional conduction block and reentrant ventricular tachyarrhythm

47 pulmonary veins and left atrium, the site of conduction block and reentry gradually shifted, as a fun

48 including discordant alternans, which caused conduction block and reentry only for the elevated I(Ca,

49 en more effective at inducing unidirectional conduction block and reentry than a single extrasystole.

50 arization that were directly responsible for conduction block and self-sustained intramural reentrant

51 ization of AF should promote regional atrial conduction block and terminate AF.

52 e development of de novo zones of functional conduction block and/or slowed conduction to create the

53 n combined with cardiomyopathy manifested by conduction blocks and arrhythmias resulting in premature

54 ested by atropine-sensitive atrioventricular conduction blocks and bradycardia that may culminate in

55 ENP2-deficient mice develop atrioventricular conduction blocks and cardiac asystole.

56 , enough under certain conditions to produce conduction blocks and initiate reentrant waves.

57 Frequent intra-PV conduction blocks and multiple wave fronts in the PVs we

58 egments, less common temporal dispersion and conduction block, and absent sural sparing.

59 explain the link between altered metabolism, conduction block, and arrhythmic risk.

60 including bradycardia and rate variability, conduction block, and blunted response to adrenergic sti

61 inantly motor, demyelinating neuropathy with conduction block, and chronic hemolysis attributed to p.

62 quantify acute inflammation, demyelination, conduction block, and later to estimate remyelination an

63 paravalvular regurgitation, atrioventricular conduction block, and mitral or coronary compromise.

64 Nerve electrophysiology revealed conduction block, and neuromuscular junctions showed mar

65 ntricular fibrillation (VF) activation rate, conduction block, and organization transmurally in pigs

66 ed in the BZ with rate-dependent slowing and conduction block, and reentry was demonstrated in one su

67 inantly motor, demyelinating neuropathy with conduction block, and secondary axonal damage attributed

68 etrograde propagation, wavefront collisions, conduction blocks, and re-entry), operating across brady

69 ers, particularly long lines of longitudinal conduction block, are more pronounced in patients with A

70 etal muscle weakness associated with cardiac conduction blocks, arrhythmias and restrictive heart fai

71 normal conduction, depressed conduction, and conduction block as [K+]o was gradually increased from 4

72 a fivefold increase in atrioventricular (AV) conduction blocks, as well as bradycardia and premature

73 dentified 45 published cases of third-degree conduction block associated with Lyme carditis in the Un

74 dn-PI3K-2Tg; DCM-2Tg) develop severe DCM and conduction block, associated with increased expression o

75 he effective refractory period and abolished conduction block at short cycle lengths (7 experiments).

76 Seven of the 22 specimens (32%) showed conduction block at surgery.

77 Distal LSPV pacing resulted in conduction block at the anterior PV-LA junction, with do

78 a result of an intermittent Wenckebach-like conduction block at the domain boundaries.

79 Activation sequence maps demonstrated conduction block at the lateral tunnel suture line, whic

80 r these deficits is loss of myelin, creating conduction block at the site of injury.

81 cemaker (PPM) placement for atrioventricular conduction block (AV block) after operative repair of pe

82 ease (ICCD) giving rise to atrio-ventricular conduction block (AVB), right bundle branch block, brady

83 ifurcation was the development of functional conduction block between the anterior or posterior right

84 achian valve and ridge (EVR) forms a line of conduction block between the IVC and coronary sinus (CS)

85 (1) The EVR forms a line of fixed conduction block between the IVC and the CS; (2) the EVR

86 ess required formation of a complete line of conduction block between the TA and the EVR, identified

87 se data support the theory that some form of conduction block between the vena cava is essential for

88 emature stimulus could induce unidirectional conduction block but only halothane reduced the critical

89 Acute amiodarone decreased conduction block by 22%, while chronic amiodarone increa

90 rol the vulnerable window for unidirectional conduction block by a single premature extrasystole.

91 ion to influence the "vulnerable window" for conduction block by subsequent extrasystoles, particular

92 al gradient in action potential duration for conduction block can be analytically derived, and once t

93 Both seizures and cardiac conduction blocks can be prevented by retigabine, a Kv7

94 ion with elevated K+, indicating that axonal conduction block cannot account for our observations.

95 reentry by encountering functional lines of conduction block caused by enhanced dispersion of refrac

96 arity points in AP phase movies and sites of conduction block (CB) as sites where an AP wavefront fai

97 cal deficits are due to mechanically induced conduction block (CB).

98 nduction block (CB2), transition from type I conduction block (CB1) to CB2, and unstable nodes.

99 large tau, we observed formations of type II conduction block (CB2), transition from type I conductio

100 Intranodal longitudinal conduction blocks coincided with interstitial fibrosis s

101 The conduction block corresponded to segmental PV-LA muscle

102 Conduction blocks crossing the core region, but not reac

103 with advantages that include confirmation of conduction block, decreased surgical morbidity, and poss

104 round a variably complete line of functional conduction block, descending on the posterior wall from

105 defects and result in atrioventricular (AV) conduction block during postnatal life.

106 of waveform variability, evidence of axonal conduction block, evidence of axonal conduction variabil

107 Evidence for frequency-dependent conduction block (FDCB) in the patients' central motor p

108 6 atrial preparations demonstrated sustained conduction block from a single ablation line.

109 Longitudinal lines of conduction block >10 mm were also associated with postop

110 A high amount of conduction block (>4%) was associated with de novo posto

111 Understanding of the pathophysiology of conduction block has evolved with better understanding o

112 had atrial fibrillation (AF) or interatrial conduction block (IAB) (P wave duration, >or=120 msec),

113 isthmus (11 of 20 patients) and 2:1 isthmus conduction block immediately preceding AF (4 of 20 patie

114 ave previously reported can cause reversible conduction block in axons.

115 report that such compounds cause reversible conduction block in both normal and demyelinated axons o

116 reflect the reversal of compression-induced conduction block in larger myelinated fibres outside the

117 ers in cowhage-induced itch: (1) a selective conduction block in myelinated fibers substantially redu

118 ntially, the vulnerability to unidirectional conduction block in response to a premature stimulus inc

119 lower SA conduction and frequent sino-atrial conduction block in Scn5a+/- SAN preparations.

120 nide (0.1 micromol/L) produced bidirectional conduction block in straight and bifurcated gap preparat

121 t the outer border of the arcs of functional conduction block in the "normal" myocardial zone and at

122 multiple PV-LA breakthroughs, with segmental conduction block in the anterior PV-LA junction.

123 st within 30 s), coincident with anterograde conduction block in the AV node.

124 iated with slowing of conduction and finally conduction block in the CCP.

125 At rapid pacing, APD maps show areas of conduction block in the failing heart.

126 +/- SD) doses of adenosine required to cause conduction block in the fast and slow pathways were 2.7

127 pathway, and progressively moved the site of conduction block in the inferior pathway proximally, thu

128 We sought to 1) correlate conduction block in the isthmus of the right atrium betw

129 After a single ablation procedure, conduction block in the lateral wall was verified in 10

130 distal generation of fasciculations, axonal conduction block in the motor unit arborization and of v

131 Linear ablation resulting in bidirectional conduction block in the TA-ER isthmus terminated spontan

132 stantial decrease in neural excitability and conduction block in vagal afferent nerves.

133 ability to precisely define reversible local conduction blocks in beating cultures without influencin

134 n conclusion, estimated activation rates and conduction block incidence during VF are not uniformly d

135 During seizures the occurrence of AV conduction blocks increased, predisposing Kv1.1-deficien

136 red separately, only atropine ameliorated AV conduction blocks, indicating that excessive parasympath

137 ted, all with evidence of distal focal motor conduction block involving the median nerve in the forea

138 ardia cycle length converged on the lines of conduction block irrespective of the stimulation site, a

139 Atrioventricular conduction block is a complication of operative repair o

140 The time to achieve conduction block is a function of the temperature set po

141 Isthmus conduction block is associated with flutter ablation suc

142 Moreover, the critical gradient for conduction block is higher for an extrasystole traveling

143 random coupling intervals, vulnerability to conduction block is proportional to their number.

144 Since the substrate for conduction block is spatial dispersion of refractoriness

145 Third-degree conduction block is the most severe form and can be fata

146 Complete AV conduction block is typically treated by implanting an e

147 ly responsible for development of functional conduction block, leading to PVT in 63% of HF wedges but

148 Increased fibrosis provides a site for conduction block, leading to the continuous generation o

149 kalemia local excitability, causing extended conduction block lines and slowed conduction in the isch

150 Before achieving conduction block, marked conduction slowing or intermitt

151 Conduction block may be both antiarrhythmic and proarrhy

152 ations of cellular electrical properties and conduction block may provide a generic mechanism for the

153 rmal conduction (7/10; reduced velocities or conduction blocks; median, 2.9 cycles/min; range, 2.1-3.

154 (CIDP) and multifocal motor neuropathy with conduction block (MMNCB).

155 e compounds was confirmed in a mouse cardiac conduction block model of arrhythmia.

156 This example of axo-axonic conduction block--neurons in one pathway inhibiting the

157 Unidirectional conduction block occurred at the junction between the AV

158 on, excitability was maintained by INa, with conduction block occurring at 95% INa inactivation.

159 arge dispersion of refractoriness leading to conduction block of a subsequent extrasystole.

160 PPI mechanism in Tritonia: prepulse-elicited conduction block of action potentials traveling in the s

161 ising from the border zone or unidirectional conduction block of paced beats.

162 Unidirectional conduction block of premature extrasystoles can lead to

163 Exit conduction block of triggered responses occurred to vary

164 precisely targeted by light to induce local conduction blocks of circular or linear shapes.

165 and that they do so, in part, by overcoming conduction block or by activating silent synapses.

166 asing neurotransmission by overcoming axonal conduction block or by increasing transmitter release.

167 ients had a severe phenotype associated with conduction block or decreased distal motor amplitude.

168 Adenosine induced SAN intranodal conduction block or microreentry in 6 of 8 dogs with HF

169 mal model of CMT, whereby weakness is due to conduction block or neuromuscular junction failure rathe

170 rhythm including temporary atrioventricular conduction block or tachycardia termination.

171 ssociated with the development of intramural conduction block or with significant changes in the acti

172 disease (sinus bradycardia, atrioventricular conduction block, or atrial arrhythmias) and dilated car

173 Energy was delivered until evidence of conduction block, or for a maximum of 10 seconds.

174 icardial breakthrough, followed by a line of conduction block parallel to the epicardial fiber orient

175 d to action potential duration alternans and conduction block preceding the transition to AF.

176 These observations show that unidirectional conduction block provides a peripheral mechanism of temp

177 ment of complete cavotricuspid isthmus (CTI) conduction block reduces typical atrial flutter recurren

178 How alternans causes the local conduction block required for initiation of spiral wave

179 visualize tissue destruction, by reversible conduction block resulting from edema surrounding lesion

180 ptly broke across a stable arc of functional conduction block, resulting in premature activation of t

181 and low-temperature tests produce reversible conduction block, suggesting minimal permanent injury.

182 esions are noted in some PVs with persistent conduction block, suggesting that lesion geometry may in

183 discordant alternans is more likely to cause conduction block than a comparable degree of preexisting

184 en in the biopsy specimens from the PVs with conduction block than in specimens from the PVs showing

185 the LA from PVs and this functional line of conduction block that resulted in LA macroreentry or for

186 In an in vitro model of conduction block, the EECT was patterned so that it conn

187 Electroporation and conduction block thresholds in papillary muscles were 28

188 rete midlateral right atrial central line of conduction block to the inferior vena cava terminated an

189 ed pitfalls in the assessment of transmitral conduction block using differential coronary sinus and l

190 The many published criteria for conduction block vary in sensitivity and specificity and

191 neuropathy, multifocal motor neuropathy with conduction block, vasculitic neuropathies, and motor neu

192 nique mechanism of channel regulation: ionic conduction block via an electrostatic barrier rather tha

193 In groups 2 and 3, isthmus conduction block was achieved during radiofrequency ener

194 Conduction block was achieved in 1 (17%) of 6 atrial pre

195 analysis revealed that complete interatrial conduction block was associated with confluent ablation

196 In STPs, pseudo-2:1 conduction block was caused by double potentials arising

197 In addition, recovery from conduction block was common in the laboratory and had a

198 Isthmus conduction block was defined using multipolar recording

199 In the present study, a mechanism for conduction block was derived from experimental studies i

200 s at a cycle length of 600 ms, until isthmus conduction block was observed (n = 14); and group 3 = ra

201 atrial pacing in sinus rhythm until isthmus conduction block was observed (n = 7).

202 ter the initial flutter termination, isthmus conduction block was observed in 9 of the 14 patients.

203 Rate-dependent conduction block was observed in a second pathway just s

204 Epicardial conduction block was observed in six of seven preparatio

205 Conduction block was obtained across 28 of 29 channels.

206 er distance into the lesion, suggesting that conduction block was overcome.

207 er distance into the lesion, suggesting that conduction block was overcome.

208 In some patients, isthmus conduction block was pacing rate dependent.

209 at cycle lengths that intermittently caused conduction block was performed at multiple sites.

210 In one of 10 patients, a complete conduction block was performed with MR imaging guidance.

211 Conduction block was present in most patients (75%; medi

212 Multiple regions of conduction block were created with a very high cardiover

213 The sites of conduction block were demonstrated to be just after the

214 Ictal asystole, bradycardia and AV-conduction block were self-limiting in all but one of th

215 To determine whether the interictal AV conduction blocks were of cardiac or neural origin, we e

216 The conduction blocks were spatiotemporally controlled by el

217 idence of double peaks (DPI), an estimate of conduction block, were calculated every 8 ms at each ele

218 f flutter recurrence after achieving isthmus conduction block, whether the block was achieved in conj

219 rsion forms the substrate for unidirectional conduction block, which is required for the initiation o

220 onor cell interface affects vulnerability to conduction block, with important implications for the de

221 formed with 0.5 nM saxitoxin led to complete conduction block within 10 min of application.

222 erminating the tachycardia and demonstrating conduction block within the atrial flutter circuit after

223 logy of heart failure and in the location of conduction block within the heart.

224 these sites on the axonal membrane can cause conduction block without other electrophysiological feat

225 e mechanisms by which rapid pacing can cause conduction block without terminating reentry.

226 uency, enabling the generation of reversible conduction blocks without the risks of entrainment.